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1 matostatin (co-containing neuropeptide Y and neuronal nitric oxide synthase).
2 )gamma signaling that leads to activation of neuronal nitric oxide synthase.
3  by influencing the phosphorylation state of neuronal nitric oxide synthase.
4 glutamate receptor (mGluR)-2 and mGluR3, and neuronal nitric oxide synthase.
5 vels of GAD(67), GAD(65), GAT-1, mGluR2, and neuronal nitric oxide synthase.
6 responses and are mediated, in part, through neuronal nitric oxide synthase.
7 19's IQ motif, binds calmodulin and inhibits neuronal nitric oxide synthase.
8 Cs differentiated into neurons and expressed neuronal nitric oxide synthase.
9 ral ischemia in vivo, MMP-9 colocalized with neuronal nitric oxide synthase.
10 d for rabbit CPR and the reductase domain of neuronal nitric oxide synthase.
11 ced production of muscle membrane-associated neuronal nitric oxide synthase.
12  interneurons ( approximately 1%) expressing neuronal nitric oxide synthase.
13 ent, but only a small proportion coexpressed neuronal nitric oxide synthase.
14 eversed by NXN-323, a selective inhibitor of neuronal nitric oxide synthase.
15 -93 and the N-methyl-d-aspartate receptor or neuronal nitric-oxide synthase.
16 ygenase or reductase domains of inducible or neuronal nitric-oxide synthases.
17 r relevant nitric oxide sources, namely, the neuronal (nitric oxide synthase 1) isoform, in sepsis an
18 , we show that FMRP regulates translation of neuronal nitric oxide synthase 1 (NOS1) in the developin
19  alpha-, beta-, and gamma-splice variants of neuronal nitric oxide synthase 1 (NOS1), and NOS1beta ex
20                             The inhibitor of neuronal nitric oxide synthase, 7-nitroindazole, also pr
21                                              Neuronal nitric oxide synthase, a component of the dystr
22 n time- and isoform-specific endothelial and neuronal nitric oxide synthase activation and nitric oxi
23    The observed early loss of subsarcolemmal neuronal nitric oxide synthase activity, onset of oxidat
24 rtex in vivo and that it acts in addition to neuronal nitric oxide synthase (alphaNOS1), an enzyme th
25                     The combination of early neuronal nitric oxide synthase and delayed inducible nit
26                      Increased expression of neuronal nitric oxide synthase and increased 3-nitrotyro
27 polarization, stimulation of CPARs activated neuronal nitric oxide synthase and production of cGMP.
28 emodeling is attributable to preservation of neuronal nitric oxide synthase and sarcoplasmic reticulu
29 ed to nonlethal 2-h serum deprivation, their neuronal nitric oxide synthase and Trx were up-regulated
30 ine acetyltransferase, tyrosine hydroxylase, neuronal nitric oxide synthase, and vasoactive intestina
31 eptor 4, glial fibrillary acidic protein and neuronal nitric-oxide synthase, and Abeta and p-Tau(Ser-
32 e elevated association of alpha1-syntrophin, neuronal nitric-oxide synthase, and alpha-dystroglycan w
33 ported that heme-dependent activation of apo-neuronal nitric oxide synthase (apo-nNOS) to the active
34 ich encodes a carboxy-terminal PDZ ligand of neuronal nitric oxide synthase, are associated with the
35 amide (6) is as potent an inhibitor of nNOS (neuronal nitric oxide synthase) as 2; selectivity for nN
36 dy identified NOS1AP (CAPON), a regulator of neuronal nitric oxide synthase, as a new target that mod
37 s confirmed by detection of sarcoglycans and neuronal nitric oxide synthase at the sarcolemma.
38 l entropy retention (CaM in complex with the neuronal nitric oxide synthase binding sequence), Lys-14
39    Next, we establish that ubiquitination of neuronal nitric-oxide synthase by the native ubiquitinat
40 the gene for carboxyl-terminal PDZ ligand of neuronal nitric oxide synthase (CAPON), making it a prim
41 re of the FAD/NADPH-containing domain of rat neuronal nitric-oxide synthase, complexed with NADP(+),
42 e, nitrergic nerve fibers lose some of their neuronal nitric oxide synthase content and function.
43 ave tested the hypothesis that inhibition of neuronal nitric oxide synthase could ameliorate the seve
44 ayed gastric emptying; expression of Kit and neuronal nitric oxide synthase decreased markedly in the
45 nometry was performed in wild-type controls, neuronal nitric oxide synthase-deficient (nNOS(-/-)) mic
46 tes from wild-type (WT), NO/redox imbalance (neuronal nitric oxide synthase-deficient mice-1 [NOS1(-/
47                            Mice deficient in neuronal nitric-oxide synthase demonstrated a decrease i
48 ovide definitive evidence that inhibition of neuronal nitric oxide synthase-derived excessive nitric
49 wnregulation of metabolism through increased neuronal nitric oxide synthase dimerization.
50           We also report that this increased neuronal nitric oxide synthase expression and increased
51                                              Neuronal nitric oxide synthase expression during ischemi
52         Akt inhibition independently reduced neuronal nitric oxide synthase expression in Schwann cel
53                                              Neuronal nitric oxide synthase expression in sciatic ner
54 ro, rosuvastatin inhibited downregulation of neuronal nitric oxide synthase expression induced by hig
55 ive oxygen species, rapidly restored Kit and neuronal nitric oxide synthase expression, and completel
56  rosuvastatin, consistent with the change of neuronal nitric oxide synthase expression.
57     Both inducible nitric oxide synthase and neuronal nitric oxide synthase expressions correlated wi
58           Herein, we describe studies of rat neuronal nitric oxide synthase FAD domains, in which the
59  defects in mice harboring a deletion of the neuronal nitric oxide synthase gene (nNOS).
60              We determined whether mice with neuronal nitric oxide synthase gene disruption (nNOS(-/-
61                                              Neuronal nitric oxide synthase group was administered wi
62                                  Control and neuronal nitric oxide synthase groups were given a cutan
63 gnificant reduction was seen in staining for neuronal nitric oxide synthase, heme oxygenase-2, tyrosi
64 uclear redox factor-1 (Ref-1), mRNA of human neuronal nitric oxide synthase (hNOS1) was maximally up-
65  greater proportion of the synaptic proteins neuronal nitric oxide synthase, Homer, and beta-catenin.
66  was conducted to determine the origin(s) of neuronal nitric oxide synthase-immunoreactive (NOS-IR) f
67 ration thus promotes increased expression of neuronal nitric oxide synthase in dural afferents, which
68 t the triptan-induced increased labelling of neuronal nitric oxide synthase in dural afferents.
69           CRF(2) colocalized with CRF(1) and neuronal nitric oxide synthase in the rat colon, LMMP, a
70  rat trigeminal dural afferents labelled for neuronal nitric oxide synthase in the trigeminal ganglio
71 munoreactivity for VIP was co-localized with neuronal nitric oxide synthase in varicose intramuscular
72  act as an inhibitor of calmodulin-dependent neuronal nitric oxide synthase in vitro, although PEP-19
73    NMDAR stimulation, well known to activate neuronal nitric oxide synthase, increases both nitrosyla
74  blocking either GluN2B-containing NMDARs or neuronal nitric oxide synthase, indicating changing role
75  highlight the potential clinical utility of neuronal nitric oxide synthase inhibition in preventing
76                                Additionally, neuronal nitric oxide synthase inhibition prevented envi
77                                          The neuronal nitric oxide synthase inhibitor AAAN (N-(4S)-4-
78              Treatment of injured sheep with neuronal nitric oxide synthase inhibitor attenuated all
79 nses were not corrected by the addition of a neuronal nitric oxide synthase inhibitor indicating that
80 uous infusion of 7-nitroindazole, a specific neuronal nitric oxide synthase inhibitor, during the fir
81 stricted analogues of a potent and selective neuronal nitric oxide synthase inhibitor, l-nitroarginin
82 ide synthase (injured, treated with specific neuronal nitric oxide synthase inhibitor, ZK 234238 (n =
83 tures, by the addition of 7-nitroindazole, a neuronal nitric oxide synthase inhibitor.
84 l (injured, treated with saline, n = 6); and neuronal nitric oxide synthase (injured, treated with sp
85    Evaluation of inducible, endothelial, and neuronal nitric-oxide synthases (iNOS, eNOS, and nNOS) r
86                         We further show that neuronal nitric oxide synthase is regulated by cholinerg
87 omalonate adducts inactivate selectively the neuronal nitric oxide synthase isoform in a manner compl
88 11b), and neuronal (neuron-specific enolase, neuronal nitric oxide synthase) markers in IR(Akita) com
89 mRNA levels, but there was small increase in neuronal nitric-oxide synthase mRNA levels in the pentos
90             Without syntrophin, utrophin and neuronal nitric oxide synthase mu (nNOSmu) fail to local
91                               Among these is neuronal nitric oxide synthase mu (nNOSmu), which requir
92 that this effect may be mediated in part via neuronal nitric oxide synthase/nitric oxide and phosphat
93 mouse model of human enteric neuropathy, the neuronal nitric oxide synthase (nNOS(-/-)) deficient mou
94 ng N-methyl-d-aspartate (NMDA) receptors and neuronal nitric oxide synthase (nNOS) activity in part m
95 I), a reversible inhibitor of both MAO-B and neuronal nitric oxide synthase (nNOS) activity.
96                            As a consequence, neuronal nitric oxide synthase (nNOS) and aquaporin-4 we
97 isualization of intracellular NO, along with neuronal nitric oxide synthase (nNOS) and cGMP immunohis
98 ic route to chiral pyrrolidine inhibitors of neuronal nitric oxide synthase (nNOS) and crystal struct
99       Using NADPH-diaphorase histochemistry, neuronal nitric oxide synthase (nNOS) and inducible nitr
100                                              Neuronal nitric oxide synthase (nNOS) and p38MAPK are st
101 eptor antagonist mecamylamine, inhibitors of neuronal nitric oxide synthase (nNOS) and soluble guanyl
102 erstood, cortical neurons immunoreactive for neuronal nitric oxide synthase (nNOS) and the neurokinin
103    Cortical interneurons, immunoreactive for neuronal nitric oxide synthase (nNOS) and the receptor N
104                      Selective inhibitors of neuronal nitric oxide synthase (nNOS) are regarded as va
105 methyl-d-aspartic acid (NMDA) receptors, and neuronal nitric oxide synthase (nNOS) at the neuronal pl
106                                          The neuronal nitric oxide synthase (nNOS) basal and calmodul
107                This paper reports a study of neuronal nitric oxide synthase (nNOS) by FTIR difference
108        The PDZ protein interaction domain of neuronal nitric oxide synthase (nNOS) can heterodimerize
109             We have previously reported that neuronal nitric oxide synthase (nNOS) can protect develo
110                          The distribution of neuronal nitric oxide synthase (nNOS) containing neurons
111 ior colliculus (SC), we studied the onset of neuronal nitric oxide synthase (nNOS) expression in the
112 gy are attributable to the secondary loss of neuronal nitric oxide synthase (nNOS) from dystrophin-de
113  defects typically cause a secondary loss of neuronal nitric oxide synthase (nNOS) from the post-syna
114 ineered mice with targeted disruption of the neuronal nitric oxide synthase (nNOS) gene established t
115                      Selective inhibition of neuronal nitric oxide synthase (nNOS) has been shown to
116 levels of nitric oxide through inhibition of neuronal nitric oxide synthase (nNOS) has the potential
117 tion of choline acetyltransferase (ChAT) and neuronal nitric oxide synthase (nNOS) immunoreactivities
118  early-born neurons containing calretinin or neuronal nitric oxide synthase (nNOS) immunoreactivities
119 aternal aggression by selectively inhibiting neuronal nitric oxide synthase (nNOS) in lactating prair
120       For further elucidation of the role of neuronal nitric oxide synthase (nNOS) in macula densa (M
121 Y), and the nitric oxide synthesizing enzyme neuronal nitric oxide synthase (nNOS) in nerve fibers of
122  and that sarcolemma-localized signalling by neuronal nitric oxide synthase (nNOS) in skeletal muscle
123 ble messenger molecule produced primarily by neuronal nitric oxide synthase (nNOS) in the central ner
124                                              Neuronal nitric oxide synthase (nNOS) in the medial preo
125 y gene, in the neurons containing VGLUT3 and neuronal nitric oxide synthase (nNOS) in the PBN followi
126                                              Neuronal nitric oxide synthase (nNOS) inhibition is a pr
127 ect was blocked by NMDA receptor (NMDAR) and neuronal nitric oxide synthase (nNOS) inhibition.
128 resent study, we examined the effects of the neuronal nitric oxide synthase (nNOS) inhibitor 7-nitroi
129 5-phosphonopentanoic acid (AP5; 100 microM), neuronal nitric oxide synthase (nNOS) inhibitor 7-nitroi
130 fluoromethylphenyl imidazole (TRIM), a novel neuronal nitric oxide synthase (nNOS) inhibitor, in thre
131 ly inhibited by either endothelial (eNOS) or neuronal nitric oxide synthase (nNOS) inhibitors and com
132 rve injury, and their effects are blocked by neuronal nitric oxide synthase (nNOS) inhibitors and M4
133 odipeptide analogues as potent and selective neuronal nitric oxide synthase (nNOS) inhibitors.
134 metabotropic glutamate receptor5 (mGluR5) on neuronal nitric oxide synthase (nNOS) interneurons.
135                                Inhibition of neuronal nitric oxide synthase (nNOS) is a promising the
136                  The C-terminal tail (CT) of neuronal nitric oxide synthase (nNOS) is a regulatory el
137                                              Neuronal nitric oxide synthase (nNOS) is a target for de
138                                              Neuronal nitric oxide synthase (nNOS) is abundantly expr
139                      Selective inhibition of neuronal nitric oxide synthase (nNOS) is an important th
140                                              Neuronal nitric oxide synthase (nNOS) is broadly express
141                                              Neuronal nitric oxide synthase (nNOS) is composed of an
142         Excess nitric oxide (NO) produced by neuronal nitric oxide synthase (nNOS) is implicated in n
143                                              Neuronal nitric oxide synthase (nNOS) is implicated in s
144                                              Neuronal nitric oxide synthase (nNOS) is induced in dors
145                                              Neuronal nitric oxide synthase (nNOS) is present in gast
146                    Electron transfer through neuronal nitric oxide synthase (nNOS) is regulated by th
147                  We reported previously that neuronal nitric oxide synthase (nNOS) is the predominant
148                  We previously reported that neuronal nitric oxide synthase (nNOS) is the predominant
149                         Recent data suggests neuronal nitric oxide synthase (nNOS) mediates the NO co
150                                              Neuronal nitric oxide synthase (nNOS) membrane delocaliz
151 aptic associated protein 102 kD (SAP102) and neuronal nitric oxide synthase (nNOS) mRNAs.
152 h targeted disruptions (knockout; KO) in the neuronal nitric oxide synthase (nNOS) or inducible (i)NO
153 ipeptide inhibitors are highly selective for neuronal nitric oxide synthase (nNOS) over the endotheli
154 sus sequence -E-S/T-X-V-COOH, also binds the neuronal nitric oxide synthase (nNOS) PDZ domain in a ma
155 indicated that inhibition or ablation of the neuronal nitric oxide synthase (nNOS) prevents the devel
156                                              Neuronal nitric oxide synthase (nNOS) produces gaseous n
157 as decrease in the dimer to monomer ratio of neuronal nitric oxide synthase (nNOS) protein and lowere
158  reperfusion, mediated via alteration of the neuronal nitric oxide synthase (nNOS) protein isoform wi
159 e of basal and calmodulin- (CaM-) stimulated neuronal nitric oxide synthase (nNOS) reduction of 2,6-d
160                             NO production by neuronal nitric oxide synthase (nNOS) requires calmoduli
161                  Double labeling showed that neuronal nitric oxide synthase (nNOS) seldom colocalized
162           The role of central and peripheral neuronal nitric oxide synthase (nNOS) splice variants in
163 eurons in vitro and analysis of mice lacking neuronal nitric oxide synthase (nNOS) suggest that NO me
164                        The secondary loss of neuronal nitric oxide synthase (nNOS) that occurs in dys
165 y S-nitrosylation after selective binding of neuronal nitric oxide synthase (nNOS) to COX-2.
166 ort that a single mutation of Leu-337 in rat neuronal nitric oxide synthase (nNOS) to His makes the e
167                                  SNTA1 links neuronal nitric oxide synthase (nNOS) to the nNOS inhibi
168 n which there was myocardial expression of a neuronal nitric oxide synthase (nNOS) transgene.
169                                  It binds to neuronal nitric oxide synthase (nNOS) via the adaptor pr
170            Expression of RET, GFRalpha1, and neuronal nitric oxide synthase (nNOS) was assessed by We
171     Here, a thioether-based inhibitor (3) of neuronal nitric oxide synthase (nNOS) was designed, and
172 r oxidative stress, and to determine whether neuronal nitric oxide synthase (nNOS) was involved in su
173 endothelial nitric oxide synthase (eNOS) and neuronal nitric oxide synthase (nNOS) were determined by
174                Short-term over-expression of neuronal nitric oxide synthase (nNOS) with adenoviral ge
175                      The X-ray structures of neuronal nitric oxide synthase (nNOS) with N(omega)-hydr
176 st studies have demonstrated the presence of neuronal nitric oxide synthase (nNOS) within the LHA, it
177 nuation is a result of altered expression of neuronal nitric oxide synthase (nNOS) within the rostral
178  hypothesis that a single allele deletion of neuronal nitric oxide synthase (nNOS) would impair the n
179 e observed a decrease in membrane-associated neuronal nitric oxide synthase (nNOS), a decrease in PSD
180                The PDZ-kinases phosphorylate neuronal nitric oxide synthase (nNOS), a known binding p
181 e aim of this study was to determine whether neuronal nitric oxide synthase (nNOS), a regulator of ne
182 ping PVN, including estrogen receptors (ER), neuronal nitric oxide synthase (nNOS), and brain-derived
183 ls of basic fibroblast growth factor (bFGF), neuronal nitric oxide synthase (nNOS), and caspase-3.
184 from muscle results in the secondary loss of neuronal nitric oxide synthase (nNOS), and NO is able to
185 in postsynaptic density protein-95 (PSD-95), neuronal nitric oxide synthase (nNOS), and the N-methyl-
186 s CHIP(+/+) cytosol to ubiquitinate purified neuronal nitric oxide synthase (nNOS), another establish
187 racer detection and immunohistochemistry for neuronal nitric oxide synthase (nNOS), choline acetyl tr
188 ergic interneurons, which express the enzyme neuronal nitric oxide synthase (nNOS), has recently been
189  collected to evaluate eNOS phosphorylation, neuronal nitric oxide synthase (nNOS), inducible nitric
190 ath seems to require the local activation of neuronal nitric oxide synthase (nNOS), itself held by sc
191 ne-related peptide (CGRP), substance P (SP), neuronal nitric oxide synthase (nNOS), neurofilament 200
192 mining CARTp with tyrosine hydroxylase (TH), neuronal nitric oxide synthase (nNOS), or choline acetyl
193 ors, voltage gated calcium channels (VGCCs), neuronal nitric oxide synthase (nNOS), oxidative stress
194                             Like that of the neuronal nitric oxide synthase (nNOS), the binding of Ca
195                Upregulation of the levels of neuronal nitric oxide synthase (nNOS), the enzyme system
196 t neonatal mice carrying a null mutation for neuronal nitric oxide synthase (nNOS), the enzyme that s
197 hat gephyrin was found to be associated with neuronal nitric oxide synthase (nNOS), the major source
198  in an increased association of DLC/PIN with neuronal nitric oxide synthase (nNOS), thereby reducing
199 dies against protein gene product (PGP) 9.5, neuronal nitric oxide synthase (nNOS), vasoactive intest
200 hich have the KXTQT recognition sequence and neuronal nitric oxide synthase (nNOS), which has the GIQ
201      This method was successfully applied to neuronal nitric oxide synthase (nNOS), which is implicat
202          Erection requires the activation of neuronal nitric oxide synthase (nNOS), which is tightly
203 rojecting choline acetyltransferase (ChAT)-, neuronal nitric oxide synthase (nNOS)-, and parvalbumin
204 vo during theta rhythm, into NGFCs of rat or neuronal nitric oxide synthase (nNOS)-Cre-tdTomato mouse
205 termine whether Type 1 diabetes (T1D) alters neuronal nitric oxide synthase (nNOS)-dependent reactivi
206                            Overproduction of neuronal nitric oxide synthase (nNOS)-derived NO is detr
207  therefore, have characterized some of these neuronal nitric oxide synthase (nNOS)-thioether inhibito
208 NO production that depended on activation of neuronal nitric oxide synthase (nNOS).
209 or (NMDAR) stimulate the production of NO by neuronal nitric oxide synthase (nNOS).
210 he myenteric plexus are neurons that contain neuronal nitric oxide synthase (nNOS).
211 om the colocalization of NMDA receptors with neuronal nitric oxide synthase (nNOS).
212 tivation of p38 MAP kinase (phospho-p38) and neuronal nitric oxide synthase (nNOS).
213 tween the reductase and oxygenase domains of neuronal nitric oxide synthase (nNOS).
214 ave established the cardioprotective role of neuronal nitric oxide synthase (nNOS).
215 y (BMD) and Duchenne muscular dystrophy lack neuronal nitric oxide synthase (nNOS).
216 the involvement of both inducible (iNOS) and neuronal nitric oxide synthase (nNOS).
217 lation to production of nitric oxide (NO) by neuronal nitric oxide synthase (nNOS).
218 ains neurons that express NMDA receptors and neuronal nitric oxide synthase (nNOS).
219          MNTB neurons express high levels of neuronal nitric oxide synthase (nNOS).
220 miR-31-dependent reduction in dystrophin and neuronal nitric oxide synthase (nNOS, also known as NOS1
221 n this study, we show that mice deficient in neuronal nitric oxide synthase (nNOS-/-) are more vulner
222                                              Neuronal nitric-oxide synthase (nNOS or NOS I) and endot
223 ks parallel to the FAD isoalloxazine ring in neuronal nitric-oxide synthase (nNOS) and is representat
224 overed that certain drugs suicide-inactivate neuronal nitric-oxide synthase (nNOS) and lead to the pr
225           Citrulline formation by both human neuronal nitric-oxide synthase (nNOS) and mouse macropha
226 We show here that during the inactivation of neuronal nitric-oxide synthase (nNOS) by AG that the pro
227  is established that suicide inactivation of neuronal nitric-oxide synthase (nNOS) by drugs and other
228        To identify whether the PDZ domain of neuronal nitric-oxide synthase (nNOS) can play such a ro
229                                              Neuronal nitric-oxide synthase (nNOS) contains a unique
230 xport occurred, paralleled by dystrophin and neuronal nitric-oxide synthase (nNOS) down-regulation an
231                                          The neuronal nitric-oxide synthase (nNOS) flavoprotein domai
232                                              Neuronal nitric-oxide synthase (nNOS) has various splici
233 metabolism-based irreversible inactivator of neuronal nitric-oxide synthase (nNOS) in vitro, causes t
234 f the NADPH-dependent reactions catalyzed by neuronal nitric-oxide synthase (nNOS) including ferricyt
235  Ca(2+)-activated calmodulin (Ca(2+)-CaM) to neuronal nitric-oxide synthase (nNOS) increases the rate
236                                              Neuronal nitric-oxide synthase (nNOS) is a constitutivel
237                       It is established that neuronal nitric-oxide synthase (nNOS) is ubiquitylated a
238                 The crystal structure of the neuronal nitric-oxide synthase (nNOS) NADPH/FAD binding
239  catalysis by tetrahydrobiopterin (H4B)-free neuronal nitric-oxide synthase (nNOS) to understand how
240 arginine, is a weak competitive inhibitor of neuronal nitric-oxide synthase (nNOS) with an apparent K
241  is established that suicide inactivation of neuronal nitric-oxide synthase (nNOS) with guanidine com
242 ly enhances SR S-nitrosylation by activating neuronal nitric-oxide synthase (nNOS).
243  endothelial nitric-oxide synthase (eNOS) or neuronal nitric-oxide synthase (nNOS).
244  BH(4) decreased the active, dimeric form of neuronal nitric-oxide synthase (nNOS).
245 oteins, a synthetic enzyme for nitric oxide (neuronal nitric oxide synthase, nNOS) and a nonphosphory
246                        Of these, only NOS I (neuronal nitric-oxide synthase; nNOS) is expressed as ca
247 of DMD, dystrophin deficiency causes loss of neuronal nitric oxide synthase (nNOSmu) from the sarcole
248 eletal muscle is the muscle-specific form of neuronal nitric oxide synthase (nNOSmu) produced by the
249 rease and mislocalization of muscle-specific neuronal nitric oxide synthase (nNOSmu), leading to func
250 ver conditions using the oxygenase domain of neuronal nitric oxide synthase (nNOSoxy) and rapid scann
251 , calretinin [CR], and parvalbumin [PV]) and neuronal nitric oxide synthase (NOS), chemical markers t
252 ain areas expressing PDE9A mRNA also contain neuronal nitric oxide synthase (NOS), the enzymatic sour
253  METH (30 mg/kg, ip) and visualized striatal neuronal nitric oxide synthase (NOS)-positive cells by i
254 is used widely as a histochemical marker for neuronal nitric oxide synthase (NOS).
255             Nitric oxide (NO) generated from neuronal nitric oxide synthase (NOS-1) in intrinsic card
256 licited by exercise training is dependent on neuronal nitric oxide synthase (NOS-1), since the NO-cGM
257 ibers of the cochlear nerve were stained for neuronal nitric oxide synthase (NOS-I) but not for sGCbe
258 g-term potentiation, but the failure to find neuronal nitric oxide synthase (NOS-I) in the dendritic
259  objectives of this study were to synthesize neuronal nitric oxide synthase (NOS-I)-selective imaging
260 ng, we compared the localization of sGC with neuronal nitric oxide synthase (NOS-I, the NO-producing
261 nce of a special auto-regulation property of neuronal nitric-oxide synthase (NOS) based on NO near-ge
262 c superoxide O(2)(.-) -producing enzyme, and neuronal nitric oxide synthase (NOS1) coimmunoprecipitat
263 a-estradiol (<10 nM) concomitantly increased neuronal nitric oxide synthase (NOS1) expression and cel
264 on and induction of constitutively expressed neuronal nitric oxide synthase (NOS1) in both human PD a
265 tric oxide (NO) derived from the activity of neuronal nitric oxide synthase (NOS1) is involved in S-n
266                                              Neuronal nitric oxide synthase (NOS1) plays key cardiac
267    Accumulating data support the notion that neuronal nitric oxide synthase (NOS1) regulates the card
268 tations, common genetic variants in CAPON, a neuronal nitric oxide synthase (NOS1) regulator, have re
269 R2) has been proposed to be an end target of neuronal nitric oxide synthase (NOS1) signalling.
270 UDP glucuronosyltransferase (UGT1), plectin, neuronal nitric oxide synthase (NOS1), and glucocorticoi
271         Here we examine the role of LepRb in neuronal nitric oxide synthase (NOS1)-expressing LebRb (
272 S1AP; also known as C-terminal PDZ ligand of neuronal nitric oxide synthase or CAPON) in dendritic pa
273 FP and SS, almost all were also positive for neuronal nitric oxide synthase or m2 muscarinic receptor
274 ant decrease in neuron counts, expression of neuronal nitric oxide synthase or smooth muscle myosin.
275   CsA did not affect cortical endothelial or neuronal nitric-oxide synthase or mRNA levels, but there
276  = 0.013), beta-dystroglycan (P = 0.025) and neuronal nitric oxide synthase (P = 0.034) expression wa
277 llel beta-strand at the same position as the neuronal nitric-oxide synthase peptide in the LC8 comple
278                                       In rat neuronal nitric oxide synthase, Phe1395 is positioned ov
279 in (DLC) (also known as protein inhibitor of neuronal nitric oxide synthase [PIN]) was significantly
280 neurons (MSN) selectively degenerate whereas neuronal nitric-oxide-synthase-positive interneurons (nN
281 ing cells of capon (C-terminal PDZ ligand of neuronal nitric-oxide synthase protein), apoptosis-assoc
282 whether AR-R 17477, a selective inhibitor of neuronal nitric oxide synthase, reduces brain injury in
283 succinate synthase, argininosuccinate lyase, neuronal nitric-oxide synthase, RNA-binding motif protei
284 rahydrobiopterin, dimeric and phosphorylated neuronal nitric oxide synthase, sarcoplasmic reticulum C
285 els, the dimerization and phosphorylation of neuronal nitric oxide synthase, sarcoplasmic reticulum C
286                  Double labeling of sGC with neuronal nitric oxide synthase showed that the two prote
287                  Double labeling of sGC with neuronal nitric oxide synthase showed that the two prote
288 in nearby capillaries via the NMDA receptors-neuronal nitric oxide synthase signaling pathway.
289 ted tat uptake, NMDA receptor activation, or neuronal nitric oxide synthase significantly reduces ens
290 ns 3 and 6, multiple PDZ protein 1, Tamalin, neuronal nitric oxide synthase, syntrophins, protein int
291                As well as the involvement of neuronal nitric-oxide synthase, the proliferative effect
292 rophin and alpha-syntrophin, which localizes neuronal nitric oxide synthase to the sarcolemma, have n
293 uitment of signalling proteins such as nNOS (neuronal nitric oxide synthase) to the DPC [4].
294 um channels and signaling molecules, such as neuronal nitric oxide synthase, to the dystrophin comple
295                                              Neuronal nitric-oxide synthase, unlike its endothelial a
296 curred in the cell somas of more than 90% of neuronal nitric oxide synthase, vasoactive intestinal pe
297                                A decrease in neuronal nitric oxide synthase, vasoactive intestinal pe
298 in-associated proteins alpha-sarcoglycan and neuronal nitric oxide synthase were also restored at the
299  between inducible nitric oxide synthase and neuronal nitric oxide synthase with renal blood flow sug
300 malian endothelial nitric oxide synthase and neuronal nitric oxide synthase, yet it is a distinct enz

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