ライフサイエンスコーパス: neurogenesis

1 ogenesis while reducing Satb2(+) upper-layer neurogenesis.

2 to elucidate the function of FXR1P in adult neurogenesis.

3 tly regulate both hippocampal blood flow and neurogenesis.

4 ional coordination during mammalian cortical neurogenesis.

5 LH-2, and in parallel to HLH-3 to promote I4 neurogenesis.

6 rtant in large mammals with respect to adult neurogenesis.

7 ect lineage progression during postembryonic neurogenesis.

8 res that sprout on neuronal dendrites during neurogenesis.

9 post-transcriptional regulators to regulate neurogenesis.

10 biting CDK7/cyclin H might similarly promote neurogenesis.

11 e of zebrafish for the study of regenerative neurogenesis.

12 w source of cerebrovascular pericytes during neurogenesis.

13 anscriptional regulator of hESC identity and neurogenesis.

14 mber of ABLKs generated during postembryonic neurogenesis.

15 d during the phases of primary and secondary neurogenesis.

16 ronic hyperglycemia on brain homeostasis and neurogenesis.

17 sing acetyl histone H3 activity and cortical neurogenesis.

18 essary for exercise to stimulate hippocampal neurogenesis.

19 shed regulator of stem cell self-renewal and neurogenesis.

20 te in cross-talk between immune response and neurogenesis.

21 hESC identity, neuroectoderm commitment and neurogenesis.

22 and investigated the effects on hippocampal neurogenesis.

23 ctions including cell junction, adhesion and neurogenesis.

24 rates neural differentiation and potentiates neurogenesis.

25 enhanced neuronal protection, and increased neurogenesis.

26 pression of active RIT1 driving robust adult neurogenesis.

27 tivity along with an increase in hippocampal neurogenesis.

28 reprogramming, as required for efficient I4 neurogenesis.

29 he DDR kinases DNA-PKcs, ATM, and ATR during neurogenesis.

30 T1, plays a critical role in IGF-1-dependent neurogenesis.

31 and the interplay between ATM and ATR during neurogenesis.

32 n controlling NSC quiescence and hippocampal neurogenesis.

33 inflammation, brain injury, autoimmunity and neurogenesis.

34 of how basal processes and endfeet influence neurogenesis.

35 ity-dependent and injury-induced hippocampal neurogenesis.

36 ulating critical genes in aNSCs during adult neurogenesis.

37 e investigated a possible role for n1-src in neurogenesis.

38 spatial expression of nrd4, a key marker of neurogenesis.

39 ining the transcriptome landscape related to neurogenesis.

40 ssion and is crucial for multiple aspects of neurogenesis.

41 c effect that has been linked to hippocampal neurogenesis.

42 R and with the subsequent alterations of NPC neurogenesis.

43 ain regions to regulate NSCs and hippocampal neurogenesis.

44 blished reports of the effects of miR-210 on neurogenesis.

45 he mechanisms that regulate pigmentation and neurogenesis.

46 (a deep layer VI marker) during upper-layer neurogenesis, a loss of Fezf2(+)/Ctip2(+) layer V neuron

47 Both FMRP and FXR2P regulate neurogenesis, a process affected in a number of neurolog

48 ation of neocortical progenitors during late neurogenesis, abnormalities in asymmetric centrosome inh

49 ATEMENT Increasing the success of endogenous neurogenesis after brain injury holds therapeutic promis

50 here critical check-points about post-stroke neurogenesis after cortical infarcts, important for the

51 egies designed to increase adult hippocampal neurogenesis (AHN) may have therapeutic potential for re

52 l knockdown of Sin3a led to reduced cortical neurogenesis, altered neuronal identity and aberrant cor

53 neurovascular tissues by combining in vitro neurogenesis and angiogenesis models using a microfluidi

54 d to optimize the gel components in terms of neurogenesis and angiogenesis.

55 ular mitochondria are critical to successful neurogenesis and are a major target of inflammatory inju

56 reverses carbofuran's inhibitory effects on neurogenesis and associated learning and memory deficits

57 ly with nonperiventricular adult hippocampal neurogenesis and basal migration of NSPCs during develop

58 onserved adverse effects of hyperglycemia on neurogenesis and brain healing in zebrafish.

59 disorders and clarify the role of Kctd13 in neurogenesis and brain size.

60 yrus, a hippocampal subdivision essential to neurogenesis and cognition, and that this impact would f

61 long-term preservation of normal hippocampal neurogenesis and cognitive and memory function, in contr

62 in astrocytes could affect adult hippocampal neurogenesis and contribute to aspects of psychiatric di

63 nd differentiation, we provide evidence that neurogenesis and differentiation can occur independently

64 We propose that the uncoupling of neurogenesis and differentiation could provide neurogeni

65 Both neurogenesis and early embryogenesis exhibit substantial

66 a major role of Pum1 and Pum2 in hippocampal neurogenesis and function.

67 g RGP behavior and proliferation dynamics in neurogenesis and glia generation remain unknown.

68 ensures the appropriate amount and timing of neurogenesis and gliogenesis in the developing hippocamp

69 d a down-regulation of genes associated with neurogenesis and glutamate receptor signaling.

70 d also in more specialized processes such as neurogenesis and growth factor signaling.

71 link the O-GlcNAc modification to mammalian neurogenesis and highlight the role of this nutrient-sen

72 in astrocytes would affect adult hippocampal neurogenesis and hippocampus-dependent behaviors.

73 Wnt/beta-catenin signaling pathway promotes neurogenesis and inhibits neurodegeneration.

74 ficiency leads to impaired adult hippocampal neurogenesis and late-onset neurodegeneration in mouse b

75 38093 treatment increases adult hippocampal neurogenesis and may provide an innovative strategy to i

76 s, but the mechanisms by which they increase neurogenesis and modulate behavior are incompletely unde

77 sterone (DHEA), a neurosteroid that promotes neurogenesis and neuron survival, and determined the tim

78 her preferred levels of drug taking enhanced neurogenesis and neuronal activation of granule cell neu

79 allel, we characterized the effects on human neurogenesis and neuronal differentiation brought about

80 e embryonic brains was sufficient to disturb neurogenesis and neuronal migration in a similar manner

81 enriched for downstream targets involved in neurogenesis and neuronal migration.

82 of established xenograft tumors, ANG-induced neurogenesis and neuroprotection, levels of pro-self-ren

83 teleost brain and have established roles in neurogenesis and neurosteroidogenesis; however, their tr

84 nity effects required for embryonic cortical neurogenesis and novel cell-autonomous Lgl1 functions co

85 al role for C5a-C5aR1 signaling in mammalian neurogenesis and provides mechanistic insight into recen

86 receptor in Ascl1-expressing cells promoted neurogenesis and reduced anxiety- and depression-like be

87 ASD-derived neurons display abnormal neurogenesis and reduced synaptogenesis leading to funct

88 f the olfactory epithelium (OE) for lifelong neurogenesis and regeneration depends on the persistence

89 autophagy plays a crucial role in regulating neurogenesis and restricting local immune response in po

90 ate the relationship between decreased adult neurogenesis and stress-induced changes in hippocampal s

91 DK8 kinase module can promote non-ectodermal neurogenesis and suggest that inhibiting CDK7/cyclin H m

92 -homolog 1 (NDE1) in human cerebral cortical neurogenesis and suggested a role in brain evolution; ho

93 dium, such that Lhx2 overexpression promotes neurogenesis and suppresses gliogenesis, whereas loss of

94 stmitotic stage and selectively controls the neurogenesis and survival of specific neuronal subtypes

95 programs in neurons controlling plasticity, neurogenesis and survival.

96 o prompted the recovery of hippocampal adult neurogenesis and synaptic plasticity and restored cognit

97 pairments and elevated stereotypy, decreased neurogenesis and synaptic plasticity, and abnormally red

98 Early brain activity is crucial for neurogenesis and the development of brain networks.

99 w epigenetic regulation contributes to adult neurogenesis and the potential impact of its dysregulati

100 n factors and levels of proteins involved in neurogenesis and the TGF-beta pathway (i.e. TGF-beta; SM

101 study the roles of these proteins in enteric neurogenesis and their cross regulation.

102 formation (APF, SOP selection), 40 hrs APF (neurogenesis), and adult antennae.

103 seizures potently modulate hippocampal adult neurogenesis, and adult-born dentate granule cells contr

104 TX is known to play a role in transcription, neurogenesis, and antiviral response.

105 enhanced brain plasticity, adult hippocampal neurogenesis, and increased levels of brain-derived neur

106 D44 play important roles in regulating adult neurogenesis, and we provide evidence that HA contribute

107 odal effects of neurotrophism, angiogenesis, neurogenesis, antiautoimmunity, and antiinflammation.

108 While a number of regulators of VM DA neurogenesis are known, processes involved in later deve

109 metabolic programs and their impact on adult neurogenesis are largely unknown.

110 Mechanisms mediating adult enteric neurogenesis are largely unknown.

111 Postnatal and adult neurogenesis are region- and modality-specific, but the

112 der patients and lower levels of hippocampal neurogenesis are the hypothesized cause.

113 Spatiotemporal variations of neurogenesis are thought to account for the evolution of

114 phase, where neurorepair mechanisms such as neurogenesis, are involved.

115 t included delayed brain differentiation and neurogenesis as well as abnormal proliferation accompany

116 d for adult neural stem cell maintenance and neurogenesis as well as the formation of ependymal cells

117 identified ID1, a HLH inhibitor of premature neurogenesis, as a target.

118 by neurodegeneration, inflammation, altered neurogenesis, as well as cognitive and memory deficits.

119 ogether suggest FTO plays important roles in neurogenesis, as well as in learning and memory.

120 deling and deacetylase (NuRD) complex at the neurogenesis-associated gene loci, and, as a consequence

121 own mechanisms for active forgetting include neurogenesis-based forgetting, interference-based forget

122 tion (astroglial and microglial activation), neurogenesis (BrdU-labeled newborn cells), and amyloidos

123 ked to this locus contains genes involved in neurogenesis but, in accordance with an earlier study, d

124 tion factor SOX2 has been implicated in otic neurogenesis, but its requirement in the specification o

125 antidepressants stimulate adult hippocampal neurogenesis, but the mechanisms by which they increase

126 lomeres are required for embryonic and adult neurogenesis, but their uncapping has surprisingly no de

127 Enhancing neurogenesis by conditional deletion of the pro-apoptoti

128 is highly conserved in animals and regulates neurogenesis by facilitating neural differentiation, yet

129 revealed that CDK-8 most likely promotes I4 neurogenesis by inhibiting the CDK-7/CYH-1 (CDK7/cyclin

130 The estrogen seems to restore neurogenesis by inhibiting the cell cycle and elevating

131 cordingly, hunger and satiety regulate adult neurogenesis by modulating the activity of this hypothal

132 erstanding the circadian regulation of adult neurogenesis can help optimize the timing of therapeutic

133 Alterations in hippocampal neurogenesis can mediate treatment of mental illness or

134 The order of neurogenesis can therefore account for the dorsoventral

135 the course of brain development by altering neurogenesis, cell death, migration, differentiation, de

136 europrogenitors are lost with increased age, neurogenesis concomitantly decreases.

137 ting the mouse olfactory bulb, where ongoing neurogenesis continually supplies new inhibitory granule

138 only known brain region where physiological neurogenesis continues into adulthood across mammalian s

139 In the present review, we evaluate how adult neurogenesis contributes to the repair and regeneration

140 l layer, with concomitant rescue of both the neurogenesis defect and hippocampal memory abnormalities

141 ntiation of NPCs, and Mbd3 depletion rescued neurogenesis defects seen in Smek1/2 knockout mice.

142 hondrial function ameliorates age-associated neurogenesis defects.

143 function as a potential target to ameliorate neurogenesis-defects in the aging hippocampus.

144 administration of S 38093 were assessed on a neurogenesis-dependent "context discrimination (CS) test

145 Combining stress and inhibition of adult neurogenesis did not have additive effects on the magnit

146 gainst carbofuran-mediated neurotoxicity and neurogenesis disruption.

147 es enriched with functional associations for neurogenesis, dopamine signaling, immune regulation and

148 rt a direct role for the importance of adult neurogenesis during abstinence in compulsive-like drug r

149 l stem and progenitor cells, but its role in neurogenesis during brain development remains unknown.

150 ceptor may be instrumental for understanding neurogenesis during eye development and for devising the

151 ptions of temporal parietal and sub-cortical neurogenesis during infancy are critical to the pathophy

152 ion of ERK signaling, or inhibition of adult neurogenesis, each blocks the ketamine-induced behaviora

153 r factor suppresses gliogenesis and promotes neurogenesis; each can substitute for loss of the other.

154 ccordingly, Mef2c-het mice exhibit decreased neurogenesis, enhanced neuronal apoptosis, and an increa

155 trophic factors along with neurotrophic and neurogenesis factors were detected; these elevations las

156 In contrast, complete inhibition of adult neurogenesis for 4 weeks led to volume reduction only in

157 With prolonged inhibition of neurogenesis for 8 or 16 weeks, volume loss spread to th

158 sible future directions for harnessing adult neurogenesis for therapeutic use.

159 age marker, during the active phase of mesDA neurogenesis from E9.5 to E14.5 during mouse development

160 ng results, the impact of Abeta pathology on neurogenesis/gliogenesis remains unclear.

161 The role of FTO in neurodevelopment and neurogenesis, however, remains largely unknown.

162 mportant epigenetic roles in mammalian adult neurogenesis; however, the precise molecular mechanisms

163 study has explored complement regulation of neurogenesis, identifying a novel relationship between t

164 euronal stem cells suggests that hippocampal neurogenesis impairment is an important factor underlyin

165 n1-src causes a striking absence of primary neurogenesis, implicating n1-src in the specification of

166 we have performed a longitudinal analysis of neurogenesis in a model of cortical ischemia in mice.

167 edictable restraint stress and inhibition of neurogenesis in a rat pharmacogenetic model.

168 Here we investigated how manipulating neurogenesis in adult mice alters excitatory synaptic tr

169 suggest that colitis promotes rapid enteric neurogenesis in adult mice and humans through differenti

170 investigate constitutive and injury-induced neurogenesis in adult vertebrates.

171 prevented carbofuran-mediated inhibition of neurogenesis in both hippocampal NSC cultures and the hi

172 nutrient availability, allowing preferential neurogenesis in brain subregions during nutrient poor co

173 at dogs are a valuable animal model of adult neurogenesis in comparative and preclinical studies.

174 d trajectories of radial glia maturation and neurogenesis in developing human telencephalon.

175 Transient inhibition of C5aR1 during neurogenesis in developing mice led to behavioral abnorm

176 s, representing a potential avenue to direct neurogenesis in developmental disorders or regenerative

177 transient Hes1 expression rescues precocious neurogenesis in Dmrta2 knockout NPCs.

178 as early determinants, promoting deep-layer neurogenesis in early cortical progenitors via input int

179 (6)A signaling also regulates human cortical neurogenesis in forebrain organoids.

180 filing, we have analysed the early stages of neurogenesis in fragile X syndrome human pluripotent ste

181 Maintaining neurogenesis in growing tissues requires a tight balance

182 estrogen replacement might partially restore neurogenesis in human premature infants.SIGNIFICANCE STA

183 y, OSNs are continually replenished by adult neurogenesis in mammals, including humans, so OSN measur

184 13 or kctd13 does not increase brain size or neurogenesis in mice or zebrafish, respectively.

185 ioral alterations and rescued abnormal adult neurogenesis in mutant DISC1 mice.

186 n proteasome function during development and neurogenesis in particular; they enable the definition o

187 and estrogen treatment reverses interneuron neurogenesis in preterm newborns by cell-cycle inhibitio

188 transcription factors known to govern adult neurogenesis in response to active RIT1 expression in th

189 cruit distinct NSC pools, allowing on-demand neurogenesis in response to physiology and environmental

190 CA3 pyramidal cells and suppression of adult neurogenesis in rodents.

191 stions about the role of sprouting and adult neurogenesis in sustaining seizure-like activity.

192 ormed by the discovery that exercise induces neurogenesis in the adult brain, with the potential to i

193 the promotion of stem cell proliferation and neurogenesis in the adult dentate gyrus.

194 equired for neural stem cell maintenance and neurogenesis in the adult lateral ventricle subventricul

195 a synthetic small molecule known to modulate neurogenesis in the adult rodent brain) during abstinenc

196 rrested alveologenesis in the lung, impaired neurogenesis in the brain and occasional myocardial fibr

197 Adult neurogenesis in the dentate gyrus (DG) is strongly influ

198 As both astrocytes and DISC1 influence adult neurogenesis in the dentate gyrus (DG) of the hippocampu

199 ABSTRACT: Exercise signals neurogenesis in the dentate gyrus of the hippocampus.

200 current dogma, there is little or no ongoing neurogenesis in the fully developed adult enteric nervou

201 ) AR-42, ameliorates the deficiency of adult neurogenesis in the granule cell layer of the dentate gy

202 Adult neurogenesis in the hippocampal subgranular zone (SGZ) i

203 s on the adult brain that relies on improved neurogenesis in the hippocampus.

204 working memory capacity via suboptimal adult neurogenesis in the hippocampus.

205 irth-dating experiments confirmed diminished neurogenesis in the MCT8-deficient cell population as we

206 show that preterm birth disrupts interneuron neurogenesis in the medial ganglionic eminence (MGE) and

207 Hence, preterm birth disrupts interneuron neurogenesis in the MGE and estrogen treatment reverses

208 Here, we show that inhibition of adult neurogenesis in the mouse dentate gyrus decreases hippoc

209 Adult neurogenesis in the olfactory epithelium is often depict

210 mice may prove useful for future studies of neurogenesis in the olfactory epithelium.

211 and that C3a receptor signalling stimulates neurogenesis in unchallenged adult mice.

212 e harmine metabolite harmol, stimulate adult neurogenesis in vitro.

213 with the host molecular machinery to impact neurogenesis in vivo is not well understood.

214 on with behavioral tests, we evaluated adult neurogenesis, including neural progenitor proliferation

215 ucing populations across different stages of neurogenesis, including radial glia-like cells, intermed

216 mature neurons, whereas genetic ablation of neurogenesis increased EPSCs in mature neurons.

217 riad changes in the brain, including reduced neurogenesis, increased synaptic aberrations, higher met

218 entate neurons increased EPSCs and prevented neurogenesis-induced synaptic suppression.

219 We propose a novel view for otic neurogenesis integrating cell dynamics whereby ingressio

220 le of radial glia cells and extends cortical neurogenesis into postnatal stages.

221 Neurogenesis is a highly regulated process in which dive

222 Specifically, we showed that neurogenesis is affected early in the APP/PS1 hippocampu

223 xercise-mediated potentiation of hippocampal neurogenesis is also diminished in RIT1 (-/-) mice.

224 Adult hippocampal neurogenesis is an important form of structural and func

225 ry is present in adult rats, even when adult neurogenesis is blocked.

226 Neurogenesis is essential to brain development and plays

227 Neurogenesis is initiated by the transient expression of

228 rstanding the molecular mechanisms governing neurogenesis is necessary for the development of transla

229 In Lmnb1-null embryos, neurogenesis is reduced, while in vivo Lmnb1 silencing i

230 The findings suggest that adult neurogenesis is required to maintain hippocampal volume

231 and accordingly, Sox2-dependent hippocampal neurogenesis is significantly blunted following IGF-1 in

232 Loss or increase in LHX2 during neurogenesis is sufficient to increase or decrease, resp

233 l dentate gyrus, a region with ongoing adult neurogenesis, is sensitive to MAGUK loss in mature anima

234 Despite extensive studies on mammalian neurogenesis, its post-transcriptional regulation remain

235 o reduced neuronal differentiation, impaired neurogenesis, learning deficits, and autism-like behavio

236 This lack of neurogenesis leaves unanswered the question of how enter

237 ble restraint stress and inhibition of adult neurogenesis led to atrophy of pyramidal cell apical den

238 Thus, during adult neurogenesis, microglia regulate the elimination (prunin

239 regarded to follow a stereotypic sequence of neurogenesis, migration, and differentiation.

240 rates, it remains unresolved which ancestral neurogenesis mode prefigures the highly divergent cytoar

241 Using inflammation-associated neurogenesis models and a transgenic approach, we aimed

242 Together these results show that neurogenesis modifies synaptic transmission to mature ne

243 Isoxazole-9 modulated neurogenesis, neuronal activation and structural plastic

244 mary cilia are crucial for early patterning, neurogenesis, neuronal maturation and survival, and tumo

245 However, neurogenesis, neuronal pruning, and neuronal enlargement

246 Postinjury neurogenesis occurs as a burst of proliferation that pea

247 the select regions of the mature brain where neurogenesis occurs.

248 r, our results show significant turnover and neurogenesis of adult enteric neurons and provide a para

249 tion of the Wnt signaling pathway to promote neurogenesis of the ENS in vitro.

250 phosphatase that has been shown to influence neurogenesis, oligodendrogenesis, and oligodendrocyte di

251 , mechanistic studies on the relationship of neurogenesis on drug seeking are limited.

252 function, in contrast to waned and abnormal neurogenesis, persistent inflammation, and functional de

253 Adult neurogenesis persists in the rodent dentate gyrus and is

254 Thus, adult neurogenesis plays a central role in the adaptive change

255 guide specification of neuronal types during neurogenesis preconfigure the brain.

256 , implicating biological pathways related to neurogenesis, programmed cell death and insulin signalin

257 ma7A as a key regulator of adult hippocampal neurogenesis, providing an example of how differential r

258 Neurogenesis-regulating miRNAs were decreased in NDAN in

259 nscriptionally regulated by NPAS3, as is the neurogenesis regulator Notch.

260 molecular mechanisms for carbofuran-impaired neurogenesis remain unknown.

261 Impaired neurogenesis resulted in a reduced number of glutamaterg

262 landscapes between mouse and human cortical neurogenesis reveals enrichment of human-specific m(6)A

263 system, neuroblasts go through two phases of neurogenesis separated by a period of mitotic quiescence

264 Neurotrophic processes including neurogenesis shown in preclinical studies may underlie t

265 ertebrate nervous system evolved to regulate neurogenesis.SIGNIFICANCE STATEMENT The Src family of no

266 halamus presents constitutive and stimulated neurogenesis, suggesting that obesity-associated hypotha

267 n Drosha-deficient hippocampal NSCs restores neurogenesis, suggesting that the Drosha/NFIB mechanism

268 evolutionarily conserved and operate during neurogenesis, suggesting that they represent a physiolog

269 Wnt/beta-catenin signaling pathway promotes neurogenesis (synapse formation and remodeling) and inhi

270 e mice show changes in genes associated with neurogenesis, synapse formation, and neuronal cell death

271 related to the central nervous system (e.g., neurogenesis, synaptic plasticity, glial cell developmen

272 d in neurodevelopmental disorders, including neurogenesis, synaptic processes and neuroimmune signali

273 In the adult brain, one of the regions where neurogenesis takes place is the subventricular zone (SVZ

274 n in the SGZ may contribute to reductions in neurogenesis that are linked to age-related decline in s

275 ids to identify specific cellular defects in neurogenesis that may explain PTEN-related macrocephaly

276 t of mRNAs related to transcription factors, neurogenesis, the cell cycle, and neuronal differentiati

277 osure has detrimental effects on hippocampal neurogenesis, the generation of new neurons from neural

278 anges span from synaptic plasticity to adult neurogenesis, the latter being highly reduced in large-b

279 Later in development, during neurogenesis, the mutation spectrum shifted toward oxida

280 Adult neurogenesis, the process of generating mature neurons f

281 onal differentiation recapitulated embryonic neurogenesis, they generated action potentials, and they

282 In contrast, ATR coordinated the DDR during neurogenesis to direct apoptosis in cycling neural proge

283 genitors to control the temporal switch from neurogenesis to gliogenesis.

284 l progenitors and the consequent decrease of neurogenesis together with the impaired survival of late

285 During neurogenesis, vertebrate and Drosophila progenitors chan

286 pathway downstream of MeCP2 that influences neurogenesis via interactions with central molecular hub

287 The circadian system may regulate adult neurogenesis via intracellular molecular clock mechanism

288 Interestingly, a milder decrease in neurogenesis was observed in heterozygotes, indicating S

289 Neurogenesis was reduced in BERKO ES cells, and oligoden

290 aracterize in vivo the function of ZFP423 in neurogenesis, we analyzed allelic murine mutants in whic

291 ablished importance of microRNAs (miRNAs) in neurogenesis, we employed isogenic human RTT patient-der

292 During deep layer neurogenesis, we show that transcription factor LHX2 bin

293 ISC1-associated behaviors and abnormal adult neurogenesis were also examined.

294 in Kmt2d(+/betaGeo) mice; similar effects on neurogenesis were observed on exogenous administration o

295 s multiple hallmarks of aging in hippocampal neurogenesis, whereas pharmacological enhancement of mit

296 ry can change the level of adult hippocampal neurogenesis, which plays an important role in regulatin

297 ed the period of Tbr1(+)/Ctip2(+) deep-layer neurogenesis while reducing Satb2(+) upper-layer neuroge

298 tein 3 (NPAS3) impairs postnatal hippocampal neurogenesis, while loss of the related protein NPAS1 pr

299 Further defining neurogenesis will improve understanding and treatment of

300 In adult neurogenesis young neurons connect to the existing netwo