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1 behaviors through the activity of the NPR-1 neuropeptide receptor.
2 regulated by natural variation in the npr-1 neuropeptide receptor.
3 n that is similar to a Phe-Met-Arg-Phe-amide neuropeptide receptor.
4 ell identity for these neuropeptides and the neuropeptide receptors.
5 in identifying novel antagonist ligands for neuropeptide receptors.
6 signaling through multiple G protein-coupled neuropeptide receptors.
8 crease in the expression of the CREB targets neuropeptide receptor 1 (NPY1R) and brain-derived neurot
10 We developed a multiscale model to bridge neuropeptide receptor-activated signaling pathway activi
12 n defined that exhibit broad specificity for neuropeptide receptors and induce apoptosis in SCLC by f
13 complex neurochemical profiles that include neuropeptides, receptors and components of fast neurotra
14 owever, dieldrin had more notable effects on neuropeptide receptors, and overlap between diazinon and
15 ty of these responses was demonstrated using neuropeptide receptor antagonists and nerve growth facto
16 gulate the MCH system, we investigated which neuropeptide receptors are expressed by MCH neurons by u
20 iations in receptor gene structure can alter neuropeptide receptor distribution and thereby contribut
23 ls, an increased expression of the mitogenic neuropeptide receptors for gastrin-releasing peptide and
25 RMG is the central site of action of the neuropeptide receptor gene npr-1, which distinguishes so
30 Similar experiments on midline-expressed neuropeptide receptor genes reveal considerable diversit
31 s express 9 neuropeptide precursor genes, 13 neuropeptide receptor genes, and 31 small-molecule neuro
32 ress in the characterization of invertebrate neuropeptide receptors, has the potential to propel neur
34 l terminus of a chimeric mutant delta opioid neuropeptide receptor is sufficient to re-route internal
35 hrough Gq and PLCbeta by autocrine-signaling neuropeptide receptors is a dominant pathway involved in
38 raging is modified by the naturally variable neuropeptide receptor npr-1, providing insights into how
39 voidance behaviour through inhibition of the neuropeptide receptor NPR-1, which we have previously sh
40 CO2 response is regulated by the polymorphic neuropeptide receptor NPR-1: animals with the N2 allele
41 to decrease adaptation on food, whereas the neuropeptide receptors NPR-1 and NPR-2 act to increase a
42 lethargus was abolished in mutants lacking a neuropeptide receptor (NPR-1) and was reduced in mutants
43 ith an array of ASI neuropeptides activating neuropeptide receptors on additional neurons involved in
46 ence in NPR-1, a predicted G-protein-coupled neuropeptide receptor related to Neuropeptide Y receptor
51 mical characterization of GLB-33, a putative neuropeptide receptor that is exclusively expressed in t
54 gene, npr-1, encodes a previously described neuropeptide receptor whose high activity in N2 promotes
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