ライフサイエンスコーパス: neuromodulin

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1 a regulatory motif originally identified in neuromodulin.

2 alanine rich C kinase substrate (MARCKS) and neuromodulin.

3 in kinase C and calmodulin binding domain of neuromodulin.

4 In cells expressing wild-type neuromodulin, a complex with calmodulin that is sensitiv

5 ed expression of the neuronal phosphoprotein neuromodulin (also known as GAP-43, F1, B-50, and p57) i

6 associated protein 43 (GAP-43; also known as neuromodulin and B-50) in axonal structural plasticity b

7 dentical to the calmodulin binding domain of neuromodulin and contained an IQ motif found in other ca

8 ween calmodulin (CaM) and the IQ domain from neuromodulin, and have developed detailed kinetic models

9 aredoxin, growth associated protein (GAP)-43/neuromodulin, and phenobarbital-induced cytochrome P450.

10 Overall, the binding pocket for neuromodulin appears to be less compact and more dynamic

11 inase substrate (MARCKS) and F1/GAP-43 (B-50/neuromodulin) are both major specific substrates for pro

12 neuronal growth-associated protein (GAP)-43 (neuromodulin, B-50, F1), which is concentrated in the gr

13 istent with the hypothesis that CaM bound to neuromodulin comprises a localized store that can be eff

14 For the 17 residue neuromodulin derived peptide, which is Ca2+ independent

15 The peripheral MP neuromodulin displays robustly with packaging by convent

16 similar to the calmodulin binding domains of neuromodulin (GAP-43) and neurogranin (RC3).

17 to modify rat brain neurogranin/RC3 (Ng) and neuromodulin/GAP-43 (Nm).

18 sequence of the calmodulin-binding domain of neuromodulin has been studied by fluorescence spectrosco

19 we analyze the role of calmodulin binding by neuromodulin in these responses.

20 Synthesis of GAP-43 (also known as neuromodulin) in neurons is induced during axon growth,

21 lped clarify the role of GAP-43 (FI, B-50 or neuromodulin) in regulating the growth state of axon ter

22 the affinities of a reporter containing the neuromodulin IQ domain are essentially identical, with K

23 the complex between calmodulin (CaM) and the neuromodulin IQ domain consists of two phases.

24 Release of CaM from the neuromodulin IQ domain therefore appears to be promoted

25 Replacement of a central Gly in the neuromodulin IQ domain with a Lys at this position in PE

26 GAP-43 (neuromodulin) is a protein kinase C substrate that is ab

27 Thus, the multitude of effects of neuromodulin noted in previous studies may result from d

28 Despite the similar IQ motif in PEP-19 and neuromodulin or neurogranin, PEP-19 was not a substrate

29 pproximately 5-fold, while the value for the neuromodulin reporter complex is increased by the same f

30 cted, but the value for the complex with the neuromodulin reporter is increased approximately 12-fold

31 Since the central Gly in the neuromodulin sequence is conserved in half of all known

32 ately before the "IQ" amino acid pair in the neuromodulin sequence with the Ala in PEP19 accounts for

33 ment of an Ala in the N-terminal half of the neuromodulin sequence with the Gln in PEP19 accounts for

34 Transfection of several mutant forms of neuromodulin shows that the effects of this protein on s

35 activator was distinct from other proteins (neuromodulin, tubulin, and beta-amyloid precursor protei

36 in kinase C and calmodulin binding domain of neuromodulin was studied using a combination of NMR, EPR

37 toylated alanine-rich C kinase substrate and neuromodulin were previously found to be in extended con

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