ライフサイエンスコーパス: neurogenic bladder

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1 ment, causing cardiac failure, diplegia, and neurogenic bladder.

2 sponse, upper gastrointestinal symptoms, and neurogenic bladder.

3 al urachal sinus, and lipomeningocele with a neurogenic bladder.

4 gy influence UTI susceptibility with the SCI-neurogenic bladder.

5 sis, and voiding dysfunctions as a result of neurogenic bladders.

6 The most common clinical findings were neurogenic bladder (100%), spastic paraplegia with vibra

7 ocedures (43% versus 15% overall), to have a neurogenic bladder (29% versus 12% overall), to have rec

8 In the management of neurogenic bladder and chronic lower urinary tract sympt

9 Neurogenic bladder and sphincter dysfunction can be mana

10 s current surgical practice in patients with neurogenic bladders and urinary incontinence.

11 Patients with diabetes mellitus, neurogenic bladder, bladder outlet obstruction, and recu

12 treatment of pediatric voiding dysfunction, neurogenic bladder, chronic lower urinary tract symptoma

13 y a description of the various causes of the neurogenic bladder, discussed in a hierarchical order st

14 For various neurogenic bladder disorders, urodynamic studies may dem

15 review of the methods available for treating neurogenic bladder disorders.

16 ers the articles that have been published on neurogenic bladder dysfunction as well as on posterior u

17 The outlook for patients with neurogenic bladder has been transformed by a combination

18 s, were not required for colonization of the neurogenic bladder in human beings.

19 rent standard of care in which to remedy the neurogenic bladder, it is still a stop-gap measure and i

20 This model will allow elucidation of SCI-neurogenic bladder-mediated changes in host response tha

21 ransplant bladder augmentation, did not have neurogenic bladders or require preoperative catheterizat

22 micturition and defecation for patients with neurogenic bladder overactivity or spinal cord lesions.

23 Neurogenic bladder predisposes to recurrent urinary trac

24 2 mutations were absent in 23 non-neurogenic neurogenic bladder probands and, of 439 families with no

25 al recessive leukodystrophy characterized by neurogenic bladder, progressive spastic gait, and periph

26 thra for evacuation (e.g. bladder exstrophy, neurogenic bladder, radiation injury, and marked urethra

27 The median age was 51 years for the onset of neurogenic bladder symptoms, 63 years for wheelchair dep

28 nmol/L), sicca complex, abnormal pupils, and neurogenic bladder; three had severe upper gastrointesti

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