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1 examination is an essential component of the neurologic examination.
2 brain death and are not a substitute for the neurologic examination.
3 brain death and are not a substitute for the neurologic examination.
4 one of many normal eye movements seen on the neurologic examination.
5 of 244 patients had peripheral neuropathy by neurologic examination.
6 urotoxicity (FACT/GOG-Ntx) questionnaire and neurologic examination.
7 gh medical history and complete physical and neurologic examination.
8 eurocritical care as they allow for frequent neurologic examinations.
9 5/8; p < 0.03), and all survivors had normal neurologic examinations.
10 serial magnetic resonance imaging scans and neurologic examinations.
11 years of age or older, were given structured neurologic examinations.
12 e brain injury is reliant on features of the neurologic examination, anatomical and physiological cha
14 on interval may be shortened, and the second neurologic examination and apnea test (or all components
15 ion interval may be shortened and the second neurologic examination and apnea test (or all components
16 and medications that can interfere with the neurologic examination and apnea testing should be disco
17 and medications that can interfere with the neurologic examination and apnea testing should be disco
19 c work-up, including a complete physical and neurologic examination and cervical spine radiographs.
20 Main Outcomes and Measures: Quantitative neurologic examination and diffusion tensor imaging perf
21 hy controls were administered a standardized neurologic examination and measures of cognition, depres
22 n were prospectively assessed (by means of a neurologic examination and the Bayley Scales of Infant D
23 completed the study without change in their neurologic examination and with activation seen in eight
25 ments, including neuropsychological testing, neurologic examination, and clinical and medical history
26 uction, central motor conduction, a clinical neurologic examination, and cognitive function were asse
27 C]PiB PET, magnetic resonance imaging (MRI), neurologic examination, and detailed cognitive testing u
28 erwent DWI, comprehensive ophthalmologic and neurologic examination, and diagnostic evaluations for t
29 sis of the clinical features in the history, neurologic examination, and neuroimaging studies led to
30 underwent assessment with a nurse interview, neurologic examination, and neuropsychological testing.
31 he hospital, impede efforts to perform daily neurologic examinations, and increase the need for tests
34 ) had "questionable" or abnormal findings on neurologic examination, as compared with 48 controls (29
37 -free controls (n = 66) underwent systematic neurologic examination by a neurologist blinded to disea
38 ave a magnetic resonance image of the brain, neurologic examination by a neurologist, and cognitive t
39 netic resonance angiography (MRA) studies, a neurologic examination by a pediatric neurologist, and c
40 here is uncertainty about the results of the neurologic examination; c) if a medication effect may be
41 here is uncertainty about the results of the neurologic examination; (c) if a medication effect may b
46 l intensity within 1 hour and who had normal neurologic examination findings, the Ottawa SAH Rule was
47 annual, standardized neuropsychological and neurologic examination findings, using criteria from the
52 iven the challenges of performing a reliable neurologic examination in children, neuroimaging might b
55 lowing protocol: a standardized physical and neurologic examination in the emergency department, cran
56 f the SPECT scan, DLB patients had a routine neurologic examination including Hoehn and Yahr grading
57 n patients with TTR-FAP underwent a complete neurologic examination, including Neuropathy Impairment
58 operative evaluation, medication history and neurologic examination, intraoperative awareness of cond
59 urosensory toxicity of oxaliplatin, detailed neurologic examination, needle electromyography (EMG), a
61 was designed to establish the reliability of neurologic examination, neuron-specific enolase (NSE), a
63 aphics, genetic testing, symptom evaluation, neurologic examination, neuropsychological bedside testi
64 nt of leukocyte galactocerebrosidase levels, neurologic examinations, neuropsychological tests, magne
65 formance was assessed using the Physical and Neurologic Examination of Subtle Signs (PANESS), with hi
67 ining written, informed consent and baseline neurologic examinations, patients were randomized to tre
72 rom baseline and achieved stable or improved neurologic examination score and Karnofsky performance s
75 infarcts on magnetic resonance imaging and a neurologic examination showing no abnormalities correspo
76 cal features, including presenting symptoms, neurologic examination signs, neuropsychological perform
77 ow Coma Scale and normal findings on a brief neurologic examination; the patients then underwent CT.
78 r predictor of intracranial injury than is a neurologic examination, though controversy continues as
84 r, point tenderness at T10-T12, and a normal neurologic examination with preserved lower extremity st
85 MRI) of the brain in the setting of a normal neurologic examination without a history or physical fin
86 olled 56 subjects after a brief survey and a neurologic examination yielding normal Subjects were dir
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