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1 B > senktide > substance P = neurokinin A > neurokinin B.
2 s to concentrations of neurokinin A (10 nM), neurokinin B (0.1 microM), substance P (1 microM), neuro
3 NA and have elucidated the putative sites of neurokinin B action in the rat central nervous system.
4 he Tac2 gene, which encodes the neuropeptide neurokinin B and its corresponding receptor neurokinin 3
6 es for their natural ligands substance P and neurokinin B but also exhibited surprisingly high affini
7 arked alterations in hypothalamic kisspeptin/neurokinin B/dynorphin (KNDy) neurons, we hypothesized t
8 are also effects at the ARH to disrupt Kiss1/neurokinin B/dynorphin neuronal function through inhibit
9 trophy of neurons expressing substance P and neurokinin B gene transcripts in the infundibular (arcua
10 ing at the NK-3 receptor homolog was [MePhe7]neurokinin B > senktide > substance P = neurokinin A > n
11 hich includes substance P, neurokinin A, and neurokinin B, have three distinct receptors: NK-1, NK-2,
13 h similar rank orders of potency of [MePhe7] neurokinin B = neurokinin B = senktide > NKA = substance
17 Patients with loss-of-function mutation in neurokinin B (NKB) and its receptor show hypogonadotropi
18 Human genetic studies have revealed that neurokinin B (NKB) and its receptor, neurokinin-3 recept
19 determine whether preprodynorphin (Dyn) and neurokinin B (NKB) are coexpressed in Kiss1 neurons in t
20 f neurons expressing the substance P (SP) or neurokinin B (NKB) genes in the human hypothalamus and b
26 derable evidence suggests that dynorphin and neurokinin B (NKB) neurons in the hypothalamic arcuate n
28 ction mutations in the genes encoding either neurokinin B (NKB) or its receptor, NK3 (NK3R), result i
29 procal plots of identical slope when [MePhe7]neurokinin B (NKB) was used as the other competition par
30 ng substance P (SP), neurokinin A (NKA), and neurokinin B (NKB), that are encoded by the tac1 (SP and
31 conservation and physiological functions of neurokinin B (NKB), we identified tachykinin (tac) and t
33 called GPR54), neurokinin B (TAC3), and the neurokinin B receptor (TACR3) result in pubertal failure
38 in receptor (KISS1R, formerly called GPR54), neurokinin B (TAC3), and the neurokinin B receptor (TACR
40 ncluding substance P (SP), neurokinin A, and neurokinin B, which are recognized for their roles in th
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