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1 s as a neurotransmitter, neuromodulator, and neurohormone.
2 ion of serotonin to melatonin, the circadian neurohormone.
3 of N-linked glycoproteins and peptides, like neurohormones.
4 and may be released into the blood to act as neurohormones.
5 nts that unload the heart or target systemic neurohormones.
6 eactive and appear to release YXFGLamides as neurohormones.
7 ation of selective neural circuits by opioid neurohormones.-
8 a neuroactive monoamine that functions as a neurohormone, a neuromodulator, and a neurotransmitter i
9 present study suggests that CRF actions as a neurohormone and as a neurotransmitter in the LC may be
12 ew studies supporting the mediating roles of neurohormones and neurotransmitters (e.g., cortisol, nor
14 We examined levels of monoamine metabolites, neurohormones, and neuropeptides in the cerebrospinal fl
15 n protects against pathological responses to neurohormones, and sustained pressure-overload stress.
17 y it has become apparent that in addition to neurohormones, another portfolio of biologically active
22 ase at neurohypophysial nerve terminals, the neurohormones arginine vasopressin (aVP) and oxytocin (O
23 nce of biologically active molecules such as neurohormones as mediators of disease progression in hea
24 ranscription factor; galanin, a hypothalamic neurohormone; BAX, a proapoptotic signaling factor; and
25 m the CNS and appears to be identical to the neurohormone bombyxin, a member of the insulin family of
26 ediately after shedding the old cuticle, the neurohormone bursicon causes the hardening and darkening
27 he mechanisms have remained enigmatic is the neurohormone bursicon, which, after the final molt, coor
29 between melatonin--a scotoperiod-responsive neurohormone closely tied to seasonal adaptation--and do
30 reasing putative pulse-time sets for a given neurohormone concentration time series; and then, recurs
31 Here, we determined the role of the stress neurohormone corticotropin-releasing factor (CRF) in str
32 Previous studies have found that the stress neurohormone corticotropin-releasing factor (CRF) inhibi
33 ated by fibers containing the stress-related neurohormone corticotropin-releasing factor (CRF), which
36 suppression of the expression of the stress neurohormone corticotropin-releasing hormone (CRH) in hy
38 Because adrenalectomy also alters release of neurohormone CRF, the present study suggests that CRF ac
39 ogical mediators of cardiac hypertrophy (eg, neurohormones, cytokines, and stretch) have been shown t
40 anosensory neurons in C. elegans release the neurohormone dopamine to promote proteostasis in epithel
41 delivered neurotransmitters and circulating neurohormones elicit a wide range of rhythmic motor outp
43 ors and are mimicked by the actions of other neurohormones (endothelin, prostaglandin F(2alpha) angio
46 ion, including cardiovascular diseases, age, neurohormones, genetics, diet, autonomic influences, and
52 beta-PDHs; Canpr-beta-PDH II appears to be a neurohormone in the SG, whereas Canpr-beta-PDH I may fun
55 ance of biologically active molecules (e.g., neurohormones) in disease progression in heart failure.
56 rving as the site of release of hypothalamic neurohormones into a plexus of hypophyseal capillaries.
58 ations for how various neuroactive drugs and neurohormones known to modulate extrasynaptic GABA(A) re
59 ncluded changes in exercise capacity, plasma neurohormones, left ventricular function, and overall HF
64 tumor necrosis factor alpha), much like the neurohormones, may represent another class of biological
65 t suggests that the cytokines, much like the neurohormones, may represent another class of biological
68 tylserotonin, the precursor of the circadian neurohormone melatonin, is catalyzed by serotonin N-acet
70 ies designed to reverse the effects of these neurohormones on the kidney have so far had limited succ
71 to Aedes aegypti triggers the release of two neurohormones, ovary ecdysteroidogenic hormone (OEH) and
72 s of heart failure M&M, but changes in these neurohormones over time are associated with correspondin
75 of heart failure, we examined whether these neurohormones predicted all-cause mortality, cardiovascu
76 okine macrophage migration inhibitory factor neurohormone receptors such as growth hormone- releasing
84 statistical estimation of unobserved in vivo neurohormone secretion and within-axis, dose-responsive
88 induction of oxidative stress transduced by neurohormones, such as angiotensin II and catecholamines
91 he modulatory effects of melatonin, a pineal neurohormone that mediates circadian and seasonal proces
92 veness of gastric vagal afferents to several neurohormones, the aim of the present study was to deter
93 of the Drosophila FMRFamide gene function as neurohormones to modulate the strength of contraction at
95 fter modification, the cDNA for the putative neurohormone was expressed in a bacterial system, and th
97 icroorganisms have evolved systems for using neurohormones, which are widely distributed throughout n
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