ライフサイエンスコーパス: neurological symptom

1 neuron populations required to ameliorate a neurological symptom.

2 and in most of the cases subsequent multiple neurological symptoms.

3 both systemic disease and, in some patients, neurological symptoms.

4 activity were observed, even after onset of neurological symptoms.

5 mild TBI, might be associated with long-term neurological symptoms.

6 e (CS) is a multisystem disorder with severe neurological symptoms.

7 with failure to thrive, immunodeficiency and neurological symptoms.

8 Seventy patients (66.0%) developed severe neurological symptoms.

9 76) and were mostly mild; only 1 neonate had neurological symptoms.

10 erature comparing PNES with other functional neurological symptoms.

11 There was absence of fever and neurological symptoms.

12 raphic imaging within 3 months after LTx for neurological symptoms.

13 ler study and can predict the development of neurological symptoms.

14 netic disorders marked by severe somatic and neurological symptoms.

15 oss of MeCP2 expression results primarily in neurological symptoms.

16 pathways that may be modulated to treat the neurological symptoms.

17 t stroke in any patient with abrupt onset of neurological symptoms.

18 ll mice before and after the onset of severe neurological symptoms.

19 with genetic syndromes that frequently have neurological symptoms.

20 y, the patient experienced an improvement in neurological symptoms.

21 nd development of some of the characteristic neurological symptoms.

22 syndrome (GLUT1-DS) leads to a wide range of neurological symptoms.

23 in some types, mental retardation and other neurological symptoms.

24 neuropil aggregate formation and ameliorates neurological symptoms.

25 creasing serum viral loads and resolution of neurological symptoms.

26 s, myelin and axonal injury, and progressive neurological symptoms.

27 aine appear to reduce the risk for transient neurological symptoms.

28 and cattle, invariably die after exhibiting neurological symptoms.

29 ging abnormalities that accompany these same neurological symptoms.

30 usually fatal disease accompanied by severe neurological symptoms.

31 lting in an autoimmune attack on neurons and neurological symptoms.

32 with recurrent thrombosis, miscarriages, and neurological symptoms.

33 ronal nuclear inclusions and appeared before neurological symptoms.

34 and subcellular disease changes may mediate neurological symptoms.

35 deficits precede the onset of psychiatric or neurological symptoms.

36 In piglets and mice, Stx can induce fatal neurological symptoms.

37 y include progressive hearing loss and other neurological symptoms.

38 green fluorescent protein (GFP) exhibited no neurological symptoms.

39 tingtin accumulation and develop progressive neurological symptoms.

40 iscordance and escape may be associated with neurological symptoms.

41 is highly correlated with the development of neurological symptoms.

42 ted, solid mesenteric tumors with no obvious neurological symptoms.

43 kers, but there was no clear relationship to neurological symptoms.

44 n shown to contribute to the age at onset of neurological symptoms.

45 aplastic carcinoma), was admitted with focal neurological symptoms.

46 since murine models fail to mirror classical neurological symptoms.

47 g to motor neuron cell death and progressive neurological symptoms.

48 with some patients manifesting non-cognitive neurological symptoms.

49 nmental and genetic factors that cause these neurological symptoms.

50 ervous system nocardiosis, 13 (43.3%) had no neurological symptoms.

51 lowly growing VT in a woman with progressive neurological symptoms.

52 w dysfunction in their absence underlies FXS neurological symptoms.

53 ll patients with meningovascular NS had only neurological symptoms.

54 lactosemia disease states manifest long-term neurological symptoms.

55 bination with abnormal copper metabolism and neurological symptoms.

56 found between key brain findings and overall neurological symptoms.

57 stations of liver disease, but often worsens neurological symptoms.

58 sorder characterized by immunodeficiency and neurological symptoms.

59 episode of acute onset atypical or nonfocal neurological symptoms.

60 atory, gastrointestinal, cardiovascular, and neurological symptoms.

61 with time, peaking during the height of the neurological symptoms (1.5E copies/mL).

62 ported a minor infection a few days prior to neurological symptoms, 2 patients presented criteria for

63 less than 10 months after the onset of their neurological symptoms (3.7 +/- 2.5 months, median +/- in

64 aining 30% had persistent hypertension (9%), neurological symptoms (4%), decreased glomerular filtrat

65 interfere with DNA repair but have only mild neurological symptoms [6-9], suggesting that the link be

66 had small-cell lung cancer (94%), multifocal neurological symptoms (78%) and a poor neurological outc

67 , 179 cancer patients without paraneoplastic neurological symptoms, 96 patients with paraneoplastic s

68 logical cases were characterized by relevant neurological symptoms, a history of a past head injury w

69 gy of FMD (and by extension other functional neurological symptoms): abnormal attentional focus, abno

70 ed; once injury is suspected on the basis of neurological symptoms, additional forms of intervention

71 ctors of mortality include presentation with neurological symptoms (adjusted odds ratio, 16.0; P<0.00

72 ions with self-report measures of functional neurological symptoms, adverse life events and PTSD symp

73 Rebound syndrome was defined as new severe neurological symptoms after ceasing fingolimod treatment

74 Seven patients developed neurological symptoms; all of them had a positive RLS (P

75 r sibling was a 70-year-old man with minimal neurological symptoms and a mild depressive disorder.

76 including an additional 10 items on specific neurological symptoms and an additional 5 items on menta

77 ain to environmental insult causes different neurological symptoms and behavioral outcomes depending

78 sclerosis (TSC), a hereditary syndrome with neurological symptoms and benign hamartoma tumours in th

79 agnosis and aggressive treatment can improve neurological symptoms and can substantially enhance a pa

80 , a site 2 neurotoxin that may induce severe neurological symptoms and cardiovascular collapse.

81 ialic acid (SA) receptor specificity, caused neurological symptoms and death in ferrets inoculated wi

82 upper and lower motor neurons (LMNs) causes neurological symptoms and dysfunctions in motor neuron d

83 neuronal and glial cells display more severe neurological symptoms and earlier death than N171-82Q mi

84 PLP-150Q mice show progressive neurological symptoms and early death, as well as age-de

85 lase inhibitor prevents them from developing neurological symptoms and extends their life span threef

86 ve resulted in the subsequent improvement of neurological symptoms and impairments.

87 individuals with GTPBP3 mutations developed neurological symptoms and MRI involvement of thalamus, p

88 nical course was complicated by debilitating neurological symptoms and multiple hospitalizations lead

89 These mice showed weight loss, progressive neurological symptoms and neurodegeneration before early

90 spectively; p < 0.0001) and experienced more neurological symptoms and other end-organ ischemic event

91 rol ligand, act in concert to delay onset of neurological symptoms and prolong the lifespan of npc1(-

92 n stress-related neuroplasticity, functional neurological symptoms and reduced insular volume was ide

93 diagnosis of multiple sclerosis is based on neurological symptoms and signs, alongside evidence of d

94 boratory-confirmed brucellosis cases who had neurological symptoms and signs, and 48 (37.5%) were dia

95 tt syndrome, a disease characterized by both neurological symptoms and systemic abnormalities.

96 We examined the neurological symptoms and the causal tumours in 50 patie

97 linical interest in patients with functional neurological symptoms and the clear shift in diagnostic

98 ated the prevalences of various non-amnestic neurological symptoms and the contributions of age of on

99 We hypothesised that functional neurological symptoms and the magnitude of childhood abu

100 erval of <4 years between the development of neurological symptoms and tumour diagnosis; (iii) exclus

101 size >5 cm, tumour crossing the midline, or neurological symptoms), and without known HIV infection,

102 size >5 cm, tumour crossing the midline, or neurological symptoms), and without known HIV infection,

103 All patients with late NS had neurological symptoms, and 2 (40%) also had ocular sympt

104 had ophthalmological symptoms, 14 (47%) had neurological symptoms, and 8 (26%) had both.

105 ncluding ichthyosis, primary hypothyroidism, neurological symptoms, and cryptorchidism.

106 ical characteristics, time to development of neurological symptoms, and modified Rankin Scale (mRS) s

107 ients manifested acute episodes of transient neurological symptoms, and neurological impairment was m

108 poor health, infectious diseases, insomnia, neurological symptoms, and suicide attempts during early

109 is, having a history of encephalitis, subtle neurological symptoms, and/or abnormal results on ancill

110 Neurological symptoms are common manifestations of Lyme

111 Functional (psychogenic) neurological symptoms are frequently encountered in neur

112 local anaesthetics with respect to transient neurological symptoms are limited and conflicting.

113 disease but they die from cancer before the neurological symptoms are manifested.

114 nt and postpartum patients with headache and neurological symptoms are often diagnosed with pre-eclam

115 Neurological symptoms are the predominant problem and th

116 (PNES) are classified with other functional neurological symptoms as 'Conversion Disorder', but ther

117 left anterior insular volume and functional neurological symptoms as measured by the Patient Health

118 ement alone (ie, pharmacological therapy for neurological symptoms as needed).

119 RBPs and associated factors lead to diverse neurological symptoms, as characterized by neurodevelopm

120 ity, and thus contribute to the emergence of neurological symptoms associated with ASD.

121 Patients can also have neurological symptoms associated with coagulopathies, im

122 However, in many cases it leads to neurological symptoms associated with haemodynamic distu

123 minergic systems in brain which may underlie neurological symptoms associated with human disorders su

124 g normal brain function, thus explaining the neurological symptoms associated with IP.

125 ed piglets had diarrhea and the severe fatal neurological symptoms associated with Stx2 intoxication.

126 after bacterial challenge, followed by fatal neurological symptoms at 48 to 96 h.

127 ich Mecp2 was deleted in brain showed severe neurological symptoms at approximately six weeks of age.

128 h MS more often show disabling and polyfocal neurological symptoms at disease onset than adults with

129 One patient (1.4%) still had major neurological symptoms at the latest follow-up.

130 inely done in patients presenting with acute neurological symptoms because this might shed light on t

131 Duration of neurological symptoms before diagnosis was 30 (interquar

132 Surprisingly, despite the lack of neurological symptoms, beta3A-deficient mouse brain poss

133 ainstem lesions on imaging, who had onset of neurological symptoms between Aug 1, 2014, and Oct 31, 2

134 This leads to severe neurological symptoms, but the mechanisms underlying bra

135 Forty percent of AIDS patients suffer from neurological symptoms, but the selective profile of dama

136 ll had a prodromal febrile illness preceding neurological symptoms by a median of 7 days (IQR 5.75-8)

137 plasmic mutant huntingtin and its associated neurological symptoms by preventing the accumulation of

138 tations range from itching and minor pain to neurological symptoms, cardiac collapse, and death.

139 eolysis, severe osteoporosis, short stature, neurological symptoms, cardiovascular defects, and polyc

140 Lesch-Nyhan syndrome encompasses a host of neurological symptoms, caused by a deficiency of the pur

141 tment with glucocorticoid analogues improved neurological symptoms characteristic of this syndrome.

142 g body of evidence indicating that transient neurological symptoms commonly occur after uneventful li

143 continually pregnant showed greatly improved neurological symptoms compared to affected females that

144 brains of infected mice, producing no major neurological symptoms, compared with the invariably leth

145 dults with low IQs than do those who develop neurological symptoms consistent with encephalopathy.

146 atients with acute Zika virus disease and no neurological symptoms (control group 2; n=70).

147 with HuAb negative patients with respect to neurological symptoms, course of the neurological disord

148 e accumulation of heparan sulfate results in neurological symptoms, culminating in extensive neurodeg

149 al diagnostic term for medically unexplained neurological symptoms (currently known as 'conversion di

150 The median delay from neurological symptom debut to first hospital contact was

151 ction, Catecholamines-up, delay the onset of neurological symptoms, dopaminergic neuron death, and mo

152 rful array of antiretroviral treatments, but neurological symptoms due to comorbid conditions, includ

153 atients with progressive subacute to chronic neurological symptoms due to congestive myelopathy cause

154 Patients often suffer from neurological symptoms due to obstruction of cranial fora

155 a possible mechanism for the development of neurological symptoms during panic attacks.

156 article reviews the treatment of functional neurological symptoms during World War I by Lewis Yealla

157 features range from none, to discrete focal neurological symptoms (eg, stroke), to insidious global

158 D50 of BoNT/C1 ad is 5 mg/kg, with transient neurological symptoms emerging at sub-lethal doses.

159 s a primary means by which seizure and other neurological symptoms ensue, our findings highlight oxLD

160 nal pain, vomiting, diarrhea, or nausea) and neurological symptoms (extremity paresthesia, arthralgia

161 Functional neurological symptoms (FNS) are commonly encountered but

162 Functional (conversion) neurological symptoms (FNS) are commonly encountered in

163 Partial improvement of the neurological symptoms following cancer removal was assoc

164 ng, and tender hepatomegaly, with a range of neurological symptoms from tinnitus, vertigo, headaches,

165 Neurocognitive function, neurological symptoms, functional independence, and heal

166 Overt neurological symptoms have a relatively late onset (15-2

167 However, recurrent episodes of focal neurological symptoms have been described after initial

168 been associated with several SCLC antigens, neurological symptoms have not been observed in patients

169 ions for RTT are limited to alleviating some neurological symptoms; hence, more effective therapeutic

170 Neurological symptoms improved in all patients who survi

171 tration of Cu(II)(atsm) delayed the onset of neurological symptoms, improved locomotive capacity and

172 with persistence of patient-reported sensory neurological symptoms in 25% of patients.

173 Cancer anteceded neurological symptoms in 4 of 28 patients.

174 present in 65% of all patients with NS, and neurological symptoms in 60%.

175 A2AR activity therapeutically can attenuate neurological symptoms in ADK deficiency.

176 A2AR activity therapeutically can attenuate neurological symptoms in ADK deficiency.

177 partly protected against the development of neurological symptoms in an experimental autoimmune ence

178 xpression leads to striking loss of advanced neurological symptoms in both immature and mature adult

179 mutation should include questions regarding neurological symptoms in both older male and female carr

180 f gene regulatory networks as a cause of the neurological symptoms in CS.

181 were associated with a greater frequency of neurological symptoms in DLBCL and BL and with parenchym

182 of one volunteer and produced mild-to-severe neurological symptoms in four others.

183 ecently described the presence of late-onset neurological symptoms in male carriers of premutation (F

184 Late-onset neurological symptoms in MBP-N51/KC mice may indicate un

185 ATA box-binding protein causes age-dependent neurological symptoms in mice and the degeneration of ce

186 (160Q) in astrocytes is sufficient to induce neurological symptoms in mice.

187 to estimate the prevalences of non-amnestic neurological symptoms in participants with ADAD.

188 why the same degree of azotemia causes fewer neurological symptoms in patients with chronic as oppose

189 ic Gaucher disease (nGD) manifests as severe neurological symptoms in patients with no effective trea

190 should be considered in the setting of acute neurological symptoms in patients with renal failure, bl

191 Perfusion abnormalities are associated with neurological symptoms in patients with SCD, whether or n

192 entiate between the specific causes of acute neurological symptoms in pregnant and post-partum patien

193 Acute neurological symptoms in pregnant and post-partum women

194 lular LB/LN-like inclusions and the onset of neurological symptoms in recipient animals.

195 (ADAD) is a rare disorder with non-amnestic neurological symptoms in some clinical presentations.

196 ciency, HIV-1 has been linked to a number of neurological symptoms in the absence of opportunistic in

197 d broadly applicable approach for localizing neurological symptoms in the setting of brain lesions.

198 etary modification partially rescued overall neurological symptoms in these mice after onset.

199 Neurological symptoms in tuberous sclerosis complex (TSC

200 ve even when administered after onset of the neurological symptoms, in young adult mice, and without

201 disorder that targets NMDARs, causing severe neurological symptoms including hallucinations, psychosi

202 A common phenotype of severe neurological symptoms including intractable epilepsy and

203 ated in adulthood, the mice displayed severe neurological symptoms including tremors and hind-limb pa

204 encephalitis, particularly in the setting of neurological symptoms, including extrapyramidal side eff

205 ges on cerebral neuroimaging and progressive neurological symptoms, including movement disorders and

206 (+) embryos by doxycycline leads to multiple neurological symptoms, including severe epilepsy and pre

207 tion of allopregnanolone delays the onset of neurological symptoms, increases Purkinje and granule ce

208 The etiology of these neurological symptoms is unclear and the function of the

209 ond phase of the disease, after the onset of neurological symptoms, magnetic resonance imaging (MRI)

210 These neurological symptoms may also be due to DST gene mutati

211 osquito-borne flavivirus, has induced severe neurological symptoms more frequently in the elderly pop

212 sented different developmental disorders and neurological symptoms - most often cerebral palsy and ep

213 In only one patient there were no neurological symptoms, most of the children presented di

214 osquito-borne flavivirus, has induced severe neurological symptoms, mostly in the elderly population.

215 rational explanation for the well-described neurological symptoms observed after allo-HSCT.

216 diseases and provides an explanation for the neurological symptoms observed in some patients with SLE

217 2 gene occur in the germ cells with onset of neurological symptoms occurring in early childhood, the

218 was strongly predictive of the occurrence of neurological symptoms (odds ratio, 204; 95% confidence i

219 The most common neurological symptom of tuberous sclerosis complex (TSC)

220 of brain ATM activity in the severity of the neurological symptoms of A-T.

221 The neurological symptoms of ataxia-telangiectasia may thus

222 ubsequent colonization of brain tissues with neurological symptoms of disease, we found similar resul

223 ording to one proposal, many psychiatric and neurological symptoms of FXS result from unchecked activ

224 ynaptic Fragile X proteins contribute to the neurological symptoms of FXS.

225 f polyglutamine proteins are involved in the neurological symptoms of glutamine repeat diseases is un

226 ant huntingtin in glia can contribute to the neurological symptoms of HD.

227 euronal communication and con-tribute to the neurological symptoms of HD.

228 f psychosis correlated with the onset of the neurological symptoms of Huntington's disease, and the a

229 attack, which is clinically defined as focal neurological symptoms of ischaemic origin resolving spon

230 thrin-mediated membrane traffic leads to the neurological symptoms of Lowe syndrome.

231 ain functions and the dynamic changes in the neurological symptoms of RTT.

232 sleep and cognition recognised alongside the neurological symptoms of seizures and autonomic instabil

233 ous organs and the mechanisms underlying the neurological symptoms of the disease remain largely unkn

234 ceptual basis for linking the behavioral and neurological symptoms of the disease with the frequently

235 ely to contribute to the pathogenesis of the neurological symptoms of TSC.

236 lformation (molar tooth sign), together with neurological symptoms of variable expressivity.

237 tently, older SCA17 mice experienced earlier neurological symptom onset and more severe Purkinje cell

238 tients with subacute stroke (3-14 days after neurological symptom onset) underwent MRI examination.

239 detergent-soluble oligomers, which preceded neurological symptom onset, and were found at equivalent

240 ne (32%) were wheelchair-bound 1 month after neurological symptom onset.

241 hyperammonia that, if untreated, results in neurological symptoms or death (ornithine transcarbamyla

242 e or reactive-like state before the onset of neurological symptoms or morphological changes.

243 Development of neurological symptoms or signs occurred in up to 30% of

244 n 5 and 20 mm in diameter without associated neurological symptoms or the need for corticosteroids.

245 ment for epidural abscesses in patients with neurological symptoms or who have not responded to medic

246 the knockout mice but did not cause obvious neurological symptoms, organomegaly, or a reduction in l

247 hemizygotes is characterized by no apparent neurological symptoms other than recurrent seizures remi

248 r mouse model of this disease, we found that neurological symptoms parallel progressive and severe ly

249 should be assessed for unexplained fever and neurological symptoms, particularly if they reside in ar

250 Focal neurological symptoms persisted in 22% of patients at di

251 Neurological symptoms preceded the cancer diagnosis in 6

252 Neurological symptoms preceded the tumour diagnosis in 6

253 dead before one year of age, with prominent neurological symptoms premortem in 26 (38%).

254 esirable potential drug for the treatment of neurological symptoms present in most lysosomal storage

255 ubiquinone) Fe-S protein 4], delays onset of neurological symptoms, reduces neuroinflammation, and pr

256 Episodes of acute atypical or nonfocal neurological symptoms, referred to as transient neurolog

257 owever, once it manifests, particularly with neurological symptoms, reversal by dietary changes or su

258 light of better understanding of functional neurological symptoms, Sacks' experiences deserve to be

259 lanced XCI, which may affect the severity of neurological symptoms seen in these patients; however, w

260 , was independent of sensorimotor functional neurological symptom severity and motor FND subtype.

261 ions with self-report measures of functional neurological symptom severity or childhood abuse.

262 nhibitor of thiamine metabolism that induces neurological symptoms similar to that of Wernicke-Korsak

263 In most patients, recovery from neurological symptoms started within a few hours after o

264 ntial carotid artery narrowing but no recent neurological symptom (stroke or transient ischaemia), th

265 ic (ie, has caused no recent stroke or other neurological symptoms), successful carotid endarterectom

266 at pain spreading along the artery can cause neurological symptoms such as vertigo and syncope.

267 (IP) is a genetic disease leading to severe neurological symptoms, such as epileptic seizures, but n

268 ion of unesterified cholesterol and multiple neurological symptoms, such as vertical supranuclear oph

269 ian of 6 days (IQR 4-10) before the onset of neurological symptoms, suggesting recent Zika virus infe

270 RIAL/METHODS: Thirty-six patients with acute neurological symptoms suggestive of PRES were included i

271 S) is a surgical procedure used to treat the neurological symptoms that are associated with certain n

272 s not cause histopathological CNS lesions or neurological symptoms that can be attributed to the viru

273 y juvenile megaloblastic anaemia, as well as neurological symptoms that may be the only manifestation

274 we presented a very rare eagle syndrome with neurological symptoms that occurred suddenly with cervic

275 racterized by rapid onset of psychiatric and neurological symptoms that often culminate in severe neu

276 ll patients had manifested acute or subacute neurological symptoms; the brain MRIs indicating diffuse

277 Neurological symptoms, time course of the disease, and i

278 itional and widely used approach for linking neurological symptoms to specific brain regions involves

279 'UK-Functional Neurological Symptoms (UK-FNS)' was an informal inaugura

280 Median age of onset of neurological symptoms was 14 years (range 0.5-55 years)

281 ospital death (P:<0.05); the presence of new neurological symptoms was a significant preoperative ris

282 lop Stx-related cerebellar lesions and fatal neurological symptoms, we show that administration of St

283 s between these metabolites and clinical and neurological symptoms were examined at both time points.

284 to the Mayo Clinic (Rochester, MN, USA) with neurological symptoms were included.

285 Mild residual neurological symptoms were present in 21.7% of patients

286 inimal protein is able to prevent or reverse neurological symptoms when introduced into MeCP2-deficie

287 vement of other neurons can cause additional neurological symptoms, which define a diverse set of com

288 r an autosomal recessive disease with severe neurological symptoms, which usually leads to death in e

289 signs (prodromes) and aura (transient focal neurological symptoms) whose origin is thought to involv

290 Of 1047 patients with neurological symptoms, whose sera or CSF were examined f

291 infect multiple avian species, resulting in neurological symptoms with unknown routes of transmissio

292 lsant-withdrawal splenial abnormalities with neurological symptoms, with gabapentin-type anticonvulsa

293 , but showed high antibody titers during the neurological symptoms, with the IgM decrease paralleling

294 Patients with development of neurological symptoms within 12 months of anti-PD-1 ther

295 umor burden manifested by the onset of focal neurological symptoms within 21 days.