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1 or without cerebrovascular disease, defined neuropathologically.
2 s have been the most commonly affected cells neuropathologically.
3 our with Parkinson's disease, both confirmed neuropathologically.
4 gnosis of multiple system atrophy, confirmed neuropathologically.
5 d those who subsequently died were evaluated neuropathologically.
6 multiple system atrophy, both clinically and neuropathologically.
7 grel dogs weighing 23 to 30 kg and validated neuropathologically.
8 were highly abundant and were found only in neuropathologically affected areas of ALS patients but n
10 renia (N=26) were compared with those from a neuropathologically and neuropsychiatrically normal elde
11 ltiple system atrophy (MSA) cases, confirmed neuropathologically, and eight age-matched controls.
12 identified in the tissue, blindly described neuropathologically, and subsequently divided into ische
13 easured functionally as dementia severity or neuropathologically as increased neuritic plaque and neu
14 ases, which were confirmed biochemically and neuropathologically as variably protease-sensitive prion
17 and Alzheimer's diseases, are characterized neuropathologically by accumulation of misfolded protein
18 progressive myoclonus epilepsy characterized neuropathologically by aggregates of abnormally structur
19 Both human and murine NPC are characterized neuropathologically by ballooned neurons distended with
20 order characterized clinically by ataxia and neuropathologically by cerebellar atrophy and granule ce
23 features, as well as cerebellar ataxia, and neuropathologically by neuronal loss, gliosis, and iron
26 odegenerative disorder that is characterized neuropathologically by the presence of neuropil aggregat
27 causes and disease symptoms, and are linked neuropathologically by the RNA binding protein TDP-43 (T
29 n hippocampal subfields in 95 clinically and neuropathologically characterized human cases of DLB, fi
30 prefrontal cortex of antemortem-assessed and neuropathologically characterized schizophrenic and comp
31 From these studies, it is clear that MCI is neuropathologically complex and cannot be understood wit
32 eresting that, in four of four patients with neuropathologically confirmed acute hypoxic changes, we
37 e United Kingdom, of which 33 cases had been neuropathologically confirmed and 2 classified as probab
39 and spinal cord samples originating from 137 neuropathologically confirmed control individuals to stu
40 fidence interval: 1.06-1.20) and presence of neuropathologically confirmed degenerative changes (that
41 d with a novel PRNP mutation associated with neuropathologically confirmed Gerstmann-Straussler-Schei
42 Center (1985-2015) and 1115 individuals with neuropathologically confirmed LOAD were included from th
43 ts (8 men, 3 women, age 64 +/- 3 years) with neuropathologically confirmed MSA and 11 control subject
45 in human post-mortem cerebellar tissue from neuropathologically confirmed PD cases and neurologicall
46 ently examined that consisted of 67 cases of neuropathologically confirmed sCJD (33 female, 34 male;
47 analyzed 108 CSF samples from patients with neuropathologically confirmed sCJD or from control patie
48 sues obtained at necropsy from patients with neuropathologically confirmed variant CJD, but not from
49 tissues at necropsy from four patients with neuropathologically confirmed vCJD and from individuals
50 cribe a family (DUK1684) with clinically and neuropathologically confirmed, autosomal dominant, non-A
51 on markers from a unique cohort of over 1600 neuropathologically defined AD cases and controls (1019
52 e units [95% CI, -1.52 to -0.02]), and lower neuropathologically defined Alzheimer disease (beta = -0
53 obulin gene is a risk factor associated with neuropathologically defined Alzheimer's disease in our p
54 enerative diseases termed synucleinopathies, neuropathologically defined by inclusions containing agg
55 lpha2-macroglobulin gene was associated with neuropathologically defined diagnosis of Alzheimer's dis
57 uantified in temporalis muscles and brain of neuropathologically diagnosed Alzheimer disease (AD) and
58 [interquartile range, 47-76 years]), CTE was neuropathologically diagnosed in 177 players (87%; media
59 rtex, and sensory cortex from clinically and neuropathologically diagnosed sporadic ALS cases and age
60 f cognitive deficits occur in AD and several neuropathologically distinct age-associated neurodegener
61 f FTD pathology, suggesting that bvFTD-SP is neuropathologically distinct from other forms of FTD.
66 ofile of 788 brain samples obtained from 101 neuropathologically healthy individuals (10 distinct bra
67 RNA expression profile of brain samples from neuropathologically healthy individuals, showed that KCT
68 ty, suggesting ischaemic demyelination, seen neuropathologically in SVD, may be an important predicto
69 istory of parkinsonism or dementia confirmed neuropathologically, including PSP (n = 24), corticobasa
70 received immunisation and who were examined neuropathologically, mean Abeta load was lower than in a
72 n applied to an array-based dataset from 150 neuropathologically normal adult human brains, our metho
74 g and expression analysis on a series of 193 neuropathologically normal human brain samples using the
77 one comprised of four brain regions from 150 neuropathologically normal samples and another comprised
78 ther comprised of ten brain regions from 134 neuropathologically normal samples, and show that by usi
81 under-reported lesions subsequently analysed neuropathologically, particularly those arising within t
84 included a chronic spinocerebellar syndrome, neuropathologically proven Leigh syndrome, and sudden de
90 cle events, we investigated the brains of 12 neuropathologically verified cases of Alzheimer's diseas
93 from neuropathologically verified discovery, neuropathologically verified replication, and clinically
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