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1 avage of a host protein-derived peptide (pro-neuropeptide Y).
2 es such as stromal cell-derived factor-1 and neuropeptide Y.
3  decarboxylase-67 (GAD67), somatostatin, and neuropeptide Y.
4 ell type-specific distribution of endogenous neuropeptide Y.
5 e pool neurons that already express GABA and neuropeptide Y.
6 amage, and compensatory changes in EAAT3 and neuropeptide Y.
7  abundant levels of tyrosine hydroxylase and neuropeptide Y.
8  estrogen receptor-alpha colocalization with neuropeptide Y.
9 so synthesize a range of peptides, including neuropeptide Y.
10 ferent orexigenic molecules: AgRP; GABA; and neuropeptide Y.
11 OM), vasoactive intestinal peptide (VIP), or neuropeptide Y.
12 lar (3V) infusions of the orexigenic peptide neuropeptide Y 3-36 in awake, freely moving rats and det
13         dNPF is an ortholog of the mammalian neuropeptide Y, a highly conserved neuromodulator that s
14 rocessed for immunocytochemistry for SST and neuropeptide Y, a neuropeptide partially coexpressed in
15          In the current study, we found that neuropeptide Y, a protein produced by neurons, affected
16                         Neurons coexpressing neuropeptide Y, agouti-related peptide, and GABA (NAG) p
17                                 Hypothalamic neuropeptide Y, agouti-related peptide, and proopiomelan
18 icted neuronal subsets (proopiomelanocortin, neuropeptide Y/agouti-related peptide, and steroidogenic
19 ng hormone neurons but has limited effect on neuropeptide Y/agouti-related protein and proopiomelanoc
20 ellular mechanism in proopiomelanocortin and neuropeptide Y/agouti-related protein neurons and links
21 ons of the arcuate nucleus, specifically the neuropeptide Y/agouti-related protein neurons and the do
22 ances the activity of agouti-related protein/neuropeptide Y (AgRP/NPY)-expressing neurons but induces
23  approaches we show this plasticity requires neuropeptide Y, an adrenal cotransmitter and the activat
24 f sympathetic nerves and tissue abundance of neuropeptide-Y, an indicator of sympathetic nerve activi
25 (-/-) mice, and other transcripts, including neuropeptide Y and activating transcription factor-3, ar
26  reduced appetite-stimulating neuropeptides, neuropeptide Y and Agouti-related peptide.
27 d by arcuate nucleus neurons that co-express neuropeptide Y and agouti-related protein (NPY/AgRP neur
28 d food intake, body weight, and hypothalamic neuropeptide Y and Agouti-related protein mRNA expressio
29 nduced hepatic FGF21, which caused increased neuropeptide Y and agouti-related protein mRNAs in the h
30 he regulation of social behavior (especially neuropeptide Y and corticotropin-releasing factor) are m
31  disorder are enhanced hippocampal levels of neuropeptide Y and EAAT3 and increased calpain proteolys
32 at co-express agouti-related protein (AgRP), neuropeptide Y and gamma-aminobutyric acid (GABA) are kn
33  control animals and had lower expression of neuropeptide Y and higher expression of proopiomelanocor
34 wn neuropeptides were not detected including neuropeptide Y and neurotensin.
35                 In the arcuate nucleus, both Neuropeptide Y and proopiomelanocortin cells expressed t
36 f brainstem and hypothalamic neurons express neuropeptide Y and proopiomelanocortin in the arcuate nu
37 ons of the hypothalamus and colocalizes with neuropeptide Y and proopiomelanocortin neurons in the ar
38  of GABAergic GAD67+ interneurons expressing neuropeptide Y and somatostatin are reduced in the hippo
39 2.2 is not required for the specification of neuropeptide Y and vasoactive intestinal polypeptide, in
40 coneogenic enzymes and elevated hypothalamic neuropeptide-Y and agouti-related protein mRNA levels.
41 n analogue of the C-terminal pentapeptide of neuropeptide Y, and a neuropeptide FF analogue) were sub
42 othalamic neuropeptides proopiomelanocortin, neuropeptide Y, and agouti-related peptide in T1D mice.
43     Ivy cells express nitric oxide synthase, neuropeptide Y, and high levels of GABA(A) receptor alph
44 -regulating proteins agouti-related peptide, neuropeptide Y, and uncoupling protein 2 in the hypothal
45 hich express the orexigenic neuronal marker, Neuropeptide-Y, and respond to fasting and leptin-induce
46                        Injections of Aplysia neuropeptide Y (apNPY) reduced food intake and slowed do
47            Agouti-related peptide (AGRP) and Neuropeptide Y are potent orexigens and are coexpressed
48 ress characteristic combinations of GABA and neuropeptide Y as cotransmitters and Lim1,2 and Nurr1 tr
49 he modulation of neurotransmitter release by neuropeptide Y, baclofen, and adenosine as revealed by [
50 the insulin promotor (Ins2) were stained for neuropeptide Y before, during, and after virally induced
51 s that synthesize agouti-related peptide and neuropeptide Y but inhibited anorexigenic neurons that s
52 e BBB-impermeable neuropeptides dalargin and neuropeptide Y chemically conjugated with FC5-Fc fusion
53 .93]) after stressful training; lower plasma neuropeptide Y concentration after stressful training (d
54 ures were heart rate, breathing rate, plasma neuropeptide Y concentration, score on the Response to S
55                                              Neuropeptide Y-containing interneurons in the dentate hi
56 eurons that are defined by the expression of neuropeptide Y::Cre (NPY::Cre) act to gate mechanical it
57                                              Neuropeptide Y detection was performed.
58 the mammalian target of rapamycin pathway in neuropeptide-Y-ergic neurons of the arcuate nucleus, and
59  A significant reduction of calbindin-, NPY (neuropeptide Y)-expressing, and cholinergic interneurons
60 ic inhibitory circuit involving a variety of neuropeptide Y-expressing interneurons.
61 endrites, whereas nitric oxide synthase- and neuropeptide Y-expressing ivy cells provided synaptic an
62                        In addition, we found neuropeptide Y expression almost exclusively in those GI
63                   Surprisingly, hypothalamic neuropeptide Y expression was completely suppressed, sug
64 nders to two model peptides, angiotensin and neuropeptide Y, following screening by solution phage pa
65 g mice showed increased levels of C-terminal neuropeptide Y fragments and increased neurogenesis.
66 europeptide F (NPF), a homolog of vertebrate neuropeptide Y, functions in feeding and coordination of
67  single nucleotide polymorphism (SNP) in the neuropeptide Y gene has been associated with elevated se
68                    Both serotonin (5-HT) and neuropeptide Y have been shown to affect a variety of ma
69 essing Drosophila neuropeptide F (dNPF), the neuropeptide Y homolog, strongly correlates with food-od
70 leasing short neuropeptide F, the Drosophila neuropeptide Y homolog.
71 used immunohistochemistry for calretinin and neuropeptide Y in 24 age- and gender-matched patients wi
72 nthesize gamma-amino-butyric acid (GABA) and neuropeptide Y in adult mice leads to starvation within
73 d release of the orexigenic neurotransmitter neuropeptide Y in response to glucose.
74 ons expressing parvalbumin, somatostatin and neuropeptide Y in the dentate gyrus, reduced aberrant ne
75  pro-opiomelanocortin/agouti-related peptide/neuropeptide Y in the hypothalamus of the rats.
76 ar responsiveness and vascular reactivity to neuropeptide Y in the offspring.
77 rvating (calretinin(+), somatostatin(+), and neuropeptide Y(+)) interneurons.
78  TH-ir decreased by 21%, and in hypothalamus neuropeptide Y-ir increased by 10% in MPH-exposed rats.
79                                   Numbers of neuropeptide Y-IR neurons were not affected.
80 osal plexus and was expressed exclusively by neuropeptide Y-IR neurons.
81                                              Neuropeptide Y is a key peptide affecting adiposity and
82  neuropeptides such as ghrelin, orexin A and neuropeptide Y is also discussed.
83          Multiple peptide systems, including neuropeptide Y, leptin, ghrelin, and others, are involve
84 P, corticotropin releasing factor (CRF), and neuropeptide Y levels in adult male Long-Evans rats defe
85 ng neuropeptide F receptor 1, a receptor for neuropeptide Y-like neuropeptide F.
86 n and attraction is thought to depend on the neuropeptide Y-like receptor NPR-1, because solitary and
87 ting cells and identified neurons expressing neuropeptide Y-like short neuropeptide F (sNPF).
88 -expressing interneurons and initiation of a neuropeptide-y-like signaling axis that promotes elevate
89 cystokinin/CB(1) cannabinoid receptor(+) and neuropeptide Y(+) local-circuit interneurons upon SAVA m
90 f both Lifeact-GFP and the SG marker protein neuropeptide Y-mCherry reveals that SGs actively translo
91  neuropeptide F, the invertebrate homolog of neuropeptide Y, modulate aggression.
92 tissue weight, fecal output, arcuate nucleus neuropeptide Y mRNA expression, plasma corticosterone, a
93 ng hormone but surprisingly has no effect on neuropeptide Y mRNA, a neuropeptide previously shown to
94                                              Neuropeptide Y neurons have been shown to be particularl
95 s, stimulation of the agouti-related protein/neuropeptide Y neurons, and activation of the hypothalam
96 lly significant difference in the density of neuropeptide Y+ neurons between subjects with autism and
97 n, corticotropin releasing hormone, galanin, neuropeptide Y, neurotensin, preproenkephalin and tachyk
98 in, dynorphin) and brain antistress systems (neuropeptide Y, nociceptin [orphanin FQ]) in drug depend
99 r, metabotropic glutamate receptors-2 mGlu2, neuropeptide-Y NPY, and mineralocorticoid receptors MR).
100 d identify the vasoconstrictive mechanism as Neuropeptide Y (NPY) acting on Y1 receptors.
101 e truncated cleaved form of neurotransmitter neuropeptide Y (NPY) actively promotes a breach of BM va
102                                              Neuropeptide Y (NPY) administration into the basolateral
103                 Bioconjugates containing the neuropeptide Y (NPY) analogue [F(7),P(34)]-NPY as target
104  In lean mice, the adipokine leptin inhibits neuropeptide Y (NPY) and agouti-related peptide (AgRP) n
105 bits neurotransmission in neurons expressing neuropeptide Y (NPY) and agouti-related peptide (AgRP) v
106            Hypothalamic neurons that express neuropeptide Y (NPY) and agouti-related protein (AgRP) a
107                                              Neuropeptide Y (NPY) and Agouti-related protein (AgRP),
108  the expression of orexigenic neuropeptides [neuropeptide Y (NPY) and agouti-related protein (AgRP)]
109  with changes in orexigenic peptides such as neuropeptide Y (NPY) and anorexigenic peptides such as a
110 s that regulate stress and reward, including neuropeptide Y (NPY) and corticotropin-releasing factor
111 s, including somatostatin (SST), tachykinin, neuropeptide Y (NPY) and cortistatin, in a pattern remin
112 was developed for the sensitive detection of neuropeptide Y (NPY) and employed in the analysis of NPY
113        LIF also regulates expression of both neuropeptide Y (NPY) and galanin following peripheral ne
114 c), containing pro-opoiomelanocortin (POMC), neuropeptide Y (NPY) and growth hormone releasing hormon
115                                              Neuropeptide Y (NPY) and its receptors (especially Y1, Y
116 up-regulating specific target genes, such as neuropeptide Y (NPY) and its Y1 and Y5 receptors (Y5Rs).
117 and modulation of neurological signals, with Neuropeptide Y (NPY) and Orexin A (OXA) offering diagnos
118                                              Neuropeptide Y (NPY) and pancreatic polypeptide (PP) con
119 ion of corticotropin releasing factor (CRF), neuropeptide Y (NPY) and proopiomelanocortin (POMC) in h
120  the lateral hypothalamus (LH) together with neuropeptide Y (NPY) and proopiomelanocortin (POMC) neur
121  the total number of interneurons expressing neuropeptide Y (NPY) and vasoactive intestinal polypepti
122                       Other peptides such as neuropeptide Y (NPY) are synthesized throughout the brai
123 sh, and here we report the identification of neuropeptide Y (NPY) as both necessary for normal daytim
124 in a structural model of the peptide hormone neuropeptide Y (NPY) bound to its human G-protein-couple
125 in the basolateral amygdaloid complex (BLA), neuropeptide Y (NPY) buffers against protracted anxiety
126 e ontogeny of proopiomelanocortin (POMC) and neuropeptide Y (NPY) cell populations, which exert oppos
127 pocretin cells respond to dynorphin, arcuate neuropeptide Y (NPY) cells respond to hypocretin], diffe
128 stimulatory connections onto the neighboring neuropeptide Y (NPY) cells.
129  strongly implicated hindbrain catecholamine/neuropeptide Y (NPY) coexpressing neurons as key mediato
130 ression of agouti-related protein (Agrp) and neuropeptide Y (Npy) coincide with the cyclic changes in
131 c (tg) mice affected primarily the levels of neuropeptide Y (NPY) compared with other neuropeptides.
132 subpopulation of CA1 interneurons expressing neuropeptide Y (NPY) contributes prominently to this dyn
133                                              Neuropeptide Y (NPY) counters stress and is involved in
134  we show that the claims of Zhou et al. that neuropeptide Y (NPY) diplotype-predicted expression is c
135          The DMH contains neurons expressing Neuropeptide Y (NPY) during specific physiological condi
136 s glucocorticoids have pronounced effects on neuropeptide Y (NPY) expression and as NPY neurons proje
137 gered molecular plasticity including ectopic neuropeptide Y (NPY) expression in granule cells, but wi
138  element binding) binding protein (CBP), and neuropeptide Y (NPY) expression in the amygdaloid brain
139 binding protein (CREB) resulted in decreased neuropeptide Y (NPY) expression in the central amygdala
140 icular and dorsomedial nuclei, and increased neuropeptide Y (NPY) expression in the hypothalamus.
141                                      Because neuropeptide Y (NPY) expression is strongly induced in D
142 pression of vGLUT1 but not vGAT, and reduced Neuropeptide Y (NPY) expression.
143 genesis with emphasis on the distribution of neuropeptide Y (NPY) expression.
144 nes and their cognate receptors belonging to neuropeptide Y (NPY) family mediate diverse biological f
145                                          The neuropeptide Y (NPY) family of peptides and receptors re
146 ucagon-like peptide-1 (GLP-1), glucagon, and neuropeptide Y (NPY) from circumvallate papillae of Tas1
147                                              Neuropeptide Y (NPY) gene expression in the hippocampal
148                 Recent studies indicate that neuropeptide Y (NPY) has a major role in the regulation
149  anxiety and other psychiatric disorders and neuropeptide Y (NPY) has emerged as a key component of a
150                                              Neuropeptide Y (NPY) has robust anxiolytic properties an
151                      SIGNIFICANCE STATEMENT: Neuropeptide Y (NPY) has robust anxiolytic properties, a
152               While recent animal studies of neuropeptide Y (NPY) have suggested a facilitator role f
153 nd that appetite-regulating systems, such as neuropeptide Y (NPY) in the arcuate nucleus (ARH) and or
154         Previous studies have suggested that neuropeptide Y (NPY) in the dorsomedial hypothalamus (DM
155             The expression of the orexigenic neuropeptide Y (NPY) in the hypothalamus to the low lept
156                However, the specific role of neuropeptide Y (NPY) in this process has not been system
157                                              Neuropeptide Y (NPY) interacts with the Y(1) receptor, N
158 ic inputs to the two populations of striatal neuropeptide Y (NPY) interneurons, plateau low threshold
159                                              Neuropeptide Y (NPY) is a 36-amino-acid peptide transmit
160                                              Neuropeptide Y (NPY) is a hypothalamic neuropeptide that
161                                              Neuropeptide Y (NPY) is a peptide known for its anti-anx
162                                              Neuropeptide Y (NPY) is a putative endogenous anxiolytic
163                                              Neuropeptide Y (NPY) is a well-established orexigenic pe
164                                              Neuropeptide Y (NPY) is an important modulatory neuropep
165                                              Neuropeptide Y (NPY) is an important peptide regulating
166                                              Neuropeptide Y (NPY) is anxiolytic and its release is in
167                                              Neuropeptide Y (NPY) is induced in peripheral tissues su
168                                              Neuropeptide Y (NPY) is one of the major neuropeptides p
169                                              Neuropeptide Y (NPY) is one of the most abundant protein
170                                              Neuropeptide Y (NPY) is one of the most widespread neuro
171 cord following inflammation or nerve injury, neuropeptide Y (NPY) is poised to regulate the transmiss
172 ported that the sympathetic neurotransmitter neuropeptide Y (NPY) is potently angiogenic, primarily t
173                                              Neuropeptide Y (NPY) is produced in the intergeniculate
174 evious observations that fluorescent-labeled neuropeptide Y (NPY) is usually released within 200 ms a
175                                              Neuropeptide Y (NPY) is widely expressed in the central
176                            ICV TTR decreased neuropeptide Y (NPY) levels in the DMH and the paraventr
177  understand whether genetic variation at the Neuropeptide Y (NPY) locus governs secretion and stress
178                                              Neuropeptide Y (NPY) mediates stress-induced obesity in
179                      Acute administration of neuropeptide Y (NPY) modulates alcohol intake in genetic
180 cordings were made from GFP (Renilla)-tagged neuropeptide Y (NPY) neurons from the arcuate nucleus of
181                                              Neuropeptide Y (NPY) neurons in both the arcuate nucleus
182 nhibitory effect of Dyn-A on arcuate nucleus neuropeptide Y (NPY) neurons mediated by both 1 and (kap
183  In contrast, kisspeptin inhibits orexigenic neuropeptide Y (NPY) neurons through an indirect mechani
184                  The DMH contains orexigenic neuropeptide Y (NPY) neurons, but the role of these neur
185 ts of intracerebroventricular application of Neuropeptide Y (NPY) on licking microstructure for sucro
186 he purpose of this study was to characterize neuropeptide Y (NPY) overflow and metabolism from isolat
187            Recent studies suggest that human neuropeptide Y (NPY) plays a prominent role in managemen
188 nt protein is expressed under control of the neuropeptide Y (NPY) promoter and striatal NPY-expressin
189  a green fluorescent protein (GFP) under the neuropeptide Y (NPY) promoter.
190 e neuropeptide F receptor NPFR1, a mammalian neuropeptide Y (NPY) receptor homolog, centrally regulat
191 he autonomic nervous system and hypothalamic neuropeptide Y (NPY) release during fasting regulates he
192                                              Neuropeptide Y (NPY) signaling in the CNS was modulated
193                                              Neuropeptide Y (NPY) stimulates feeding and weight gain,
194          In the current study, we found that neuropeptide Y (NPY) suppressed monocyte recruitment to
195 on requires simultaneous withdrawal of tonic neuropeptide Y (NPY) sympathoinhibition.
196           It has been speculated this alters neuropeptide Y (NPY) synthesis, trafficking, or secretio
197              Functional genetic variation of neuropeptide Y (NPY) was recently identified as a source
198  fibers coexpress the inhibitory transmitter neuropeptide Y (NPY) with glutamate.
199 udied the role of alpha(2)-adrenoceptors and neuropeptide Y (NPY) Y(1) receptors in mediating vasocon
200 r-NH(2)](2), 1, to be a moderately selective neuropeptide Y (NPY) Y(4) receptor agonist.
201 We applied this BiFC system to study example neuropeptide Y (NPY) Y1 receptor dimers.
202 d the role of alpha-adrenergic receptor- and neuropeptide Y (NPY) Y1 receptor-mediated vasoconstricti
203                                Activation of neuropeptide Y (NPY) Y1 receptors (Y1r) in the rat basol
204    Normal breast tissue mainly expresses the neuropeptide Y (NPY) Y2 receptor whereas primary human b
205 ations of GABAergic interneurones expressing neuropeptide Y (NPY)(+), parvalbumin (PV)(+) and tyrosin
206  neurons [gamma-aminobutyric acid (GABA)(+), neuropeptide Y (NPY)(+), proopiomelanocortin (POMC)(+),
207                                              Neuropeptide Y (NPY), a 36 aa peptide, regulates stress
208                                              Neuropeptide Y (NPY), a brain neuromodulator that has be
209                         Double labeling with neuropeptide Y (NPY), a marker for vasomotor neurons, re
210 study, we identified increased expression of neuropeptide Y (NPY), a pleiotropic growth factor which
211 have studied the subcellular distribution of neuropeptide Y (NPY), a prototypical and broadly express
212                                              Neuropeptide Y (NPY), a stress modulatory transmitter, i
213         Our previous studies have identified neuropeptide Y (NPY), a sympathetic neurotransmitter exp
214 In birds, CART inhibits food intake, whereas neuropeptide Y (NPY), a well-known orexigenic peptide, s
215 ry indicate that cortical astrocytes contain neuropeptide Y (NPY), a widespread neuronal transmitter.
216  from chemotherapy-induced cell death, while neuropeptide Y (NPY), acting via its Y2 receptor (Y2R),
217 c neuron markers proopiomelanocortin (POMC), neuropeptide Y (NPY), agouti-related peptide (AGRP), som
218 ment, hypothalamic mRNA expression levels of neuropeptide Y (NPY), agouti-related protein (AGRP), pro
219                    We studied the actions of neuropeptide Y (NPY), an abundant neocortical neuropepti
220 se changes are regulated postsynaptically by neuropeptide Y (NPY), an adrenal cotransmitter.
221 ted whether VPA influences the expression of neuropeptide Y (NPY), an endogenous anticonvulsant.
222 creased mRNA levels of hypothalamic GHS-R1a, neuropeptide Y (NPY), and agouti-related protein (AgRP),
223  (CB), parvalbumin (PV), calretinin (CR) and neuropeptide Y (NPY), and somatostatin (SOM) and glial f
224  augments the SCN clock-resetting effects of neuropeptide Y (NPY), another neurochemical correlate of
225 ctor 3 (ATF3), the neuropeptides galanin and neuropeptide Y (NPY), brain-derived neurotrophic factor
226 scription level: monoamine oxidase A (MAOA), neuropeptide Y (NPY), endothelial nitric oxide synthase
227 -melanocyte-stimulating hormone (alpha-MSH), neuropeptide Y (NPY), glutamate, and GABA from first-ord
228 ), galanin, gastrin-releasing peptide (GRP), neuropeptide Y (NPY), nitric oxide synthase (NOS), serot
229 ed by either gamma-aminobutyric acid (GABA), neuropeptide Y (NPY), or beta-endorphin receptor blockad
230 Immunohistochemistry for somatostatin (SOM), neuropeptide Y (NPY), parvalbumin (PV), cholecystokinin
231 vels of genes that regulate appetite, namely neuropeptide Y (NPY), pro-opiomelanocortin (POMC) and th
232 he well-characterized neuropeptides, such as neuropeptide Y (NPY), proopiomelanocortin (POMC), orexin
233 ent with PEDF + DHA induced transcription of neuropeptide y (npy), small proline-rich protein 1a (spr
234 GFP) in a subset of neurons that colocalized neuropeptide Y (NPY), somatostatin (SST), and GABA for w
235 xp4 expression induces ectopic expression of neuropeptide Y (Npy), which has been reported to be pres
236  effects were due to disrupted signalling of neuropeptide Y (NPY), which is known to mediate non-phot
237 ar interneurons of the dentate gyrus express neuropeptide Y (NPY), which modulates granule cell activ
238                       One relevant target is neuropeptide Y (NPY), which regulates both stress and fo
239 l transduction pathway modulating release of neuropeptide Y (NPY), which stimulates OE stem cell acti
240 cell activation and promotes the activity of neuropeptide Y (NPY)- and agouti-related peptide (AgRP)-
241 nterneurons express col19a1 mRNA; subsets of neuropeptide Y (NPY)-, somatostatin (Som)-, and calbindi
242 e investigated the properties of neostriatal neuropeptide Y (NPY)-expressing interneurons in transgen
243 1 (GAD1) in either cholecystokinin (CCK)- or neuropeptide Y (NPY)-expressing interneurons.
244 on, appeared to be precipitated by a loss of neuropeptide Y (NPY)-mediated local circuit inhibition a
245 ood attraction to aversion is regulated by a neuropeptide Y (NPY)-related brain signaling peptide.
246 expressing agouti-related peptide (AgRP) and neuropeptide Y (Npy).
247 ropeptides agouti-related protein (AgRP) and neuropeptide Y (NPY).
248 e-like ethanol drinking to study the role of neuropeptide Y (NPY).
249 r producing the neuropeptides enkephalin and neuropeptide Y (NPY).
250 III are GABAergic, and some of these express neuropeptide Y (NPY).
251 rsors and express neuronal proteins, such as neuropeptide Y (NPY).
252 n enzyme that truncates the neurotransmitter neuropeptide Y (NPY).
253 F co-receptors GFRalpha1 and RET, as well as neuropeptide Y (NPY).
254  release of the anticonvulsant neuropeptide, neuropeptide Y (NPY).
255 nal white adipose tissue that is mediated by neuropeptide Y (NPY).
256 e stimulating hormone (alpha-MSH) as well as neuropeptide Y (NPY).
257 ented for the expression of a third peptide, neuropeptide Y (NPY).
258  increase in haemoglobin, catecholamines and neuropeptide Y (NPY).
259 asting discharge rates of fluorescent-tagged neuropeptide-Y (NPY) (within 200 ms) and tissue plasmino
260  that express pro-opiomelanocortin (POMC) or neuropeptide-Y (NPY) and agouti-related protein (AgRP).
261 escence microscopy of lumen-targeted probes (neuropeptide Y [NPY]-pH-insensitive yellow fluorescent p
262 ridization to localize appetite-stimulating (neuropeptide Y, NPY; agouti-related protein, AGRP) and a
263 cal activity: the N-terminal region of human neuropeptide Y (NPY1-9, Tyr(1)-Pro(2)-Ser(3)-Lys(4)-Pro(
264 tostatin, vasoactive intestinal polypeptide, neuropeptide Y, or cholecystokinin (antigens commonly co
265                       Vascular reactivity to neuropeptide Y (P<0.05) and electrical field stimulation
266              Furthermore, loss of calbindin, neuropeptide Y, parvalbumin, and GAD65-positive interneu
267 r morphology and expression of somatostatin, neuropeptide Y, parvalbumin, or calretinin, we infer tha
268                                          The neuropeptide Y pathway acts together with SRB-13 to anta
269                  The possible involvement of neuropeptide Y pathways during 2DG-induced expression of
270 creatic polypeptide (PPY) are members of the neuropeptide Y peptide family.
271 e submucosal plexus, DOReGFP was detected in neuropeptide Y-positive secretomotor and vasodilator neu
272                                      Loss of neuropeptide Y prevents a fasting-induced increase in ep
273 pressing green fluorescent protein under the neuropeptide Y promoter, we find that, across all layers
274 tance P, calcitonin gene-related peptide, or neuropeptide Y protein expression in DRGs and spinal cor
275                         Lower mRNA levels of neuropeptide Y receptor 1 (NPY1R) and NPY5R but not NPY
276 boratory-derived, mutation controlled by the neuropeptide Y receptor homolog npr-1 can affect dauer l
277                                          The neuropeptide Y receptor homolog, NPR-1, and an inhibitor
278  multiple signaling molecules, including the neuropeptide Y receptor NPR-1 and the calcineurin subuni
279                                          The neuropeptide Y receptor signaling pathway has been impli
280 on and this effect was blocked by a specific neuropeptide Y receptor Y1 (NPY1R) antagonist.
281 ion, although this was blocked by a specific neuropeptide Y receptor Y1 receptor antagonist, suggesti
282 ne, which encodes a homolog of the mammalian neuropeptide Y receptor.
283 coupled receptor (GPCR) related to mammalian neuropeptide Y receptors, functions to suppress innate i
284 uantification of neuronal glucocorticoid and neuropeptide Y receptors.
285  Y1 receptor (Y1R) and Y5 receptor (Y5R) for neuropeptide Y share similar actions in the regulation o
286 ive deficits, as well as endocannabinoid and neuropeptide Y system alterations and altered circadian
287                                          The neuropeptide Y system consists of several neuropeptides
288 er was monitored by observing DCV-associated neuropeptide Y tagged with a fluorescent protein.
289 receptor systems (bombesin, neurotensin, and neuropeptide-Y) that offer high potential in the field o
290 d tumor necrosis factor, whereas addition of neuropeptide Y to wild-type macrophages attenuates the r
291 s-associated increase of immunoreactivity of neuropeptide Y, tyrosine hydroxylase, and somatostatin.
292         In contrast, interneurons containing neuropeptide Y, vasoactive intestinal peptide, or the 5-
293                                              Neuropeptide Y was elevated in the caudate-putamen.
294  hypothalamic expression of the orexic gene, neuropeptide Y, was lower and expression of the anorexic
295  (AgRP) neurons (which also release AgRP and neuropeptide Y), we generated mice with an AgRP neuron-s
296  (AgRP) neurons (which also release AgRP and neuropeptide Y), we generated mice with an AgRP neuron-s
297 ropeptide F (sNPF), an ortholog of mammalian neuropeptide Y, which we show here is a direct target of
298                     The results suggest that neuropeptide Y Y1R differentially expressed in the limbi
299                                  The role of neuropeptide Y Y2 receptor (Y2R) in human diseases such
300 ereomeric mixture) is a potent and selective neuropeptide Y Y4 receptor agonist.

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