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1 erived neuropeptides such as substance P and neurokinin A.
2 ncodes substance P and a related tachykinin, neurokinin A.
3 ion and nuclear translocation in response to neurokinin A.
4 roM), whereas responses to concentrations of neurokinin A (10 nM), neurokinin B (0.1 microM), substan
7 ance P, serotonin, gamma-amino-butyric acid, neurokinins A and B, neurotensin, neuropeptide Y, peptid
8 (50) values were determined for substance P, neurokinin A, and galanin and were found to correspond w
10 ogues of SP, and the NK-2 endogenous ligand, neurokinin A, and is coupled to the phospholipase C path
11 f neuropeptides, which includes substance P, neurokinin A, and neurokinin B, have three distinct rece
12 of neuropeptides including substance P (SP), neurokinin A, and neurokinin B, which are recognized for
13 binding of the three peptides, substance P, neurokinin A, and propionyl[Met(O(2))(11)]SP(7-11), to t
14 er of labeled neurons containing Substance P/Neurokinin A (approximately 20% of total labeled neurons
15 pigs, few stained positively for Substance P/Neurokinin A (approximately 3% of total labeled neurons)
16 related neurokinin peptides, substance P and neurokinin A, are found in abundance within the substant
17 reprotachykinin A gene, which encodes SP and neurokinin A, are resistant to kainate excitoxicity.
21 these studies indicated that substance P and neurokinin A can directly induce nerve growth factor mRN
22 th Sendai virus on the number of Substance P/Neurokinin A-containing vagal afferent neurons whose cel
25 uge NANCi responses, TSM was contracted with neurokinin A in the presence of atropine, propranolol, a
28 ceptors were able to bind to substance P and neurokinin A ligand with similar affinities; however, th
29 ll mice are caused by the extinction of a SP/neurokinin A-mediated signaling pathway that is activate
30 , Lys-bradykinin, Lys-(des-Arg9)-bradykinin, neurokinin A, Met-Lys-bradykinin, histatin 8, and a myos
31 er naturally occurring tachykinins including neurokinin A, neuropeptide K, neuropeptide gamma, eledoi
33 f neuropeptides, including substance P (SP), neurokinin A (NKA), and neurokinin B (NKB), that are enc
35 uperfusion of different tachykinin agonists (neurokinins A (NKA) and B (NKB), SP, and senktide) gave
37 effect of the neuropeptides, substance P and neurokinin A, on nerve growth factor expression in human
38 served with application of either [beta-Ala8]neurokinin A or senktide, selective NK2 and NK3 receptor
39 s there were significantly fewer Substance P/Neurokinin A positive neurons in nodose ganglia from vir
43 C1 (encoding the tachykinins substance P and neurokinin A) to be strongly activated by LP within the
44 ing of fluorescently labeled substance P and neurokinin A was tested on the human astrocytoma cell li
46 Fluorescent and radiolabeled substance P and neurokinin A were internalized similarly by cells expres
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