ライフサイエンスコーパス: neuromuscular junction

1 of glutamate receptor clusters at the larval neuromuscular junction.

2 endent synaptic plasticity at the Drosophila neuromuscular junction.

3 lutamatergic transmission also occurs at the neuromuscular junction.

4 ructural and functional abnormalities at the neuromuscular junction.

5 ise from impaired signal transmission at the neuromuscular junction.

6 teins essential for neurotransmission at the neuromuscular junction.

7 ity-evoked peptide release at the Drosophila neuromuscular junction.

8 presynaptic microtubule stabilization at the neuromuscular junction.

9 r structural modifications at the Drosophila neuromuscular junction.

10 rol structure and function of the Drosophila neuromuscular junction.

11 aintaining proper synaptic morphology at the neuromuscular junction.

12 iched in skeletal muscles, especially at the neuromuscular junction.

13 apy of impaired synaptic transmission at the neuromuscular junction.

14 of proteins can produce defects at the human neuromuscular junction.

15 Dsychronic (DYSC), at the Drosophila larval neuromuscular junction.

16 plasma membranes of certain neurons and the neuromuscular junction.

17 muscles, resulting in poor formation of the neuromuscular junction.

18 echanism for graded locomotor control at the neuromuscular junction.

19 ation of muscle mass after disruption of the neuromuscular junction.

20 tricted to muscle and, in particular, to the neuromuscular junction.

21 thway to changes in synaptic function in the neuromuscular junction.

22 g inhibition of acetylcholine release at the neuromuscular junction.

23 acophony protein in whole animals and at the neuromuscular junction.

24 glycosylation for proper functioning of the neuromuscular junction.

25 ripheral cholinergic synapses, including the neuromuscular junction.

26 tamatergic synapses of the Drosophila larval neuromuscular junction.

27 endent synaptic remodeling at the Drosophila neuromuscular junction.

28 citatory synaptic transmission at the larval neuromuscular junction.

29 ue, probably around the nerve endings of the neuromuscular junction.

30 llite cells and within motor neurons via the neuromuscular junction.

31 eostatic synaptic compensation at the larval neuromuscular junction.

32 l synaptic-function at the Drosophila larval neuromuscular junction.

33 defects in the structure and function of the neuromuscular junction.

34 locking acetylcholine receptors at the mouse neuromuscular junction.

35 s glutamatergic synaptogenesis in developing neuromuscular junctions.

36 he highly reliable transmission at zebrafish neuromuscular junctions.

37 the sciatic nerve, and axon terminals of the neuromuscular junctions.

38 evoked ACh release at Caenorhabditis elegans neuromuscular junctions.

39 by distinct synaptic scaffolds at C. elegans neuromuscular junctions.

40 ed release at Drosophila melanogaster embryo neuromuscular junctions.

41 functional defects at a subset of vulnerable neuromuscular junctions.

42 Gs, and reduced complement activation at the neuromuscular junctions.

43 uggesting an effect on synaptogenesis beyond neuromuscular junctions.

44 ities, and defects of neural transmission at neuromuscular junctions.

45 rupts synaptic structure and function at the neuromuscular junctions.

46 e expression and the formation of functional neuromuscular junctions.

47 omyograms and by intracellular recordings at neuromuscular junctions.

48 ressive motor neuron loss and denervation of neuromuscular junctions.

49 that inhibits acetylcholine (ACh) release at neuromuscular junctions.

50 autoimmune disorder that selectively targets neuromuscular junctions.

51 ty at active zones of axon terminals at frog neuromuscular junctions.

52 inhibits nerve regeneration and destabilizes neuromuscular junctions.

53 l muscle by motor neurons occurs through the neuromuscular junction, a cholinergic synapse essential

54 g all Shank proteins and used the Drosophila neuromuscular junction, a model glutamatergic synapse, t

55 or may be caused indirectly by neuronal and neuromuscular junction abnormalities.

56 Several neural systems, including the neuromuscular junction and climbing fiber innervation of

57 The neuromuscular junction and CNS neurons share these featu

58 ential for normal synaptic plasticity at the neuromuscular junction and for muscle strength, enduranc

59 diate fast chemical neurotransmission at the neuromuscular junction and have diverse signalling roles

60 xons during both postnatal maturation of the neuromuscular junction and myofiber reinnervation after

61 mulation of monosynaptic transmission at the neuromuscular junction and reliable activation of a gust

62 t the postsynaptic membrane of glutamatergic neuromuscular junctions and controls multiple parameters

63 h a less compact basement membrane, immature neuromuscular junctions and dysfunctional muscle predisp

64 eostasis at the Drosophila, mouse, and human neuromuscular junctions and emerging parallels at synapt

65 nerating across scar tissue, rebuilt healthy neuromuscular junctions and enhanced motor functional re

66 ceptors (AChRs) mediate signalling at mature neuromuscular junctions and fetal-type AChRs are necessa

67 sympathetic neurons make close contact with neuromuscular junctions and form a network in skeletal m

68 er axonal elongation in an in vitro model of neuromuscular junctions and hastened recovery after peri

69 the age-dependent innervation status of the neuromuscular junctions and mutant tau expression in Tg3

70 cation, caused by the massive elimination of neuromuscular junctions and pruning of axonal branches.

71 in motor units such as spinal motor neurons, neuromuscular junctions and skeletal muscle.

72 r to infection of peripheral synapses (i.e., neuromuscular junction) and infection of peripheral tiss

73 evels, the propriospinal projection network, neuromuscular junction, and central pattern generator, p

74 disease, neurofibromatosis, diseases of the neuromuscular junction, and Charcot-Marie Tooth disease

75 zed acetylcholine receptor expression at the neuromuscular junction, and epithelial morphogenesis.

76 gy, and homeostatic plasticity at the larval neuromuscular junction, and impaired olfactory habituati

77 ysiology in identifying abnormalities of the neuromuscular junction, and therefore leading investigat

78 ured neurons, Drosophila melanogaster larval neuromuscular junctions, and mammalian tissue.

79 e respiratory rhythm generator and diaphragm neuromuscular junctions appeared normal.

80 th a reduction of AChR protein levels at the neuromuscular junction (approximately 25%) in Dok-7-siRN

81 evelopment and evoked function of the larval neuromuscular junction are surprisingly normal, but the

82 eon mutants, postsynaptic specializations of neuromuscular junctions are dramatically expanded, inclu

83 use models of both diseases, suggesting that neuromuscular junctions are highly vulnerable from the v

84 Neuromuscular junctions are primary pathological targets

85 Using Drosophila neuromuscular junction as a model synapse, we describe t

86 Using Caenorhabditis elegans neuromuscular junction as a model synapse, we uncovered

87 In conclusion, this study identifies the neuromuscular junction as a target of the sympathetic ne

88 its symptomatic treatment suggests that this neuromuscular junction assay has significant potential f

89 These formed 6618 synapses including 1772 neuromuscular junctions, augmented by 1206 gap junctions

90 hed larval peristaltic contractions, loss of neuromuscular junction bouton structures, impaired olfac

91 s that block neurotransmitter release at the neuromuscular junction by cleaving SNAREs (soluble N-eth

92 induces synaptic potentiation at the larval neuromuscular junction by increasing synaptic vesicle (S

93 active controlling synaptic growth at larval neuromuscular junctions by facilitating Ca(2+) release f

94 ion of the mutant channels at the Drosophila neuromuscular junction causes abnormally elevated evoked

95 At Caenorhabditis elegans neuromuscular junctions, clustering of levamisole-sensit

96 s by electromyography found that a defective neuromuscular junction component is not always present.

97 aterals to reinnervate previously denervated neuromuscular junctions concurrently with expression of

98 motor neuron soma and maintaining functional neuromuscular junction connections are both essential el

99 cal deficits in synapse growth at the larval neuromuscular junction consistent with a dominant-negati

100 residue (p.Pro308Leu) produced a presynaptic neuromuscular junction defect and a dominant hereditary

101 Although different SMA models have shown neuromuscular junction defects and/or motor axon defects

102 e lacking miR-218 die neonatally and exhibit neuromuscular junction defects, motoneuron hyperexcitabi

103 ne sulfate was also observed when axonal and neuromuscular junction degeneration have already occurre

104 taline sulfate prevents defects in axons and neuromuscular junction degeneration in a dose-dependent

105 de preserved ganglioside distribution at the neuromuscular junction, delayed disease onset, improved

106 ons of transgenic SOD1(G93A) ALS mice delays neuromuscular junction denervation by inducing axonal sp

107 However, it was not effective at preventing neuromuscular junction denervation in a mutant SOD1(G93A

108 nic nerve axonal degeneration, and diaphragm neuromuscular junction denervation, and resulted in redu

109 uency stimulation, both wild-type and mutant neuromuscular junctions depress to steady-state response

110 a genetic screen for mutants with defective neuromuscular junction development, we identify a hypomo

111 In rsu-1 mutants, neuromuscular junctions differentiate as in the wild typ

112 ented with an autosomal-dominant presynaptic neuromuscular junction disorder resembling Lambert-Eaton

113 roteins have discrete roles localised to the neuromuscular junction, DPAGT1 is ubiquitously expressed

114 equirement for MYO9A in the formation of the neuromuscular junction during development.

115 ticide poisoning, including onset of delayed neuromuscular junction dysfunction during the cholinergi

116 Strength and reliability of the neuromuscular junction emerge as a result of its assembl

117 expression of genes encoding proteins of the neuromuscular junction, especially acetylcholine recepto

118 Motor neurons regulate neuromuscular junction formation by using agrin to stimu

119 mmed cell and tissue death, neuromaturation, neuromuscular junction formation, and neuron cell fate d

120 als between neuron and muscle, which lead to neuromuscular junction formation, dysfunction of which i

121 as reduced motor axon branching and abnormal neuromuscular junction formation.

122 ein 4 (Lrp4), a protein that is critical for neuromuscular junction formation.

123 lcholine receptor (AChR) clustering and NMJ (neuromuscular junction) formation.

124 ction and preserved motor neurons as well as neuromuscular junctions from degeneration.

125 earance occurred at motor nerve terminals of neuromuscular junctions, from where anti-ganglioside ant

126 nervous system synapses and mouse diaphragm neuromuscular junctions fully intoxicated by BoNT seroty

127 P4-agrin interaction, which is essential for neuromuscular junction function.

128 elease, whereas muscle-derived Gbb regulates neuromuscular junction growth.

129 ed axon terminals of active and resting frog neuromuscular junctions has led to the conclusion that u

130 m muscle nuclei to postsynaptic sites at the neuromuscular junction in Drosophila.

131 ectopic synapse refinement at the developing neuromuscular junction in Drosophila.

132 presynaptic neurotransmitter release at the neuromuscular junction in Drosophila.

133 s unable to rescue structural defects at the neuromuscular junction in fragile x mental retardation 1

134 Schwann cells cocultured with neurons and at neuromuscular junction in vivo through the MAPK pathway.

135 Furthermore, we compare the structure of neuromuscular junctions in 4- and 8-dpf zebrafish larvae

136 chitecture of larval zebrafish (Danio rerio) neuromuscular junctions in three dimensions.

137 ylcholine receptors for the establishment of neuromuscular junctions in vitro.

138 ological recordings in the Drosophila larval neuromuscular junction, in the presence of the use-depen

139 ch-dependent morphological phenotypes at the neuromuscular junction including synaptic size and prese

140 Our studies at the Drosophila neuromuscular junction indicate that many synaptic defec

141 Maintenance of neuromuscular junction innervation during the course of

142 nescent mitochondria in their motor axons or neuromuscular junctions; instead, they contain far fewer

143 The neuromuscular junction is known as a strong and reliable

144 hat axon branch loss at the developing mouse neuromuscular junction is mediated by branch-specific mi

145 trically localized, synaptic transmission at neuromuscular junctions is impaired and there is a reduc

146 n ortholog that is normally localized to the neuromuscular junction, is naturally upregulated in DMD

147 transmission, originally established at the neuromuscular junction, is that the time course of trans

148 ession of wild-type PrP(C) at the Drosophila neuromuscular junction leads to enhanced synaptic respon

149 nd on the presynaptic nerve terminals at the neuromuscular junction level, but not on the axonal trac

150 a peripheral pathological event resulting in neuromuscular junction loss and motoneuron (MN) degenera

151 sm underlying impaired neurotransmission and neuromuscular junction maintenance in SMA.

152 iate forms of SMA presenting with a delay in neuromuscular junction maturation and a decrease in the

153 12 d to a mean of 456 d, with improvement in neuromuscular junction morphology, down-regulation of tr

154 In the periphery, at neuromuscular junctions, motoneurons are known to enlarg

155 During the development of the neuromuscular junction, motor axons induce the clusterin

156 e of exocytosis in isolated nerve terminals, neuromuscular junctions, neuroendocrine cells and in hip

157 ments, anxiety, hippocampal LTP deficits and neuromuscular junction (NMJ) abnormalities, characterize

158 impaired synaptic transmission at the larval neuromuscular junction (NMJ) and in the adult.

159 aladaptive changes within motor neurons, the neuromuscular junction (NMJ) and muscle fibres.

160 riggering of neurotransmitter release at the neuromuscular junction (NMJ) are thought to be removed.

161 The neuromuscular junction (NMJ) consists of a tripartite sy

162 t GlyRS leads to motor deficits, progressive neuromuscular junction (NMJ) denervation and pre-synapti

163 sive loss of motor neurons (MNs) preceded by neuromuscular junction (NMJ) denervation.

164 hR clustering, a complete reversal of normal neuromuscular junction (NMJ) development where AChR clus

165 anifesting deficits in coordinated movement, neuromuscular junction (NMJ) development, synaptic glyco

166 ever, SMA's hallmark MN pathology, including neuromuscular junction (NMJ) disruption and sensory-moto

167 al of Motor Neuron (SMN) protein, leading to neuromuscular junction (NMJ) dysfunction and spinal moto

168 mulate the LRP4-MuSK receptor in muscles for neuromuscular junction (NMJ) formation.

169 SK is one of the key molecules orchestrating neuromuscular junction (NMJ) formation.

170 osophila peripheral nervous system (PNS) and neuromuscular junction (NMJ) have been identified as pla

171 complex and regulates its expression at the neuromuscular junction (NMJ) in Drosophila.

172 ia, the terminal Schwann cells (SCs), at the neuromuscular junction (NMJ) in mice.

173 ve recently reported on the pathology of the neuromuscular junction (NMJ) in Pompe disease, reflectin

174 ed and retained at postsynaptic sites at the neuromuscular junction (NMJ) in vivo remains largely unk

175 and concurrent morphological changes at the neuromuscular junction (NMJ) indicative of denervation.

176 arinic acetylcholine receptors at the lizard neuromuscular junction (NMJ) induces a biphasic modulati

177 ctions requires neural activity; because the neuromuscular junction (NMJ) is a cholinergic synapse, a

178 The Drosophila neuromuscular junction (NMJ) is a powerful system for st

179 The neuromuscular junction (NMJ) is a synapse between motor

180 The neuromuscular junction (NMJ) is a synapse formed between

181 The neuromuscular junction (NMJ) is a tripartite synapse tha

182 hether weakened synaptic transmission at the neuromuscular junction (NMJ) is an aspect of CMT2D.

183 The Drosophila neuromuscular junction (NMJ) is capable of rapidly buddi

184 SIGNIFICANCE STATEMENT: The neuromuscular junction (NMJ) is critical for all volunta

185 The neuromuscular junction (NMJ) is one of the best-studied

186 A hallmark of the neuromuscular junction (NMJ) is the high density of acet

187 The neuromuscular junction (NMJ) is the synapse between a mo

188 While the morphology of the neuromuscular junction (NMJ) is typically affected by ne

189 Here, we test the roles of Mmp at the neuromuscular junction (NMJ) model synapse in the reduct

190 We used alpha-bungarotoxin to compare neuromuscular junction (NMJ) morphology in healthy (wild

191 ling stabilizes synaptic transmission at the neuromuscular junction (NMJ) of Drosophila, mice, and hu

192 electron microscopy at the adult Drosophila neuromuscular junction (NMJ) of ventral abdominal muscle

193 Analysis of the underlying neuromuscular junction (NMJ) pathology revealed that lat

194 Denervation of the neuromuscular junction (NMJ) precedes the loss of motor

195 The Caenorhabditis elegans neuromuscular junction (NMJ) provides a genetically trac

196 The Drosophila larval glutamatergic neuromuscular junction (NMJ) represents a powerful synap

197 in SynTgSod1(-/-) mice, and degeneration of neuromuscular junction (NMJ) structure and function occu

198 At the glutamatergic neuromuscular junction (NMJ) synapse, we find that Notum

199 logy of presynaptic arbors at the Drosophila neuromuscular junction (NMJ) through a postsynaptic mech

200 The postsynaptic apparatus of the neuromuscular junction (NMJ) traps and anchors acetylcho

201 TeNT targets the neuromuscular junction (NMJ) with high affinity, yet the

202 At the Drosophila neuromuscular junction (NMJ), a retrograde BMP signal fu

203 tes synaptic growth at the Drosophila larval neuromuscular junction (NMJ), as a potential Repo target

204 The Drosophila larval neuromuscular junction (NMJ), at which glutamate acts as

205 calized with synaptophysin, a marker for the neuromuscular junction (NMJ), in >50% of the cells.

206 At the Drosophila neuromuscular junction (NMJ), Mgat1 mutants display sele

207 At the neuromuscular junction (NMJ), overexpression of either w

208 growth of new synaptic boutons at the larval neuromuscular junction (NMJ), providing a model system t

209 Here we show that in the Drosophila neuromuscular junction (NMJ), the endocytic scaffolding

210 At the Drosophila neuromuscular junction (NMJ), the loss of retrograde, tr

211 le specific receptor tyrosine kinase) at the neuromuscular junction (NMJ), thereby preventing fragmen

212 Focusing on the Drosophila neuromuscular junction (NMJ), we find that the AZ cytoma

213 s to the postsynaptic side of the Drosophila neuromuscular junction (NMJ), where it is required for t

214 c receptor function at the Drosophila larval neuromuscular junction (NMJ).

215 motoneurons and motor nerve terminals of the neuromuscular junction (NMJ).

216 dysfunction and progressive weakening of the neuromuscular junction (NMJ).

217 (SSR) at the Drosophila melanogaster larval neuromuscular junction (NMJ).

218 cided with establishment of the fully mature neuromuscular junction (NMJ).

219 growth of the Drosophila melanogaster larval neuromuscular junction (NMJ).

220 naptic vesicle endocytosis at the Drosophila neuromuscular junction (NMJ).

221 s (TSCs) are key components of the mammalian neuromuscular junction (NMJ).

222 G) is the most common disorder affecting the neuromuscular junction (NMJ).

223 c neurotransmitter release at the Drosophila neuromuscular junction (NMJ).

224 red for normal formation and function of the neuromuscular junction (NMJ).

225 is crucial for vesicle fusion at presynaptic neuromuscular junction (NMJ).

226 e morphogenic protein (BMP) signaling at the neuromuscular junction (NMJ).

227 le degeneration, denervation, neuromuscular [neuromuscular junction (NMJ)] abnormalities, and axonal

228 We show that at larval neuromuscular junctions (NMJ), motor neuron expression o

229 nges in mouse models of fALS are seen in the neuromuscular junctions (NMJs) and lower motor neurons,

230 Neuromuscular junctions (NMJs) are critical for survival

231 ar architecture of active zones in mammalian neuromuscular junctions (NMJs) at sub-diffraction limite

232 As mammals age, their neuromuscular junctions (NMJs) gradually change their fo

233 c screen for aberrant synaptic growth at the neuromuscular junctions (NMJs) in Drosophila, we identif

234 ctin lead to grossly distorted morphology of neuromuscular junctions (NMJs) in patients suffering fro

235 In the present study we used the CM9 neuromuscular junctions (NMJs) in the adult Drosophila t

236 rate significant pathological alterations at neuromuscular junctions (NMJs) of the diaphragm and tibi

237 Schwann cells (SCs) at neuromuscular junctions (NMJs) play active roles in syna

238 tical for the development and maintenance of neuromuscular junctions (NMJs) remains largely unknown.

239 During development, mammalian neuromuscular junctions (NMJs) transit from multiple-inn

240 CMNs form anatomically mature and functional neuromuscular junctions (NMJs) when cocultured with chic

241 ility of motor neurons and their output, the neuromuscular junctions (NMJs), has been considered a ke

242 e the morphology of their previously damaged neuromuscular junctions (NMJs), suggesting that the bene

243 In mouse neuromuscular junctions (NMJs), we find that lack of Cpl

244 pruning also occurs at embryonic Drosophila neuromuscular junctions (NMJs), where low-frequency Ca(2

245 ive disorders resulting from degeneration of neuromuscular junctions (NMJs), which form the connectio

246 king of a pHluorin-tagged Syt4 at Drosophila neuromuscular junctions (NMJs).

247 f SMN deficiency are profound defects of the neuromuscular junctions (NMJs).

248 maintenance depends on motor innervation at neuromuscular junctions (NMJs).

249 removal of supernumerary axonal inputs from neuromuscular junctions (NMJs).

250 d, as occurs normally, to newly reinnervated neuromuscular junctions (NMJs).

251 ads to a severe depletion of mitochondria in neuromuscular junctions (NMJs).

252 vity and the structure and function of their neuromuscular junctions (NMJs).

253 In this report we show in the neuromuscular junction of Caenorhabditis elegans that ap

254 c currents frequently occur in bursts at the neuromuscular junction of Caenorhabditis elegans.

255 local concentration of acetylcholine at the neuromuscular junction of frog cutaneous pectoris muscle

256 ion in the cholinergic nervous system and in neuromuscular junctions of animals.

257 We show for the first time that neuromuscular junctions of the extraocular muscles (resp

258 Conversely, neuromuscular junctions of the lumbrical muscles of the

259 l adhesion kinase or FAK), is reduced in the neuromuscular junctions of Top3beta mutant flies.

260 erturbed were genes/pathways associated with neuromuscular junction patency (providing molecular evid

261 roved neuromuscular function and ameliorated neuromuscular junction pathology in SMA mice.

262 n affected: skeletal muscle, cardiac muscle, neuromuscular junction, peripheral nerve, or central ner

263 y, we examined the relative contributions of neuromuscular junction physiology and the motor program

264 on and motor defects, but not the defects in neuromuscular junction physiology.

265 og: Futsch, which is expressed at the larval neuromuscular junction, presynaptically only.

266 y autoantibodies that target proteins at the neuromuscular junction, primarily the acetylcholine rece

267 The scaffolding protein at the neuromuscular junction, rapsyn, enables clustering of ni

268 es motor neuron regeneration and accelerates neuromuscular junction re-innervation.

269 targeted to synaptic sites at the vertebrate neuromuscular junction remains unclear.

270 arcopenia-related functional denervation and neuromuscular junction remodeling), protein degradation,

271 Uptake at the neuromuscular junction represents a major unexpected pat

272 e overextension of axons and mistargeting of neuromuscular junctions, resulting in uncoordinated loco

273 confocal microscopic analysis of the larval neuromuscular junction reveals no gross abnormalities, s

274 y when not confronted by debris and reoccupy neuromuscular junction sites efficiently.

275 well as the need to look outside traditional neuromuscular junction-specific proteins for further con

276 Moreover, compared with the neuromuscular junction, steps regulating the synaptogeni

277 release from motor neurons at the fly larval neuromuscular junction, suggesting an increase in excita

278 in SMA changes appeared concomitantly at the neuromuscular junction, suggesting that mechanisms of ne

279 P2B(Intron5), at the Drosophila melanogaster neuromuscular junction synapse revealed synaptic overgro

280 sted synaptic facilitation and depression in neuromuscular junction synapses that use exclusively CaV

281 keletal muscle builds a functional all-human neuromuscular junction that can be triggered to twitch u

282 Loss of TMEM184b causes swellings at neuromuscular junctions that become more numerous with a

283 ation of the actin cytoskeleton in axons and neuromuscular junctions to protect motoneurons in SMA.

284 n was slackened mechanically by bringing the neuromuscular junction toward the central nervous system

285 of surviving motor units and instability of neuromuscular junction transmission.

286 ophysiological and morphological deficits of neuromuscular junctions upon sympathectomy and in myasth

287 ChE) are anchored in the basal lamina of the neuromuscular junction using a collagen-like tail subuni

288 c proteoglycan with critical function at the neuromuscular junction was previously found mutated in m

289 Green) uptake in the presynaptic terminal of neuromuscular junctions was restored to control levels i

290 utamatergic synapse of the Drosophila larval neuromuscular junction, we find that presynaptic depress

291 At the neuromuscular junction, we showed mEPP amplitudes and fr

292 ed postsynaptic scaffold proteins within the neuromuscular junction were completely eliminated.

293 Tongue muscle neuromuscular junctions were also spared in both animal

294 The development and maturation of neuromuscular junctions were not disrupted in Tg30 mice,

295 annel is highly expressed at the presynaptic neuromuscular junction where it contributes to action po

296 microscopy was used to image postnatal mouse neuromuscular junctions where elimination is well studie

297 Here experiments at the Drosophila neuromuscular junction, where DCVs contain neuropeptides

298 ological features of disease manifest at the neuromuscular junction, where significant denervation oc

299 vesicle recycling pathways at complexin null neuromuscular junctions, where spontaneous release is dr

300 ontaneous postsynaptic current bursts at the neuromuscular junction, which are physiological signals