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1 avage of a host protein-derived peptide (pro-neuropeptide Y).
2 es such as stromal cell-derived factor-1 and neuropeptide Y.
3 decarboxylase-67 (GAD67), somatostatin, and neuropeptide Y.
4 ell type-specific distribution of endogenous neuropeptide Y.
5 e pool neurons that already express GABA and neuropeptide Y.
6 amage, and compensatory changes in EAAT3 and neuropeptide Y.
7 abundant levels of tyrosine hydroxylase and neuropeptide Y.
8 estrogen receptor-alpha colocalization with neuropeptide Y.
9 so synthesize a range of peptides, including neuropeptide Y.
10 ferent orexigenic molecules: AgRP; GABA; and neuropeptide Y.
11 OM), vasoactive intestinal peptide (VIP), or neuropeptide Y.
12 lar (3V) infusions of the orexigenic peptide neuropeptide Y 3-36 in awake, freely moving rats and det
14 rocessed for immunocytochemistry for SST and neuropeptide Y, a neuropeptide partially coexpressed in
18 icted neuronal subsets (proopiomelanocortin, neuropeptide Y/agouti-related peptide, and steroidogenic
19 ng hormone neurons but has limited effect on neuropeptide Y/agouti-related protein and proopiomelanoc
20 ellular mechanism in proopiomelanocortin and neuropeptide Y/agouti-related protein neurons and links
21 ons of the arcuate nucleus, specifically the neuropeptide Y/agouti-related protein neurons and the do
22 ances the activity of agouti-related protein/neuropeptide Y (AgRP/NPY)-expressing neurons but induces
23 approaches we show this plasticity requires neuropeptide Y, an adrenal cotransmitter and the activat
24 f sympathetic nerves and tissue abundance of neuropeptide-Y, an indicator of sympathetic nerve activi
25 (-/-) mice, and other transcripts, including neuropeptide Y and activating transcription factor-3, ar
27 d by arcuate nucleus neurons that co-express neuropeptide Y and agouti-related protein (NPY/AgRP neur
28 d food intake, body weight, and hypothalamic neuropeptide Y and Agouti-related protein mRNA expressio
29 nduced hepatic FGF21, which caused increased neuropeptide Y and agouti-related protein mRNAs in the h
30 he regulation of social behavior (especially neuropeptide Y and corticotropin-releasing factor) are m
31 disorder are enhanced hippocampal levels of neuropeptide Y and EAAT3 and increased calpain proteolys
32 at co-express agouti-related protein (AgRP), neuropeptide Y and gamma-aminobutyric acid (GABA) are kn
33 control animals and had lower expression of neuropeptide Y and higher expression of proopiomelanocor
36 f brainstem and hypothalamic neurons express neuropeptide Y and proopiomelanocortin in the arcuate nu
37 ons of the hypothalamus and colocalizes with neuropeptide Y and proopiomelanocortin neurons in the ar
38 of GABAergic GAD67+ interneurons expressing neuropeptide Y and somatostatin are reduced in the hippo
39 2.2 is not required for the specification of neuropeptide Y and vasoactive intestinal polypeptide, in
40 coneogenic enzymes and elevated hypothalamic neuropeptide-Y and agouti-related protein mRNA levels.
41 n analogue of the C-terminal pentapeptide of neuropeptide Y, and a neuropeptide FF analogue) were sub
42 othalamic neuropeptides proopiomelanocortin, neuropeptide Y, and agouti-related peptide in T1D mice.
43 Ivy cells express nitric oxide synthase, neuropeptide Y, and high levels of GABA(A) receptor alph
44 -regulating proteins agouti-related peptide, neuropeptide Y, and uncoupling protein 2 in the hypothal
45 hich express the orexigenic neuronal marker, Neuropeptide-Y, and respond to fasting and leptin-induce
48 ress characteristic combinations of GABA and neuropeptide Y as cotransmitters and Lim1,2 and Nurr1 tr
49 he modulation of neurotransmitter release by neuropeptide Y, baclofen, and adenosine as revealed by [
50 the insulin promotor (Ins2) were stained for neuropeptide Y before, during, and after virally induced
51 s that synthesize agouti-related peptide and neuropeptide Y but inhibited anorexigenic neurons that s
52 e BBB-impermeable neuropeptides dalargin and neuropeptide Y chemically conjugated with FC5-Fc fusion
53 .93]) after stressful training; lower plasma neuropeptide Y concentration after stressful training (d
54 ures were heart rate, breathing rate, plasma neuropeptide Y concentration, score on the Response to S
56 eurons that are defined by the expression of neuropeptide Y::Cre (NPY::Cre) act to gate mechanical it
58 the mammalian target of rapamycin pathway in neuropeptide-Y-ergic neurons of the arcuate nucleus, and
59 A significant reduction of calbindin-, NPY (neuropeptide Y)-expressing, and cholinergic interneurons
61 endrites, whereas nitric oxide synthase- and neuropeptide Y-expressing ivy cells provided synaptic an
64 nders to two model peptides, angiotensin and neuropeptide Y, following screening by solution phage pa
65 g mice showed increased levels of C-terminal neuropeptide Y fragments and increased neurogenesis.
66 europeptide F (NPF), a homolog of vertebrate neuropeptide Y, functions in feeding and coordination of
67 single nucleotide polymorphism (SNP) in the neuropeptide Y gene has been associated with elevated se
69 essing Drosophila neuropeptide F (dNPF), the neuropeptide Y homolog, strongly correlates with food-od
71 used immunohistochemistry for calretinin and neuropeptide Y in 24 age- and gender-matched patients wi
72 nthesize gamma-amino-butyric acid (GABA) and neuropeptide Y in adult mice leads to starvation within
74 ons expressing parvalbumin, somatostatin and neuropeptide Y in the dentate gyrus, reduced aberrant ne
78 TH-ir decreased by 21%, and in hypothalamus neuropeptide Y-ir increased by 10% in MPH-exposed rats.
84 P, corticotropin releasing factor (CRF), and neuropeptide Y levels in adult male Long-Evans rats defe
86 n and attraction is thought to depend on the neuropeptide Y-like receptor NPR-1, because solitary and
88 -expressing interneurons and initiation of a neuropeptide-y-like signaling axis that promotes elevate
89 cystokinin/CB(1) cannabinoid receptor(+) and neuropeptide Y(+) local-circuit interneurons upon SAVA m
90 f both Lifeact-GFP and the SG marker protein neuropeptide Y-mCherry reveals that SGs actively translo
92 tissue weight, fecal output, arcuate nucleus neuropeptide Y mRNA expression, plasma corticosterone, a
93 ng hormone but surprisingly has no effect on neuropeptide Y mRNA, a neuropeptide previously shown to
95 s, stimulation of the agouti-related protein/neuropeptide Y neurons, and activation of the hypothalam
96 lly significant difference in the density of neuropeptide Y+ neurons between subjects with autism and
97 n, corticotropin releasing hormone, galanin, neuropeptide Y, neurotensin, preproenkephalin and tachyk
98 in, dynorphin) and brain antistress systems (neuropeptide Y, nociceptin [orphanin FQ]) in drug depend
99 r, metabotropic glutamate receptors-2 mGlu2, neuropeptide-Y NPY, and mineralocorticoid receptors MR).
101 e truncated cleaved form of neurotransmitter neuropeptide Y (NPY) actively promotes a breach of BM va
104 In lean mice, the adipokine leptin inhibits neuropeptide Y (NPY) and agouti-related peptide (AgRP) n
105 bits neurotransmission in neurons expressing neuropeptide Y (NPY) and agouti-related peptide (AgRP) v
108 the expression of orexigenic neuropeptides [neuropeptide Y (NPY) and agouti-related protein (AgRP)]
109 with changes in orexigenic peptides such as neuropeptide Y (NPY) and anorexigenic peptides such as a
110 s that regulate stress and reward, including neuropeptide Y (NPY) and corticotropin-releasing factor
111 s, including somatostatin (SST), tachykinin, neuropeptide Y (NPY) and cortistatin, in a pattern remin
112 was developed for the sensitive detection of neuropeptide Y (NPY) and employed in the analysis of NPY
114 c), containing pro-opoiomelanocortin (POMC), neuropeptide Y (NPY) and growth hormone releasing hormon
116 up-regulating specific target genes, such as neuropeptide Y (NPY) and its Y1 and Y5 receptors (Y5Rs).
117 and modulation of neurological signals, with Neuropeptide Y (NPY) and Orexin A (OXA) offering diagnos
119 ion of corticotropin releasing factor (CRF), neuropeptide Y (NPY) and proopiomelanocortin (POMC) in h
120 the lateral hypothalamus (LH) together with neuropeptide Y (NPY) and proopiomelanocortin (POMC) neur
121 the total number of interneurons expressing neuropeptide Y (NPY) and vasoactive intestinal polypepti
123 sh, and here we report the identification of neuropeptide Y (NPY) as both necessary for normal daytim
124 in a structural model of the peptide hormone neuropeptide Y (NPY) bound to its human G-protein-couple
125 in the basolateral amygdaloid complex (BLA), neuropeptide Y (NPY) buffers against protracted anxiety
126 e ontogeny of proopiomelanocortin (POMC) and neuropeptide Y (NPY) cell populations, which exert oppos
127 pocretin cells respond to dynorphin, arcuate neuropeptide Y (NPY) cells respond to hypocretin], diffe
129 strongly implicated hindbrain catecholamine/neuropeptide Y (NPY) coexpressing neurons as key mediato
130 ression of agouti-related protein (Agrp) and neuropeptide Y (Npy) coincide with the cyclic changes in
131 c (tg) mice affected primarily the levels of neuropeptide Y (NPY) compared with other neuropeptides.
132 subpopulation of CA1 interneurons expressing neuropeptide Y (NPY) contributes prominently to this dyn
134 we show that the claims of Zhou et al. that neuropeptide Y (NPY) diplotype-predicted expression is c
136 s glucocorticoids have pronounced effects on neuropeptide Y (NPY) expression and as NPY neurons proje
137 gered molecular plasticity including ectopic neuropeptide Y (NPY) expression in granule cells, but wi
138 element binding) binding protein (CBP), and neuropeptide Y (NPY) expression in the amygdaloid brain
139 binding protein (CREB) resulted in decreased neuropeptide Y (NPY) expression in the central amygdala
140 icular and dorsomedial nuclei, and increased neuropeptide Y (NPY) expression in the hypothalamus.
144 nes and their cognate receptors belonging to neuropeptide Y (NPY) family mediate diverse biological f
146 ucagon-like peptide-1 (GLP-1), glucagon, and neuropeptide Y (NPY) from circumvallate papillae of Tas1
149 anxiety and other psychiatric disorders and neuropeptide Y (NPY) has emerged as a key component of a
153 nd that appetite-regulating systems, such as neuropeptide Y (NPY) in the arcuate nucleus (ARH) and or
158 ic inputs to the two populations of striatal neuropeptide Y (NPY) interneurons, plateau low threshold
171 cord following inflammation or nerve injury, neuropeptide Y (NPY) is poised to regulate the transmiss
172 ported that the sympathetic neurotransmitter neuropeptide Y (NPY) is potently angiogenic, primarily t
174 evious observations that fluorescent-labeled neuropeptide Y (NPY) is usually released within 200 ms a
177 understand whether genetic variation at the Neuropeptide Y (NPY) locus governs secretion and stress
180 cordings were made from GFP (Renilla)-tagged neuropeptide Y (NPY) neurons from the arcuate nucleus of
182 nhibitory effect of Dyn-A on arcuate nucleus neuropeptide Y (NPY) neurons mediated by both 1 and (kap
183 In contrast, kisspeptin inhibits orexigenic neuropeptide Y (NPY) neurons through an indirect mechani
185 ts of intracerebroventricular application of Neuropeptide Y (NPY) on licking microstructure for sucro
186 he purpose of this study was to characterize neuropeptide Y (NPY) overflow and metabolism from isolat
188 nt protein is expressed under control of the neuropeptide Y (NPY) promoter and striatal NPY-expressin
190 e neuropeptide F receptor NPFR1, a mammalian neuropeptide Y (NPY) receptor homolog, centrally regulat
191 he autonomic nervous system and hypothalamic neuropeptide Y (NPY) release during fasting regulates he
199 udied the role of alpha(2)-adrenoceptors and neuropeptide Y (NPY) Y(1) receptors in mediating vasocon
202 d the role of alpha-adrenergic receptor- and neuropeptide Y (NPY) Y1 receptor-mediated vasoconstricti
204 Normal breast tissue mainly expresses the neuropeptide Y (NPY) Y2 receptor whereas primary human b
205 ations of GABAergic interneurones expressing neuropeptide Y (NPY)(+), parvalbumin (PV)(+) and tyrosin
206 neurons [gamma-aminobutyric acid (GABA)(+), neuropeptide Y (NPY)(+), proopiomelanocortin (POMC)(+),
210 study, we identified increased expression of neuropeptide Y (NPY), a pleiotropic growth factor which
211 have studied the subcellular distribution of neuropeptide Y (NPY), a prototypical and broadly express
214 In birds, CART inhibits food intake, whereas neuropeptide Y (NPY), a well-known orexigenic peptide, s
215 ry indicate that cortical astrocytes contain neuropeptide Y (NPY), a widespread neuronal transmitter.
216 from chemotherapy-induced cell death, while neuropeptide Y (NPY), acting via its Y2 receptor (Y2R),
217 c neuron markers proopiomelanocortin (POMC), neuropeptide Y (NPY), agouti-related peptide (AGRP), som
218 ment, hypothalamic mRNA expression levels of neuropeptide Y (NPY), agouti-related protein (AGRP), pro
221 ted whether VPA influences the expression of neuropeptide Y (NPY), an endogenous anticonvulsant.
222 creased mRNA levels of hypothalamic GHS-R1a, neuropeptide Y (NPY), and agouti-related protein (AgRP),
223 (CB), parvalbumin (PV), calretinin (CR) and neuropeptide Y (NPY), and somatostatin (SOM) and glial f
224 augments the SCN clock-resetting effects of neuropeptide Y (NPY), another neurochemical correlate of
225 ctor 3 (ATF3), the neuropeptides galanin and neuropeptide Y (NPY), brain-derived neurotrophic factor
226 scription level: monoamine oxidase A (MAOA), neuropeptide Y (NPY), endothelial nitric oxide synthase
227 -melanocyte-stimulating hormone (alpha-MSH), neuropeptide Y (NPY), glutamate, and GABA from first-ord
228 ), galanin, gastrin-releasing peptide (GRP), neuropeptide Y (NPY), nitric oxide synthase (NOS), serot
229 ed by either gamma-aminobutyric acid (GABA), neuropeptide Y (NPY), or beta-endorphin receptor blockad
230 Immunohistochemistry for somatostatin (SOM), neuropeptide Y (NPY), parvalbumin (PV), cholecystokinin
231 vels of genes that regulate appetite, namely neuropeptide Y (NPY), pro-opiomelanocortin (POMC) and th
232 he well-characterized neuropeptides, such as neuropeptide Y (NPY), proopiomelanocortin (POMC), orexin
233 ent with PEDF + DHA induced transcription of neuropeptide y (npy), small proline-rich protein 1a (spr
234 GFP) in a subset of neurons that colocalized neuropeptide Y (NPY), somatostatin (SST), and GABA for w
235 xp4 expression induces ectopic expression of neuropeptide Y (Npy), which has been reported to be pres
236 effects were due to disrupted signalling of neuropeptide Y (NPY), which is known to mediate non-phot
237 ar interneurons of the dentate gyrus express neuropeptide Y (NPY), which modulates granule cell activ
239 l transduction pathway modulating release of neuropeptide Y (NPY), which stimulates OE stem cell acti
240 cell activation and promotes the activity of neuropeptide Y (NPY)- and agouti-related peptide (AgRP)-
241 nterneurons express col19a1 mRNA; subsets of neuropeptide Y (NPY)-, somatostatin (Som)-, and calbindi
242 e investigated the properties of neostriatal neuropeptide Y (NPY)-expressing interneurons in transgen
244 on, appeared to be precipitated by a loss of neuropeptide Y (NPY)-mediated local circuit inhibition a
245 ood attraction to aversion is regulated by a neuropeptide Y (NPY)-related brain signaling peptide.
259 asting discharge rates of fluorescent-tagged neuropeptide-Y (NPY) (within 200 ms) and tissue plasmino
260 that express pro-opiomelanocortin (POMC) or neuropeptide-Y (NPY) and agouti-related protein (AgRP).
261 escence microscopy of lumen-targeted probes (neuropeptide Y [NPY]-pH-insensitive yellow fluorescent p
262 ridization to localize appetite-stimulating (neuropeptide Y, NPY; agouti-related protein, AGRP) and a
263 cal activity: the N-terminal region of human neuropeptide Y (NPY1-9, Tyr(1)-Pro(2)-Ser(3)-Lys(4)-Pro(
264 tostatin, vasoactive intestinal polypeptide, neuropeptide Y, or cholecystokinin (antigens commonly co
267 r morphology and expression of somatostatin, neuropeptide Y, parvalbumin, or calretinin, we infer tha
271 e submucosal plexus, DOReGFP was detected in neuropeptide Y-positive secretomotor and vasodilator neu
273 pressing green fluorescent protein under the neuropeptide Y promoter, we find that, across all layers
274 tance P, calcitonin gene-related peptide, or neuropeptide Y protein expression in DRGs and spinal cor
276 boratory-derived, mutation controlled by the neuropeptide Y receptor homolog npr-1 can affect dauer l
278 multiple signaling molecules, including the neuropeptide Y receptor NPR-1 and the calcineurin subuni
281 ion, although this was blocked by a specific neuropeptide Y receptor Y1 receptor antagonist, suggesti
283 coupled receptor (GPCR) related to mammalian neuropeptide Y receptors, functions to suppress innate i
285 Y1 receptor (Y1R) and Y5 receptor (Y5R) for neuropeptide Y share similar actions in the regulation o
286 ive deficits, as well as endocannabinoid and neuropeptide Y system alterations and altered circadian
289 receptor systems (bombesin, neurotensin, and neuropeptide-Y) that offer high potential in the field o
290 d tumor necrosis factor, whereas addition of neuropeptide Y to wild-type macrophages attenuates the r
291 s-associated increase of immunoreactivity of neuropeptide Y, tyrosine hydroxylase, and somatostatin.
294 hypothalamic expression of the orexic gene, neuropeptide Y, was lower and expression of the anorexic
295 (AgRP) neurons (which also release AgRP and neuropeptide Y), we generated mice with an AgRP neuron-s
296 (AgRP) neurons (which also release AgRP and neuropeptide Y), we generated mice with an AgRP neuron-s
297 ropeptide F (sNPF), an ortholog of mammalian neuropeptide Y, which we show here is a direct target of
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