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1 ing activity from the ventral midline to the neurogenic ectoderm.
2 ential cell fates in adjacent regions of the neurogenic ectoderm.
3 tivator also drive expression throughout the neurogenic ectoderm.
4 ted expression within ventral regions of the neurogenic ectoderm.
5 toderm within the ventral-most region of the neurogenic ectoderm.
6 e precise lines of sim expression within the neurogenic ectoderm.
7 pression in the anterior-most regions of the neurogenic ectoderm.
8 eristic of the ciliated band, which contains neurogenic ectoderm.
9 y rescues epidermal development in overlying neurogenic ectoderm.
11 istinction between tissues such as mesoderm, neurogenic ectoderm and dorsal ectoderm in the developin
12 itiates the differentiation of the mesoderm, neurogenic ectoderm and dorsal ectoderm in the precellul
14 on of midline cells causes a failure to form neurogenic ectoderm and Ph-sim RNAi results in severely
15 in severely dorsalized embryos lacking both neurogenic ectoderm and the appendage-bearing lateral ec
16 the animal pole domain (APD), which contains neurogenic ectoderm, and can oppose Wnt and Nodal signal
17 orsal-ventral (DV) axis to specify mesoderm, neurogenic ectoderm, and dorsal ectoderm cell fates.
20 lda drives expression within the presumptive neurogenic ectoderm, but they also demonstrate that regu
21 EGFR is activated in the ventral midline and neurogenic ectoderm by the Spitz ligand, which is proces
22 responses, but its specification of ventral neurogenic ectoderm demands a relatively high-threshold
26 ct gene expression in ventral regions of the neurogenic ectoderm in response to intermediate levels o
27 epressor, Brinker, which is expressed in the neurogenic ectoderm in response to the maternal Dorsal g
29 al specification of the mesoderm and ventral neurogenic ectoderm is highly conserved in flies and mos
32 lation of neural-epidermal fate decisions in neurogenic ectoderm of Drosophila and somitogenesis in v
36 o investigate transcriptional control during neurogenic ectoderm specification, we examined divergenc
38 ulated by the Dorsal gradient in the ventral neurogenic ectoderm (vNE) of the early Drosophila embryo
39 ipe induces ectopic expression of sim in the neurogenic ectoderm where there are low levels of the Do
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