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1 mammalian paralogs, PEX11beta, results in a neurological disorder.
2 an abnormal cortical event that underlies a neurological disorder.
3 Epilepsy is a serious and common neurological disorder.
4 argeting the BBB can influence the course of neurological disorder.
5 eric methodology can be applied to study any neurological disorder.
6 ation commonly seen in psychiatric and other neurological disorders.
7 iated signaling are associated with numerous neurological disorders.
8 ciated complications, including allergic and neurological disorders.
9 que platforms to model brain development and neurological disorders.
10 associated with various types of cancers and neurological disorders.
11 these areas as biomarkers for behavioral and neurological disorders.
12 ession with synaptic deficits in a number of neurological disorders.
13 roles in brain development, homeostasis, and neurological disorders.
14 nsmitter release and have been implicated in neurological disorders.
15 ltered networks in zebrafish models of human neurological disorders.
16 , inflammatory and autoimmune disorders, and neurological disorders.
17 lvement in diseases as diverse as cancer and neurological disorders.
18 these molecules and their crucial impact on neurological disorders.
19 es may be responsible for various aspects of neurological disorders.
20 to be common to astrocyte reactions in other neurological disorders.
21 decline of which is a major feature of many neurological disorders.
22 temic delivery of drugs for the treatment of neurological disorders.
23 nociception and are implicated in a range of neurological disorders.
24 whose functions may be compromised in human neurological disorders.
25 mediates excitotoxicity associated with many neurological disorders.
26 tical, early event in many acute and chronic neurological disorders.
27 ered in the diagnosis and study of childhood neurological disorders.
28 roach to the treatment of stroke and related neurological disorders.
29 rs and effectively facilitate BBB closure in neurological disorders.
30 modalities for the treatment of cancers and neurological disorders.
31 beneficial for the studies of T2D as well as neurological disorders.
32 get of therapeutic intervention for relevant neurological disorders.
33 Repeat expansions cause dominantly inherited neurological disorders.
34 inding proteins have been linked to numerous neurological disorders.
35 rt is a characteristic feature of a range of neurological disorders.
36 ts association with microcephaly and serious neurological disorders.
37 review the use of ASOs for the treatment of neurological disorders.
38 GABA currents is associated with a number of neurological disorders.
39 mediators in neoplastic, cardiovascular and neurological disorders.
40 peutic value against pain and probably other neurological disorders.
41 .3 function has been associated with several neurological disorders.
42 TLs located near GWAS signals for immune and neurological disorders.
43 and provide new therapeutic perspectives for neurological disorders.
44 iates, and three control cases with no known neurological disorders.
45 many therapeutic strategies aiming to treat neurological disorders.
46 diting as a potential therapy to treat human neurological disorders.
47 nic mechanisms on the basis of PRRT2-related neurological disorders.
48 ons (20-50 Hz) have been observed in several neurological disorders.
49 ment of multiple sclerosis and other chronic neurological disorders.
50 treatment for refractory epilepsy and other neurological disorders.
51 ub-threshold GWAS peaks in immunological and neurological disorders.
52 opment, modulate behavior, and contribute to neurological disorders.
53 tive ubiquitination is implicated in several neurological disorders.
54 ered before as a therapeutic alternative for neurological disorders.
55 apeutically in treating neuropathic pain and neurological disorders.
56 lead to a broad range of slowly progressive neurological disorders.
57 teract Alzheimer's disease and several other neurological disorders.
58 h deficits in the balance contribute to many neurological disorders.
59 arch processes of key regulators involved in neurological disorders.
60 ty associated with various acute and chronic neurological disorders.
61 ind the neuropsychiatric sequelae of certain neurological disorders.
62 ormalities shared by RTT and other postnatal neurological disorders.
63 aking are evident in several psychiatric and neurological disorders.
64 ne disease (MSUD), autism, and other related neurological disorders.
65 n emerging therapeutic target for cancer and neurological disorders.
66 rapeutic targets for many cardiovascular and neurological disorders.
67 with hemorrhagic stroke, seizures, and other neurological disorders.
68 (FDA) licensure 2005-2012 inclusive in seven neurological disorders.
69 S on the etiology and pathology of different neurological disorders.
70 he nervous system and are implicated in many neurological disorders.
71 n B1 in the oxidative damage associated with neurological disorders.
72 in neuronal positioning lead to a variety of neurological disorders.
73 sing sleep may benefit patients with certain neurological disorders.
74 source of cells for regenerative medicine in neurological disorders.
75 blood-brain barrier (BBB) for treating many neurological disorders.
76 bunits have been associated with an array of neurological disorders.
77 mportant therapeutic target for a variety of neurological disorders.
78 and, potentially, selective vulnerability in neurological disorders.
79 nds as interesting approach for treatment of neurological disorders.
80 ssociated with numerous diseases, especially neurological disorders.
81 st care for patients with a range of chronic neurological disorders.
82 f intracellular vesicles and are involved in neurological disorders.
83 olic and cardiovascular diseases, as well as neurological disorders.
84 cteristic feature of several psychiatric and neurological disorders.
85 mutation has previously been associated with neurological disorders.
86 protection in numerous preclinical models of neurological disorders.
87 entified in numerous patients with mental or neurological disorders.
88 e endoplasmic reticulum, develop spontaneous neurological disorders.
89 hat are modified during learning, aging, and neurological disorders.
90 d therapeutic procedures for a wide range of neurological disorders.
91 treating SRS and other polyamine-associated neurological disorders.
92 basal ganglia function and the treatment of neurological disorders.
93 ng novel therapeutic strategies for treating neurological disorders.
94 of local translation is associated with many neurological disorders.
95 on is an early and prominent feature of many neurological disorders.
96 or serious adverse events were postoperative neurological disorders (4 patients) and postoperative re
99 otein 2 (MeCP2) cause Rett syndrome (RTT), a neurological disorder affecting cognitive development, r
102 children with sudden onset of a progressive neurological disorder and regression of developmental mi
103 amples from 25 patients with noninflammatory neurological disorders and 9 with neuroborreliosis serve
104 ere, we provide as-MoCA that is valid across neurological disorders and can be administered in approx
106 with the pathogenesis of inherited monogenic neurological disorders and complex multifactorial diseas
109 h tell us more about potentially devastating neurological disorders and extend our understanding of c
110 errant synaptic activity is observed in many neurological disorders and has been suggested as an impo
111 represents a novel target in paraneoplastic neurological disorders and has high predictive value for
112 ptors are also implicated in psychiatric and neurological disorders and have received considerable at
114 tive and behavioral symptoms associated with neurological disorders and neuropsychiatric disease.
116 the emerging idea of a systemic influence to neurological disorders and point to the need for future
117 elian Genomics, and US National Institute of Neurological Disorders and Stroke (NINDS) grant R01NS058
118 ipants who started the National Institute of Neurological Disorders and Stroke Exploratory Trials in
119 w Group meeting of the National Institute of Neurological Disorders and Stroke, emphasizes the intima
121 Institutes of Health, National Institute of Neurological Disorders and Stroke, National Center for A
122 al Institute on Aging, National Institute of Neurological Disorders and Stroke, the Michael J Fox Fou
124 for uncovering the genetic causes of complex neurological disorders and the fundamental mechanisms un
125 y (SCI) remains one of the most debilitating neurological disorders and the majority of SCI patients
126 cognitive function, may underpin a number of neurological disorders and thereby highlight the importa
127 atory optic neuropathies-are associated with neurological disorders and thus are often diagnosed and
128 tion is increasingly being employed to treat neurological disorders and to enhance human performance.
129 in acid-mediated neuronal injury and various neurological disorders, and a promising lead for the dev
130 ic diseases, including inherited myopathies, neurological disorders, and cancer, PI-converting enzyme
133 bility exists in the presentation of complex neurological disorders, and the study of single nucleoti
137 ds, represent promising therapeutic tools in neurological disorder as well as for relief of pain or i
138 arizations (SDs) are recognized as actors in neurological disorders as diverse as migraine and trauma
139 QA 10-40microM is a clear indicator of many neurological disorders as well as deficiency of vitamin
140 QA ~10-40microM is a clear indicator of many neurological disorders as well as deficiency of vitamin
141 Migraine is a highly prevalent and disabling neurological disorder associated with a wide range of ps
144 expression is markedly decreased in various neurological disorders associated with epileptiform acti
145 GCPII) is effective in preclinical models of neurological disorders associated with excessive activat
147 DGRG1 (G1; also known as GPR56) underlie the neurological disorder bilateral frontoparietal polymicro
148 of tonic inhibition are associated with some neurological disorders but there are no specific ligands
149 ptor (NMDAR) activity contributes to several neurological disorders, but direct antagonism is poorly
150 a central factor in the development of many neurological disorders, but much less is known about the
151 Ion channel dysfunction causes a range of neurological disorders by altering transmembrane ion flu
152 pment of improved gene therapies for various neurological disorders by exploring why microglia destro
153 ls that share transcriptomic signatures with neurological disorders, by exposing mouse cortical neuro
155 ts and in experimental models mimicking this neurological disorder can be classified, based on their
156 model HD and show that the progression of a neurological disorder can be predicted for individual pa
157 e models of Rett syndrome (RTT), a postnatal neurological disorder caused by loss-of-function mutatio
162 dy point to a potential mechanism underlying neurological disorders caused by hnRNP U mutations.
163 sorders are severe, usually life-shortening, neurological disorders caused by nucleotide expansions,
164 spongiform encephalopathies (TSEs) are fatal neurological disorders caused by prions, which are compo
165 own to be critical for combating a number of neurological disorders caused by protein aggregation, su
166 eizures.SIGNIFICANCE STATEMENT Epilepsy is a neurological disorder characterized by an imbalance betw
167 ough syndrome (CMCS), an autosomal recessive neurological disorder characterized by early-onset senso
168 red EIB in humans, we focused on epilepsy, a neurological disorder characterized by hyperexcitable ne
169 the different forms of inherited dystonia, a neurological disorder characterized by involuntary, disa
170 Chorea-acanthocytosis (ChAc) is a fatal neurological disorder characterized by red blood cell ac
171 MECP2 cause Rett syndrome (RTT), an X-linked neurological disorder characterized by regressive loss o
172 resected from individuals with MTLE, a major neurological disorder characterized by seizures, excitot
173 ditary spastic paraplegias are heterogeneous neurological disorders characterized by a pyramidal synd
174 ive stimulation methods for the treatment of neurological disorders characterized by abnormal neurona
177 ycoconjugates is known to be associated with neurological disorders, CNS injury, and brain cancer.
178 are associated with the autosomal-recessive neurological disorder Cockayne syndrome, which is charac
179 e will be summarized, and finally, we review neurological disorders commonly associated with cardiova
180 ic roles of genes influencing both sleep and neurological disorders could lead to new treatment strat
181 eview we will present evidence from multiple neurological disorders demonstrating that changes in ast
182 dered a characteristic feature of a group of neurological disorders described as synucleinopathies.
183 n 2,141 patients who had rapidly progressive neurological disorders, determined diagnostic sensitivit
184 patients with OMS at a center for autoimmune neurological disorders done between January 2013 and Sep
187 rcolepsy can be confused with those of other neurological disorders (eg, epilepsy), posing diagnostic
188 m HAART still develop various HIV-associated neurological disorders, even when the viral load is low.
190 ents are predisposing factors for functional neurological disorder (FND) and post-traumatic stress di
191 ealth-related quality of life) in functional neurological disorder (FND), and the salience network is
192 s an essential factor in the pathogenesis of neurological disorders following acute systemic inflamma
195 mosquito-borne Zika virus was a cause of the neurological disorder Guillain-Barre syndrome (GBS) and
196 V), a re-emerging flavivirus associated with neurological disorders, has spread rapidly to more than
198 including various cancers, developmental and neurological disorders, however, the shared biology and
199 GLRB gene mutations are associated with the neurological disorder hyperekplexia characterized by a g
201 Cys and Tyr429*-induced severe, early-onset, neurological disorders in distinct human populations.
203 ew mechanistic hint to previously recognized neurological disorders in patients with inherited DNA-mi
204 the motor cortex to regain locomotion after neurological disorders in rats suggests that cortical en
205 has important implications for understanding neurological disorders in which one form of sensory atte
206 may extend to multiple neuropsychiatric and neurological disorders in which RELN deficiency is a com
207 with sleep disorders are also implicated in neurological disorders in which sleep abnormalities are
208 s are examples such as cardiovascular decay, neurological disorders including Alzheimer's disease, an
209 subject to change in synaptic plasticity and neurological disorders including autism spectrum disorde
210 ngers (NHEs) NHE6 and NHE9 are implicated in neurological disorders including Christianson syndrome,
211 ehavior is disrupted in neuropsychiatric and neurological disorders including developmental disorders
212 Aberrant KCC2 function contributes to human neurological disorders including epilepsy and neuropathi
214 common neuropathological feature in several neurological disorders, including amyotrophic lateral sc
215 and synapse impairments are features of many neurological disorders, including autism spectrum disord
216 the tubulins, are known to cause an array of neurological disorders, including lissencephaly, polymic
217 in some models of monogenic loss-of-function neurological disorders, including loss of MeCP2 in Rett
218 sociated with the pathophysiology of various neurological disorders, including multiple sclerosis.
219 ns, and they have important implications for neurological disorders, including Parkinson's disease an
220 lepsy, neurodegenerative diseases, and other neurological disorders, including schizophrenia and atte
221 X7R) provides neuroprotection toward various neurological disorders, including stroke, traumatic brai
222 undergo important phenotypic changes in many neurological disorders, including strokes, trauma, infla
224 , the neuropathy-specific Quality of Life in Neurological Disorders instrument, and the short-form Mc
225 ility of differentiating it from other acute neurological disorders is likely to improve as we learnt
226 t syndrome (RTT; OMIM 312750), a progressive neurological disorder, is caused by mutations in methyl-
229 nfantile hypertrophic cardiomyopathy and the neurological disorder Leigh syndrome result in impaired
230 duals can also experience hemorrhagic fever, neurological disorders, liver failure, and blindness, wh
231 s of AMPK with the development of cancer and neurological disorders, making it a potential therapeuti
233 1L, PTPRN2, CMTM8 and CELF2, associated with neurological disorders, NGFR and HLA-DRB6, associated wi
235 ka virus (ZIKV) infection is associated with neurological disorders of both the CNS and peripheral ne
236 emerging as a critical mechanism underlying neurological disorders of various etiologies, yet its ro
237 however, our understanding of the effects of neurological disorders on the cardiovascular system has
239 se viruses prove to be pathogenic in cancer, neurological disorders, or other diseases associated wit
241 cent cluster of microcephaly cases and other neurological disorders reported in Brazil a global publi
244 nesis and may shed light on MAP7 function in neurological disorders.SIGNIFICANCE STATEMENT Neurons co
245 d function of meningeal lymphatic vessels in neurological disorders, specifically multiple sclerosis
246 ncluding rare monogenic diseases and complex neurological disorders such as Alzheimer's disease and b
248 pes has been implicated in a wide variety of neurological disorders such as Alzheimer's disease, schi
249 se elevated level in serum is marker of many neurological disorders such as Alzheimer's, Huntington's
252 eteriorates, and these defects can result in neurological disorders such as hereditary spastic parapl
253 synaptic plasticity imbalance that underlies neurological disorders such as intellectual disability.
254 de new insights into the dynamical causes of neurological disorders such as Parkinson's disease and S
255 inhibitors, which can be valuable assets for neurological disorders such as Parkinson's disease, due
256 ry important for aiding our understanding of neurological disorders such as schizophrenia, functional
257 gy as well as in the pathogenesis of several neurological disorders such as stroke, spinal cord injur
259 as attractive drug targets for treatments of neurological disorders, such as Alzheimer's disease and
260 and its potential easily translates to other neurological disorders, such as Alzheimer's disease, in
261 s mutations in glypicans are associated with neurological disorders, such as autism and schizophrenia
262 neuronal activity, are associated with many neurological disorders, such as epilepsy, Parkinson's di
263 f Computational Psychiatry's applications to neurological disorders, such as Parkinson's disease, and
264 in perturbations are associated with various neurological disorders, such as Parkinson's, autism, and
265 ment observed in a number of psychiatric and neurological disorders, such as schizophrenia and Alzhei
266 twork and cognitive deficits associated with neurological disorders, such as schizophrenia, that resu
267 paired motivation is present in a variety of neurological disorders, suggesting that decreased motiva
268 sociated tremor/ataxia syndrome (FXTAS) is a neurological disorder that affects premutation carriers
269 rls who developed a peculiar and devastating neurological disorder that later came to bear his name.
270 ATEMENTSCN8A encephalopathy is a devastating neurological disorder that results from de novo mutation
271 sis of several developmental and adult-onset neurological disorders that affect the hippocampus, sugg
272 thus reveal a novel therapeutic strategy for neurological disorders that are accompanied by Kv2.1-fac
273 re its pathological roles in psychiatric and neurological disorders that have long been underestimate
274 Although multiple sclerosis is a common neurological disorder, the origin of the autoimmune resp
276 main causes: overlap between psychiatric and neurological disorders; the grouping of suicide and self
277 n some brain regions and involved in various neurological disorders, their functional interactions ha
278 ive stress is a pathological feature of many neurological disorders; therefore, utilizing proteins th
279 in mutations have been implicated in various neurological disorders, these findings also point toward
282 energy metabolism is important in almost all neurological disorders, to which a decrease in the capac
283 n abnormal intracellular inositol levels and neurological disorders, very little is known about the r
284 In brain slices of a mouse model of this neurological disorder, we demonstrate, characterize, and
285 al examples of well-characterized RNA-driven neurological disorders, we demonstrate how designed smal
286 t of central nervous system trauma and other neurological disorders where p75 neurotrophin receptor s
287 the cortex, a region of importance for most neurological disorders, where dendrites of each neuronal
288 ing, brain-computer interfaces, treatment of neurological disorders, where high resolution and simult
290 gated tau protein is associated with over 20 neurological disorders, which include Alzheimer's diseas
291 Huntington's disease (HD) is a progressive neurological disorder whose non-motor symptoms include s
292 These results have implications for other neurological disorders whose pathology includes IF accum
293 disease (CMT) is the most commonly inherited neurological disorder with a prevalence of 1 in 2500 peo
295 estless legs syndrome is a prevalent chronic neurological disorder with potentially severe mental and
296 a suggest that RTT should be classified as a neurological disorder with systemic metabolic components
298 Synuclein (alpha-syn) has been implicated in neurological disorders with parkinsonism, including Park
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