ライフサイエンスコーパス: neuronal protection

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1 th the goal to decipher its critical role in neuronal protection.

2 normal clearance of glutamate and providing neuronal protection.

3 ependent transcriptional activity to promote neuronal protection.

4 e attenuation of glial Ca2+ signals leads to neuronal protection.

5 ve been implicated in leukocyte adhesion and neuronal protection.

6 es and is involved in growth factor-mediated neuronal protection.

7 atment with human albumin may provide direct neuronal protection.

8 tine (ALC) is a natural compound involved in neuronal protection.

9 e whether endogenous BDNF may play a role in neuronal protection after ganglion cell trauma, BDNF exp

10 se and provide evidence that IFNbeta confers neuronal protection against HIVgp120 toxicity.

11 at delta-opioid receptors may play a role in neuronal protection against hypoxic/ischemic or glutamat

12 w that pan-HDAC inhibition not only promotes neuronal protection against oxidative stress, a common m

13 CO provides neuronal protection against stresses such as stroke and

14 .Akt(K179A)) resulted in loss of PS-mediated neuronal protection, Akt activation, and Bad and Mdm2 ph

15 mice may serve as a useful model system for neuronal protection and apoptosis in traumatic brain inj

16 vast number of cellular processes, including neuronal protection and hypotension.

17 Furthermore, ATA afforded significant neuronal protection and prevented DNA fragmentation.

18 dendrites and axon hillocks and function in neuronal protection and stabilize synapses.

19 ted with decreased immune response, enhanced neuronal protection, and increased neurogenesis.

20 cium on its target cells: neuronal survival, neuronal protection, and synaptic plasticity.

21 It also reduced TPC-induced neuronal protection (e.g. dead cells: 68.2 +/- 5.2% vs.

22 Furthermore, IRF-3-dependent neuronal protection from virus-mediated cytopathology oc

23 noids are involved in synaptic signaling and neuronal protection; however, our understanding of the m

24 uld slow amyloid plaque deposition or confer neuronal protection in a mouse model of AD.

25 sion is an important downstream component of neuronal protection in female stroke.

26 tination may be a signal for aggregation and neuronal protection in PD, which may be relevant for oth

27 age chains, leading to LRRK2 aggregation and neuronal protection in primary neurons and a Drosophila

28 tial for therapeutic exploitation to provide neuronal protection in situations of oxidative stress.

29 lin-like (SHBG) domain of PS is critical for neuronal protection in vitro and in vivo.

30 rial fission and fragmentation and conferred neuronal protection in vivo and in culture.

31 expressed transcription factor whose role in neuronal protection is now well established.

32 nfluenced the degree of plaque clearance and neuronal protection; (iv) high affinity of the antibody

33 These findings paralleled dopaminergic neuronal protection, normalized striatal neurotransmitte

34 These findings paralleled dopaminergic neuronal protection, normalized striatal neurotransmitte

35 These findings paralleled dopaminergic neuronal protection, normalized striatal neurotransmitte

36 se antagonists after SE provided significant neuronal protection raising the potential for a glial co

37 ing correlated with a clearance response and neuronal protection, whereas the ability of antibodies t

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