ライフサイエンスコーパス: neuromodulation

1 including regions implicated in learning and neuromodulation.

2 anging from disease diagnosis to optogenetic neuromodulation.

3 ronger synaptic changes, possibly because of neuromodulation.

4 n relation to dopaminergic and noradrenergic neuromodulation.

5 ing inflammation, gastric acid secretion and neuromodulation.

6 hat include ongoing spontaneous activity and neuromodulation.

7 task-positive) network (ACN), and changes in neuromodulation.

8 ed outputs after different sensory inputs or neuromodulation.

9 responses that is attenuated by cholinergic neuromodulation.

10 ea share a common dependence on serotonergic neuromodulation.

11 just after birth that exhibit strong GABA(B) neuromodulation.

12 All nervous systems are subject to neuromodulation.

13 ts, increasing interest has focused on focal neuromodulation.

14 ntain motor network output during protracted neuromodulation.

15 t networks into different regimes via direct neuromodulation.

16 n priming may involve persistent peptidergic neuromodulation.

17 ment patterns out of a background of diffuse neuromodulation.

18 a model preparation for the investigation of neuromodulation.

19 es set by a background of diffuse descending neuromodulation.

20 aptic strength, in neuronal structure and in neuromodulation.

21 ts of excitable cell signal transduction and neuromodulation.

22 by cholinergic stimulation, a common form of neuromodulation.

23 gly influenced by repetitive stimulation and neuromodulation.

24 ta and applied endeavors, including targeted neuromodulation.

25 y, and the rate of this firing is subject to neuromodulation.

26 o 16.5; P < .001) than treatment with sacral neuromodulation.

27 ting synaptic strength during plasticity and neuromodulation.

28 ronized cortical activity, and noradrenergic neuromodulation.

29 eflect aging-induced changes in dopaminergic neuromodulation.

30 al resolution are critical to understand ACh neuromodulation.

31 rties of cortical neurons without changes in neuromodulation.

32 Cholinergic neuromodulation, a candidate mechanism for aspects of at

33 rinsic membrane properties but the extent of neuromodulation across the two systems has not been wide

34 uring learning and demonstrate that top-down neuromodulation acts on adult-born neuron survival to mo

35 Knowledge of how cholinergic neuromodulation acts on neurochemically identical but mo

36 Given that cognitive training and neuromodulation affect neuroplasticity, their combinatio

37 Thus, neuromodulation allows for a dynamic adjustment of axona

38 rtant for signal integration and that axonal neuromodulation allows for a dynamic adjustment of signa

39 and nerve types involved in allergen-induced neuromodulation among different organ systems, but gener

40 nds of this operating range, suggesting that neuromodulation among thermosensory neurons maintains co

41 aps in the current understanding of dopamine neuromodulation and aging brain functions and suggest av

42 point to a relationship between pupil-linked neuromodulation and behavioral variability.

43 ad ranging, from pharmacotherapy to invasive neuromodulation and experimental gene and stem cell ther

44 signaling affects neuronal connectivity and neuromodulation and have identified AKT as a key signali

45 ptic transmission in addition to peptidergic neuromodulation and identify acetylcholine as a key tran

46 ave shown that VIP(+) cells are sensitive to neuromodulation and increase their firing during locomot

47 fferences in gating behavior, sensitivity to neuromodulation and interactions with extracellular matr

48 preferred frequencies, which are subject to neuromodulation and may interact to shape network oscill

49 Neuromodulation and neuronal plasticity act to reconfigu

50 od pressure regulation, but also involved in neuromodulation and neuroprotection.

51 urinary incontinence are treated with sacral neuromodulation and onabotulinumtoxinA with limited comp

52 rameters can nonetheless respond reliably to neuromodulation and other global perturbations.

53 s the current evidence for the use of sacral neuromodulation and percutaneous tibial nerve stimulatio

54 Both sacral neuromodulation and percutaneous tibial nerve stimulatio

55 rgic signaling reveals a novel mechanism for neuromodulation and represents an unexplored target for

56 y the interaction between diffuse descending neuromodulation and specific and focused local synaptic

57 is review examines the relationships between neuromodulation and synaptic plasticity, focusing on the

58 ically involved in neuron-glia interactions, neuromodulation and synaptic plasticity.

59 oss cortical areas, for long-term studies of neuromodulation and targeted cortical plasticity, and fo

60 eceptor antagonist methysergide blocked this neuromodulation and the swimming behavior.

61 ture of (co)activations in the DMN, ACN, and neuromodulation, and accompanied by a decreased rate of

62 s play important roles in neurotransmission, neuromodulation, and hormonal signaling.

63 n" mechanisms originating from the brain and neuromodulation, and how pain affects the sickle microen

64 for the study of central pattern generation, neuromodulation, and network dynamics.

65 isorder, including the use of animal models, neuromodulation, and pharmacoimaging studies.

66 s, factors influencing neurotransmission and neuromodulation, and proteins involved in the circadian

67 ercutaneous tibial nerve stimulation, sacral neuromodulation, and surgical procedures for stress inco

68 ight the importance of neuronal dynamics and neuromodulation, and the existence of parallel circuits.

69 We engineered a transdermal neuromodulation approach that targets peripheral (crania

70 Non-invasive and invasive neuromodulation approaches also show promise as both acu

71 peptide-based mechanisms, and entirely novel neuromodulation approaches illustrate that much can be d

72 of therapy, and in those for whom it cannot, neuromodulation approaches, such as occipital nerve stim

73 psychiatric disorders toward more responsive neuromodulation approaches.

74 However, the mechanisms underlying neuromodulation are made complex by the diversity of neu

75 pe calcium channels, breathing, sighing, and neuromodulation are severely compromised, leading to ear

76 control their activity and susceptibility to neuromodulation are unknown.

77 ivable that natural selection might act upon neuromodulation as a mechanism for sculpting the behavio

78 ngs indicate that cholinergic and adrenergic neuromodulation associated with the behavioral state of

79 erapeutic effect, including: (a) via genuine neuromodulation, (b) via non-specific placebo effects an

80 holds promise for high-efficacy personalized neuromodulations based on individual local neurodynamics

81 uals with addiction, might benefit from such neuromodulation-based approaches.

82 Rats are commonly used to study respiratory neuromodulation, but rodent sleep is characterized by a

83 states such as alertness can be governed by neuromodulation, but the underlying mechanisms and cell

84 cillations can be explained as a function of neuromodulation by acetylcholine (ACh) and norepinephrin

85 Thus, the distinct neuromodulation by ACh in these circuits could underlie

86 network as CCKBCs are highly susceptible to neuromodulation by local and subcortically generated sig

87 hensive mapping of circuit and network-level neuromodulation by NAc-DBS, which should facilitate our

88 One of the best examples of such neuromodulation by neuroestrogens concerns the acute reg

89 We tested the idea whether cardiac neuromodulation by nNOS could be sustained by long-term

90 ons transform previously suggestive roles of neuromodulation by peptides in TB cells to more concrete

91 These results demonstrate that neuromodulation by PKA and PKC is caused by their enhanc

92 extends previous influential theories of LC neuromodulation by proposing specific mechanisms for how

93 The dorsal rostral pons may be a locus of neuromodulation by suboccipital stimulation.

94 We mimicked DAergic neuromodulation by systemic injection of L-DOPA and Carb

95 Neuromodulation can be defined as a biophysical process

96 evidence that octopamine- and tyramine-based neuromodulation can be mediated by astrocytes, and demon

97 parameters, raising the question of whether neuromodulation can be reliable across individuals with

98 stitute an integration node at which OAergic neuromodulation can bias the output of P1 neurons to fav

99 de new insight into the complex issue of how neuromodulation can coordinate situation-specific behavi

100 Neuromodulation can dynamically alter neuronal and synap

101 s suggest that reduced levels of cholinergic neuromodulation can mediate an attentional bias toward r

102 ackground activity, global brain states, and neuromodulation can powerfully influence synaptic transm

103 t include projection neurons targeting known neuromodulation centers in the brain.

104 Neuromodulation changes how neural circuits process info

105 Neuromodulation changes the cellular and synaptic proper

106 Neuromodulation confers flexibility to anatomically-rest

107 Neuromodulation confers operational flexibility on motor

108 (CONtrol of Faecal Incontinence using Distal NeuromodulaTion [CONFIDeNT]) in 17 specialist hospital u

109 Coordinated reset neuromodulation consists of the application of consecuti

110 is favoured by current data, how cholinergic neuromodulation contributes to gamma oscillation product

111 Cholinergic neuromodulation controls long-term synaptic plasticity u

112 Our results provide a mechanism for how neuromodulation controls the gain and signal-to-noise ra

113 In neuromodulation, dcaps mutants provide the first genetic

114 Dopaminergic neuromodulation declines with age, suggesting that incen

115 ing an organizing principle that cholinergic neuromodulation depends critically on neurochemical iden

116 Neuromodulation device revisions and removals occurred i

117 facilitated by cholinergic and noradrenergic neuromodulation during REM sleep.

118 Incorporating this bidirectional neuromodulation-enabled correlational synaptic learning

119 Thus, our findings show that cholinergic neuromodulation enhances attentional selection via an im

120 The success of DBS and other forms of neuromodulation for neuropsychiatric disorders is the re

121 se of local field potentials for closed-loop neuromodulation for OCD.

122 and efficacious than conventional continuous neuromodulation for PD.

123 of the amygdala (CeA) is a critical site of neuromodulation for processing of bladder nociception.

124 use of onabotulinum toxin A and sacral nerve neuromodulation for the treatment of overactive bladder

125 age further development of coordinated reset neuromodulation for treating motor symptoms in Parkinson

126 on to central nervous integration, and cover neuromodulation from the molecular to the behavioral lev

127 This potentially extends the locus of neuromodulation from the nominal target to afferent brai

128 This switch in polarity of Ucn I-mediated neuromodulation, from a negative to positive regulation

129 tical local circuits, that links cholinergic neuromodulation, gamma rhythmicity, and attentional sele

130 nence per day than did the 174 in the sacral neuromodulation group (-3.9 vs -3.3 episodes per day; me

131 Neuromodulation has a role in the treatment of nonobstru

132 Cancer borealis , a premier model system for neuromodulation, has not been characterized.

133 However, disturbances in neuromodulation have also been associated with pathologi

134 point, some technical aspects of the use of neuromodulation have become more standardized and the ne

135 first time, these data show that cerebellar neuromodulation impacts activation patterns specifically

136 ic stimulation (TMS) would reduce appetitive neuromodulation in a manner similar to MDD.

137 Here we review the literature on the use of neuromodulation in addictive disorders to highlight prog

138 duction of norepinephrine and re-establishes neuromodulation in alpha7 nicotinic acetylcholine recept

139 n of the therapeutic potential of cerebellar neuromodulation in ASD may be warranted.

140 rable interest and development of the use of neuromodulation in colorectal surgery and much of the li

141 her onabotulinumtoxinA is superior to sacral neuromodulation in controlling refractory episodes of ur

142 evidence for the selectivity of cholinergic neuromodulation in GABAergic interneurons and identifies

143 rom recent clinical studies of bioelectronic neuromodulation in inflammatory and autoimmune diseases.

144 However, the cellular mechanisms of DA neuromodulation in neocortex are not well understood.

145 Cholinergic neuromodulation in neocortical networks is required for

146 rstand the mechanisms underlying cholinergic neuromodulation in OB, we developed a biophysical model

147 Our results suggest possible targets for neuromodulation in obesity (ie superior frontal gyrus) a

148 assessment of the use of LFP for closed-loop neuromodulation in OCD.

149 sory systems, highlighting the importance of neuromodulation in shaping feature extraction at all lev

150 have potential implications for therapeutic neuromodulation in similar epileptic conditions associat

151 examine the receptor basis for serotonergic neuromodulation in the antennal lobe of Manduca sexta.

152 nnabinoids) is a recently discovered form of neuromodulation in the brain.

153 ng vascular tone in blood vessels as well as neuromodulation in the brain.

154 Lastly, I consider neuromodulation in the context of strategic action choic

155 receptor (KOR), is thought to be involved in neuromodulation in the dentate gyrus.

156 ron classes undergo differential cholinergic neuromodulation in the hippocampus.

157 and the potential benefit and limitations of neuromodulation in the management of this arrhythmia.

158 One implication is that targeting neuromodulation in the medial amygdala could potentially

159 its Special Feature, provides an overview of neuromodulation in two neuronal circuits that both produ

160 at is important for dopamine homeostasis and neuromodulation in vivo.

161 safely expressed, and could mediate optical neuromodulation, in primate neocortex over many months.

162 fMRI can provide a signature of dopaminergic neuromodulation, indicating that the application of mult

163 suggest a new therapeutic approach based on neuromodulation, instead of direct inhibition, of the NM

164 opments have been accompanied by advances in neuromodulation interventions, both invasive as deep bra

165 Neuromodulation interventions, such as intrathecal baclo

166 Cholinergic neuromodulation is a candidate mechanism for aspects of

167 We conclude that neuromodulation is an important factor shaping the topog

168 Neuromodulation is an important regulatory feature of ma

169 Neuromodulation is an increasingly accepted treatment fo

170 wever, determining the mechanisms underlying neuromodulation is challenging without knowledge of the

171 Here we report that the presence of neuromodulation is correlated with the production of a b

172 endogenous peptides and amines, but whether neuromodulation is critical to the expression of a rhyth

173 The majority of this neuromodulation is mediated by delta, not mu-opioid, rec

174 Neuromodulation is often thought to have a static, gain-

175 Research suggests the action of neuromodulation is on the afferent pathway, though it re

176 ne (DA) system in normal cognitive aging, DA neuromodulation is one plausible mechanism.

177 ment for depression, but today, the field of neuromodulation is rapidly changing with the advent of n

178 Neuromodulation is required for cortical plasticity, but

179 One striking characteristic of neuromodulation is that it can configure a neural circui

180 supports the idea that in some subjects this neuromodulation is, for reasons poorly understood, upreg

181 Previous studies of neuromodulation largely focused on immediate actions of

182 s might be weakened in a heightened state of neuromodulation like that provoked by triggers of migrai

183 ealed a TBS-induced inhibition of appetitive neuromodulation, manifest in a diminished startle respon

184 induced atrial arrhythmias, suggesting that neuromodulation may be helpful in controlling AF.

185 For these women, neuromodulation may counteract the inhibitory effects of

186 Thus, neuromodulation may provide a mechanism that enables spe

187 , in conscious animals, a novel mechanism of neuromodulation mediated by the carotid chemoreceptors a

188 current stimulation (tACS) is a noninvasive neuromodulation method that uses weak sinusoidal electri

189 imulation (tRNS), a painless and more direct neuromodulation method was shown to further promote cogn

190 erspecific variation in sensitization and SN neuromodulation might be correlated with variation in th

191 on has emerged as a major mechanism by which neuromodulation might enable long-term synaptic modifica

192 Further, right posterolateral cerebellar neuromodulation modifies behavior during predictive lang

193 g interest in brain stimulation as a form of neuromodulation, much remains unknown about the network-

194 U of onabotulinumtoxinA (n = 192) or sacral neuromodulation (n = 189).

195 iefly compared tDCS with other techniques of neuromodulation, namely deep brain stimulation, vagal ne

196 ch, using the key words 'Epilepsy Surgery', 'Neuromodulation', 'Neuroablation', 'Advances', between 2

197 urrent stimulation (tDCS)-induced analgesia, neuromodulation occurs through a top-down process that d

198 Neuromodulation of arousal states ensures that an animal

199 molecular and circuit properties underlying neuromodulation of arousal states such as sleep and wake

200 Typically, the neuromodulation of biochemical signaling and biophysics

201 e raphe nuclei, in addition to their role in neuromodulation of brain states, are also involved in fa

202 elineate the complexity and heterogeneity of neuromodulation of cerebral cortex by cholinergic stimul

203 e highlight recent advances in understanding neuromodulation of Drosophila innate behaviors, with a s

204 Advances in our understanding of neuromodulation of GnRH neurons and synchronization of t

205 Cholinergic neuromodulation of hippocampal circuitry promotes networ

206 Differential 5-HT neuromodulation of MCs across the MOB and AOB could cont

207 ic ganglion (STG), is an important model for neuromodulation of motor networks.

208 Neuromodulation of olfactory circuits by acetylcholine (

209 aplasticity mechanism that may contribute to neuromodulation of plasticity in other cortical circuits

210 In this study, we asked how it shapes neuromodulation of postsynaptic responses.

211 acetylcholine) having distinct roles in the neuromodulation of prefrontal cortical function.

212 Here neuromodulation of RCrusI in neurotypical humans resulte

213 sodium currents, regulates excitability and neuromodulation of RTN neurons and CO2-stimulated breath

214 Neuromodulation of self-amplifying circuits directs cont

215 homeostasis, and energy balance, as well as neuromodulation of social behavior, stress regulation, a

216 tors of pain, and possibly to guide targeted neuromodulation of specific brain regions for therapeuti

217 Here, we demonstrate that sequential neuromodulation of STDP by acetylcholine and dopamine of

218 Thus, temporally sequenced neuromodulation of STDP enables associations to be made

219 citation/inhibition balance, we examined the neuromodulation of STDP in FS cells of mouse visual cort

220 These results demonstrate that neuromodulation of synapses involves complex and interac

221 Although neuromodulation of synapses is extensively documented, i

222 Neuromodulation of synaptic plasticity by 17beta-estradi

223 efferent cVNS, specifically as it relates to neuromodulation of systemic inflammation.

224 results are consistent with a role of MOR in neuromodulation of the auditory periphery.

225 Neuromodulation of the axon dislocated the site of initi

226 knowledge about the mechanism of EBS and the neuromodulation of the human brain.

227 ge, the first known physiological target for neuromodulation of the innate immune responses and a pot

228 thways, enhanced at the peripheral level via neuromodulation of the NMJ.

229 Here, we have studied SDF-1 alpha-mediated neuromodulation of the stratum lacunosum-moleculare by d

230 We show that neuromodulation of the sub-threshold motor state of exci

231 Here we show that coordinated reset neuromodulation of the subthalamic nucleus has both acut

232 Furthermore neuromodulation of the vagus nerve can be used in the tr

233 g within a neural network impacts subsequent neuromodulation of those synapses.

234 ehavioral observations suggest that dopamine neuromodulation of UNC-7 ensures attenuation of recursiv

235 Neuromodulation offers promise for managing both storage

236 racterized the effects of endogenous central neuromodulation on correlated fluctuations during rest i

237 This is the first report on the role of neuromodulation on SSA, and the results contribute to ou

238 With high temporal precision and reversible neuromodulation, optogenetics promises to improve existi

239 NCS-382 suggests that GHB may play a role in neuromodulation or neurotransmission in frontal brain ar

240 tudies such as the concept of specificity in neuromodulation or of receptors acting as sensors instea

241 esity and how targeting the vagus nerve with neuromodulation or pharmacology can be used as a therape

242 ophysiological signals and rs-fMRI via a new neuromodulation paradigm, which exploits these power syn

243 crucial to determining the scale of induced neuromodulation, particularly when attempting to modulat

244 mechanism by which cocaine, acting on a key neuromodulation pathway, modifies the coincidence detect

245 activity is likely modulated through diffuse neuromodulation pathways that govern arousal states and

246 ing synaptic activity facilitates cumulative neuromodulation, potentially reversing endogenous synapt

247 tic tools in vivo, we show that serotonergic neuromodulation prominently inhibits the spontaneous ele

248 nectivity between neurons, coordinated reset neuromodulation reduces pathological synchronization, a

249 We examined whether dopaminergic neuromodulation regulates activity-dependent glutamaterg

250 m body functions like a switchboard in which neuromodulation reroutes the same odor signal to differe

251 Previous neuromodulation research has demonstrated that disruptio

252 with onabotulinumtoxinA compared with sacral neuromodulation resulted in a small daily improvement in

253 ers a minimally invasive but high efficiency neuromodulation scheme with potential applications in co

254 echanisms of state dependence resulting from neuromodulation, sensory input, and experience.

255 Cortical neuromodulation sets different cortical and thalamic sta

256 names are used for this class of treatments: neuromodulation, somatic therapies, brain stimulation te

257 mechanism underlying spike timing-dependent neuromodulation (STDN) was investigated in the opisthobr

258 deep brain stimulation or other target-based neuromodulation strategies for treatment-resistant depre

259 tablished a rationale for testing a targeted neuromodulation strategy, deep brain stimulation, for tr

260 ive models, and model-based fMRI analyses of neuromodulation-strive to move beyond statistical charac

261 tative biomarkers for objectively evaluating neuromodulation success and for guiding deep brain stimu

262 cause the brain contains a chemical "map" of neuromodulation superimposed upon its synaptic connectiv

263 insic physiology, feedforward sensory input, neuromodulation, synaptic output, and functional role of

264 1) to receive either PTNS (via the Urgent PC neuromodulation system) or sham stimulation (via a trans

265 imulation (tDCS) is an emerging non-invasive neuromodulation technique that applies mA currents at th

266 magnetic stimulation (rTMS) is a noninvasive neuromodulation technique that has been closely examined

267 tion (DBS) is one of the most invasive focal neuromodulation techniques available; data have supporte

268 The neuromodulation techniques of DBS and neurofeedback, whi

269 Applications of functional neuromodulation techniques to BCI systems would allow fo

270 ents a novel mechanism of adenosine-mediated neuromodulation that could contribute to the regulation

271 Presynaptic inhibition is a form of neuromodulation that interacts with activity-dependent s

272 terface and delivered electrochemical spinal neuromodulation that restored locomotion after paralyzin

273 Neuromodulation, the alteration of individual neuron res

274 the network and may change as a function of neuromodulation, the balance or stochasticity of synapti

275 earch, brain-machine interfaces and clinical neuromodulation therapies for decades.

276 elieve these results justify further work on neuromodulation therapies targeting the affective sphere

277 ted by the ongoing testing of novel invasive neuromodulation therapies, notably, deep brain stimulati

278 and facilitate the design of more effective neuromodulation therapies.

279 to pharmacologic, cognitive, behavioral, and neuromodulation therapies.

280 e targets and patient-specific protocols for neuromodulation therapy.

281 whether these photoreceptors are subject to neuromodulation through intracellular cAMP-related signa

282 Women who respond best to neuromodulation through sacral nerve stimulation are tho

283 ctrical stimulation pulses may contribute to neuromodulation, thus warrant explicit attention in ther

284 Here we combined neuroimaging and neuromodulation to provide evidence that the cerebellum

285 Our results support the emerging paradigm of neuromodulation to treat AF.

286 a will open new opportunities, especially in neuromodulation, to treat pathologies of the lower urina

287 an unbiased and brain-wide manner, sites of neuromodulation under different conditions in the Drosop

288 Neuromodulation underlies many behavioral states and has

289 logy, and after an initial setup, ultrasonic neuromodulation (UNMOD) can be implemented in less than

290 yer-specific circuit effects of dopaminergic neuromodulation using current source density (CSD) analy

291 Neuromodulation using transcranial magnetic stimulation

292 w by analysis of sodium channel mutants that neuromodulation via PKA and PKC enhances intrinsic slow

293 This neuromodulation was accompanied by reductions in motivat

294 Moreover, neuromodulation was significantly altered as muscarine a

295 ns functional and behavorial outcomes during neuromodulation, we first combined tDCS and a tonic pain

296 strongly correlated with serotonergic (5-HT) neuromodulation, we hypothesized that the observed inter

297 his caused a complicated temporal pattern of neuromodulation when DSI and VSI-B were stimulated to fi

298 In this paper, learning takes place via neuromodulation, which allows agents to selectively chan

299 Here, I review two examples of neuromodulation within a specific microcircuit of the hi

300 nce suggests the membrane PSA is involved in neuromodulation within the central nervous system and in