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2 his study is to determine the reliability of neurohypophyseal abnormalities on magnetic resonance ima
10 essin (VP) cause autosomal dominant familial neurohypophyseal diabetes insipidus (adFNDI), a rare inh
16 sin parents and three children affected with neurohypophyseal diabetes insipidus, suggesting autosoma
18 m both in the cardiovascular response to the neurohypophyseal hormone arginine vasopressin (AVP) and
19 ized that combine into a single molecule the neurohypophyseal hormone oxytocin and the potent vasopre
22 tive resistant or R-type Ca2+ current in rat neurohypophyseal nerve terminals, but concentrations of
25 that the phylogenetic plasticity of central neurohypophyseal peptide receptor expression may contrib
28 Physiological activation of the hypothalamo-neurohypophyseal system (HNS) by dehydration results is
29 VP) release from explants of the hypothalamo-neurohypophyseal system (HNS), but the response is not s
30 cin release from explants of the hypothalamo-neurohypophyseal system (HNS), two hypotheses were teste
31 we show expression in the brain hypothalamo-neurohypophyseal system (HNS), wherein upregulation foll
32 reactive fibers, were located throughout the neurohypophyseal tract and within the posterior pituitar
33 ers were evident outside of the hypothalamic-neurohypophyseal tract, a plexus of fibers in the latera
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