ライフサイエンスコーパス: neurohypophysial

1 icate that taurine is mainly concentrated in neurohypophysial astrocytes, which are known to engulf t

2 the release of oxytocin or vasopressin from neurohypophysial axon terminals in either young or old r

3 ivity limits action potential propagation in neurohypophysial axons.

4 Ethanol reversibly increased neurohypophysial BK channel activity (EC50, 22 mM; maxim

5 nM iberiotoxin (NPo, 5% of control), whereas neurohypophysial BK channels are insensitive to charybdo

6 those reported for the action of ethanol on neurohypophysial BK channels studied in native membrane,

7 , as opposed to the slow gating (type II) of neurohypophysial BK channels.

8 s 273 nM, as opposed to >1.53 microM for the neurohypophysial channel, indicating the higher Ca2+ sen

9 us (SON) was investigated in the hypothalamo-neurohypophysial explant of male rats.

10 plays an important role in the regulation of neurohypophysial function, and blockade of this enzyme c

11 ans and non-human animals indicates that the neurohypophysial hormone oxytocin (OT) evolved to serve

12 Arginine vasopressin (AVP) is a neurohypophysial hormone regulating hydromineral homeost

13 ents which may regulate the daily changes in neurohypophysial hormone secretion.

14 e, mediating osmotically evoked drinking and neurohypophysial hormone secretion.

15 in reward, and its possible role as a third neurohypophysial hormone, we examined the brain distribu

16 receptors play no role in the modulation of neurohypophysial K+ channels.

17 that dopamine type 4 (D4) receptors modulate neurohypophysial K+ current, so this study initially tes

18 on with the [Ca2+]i regulatory properties of neurohypophysial nerve endings may explain both the depl

19 in stimulates Ca(2+)-dependent exocytosis in neurohypophysial nerve endings through receptor interact

20 als, sildenafil enhanced the excitability of neurohypophysial nerve terminals and increased the actio

21 a2+ channel inactivation was investigated in neurohypophysial nerve terminals by using patch-clamp te

22 hibit voltage-gated Na+ currents in isolated neurohypophysial nerve terminals in a concentration- and

23 ion mechanisms, which in the present case of neurohypophysial nerve terminals would lead to the enhan

24 ction potential-evoked exocytotic release at neurohypophysial nerve terminals, the neurohormones argi

25 AVP) and oxytocin (OT) release from isolated neurohypophysial (NH) terminals of the rat were investig

26 en described previously in peptide-secreting neurohypophysial (NHP) nerve terminals.

27 e identified two distinct populations of rat neurohypophysial (NHP) terminals distinguished by size,

28 s throughout the evolutionary lineage of the neurohypophysial peptide hormone receptor family of G-pr

29 tein coupled receptors that are activated by neurohypophysial peptide hormones, including vasopressin

30 Emerging evidence suggests a role of the neurohypophysial peptide oxytocin (OT) in the modulation

31 cing evidence for somatodendritic release of neurohypophysial peptides in the hypothalamus, there is

32 This caecilian species, therefore, possesses neurohypophysial peptides that are similar in their stru

33 The characterization and distribution of neurohypophysial peptides, however, has not been describ

34 epolarization and thereby depress release of neurohypophysial peptides.

35 Purified neurohypophysial plasma membranes not only had a single

36 an enhance the use-dependent facilitation of neurohypophysial secretion.

37 Ion channels from bovine neurohypophysial secretory granules (NSG) were incorpora

38 O on oxytocin secretion from the hypothalamo-neurohypophysial system (HNS) during water deprivation.

39 Studies in the hypothalamo-neurohypophysial system (HNS) have suggested that neural

40 The hypothalamic-neurohypophysial system (HNS) is an established preparat

41 nocellular neurons (MCNs) of the hypothalamo-neurohypophysial system are the only neuronal phenotypes

42 mical products in regulating the hypothalamo-neurohypophysial system during dehydration.

43 activation of the magnocellular hypothalamo-neurohypophysial system induces a coordinated astrocytic

44 Specifically, in murine neurohypophysial terminals (NHT), these events, termed C

45 metabolite of ATP, inhibits VP release from neurohypophysial terminals and adenosine receptors (AR)

46 Neurohypophysial terminals exhibited, besides L- and N-t

47 T) and vasopressin (VP) hormone release from neurohypophysial terminals is controlled by the firing p

48 sin-containing and large oxytocin-containing neurohypophysial terminals may contribute to their obser

49 he P/Q-channel class, suggesting that in the neurohypophysial terminals this current is mediated by a

50 ages of voltage-dependent Ca(2+) influx into neurohypophysial terminals were captured after excitatio

51 ave no effect on calcium currents (I(Ca)) in neurohypophysial terminals when recorded using the class

52 us Q-channels are present on a subset of the neurohypophysial terminals where, in combination with N-

53 nd on peptide release from rat permeabilized neurohypophysial terminals.

54 e of Ca(2+) current in oxytocin release from neurohypophysial terminals.

55 ibute to the control of hormone release from neurohypophysial terminals.