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1 he circadian clock and processes involved in neuronal plasticity.
2 brain regions with suspected neurogenesis or neuronal plasticity.
3 ases and phosphatases involved in modulating neuronal plasticity.
4 f Egr2/Krox20 further because of its role in neuronal plasticity.
5 ed salience of cues through learning-related neuronal plasticity.
6 one important mechanism maintaining lifelong neuronal plasticity.
7 neurotrophic factor (BDNF) as a mediator of neuronal plasticity.
8 prominent role of REM sleep in sleep-related neuronal plasticity.
9 ponse to an acute challenge, also reflecting neuronal plasticity.
10 ibits nicotinic receptor function and limits neuronal plasticity.
11 tical role in cortical neurons to facilitate neuronal plasticity.
12 , regulates many neural functions, including neuronal plasticity.
13 a strong link exists between sleep need and neuronal plasticity.
14 ociated with brain-specific genes related to neuronal plasticity.
15 the brain that may underlie cocaine-induced neuronal plasticity.
16 in regulating AMPA receptor trafficking and neuronal plasticity.
17 nt motor learning model to study theories of neuronal plasticity.
18 ght the significance of small RNA control in neuronal plasticity.
19 rtments during activity is a key mediator of neuronal plasticity.
20 ent gene regulation controls many aspects of neuronal plasticity.
21 leus during transcription-dependent forms of neuronal plasticity.
22 nt stress may occur via mechanisms involving neuronal plasticity.
23 iced mRNA by microRNA in drug adaptation and neuronal plasticity.
24 ively regulating memory stabilization during neuronal plasticity.
25 are crucial for numerous processes including neuronal plasticity.
26 critical roles in the mechanisms underlying neuronal plasticity.
27 aumatic brain injury (TBI) is partly through neuronal plasticity.
28 in neurodegenerative disease, epilepsy, and neuronal plasticity.
29 regulate angiogenesis, axonal guidance, and neuronal plasticity.
30 nel (Kcnd2) as cell type-specific markers of neuronal plasticity.
31 thway, a signaling cascade known to regulate neuronal plasticity.
32 -driven transcription programs that underlie neuronal plasticity.
33 of ion channels by phosphorylation underlies neuronal plasticity.
34 e conjoint influence of these two factors on neuronal plasticity.
35 gy field and will spread in areas related to neuronal plasticity.
36 ould mediate tolerance to alcohol, a form of neuronal plasticity.
37 good target for influencing neurogenesis and neuronal plasticity.
38 ynapse is thought to be a key determinant of neuronal plasticity.
39 r trafficking is important for many forms of neuronal plasticity.
40 drites is thought to be underlying long-term neuronal plasticity.
41 in axon guidance, synaptic development, and neuronal plasticity.
42 ction and provides a model for understanding neuronal plasticity.
43 otoreceptor survival or in the regulation of neuronal plasticity.
44 hannels may play a role in calcium-dependent neuronal plasticity.
45 ggesting a role in RAS-mediated postsynaptic neuronal plasticity.
46 ffects of D(1) and D(2) receptor blockade on neuronal plasticity.
47 ions, including neuronal differentiation and neuronal plasticity.
48 this receptor expression is associated with neuronal plasticity.
49 ation in expression of genes associated with neuronal plasticity.
50 ressed in the CNS during events that require neuronal plasticity.
51 and activation that can explain its role in neuronal plasticity.
52 CREB-mediated transcription is important for neuronal plasticity.
53 een shown to play a role in certain forms of neuronal plasticity.
54 ty in the hippocampus that may help regulate neuronal plasticity.
55 elevation is closely associated with altered neuronal plasticity.
56 indeed interfere with functions critical for neuronal plasticity.
57 proteins like PC4 play a significant role in neuronal plasticity.
58 nses of MAPK signaling in activity-dependent neuronal plasticity.
59 rotrophic factor (BDNF) levels as a proxy of neuronal plasticity.
60 and memory, amyloid protein processing, and neuronal plasticity.
61 is used to study dopamine-receptor-mediated neuronal plasticity.
62 of diverse transcription-dependent forms of neuronal plasticity.
63 roduce a supersensitive form of D1- mediated neuronal plasticity.
64 al nets and concerned with the modulation of neuronal plasticity.
65 itical role in the formation of long lasting neuronal plasticity.
66 lation are likely to serve as an effector in neuronal plasticity.
67 suggests roles for subtoxic levels of MLP in neuronal plasticity.
68 es long-term potentiation and other forms of neuronal plasticity.
69 d a critical role in cognitive functions and neuronal plasticity.
70 p codon of mRNAs, including genes related to neuronal plasticity.
71 tiation, mitogenesis, tumor suppression, and neuronal plasticity.
72 near the start of the axon that is a site of neuronal plasticity.
73 eptor concentration, indicating some form of neuronal plasticity.
74 es for segregation between distinct modes of neuronal plasticity.
75 mechanisms of hormone-dependent behavior and neuronal plasticity.
76 f evidence suggest that CDKL5 is involved in neuronal plasticity.
77 ained responding, relapse-like behavior, and neuronal plasticity.
78 tinction of ethanol cues in association with neuronal plasticity.
79 versive state of withdrawal and BDNF-induced neuronal plasticity.
80 at have long term impacts on activity driven neuronal plasticity.
81 imal models, largely because of processes of neuronal plasticity.
82 g insight into signaling pathways underlying neuronal plasticity.
83 affolds, and signaling proteins required for neuronal plasticity.
84 hing an epigenetic landscape compatible with neuronal plasticity.
85 emporal sequence, induces different forms of neuronal plasticity.
86 plays an important role in several forms of neuronal plasticity.
87 ion of L-lactate as a signaling molecule for neuronal plasticity.
88 ptors is the pivotal event for initiation of neuronal plasticity.
89 d gene expression are critical for long-term neuronal plasticity.
90 yloid pathologic abnormalities and increased neuronal plasticity.
91 riatum is an important site for drug-induced neuronal plasticity.
92 and is expected to contribute to homeostatic neuronal plasticity.
93 onded to detoxification, probably reflecting neuronal plasticity.
94 of gene expression during activity-dependent neuronal plasticity.
95 dulating glutamatergic neurotransmission and neuronal plasticity.
96 possible point of intervention to facilitate neuronal plasticity.
98 of nNOS may contribute to the development of neuronal plasticity after specific types of peripheral n
99 BC (ChABC) has striking effects on promoting neuronal plasticity after spinal cord injury (SCI), but
100 ir cerebral cortex nor indices of structural neuronal plasticity along extreme time-scales have been
102 aviors by evaluating how the PFC affects LAT neuronal plasticity and activity that is evoked by previ
107 synapses are essential for the induction of neuronal plasticity and cognitive processes in animals.
110 DNA methylation, are essential regulators of neuronal plasticity and experience-driven behavioral cha
112 spines are dynamic structures which regulate neuronal plasticity and have crucial roles in myriad bra
113 mpal gene expression in pathways involved in neuronal plasticity and increased levels of norepinephri
114 f cannabinoid-mediated short-term retrograde neuronal plasticity and is found in numerous brain regio
116 previously unknown form of steroid-mediated neuronal plasticity and may be an initial step in establ
117 cellular resilience as well as synaptic and neuronal plasticity and may influence mood and affective
118 ry roles for CRFRs in an AD-relevant form of neuronal plasticity and may link datasets documenting al
121 To investigate the role of this kinase in neuronal plasticity and memory, we generated transgenic
123 ion factor Mef2 regulates activity-dependent neuronal plasticity and morphology in mammals, and clock
124 alpha-synuclein has been implicated in both neuronal plasticity and neurodegenerative disease, but i
125 m may be the result of underlying defects in neuronal plasticity and ongoing problems with synaptic s
126 ciated with odor stimulation on second order neuronal plasticity and phenotype in the olfactory syste
127 en activator (tPA), a protease implicated in neuronal plasticity and seizures, is induced in the limb
128 gliosis, and provides beneficial effects for neuronal plasticity and survival and attenuation of micr
129 expression is critical for certain forms of neuronal plasticity and survival in the mammalian nervou
130 effects on signaling pathways that regulate neuronal plasticity and survival may be a factor in the
131 activity, neurotrophic factors implicated in neuronal plasticity and survival, and oxidative stress.
132 , GR and AKT/PI3K pathways, known to promote neuronal plasticity and survival, as well as reported to
133 spartate receptors (NMDARs) are critical for neuronal plasticity and survival, whereas their excessiv
135 actions of BDNF and glucocorticoids promote neuronal plasticity and that disruption in either pathwa
136 that exposure to LAN influences behavior and neuronal plasticity and that this effect is likely media
137 s only weakly inhibited by GABA and shows no neuronal plasticity and the other subgroup is strongly i
138 ne knock-out models is always complicated by neuronal plasticity and the potential for restructuring
139 tions paired with rehabilitation can enhance neuronal plasticity and thereby improve functional recov
140 in syntaxin-1A causes abnormal regulation of neuronal plasticity and vesicle recycling and that the a
146 odegenerative diseases are closely linked to neuronal plasticity, and induce changes in adult neuroge
148 ses which regulate synaptic transmission and neuronal plasticity, and possibly underlie memory format
150 that the molecules involved in regulation of neuronal plasticity are also involved in the mode of act
152 in the brain as a regulator of synaptic and neuronal plasticity as well as its etiological role in t
153 h receptors (nAChRs) have been implicated in neuronal plasticity as well as neurodevelopmental, neuro
154 In addition, PFC stimulation blocked LAT neuronal plasticity associated with an affective conditi
156 ent brain, these molecules are implicated in neuronal plasticity associated with learning and memory.
157 ene expression are thought to be involved in neuronal plasticity associated with learning and memory.
158 mmatory indices, glutamatergic function, and neuronal plasticity at the cellular and circuitry level
159 to fear-associated responses by facilitating neuronal plasticity at the neuron-matrix interface.
160 important implications for studies of adult neuronal plasticity because it indicates that changes in
161 keleton is critical for neurodevelopment and neuronal plasticity, but how neurons spatially control a
162 need is affected by developmental stage and neuronal plasticity, but the underlying mechanisms remai
163 Estrogens are known to have broad effects on neuronal plasticity, but their specific role in neuronal
165 dation protein (FMRP) is thought to regulate neuronal plasticity by limiting dendritic protein synthe
166 re we show that this potential mechanism for neuronal plasticity can also be exploited in a rapid and
167 ar calcium signaling, energy metabolism, and neuronal plasticity can be influenced by inducing axonal
172 n essential substrate for the behavioral and neuronal plasticity driven by reward-related experiences
173 important implications for the regulation of neuronal plasticity during neuronal development and in r
175 d postsynaptic signaling pathways related to neuronal plasticity (e.g., dendritic spine formation).
176 to neocortical development promoting greater neuronal plasticity early in postnatal life preceded the
177 acy of phosphodiesterase (PDE) inhibitors as neuronal plasticity enhancers, little is known about the
179 tor with implications in neuroprotection and neuronal plasticity following NMDA receptor activation.
180 imal and after a 1-week delay, and implicate neuronal plasticity from unlesioned areas of the central
183 pared to the developing visual system, where neuronal plasticity has been well characterized at multi
184 the changes in gene expression that underlie neuronal plasticity, has attracted the attention of both
186 n is an essential component of many forms of neuronal plasticity, however, the intracellular mechanis
192 re provide further evidence for synaptic and neuronal plasticity in ipsi-lesional MVN neurons during
193 tivation of PAF-r signaling induces aberrant neuronal plasticity in LE and leads to chronic dysfuncti
196 neuronal correlates underlying tDCS-induced neuronal plasticity in older adults and thus might help
197 describe a recently identified mechanism of neuronal plasticity in primary afferent nociceptive nerv
205 ized by a high level of experience-dependent neuronal plasticity in the central nervous system and se
206 , neuronal coupling represents a new form of neuronal plasticity in the DRG and contributes to pain h
210 drugs of abuse and/or its role in long-term neuronal plasticity in the striatum may provide a new ro
212 ral months of life are a critical period for neuronal plasticity in the visual cortex during which an
214 opiate administration activates BDNF-related neuronal plasticity in the VTA that is necessary for bot
215 develop a normal baseline by which to assess neuronal plasticity in this corticostriatal system in ra
216 Techniques to induce activity-dependent neuronal plasticity in vivo allow the underlying signali
217 e induction and maintenance of some forms of neuronal plasticity, in hippocampus-dependent and hippoc
219 ivate several kinases that are important for neuronal plasticity, including Ca2+/calmodulin-dependent
220 lutamate receptors triggers various forms of neuronal plasticity, including cerebellar long-term depr
221 iew of the critical role of CREB and BDNF in neuronal plasticity, including learning and memory, the
222 (CaMKII) are required for numerous forms of neuronal plasticity, including long-term potentiation (L
223 receptor TrkB are crucial for many forms of neuronal plasticity, including structural long-term pote
224 ied numerous Mef2 target genes implicated in neuronal plasticity, including the cell-adhesion gene Fa
226 oral responses to swim stress as well as the neuronal plasticity involved in long-term behavioral cha
227 ent with the hypothesis that PbTx-2-enhanced neuronal plasticity involves NMDAR-dependent signaling.
228 be important forms of short-term retrograde neuronal plasticity involving endocannabinoids (eCB) and
229 and inhibition (DSI) are forms of short-term neuronal plasticity involving postsynaptic release of an
230 ls were also observed in an in vivo model of neuronal plasticity involving Zif268 induction: the effe
233 tifying the mechanisms of activity-dependent neuronal plasticity is crucial to finding therapeutic in
235 In cats, electrophysiologically assessed neuronal plasticity is minimal until approximately 3 wee
237 P).SIGNIFICANCE STATEMENT Activity-dependent neuronal plasticity is the cellular basis for learning a
238 n, an activity-dependent gene that regulates neuronal plasticity, is decreased by chronic unpredictab
239 cits, and the detection of memory underlying neuronal plasticity, is greatly impeded by a lack of pre
240 nin, an evolutionarily conserved mediator of neuronal plasticity, is responsible for this behavioral
242 ain region-specific changes in signaling and neuronal plasticity may be critical components in develo
243 different mechanisms and that NO effects on neuronal plasticity may involve G-kinase II regulation o
244 tory and/or inhibitory neurotransmission and neuronal plasticity may lead to aberrant functional conn
245 eurite extension and branching are important neuronal plasticity mechanisms that can lead to the addi
248 eport that effects of calorie restriction on neuronal plasticity, memory and social behavior are abol
249 (tPA), an extracellular protease involved in neuronal plasticity, modulates the biochemical and behav
250 Aergic and cholinergic systems influence the neuronal plasticity necessary for repetition priming.
251 findings reveal a critical role for CRMP2 in neuronal plasticity, neural function and behavioural mod
252 the behaviors and related cellular signaling/neuronal plasticity/neuroendocrine alterations in the de
253 function in a cooperative manner to regulate neuronal plasticity, neurogenesis, and neuronal survival
254 g visual system in modulating two aspects of neuronal plasticity: neurotransmitter expression and top
255 hese data suggest that exercise may increase neuronal plasticity not only in controls, but also in su
256 ynaptic transmission, causing impairments of neuronal plasticity observed in Fmr1 knock-out mice and
257 humans and rodents suggest that significant neuronal plasticity occurs in areas surrounding the cort
258 of two latitudes would have an impact on the neuronal plasticity of the song-control system in songbi
259 protein phosphorylation, a critical step in neuronal plasticity, on the level and duration of membra
262 2+)](i) is directly correlated with impaired neuronal plasticity; or (3) the previously observed age-
263 ption regulatory factors are induced in many neuronal plasticity paradigms, it is still unclear what
267 contingent reinforcement and the underlying neuronal plasticity resulted from the association of rei
268 the DAT may represent an extreme example of neuronal plasticity resulting from long-term psychostimu
273 morphogenetic events, such as axon guidance, neuronal plasticity, spine maturation, and synaptogenesi
275 the pathophysiology of schizophrenia and in neuronal plasticity suggests that facilitation of NMDAR
276 aling cascades, which play critical roles in neuronal plasticity, survival, and neurite outgrowth.
277 are poorly understood, but regulators of the neuronal plasticity that accompany learning, such as neu
279 ther, these findings identify novel forms of neuronal plasticity that contribute to granule cell dysm
281 KII) is thought to be a critical mediator of neuronal plasticity that links transiently triggered Ca(
282 in the ability of cocaine to induce forms of neuronal plasticity that may contribute to addiction.
283 ate excitation may represent a novel form of neuronal plasticity that regulates activity and excitabi
284 K activity contributes, at least in part, to neuronal plasticity that underlies D(2) dopamine recepto
286 13), as a candidate gene for critical-period neuronal plasticity, the expression of which is regulate
287 an astrocyte-derived protease can influence neuronal plasticity through an extracellular matrix inde
288 t a novel pharmacologic strategy to regulate neuronal plasticity through an NMDA receptor and Src fam
289 present a pharmacologic strategy to regulate neuronal plasticity through NMDAR-dependent mechanisms.
290 also depend on a slow genomic regulation of neuronal plasticity underlying behavioral activation and
291 pamine and glutamate systems involved in the neuronal plasticity underlying cocaine craving and relap
292 c) is an immediate early gene that modulates neuronal plasticity underlying learning and memory.
293 This behaviorally induced expression of neuronal plasticity uses an NMDAR-dependent, LTP-like me
294 induced, experience-dependent expression of neuronal plasticity was blocked by the NMDA(R) antagonis
296 ts also plays a postnatal role in regulating neuronal plasticity, we modulated BMP signaling in mice
297 II isoforms have markedly different roles in neuronal plasticity, with CaMKIIalpha regulating synapti
298 in rodents and also the capacity to modulate neuronal plasticity within the medial nucleus of the amy
299 Repeated psychostimulant exposure induces neuronal plasticity within the mesolimbic dopamine syste
300 and an immediate early gene associated with neuronal plasticity, zif268, in groups of rats selected
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