ライフサイエンスコーパス: neuronal intranuclear inclusion

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1 d characteristic lentiform ub-immunoreactive neuronal intranuclear inclusions.

2 PGRN) gene, particularly in those cases with neuronal intranuclear inclusions.

3 ressive, gender-specific motor deficits, and neuronal intranuclear inclusions.

4 n Huntington's disease, with the presence of neuronal intranuclear inclusions.

5 rites in both grey and white matter and also neuronal intranuclear inclusions.

6 ion, particularly visceral neuropathies with neuronal intranuclear inclusions.

7 e disorders, including some characterized by neuronal intranuclear inclusions.

8 rrelate with alterations in the formation of neuronal intranuclear inclusions.

9 Neuronal intranuclear inclusions and cytoplasmic aggrega

10 ma-positive neuronal cytoplasmic inclusions, neuronal intranuclear inclusions and neurites were recor

11 d limb ataxia, weight loss, premature death, neuronal intranuclear inclusions, and decreased tyrosine

12 Neuronal intranuclear inclusions are found in the brains

13 Neuronal intranuclear inclusions are present in affected

14 We suggest that neuronal intranuclear inclusions are the common neuropat

15 Striatal neuronal intranuclear inclusion burden was similar betwe

16 Neuronal intranuclear inclusion disease (NIID) is a mult

17 Neuronal intranuclear inclusions have been found in the

18 ein (CBP) and mSin3a, and CBP to localize to neuronal intranuclear inclusions in a transgenic mouse m

19 cts is directly relevant to the formation of neuronal intranuclear inclusions in Huntington's disease

20 The discovery last year of neuronal intranuclear inclusions in Huntington's disease

21 cle pathology and by the late development of neuronal intranuclear inclusions in spinal neurons.

22 deficits, weight loss, cerebral atrophy, and neuronal intranuclear inclusions in the R6/2 transgenic

23 Neuronal intranuclear inclusion (NII) formation and cell

24 Patient autopsy material reveals neuronal intranuclear inclusions (NII) in affected regio

25 agment of mutant huntingtin was localized to neuronal intranuclear inclusions (NIIs) and dystrophic n

26 brain and hastened both the presentation of neuronal intranuclear inclusions (NIIs) and the onset of

27 (AF) as early as 9 weeks of age and striatal neuronal intranuclear inclusions (NIIs) by 20 weeks.

28 Neuronal intranuclear inclusions (NIIs) characteristical

29 The cellular localization and development of neuronal intranuclear inclusions (NIIs) in cortex and st

30 The transgenic mice described here develop neuronal intranuclear inclusions (NIIs), a hallmark of S

31 criptional dysregulation and accumulation of neuronal intranuclear inclusions (NIIs).

32 analysis demonstrates widespread ubiquinated neuronal intranuclear inclusions (NIIs).

33 ith development of both disease symptoms and neuronal intranuclear inclusions (NIIs).

34 t death, other than widespread ubiquitinated neuronal intranuclear inclusions (NIIs).

35 neurites, and in some familial cases UBQ-ir neuronal intranuclear inclusions of a lentiform appearan

36 ities, reactive gliosis and the formation of neuronal intranuclear inclusions predominating in the st

37 hin the hippocampus mirror the appearance of neuronal intranuclear inclusions, suggesting a relations

38 ic mouse models of Huntington's disease with neuronal intranuclear inclusions, the identification of

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