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1 erived neuropeptides such as substance P and neurokinin A.
2 ncodes substance P and a related tachykinin, neurokinin A.
3 ion and nuclear translocation in response to neurokinin A.
4 roM), whereas responses to concentrations of neurokinin A (10 nM), neurokinin B (0.1 microM), substan
5  or an NK(3) tachykinin receptor antagonist (Neurokinin A 4-10; 100 nM to 5 microM).
6 duced by 10 microM acetylcholine, 0.1 microM neurokinin A and 70 mM potassium.
7 ance P, serotonin, gamma-amino-butyric acid, neurokinins A and B, neurotensin, neuropeptide Y, peptid
8 (50) values were determined for substance P, neurokinin A, and galanin and were found to correspond w
9                                 Substance P, neurokinin A, and galanin were labeled with Cy5 and were
10 ogues of SP, and the NK-2 endogenous ligand, neurokinin A, and is coupled to the phospholipase C path
11 f neuropeptides, which includes substance P, neurokinin A, and neurokinin B, have three distinct rece
12 of neuropeptides including substance P (SP), neurokinin A, and neurokinin B, which are recognized for
13  binding of the three peptides, substance P, neurokinin A, and propionyl[Met(O(2))(11)]SP(7-11), to t
14 er of labeled neurons containing Substance P/Neurokinin A (approximately 20% of total labeled neurons
15 pigs, few stained positively for Substance P/Neurokinin A (approximately 3% of total labeled neurons)
16 related neurokinin peptides, substance P and neurokinin A, are found in abundance within the substant
17 reprotachykinin A gene, which encodes SP and neurokinin A, are resistant to kainate excitoxicity.
18                                   [beta-Ala8]Neurokinin A ([beta-Ala8]NKA; 10 nM), an NK-2 selective
19                              Substance P and neurokinin A bind to the reconstituted receptor in a bip
20 lockade of the facilitation of the reflex by neurokinin A but not substance P in normal rats.
21 these studies indicated that substance P and neurokinin A can directly induce nerve growth factor mRN
22 th Sendai virus on the number of Substance P/Neurokinin A-containing vagal afferent neurons whose cel
23 Phe7]neurokinin B > senktide > substance P = neurokinin A > neurokinin B.
24                       The role of endogenous neurokinin A in changes in the excitability of spinal ne
25 uge NANCi responses, TSM was contracted with neurokinin A in the presence of atropine, propranolol, a
26       These results indicate that endogenous neurokinin A increases the excitability of spinal neuron
27 s from peripheral release of substance P and neurokinin A, is almost absent in the mutant mice.
28 ceptors were able to bind to substance P and neurokinin A ligand with similar affinities; however, th
29 ll mice are caused by the extinction of a SP/neurokinin A-mediated signaling pathway that is activate
30 , Lys-bradykinin, Lys-(des-Arg9)-bradykinin, neurokinin A, Met-Lys-bradykinin, histatin 8, and a myos
31 er naturally occurring tachykinins including neurokinin A, neuropeptide K, neuropeptide gamma, eledoi
32                         Substance P (SP) and neurokinin-A (NK-A) are derived from the preprotachykini
33 f neuropeptides, including substance P (SP), neurokinin A (NKA), and neurokinin B (NKB), that are enc
34 ch encodes the peptides substance P (SP) and neurokinin A (NKA).
35 uperfusion of different tachykinin agonists (neurokinins A (NKA) and B (NKB), SP, and senktide) gave
36 ently mediate the effects of substance P and neurokinin A on human versus rat DA neurons.
37 effect of the neuropeptides, substance P and neurokinin A, on nerve growth factor expression in human
38 served with application of either [beta-Ala8]neurokinin A or senktide, selective NK2 and NK3 receptor
39 s there were significantly fewer Substance P/Neurokinin A positive neurons in nodose ganglia from vir
40  tachykinin receptors (substance P receptor, neurokinin A receptor, and neurokinin B receptor).
41 inese hamster ovary cells to substance P and neurokinin A, respectively.
42                              Substance P and neurokinin A stimulated a rapid increase in intracellula
43 C1 (encoding the tachykinins substance P and neurokinin A) to be strongly activated by LP within the
44 ing of fluorescently labeled substance P and neurokinin A was tested on the human astrocytoma cell li
45          Despite its inability to respond to neurokinin A, we found that NK1-betaArr1 expression caus
46 Fluorescent and radiolabeled substance P and neurokinin A were internalized similarly by cells expres

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