ライフサイエンスコーパス: neuroplasticity

1 l and activity of cholinergic nerves (termed neuroplasticity).

2 ey pathways involved in neurodevelopment and neuroplasticity.

3 nt dental implant placement can reverse this neuroplasticity.

4 valuable model disorder to study structural neuroplasticity.

5 ly G-member2 (ABCG2) have important roles in neuroplasticity.

6 ng neuroprotection, axonal regeneration, and neuroplasticity.

7 gh maladaptations in glutamate signaling and neuroplasticity.

8 dependent reorganization allowing for guided neuroplasticity.

9 characterize perinatal temporal dynamics in neuroplasticity.

10 manization of Foxp2 on learning and striatal neuroplasticity.

11 is linked with decreased activity-dependent neuroplasticity.

12 ange factor, modulates PSD-95-NR2B-dependent neuroplasticity.

13 oderate disease through experience-dependent neuroplasticity.

14 e functions by the long-term manipulation of neuroplasticity.

15 vel cognition by the long-term modulation of neuroplasticity.

16 ify factors responsible for adult peripheral neuroplasticity.

17 nct sociosexual behaviors and the associated neuroplasticity.

18 ence for localized mechanisms of hippocampal neuroplasticity.

19 e and as a key signal for dependence-related neuroplasticity.

20 dulated by genetically driven variability in neuroplasticity.

21 ue to induce long-term depression (LTD)-like neuroplasticity.

22 and function, and regulate cell survival and neuroplasticity.

23 onstrated that chronic pain causes long-term neuroplasticity.

24 ructural changes-that is, the brain displays neuroplasticity.

25 y be a significant modulator of this altered neuroplasticity.

26 ain and dampen stress-induced alterations of neuroplasticity.

27 e impairment, suggesting adaptive structural neuroplasticity.

28 pathy, is accompanied by maladaptive central neuroplasticity.

29 rophic factor (BDNF), proteins essential for neuroplasticity.

30 ation in response to increasing evidence for neuroplasticity.

31 ct involves glycineB/NMDA receptor-dependent neuroplasticity.

32 or the sustained synaptic changes underlying neuroplasticity.

33 d as a key aspect of this experience-induced neuroplasticity.

34 ators of neuronal survival and developmental neuroplasticity.

35 m the basic neuroscience of learning-induced neuroplasticity.

36 ment of the central nervous system and adult neuroplasticity.

37 conditions and are sites of stress-mediated neuroplasticity.

38 own to be important for experience dependent neuroplasticity.

39 ith neuroinflammatory signaling and impaired neuroplasticity.

40 ation, indicating long-term training-related neuroplasticity.

41 -PKA-CREB and cAMP-ERK1/2-CREB signaling and neuroplasticity.

42 re during rescue assays in mice deficient in neuroplasticity.

43 eptor subunit 2 (GluN2) that is critical for neuroplasticity.

44 DHA) 30 min after spinal cord injury induces neuroplasticity.

45 e effects of mucosal mediators on intestinal neuroplasticity.

46 beneficial effects in SCI via enhancement of neuroplasticity.

47 urvivors, with evidence for training-related neuroplasticity.

48 to be within the classic critical period for neuroplasticity [7, 8].

49 ne-bound astrocyte growth inhibitor to limit neuroplasticity, activity-dependent axonal sprouting, an

50 Previous studies demonstrate distinct neuroplasticity adaptations in the two medium spiny neur

51 We examined how a mediator of hippocampal neuroplasticity, adult neurogenesis in the subgranular z

52 ent-related brain potentials (ERPs) to assay neuroplasticity after auditory conditioning in chronic s

53 epresent a strategy for inducing respiratory neuroplasticity after declines in respiratory function t

54 l for rehabilitation, as it shows heightened neuroplasticity, allowing sensorimotor functions to re-m

55 us accumbens, but it is unknown whether this neuroplasticity alters the effect of a subsequent cocain

56 or in the expression of proteins involved in neuroplasticity, an effect mediated through cGMP, PKG, a

57 physical activity has been shown to promote neuroplasticity and an anti-inflammatory state in the ad

58 functional relationship between cholinergic neuroplasticity and ASM contractile phenotypes that oper

59 o) enhancements in hippocampal cognition and neuroplasticity and can alleviate hippocampal cognitive

60 enhance opportunities for the translation of neuroplasticity and circuit retraining research into eff

61 tients with mood disorders who have impaired neuroplasticity and cognition.

62 e improves cognitive functions by modulating neuroplasticity and cortical excitability in nonsmoking

63 clude increased neurogenesis, enhancement of neuroplasticity and deployment of a successful endoplasm

64 ecific genetic interactions related to brain neuroplasticity and development, and that these AKT1 eff

65 rting AKT1's role in transducing hippocampal neuroplasticity and dopaminergic processes, we found epi

66 immediate early genes and genes involved in neuroplasticity and epigenetic modifications.

67 gnaling, and are thus essential for striatal neuroplasticity and fear and anxiety behavior.

68 at should follow early diagnosis to optimize neuroplasticity and function.

69 We now show that neuroplasticity and functional recovery after stroke is

70 n energy metabolism and processes related to neuroplasticity and growth may be involved in the develo

71 dings advance our understanding of long-term neuroplasticity and have general implications for the de

72 n as environmental enrichment, can stimulate neuroplasticity and improve hippocampal function and per

73 is study advances our understanding of human neuroplasticity and its genetic underpinnings following

74 hippocampal neurons play important roles in neuroplasticity and learning and memory.

75 ays of VU0409551 treatment could reverse the neuroplasticity and learning deficits, respectively, in

76 ty patterns (Arc expression) associated with neuroplasticity and memory.

77 rotein (FMRP), thought to play a key role in neuroplasticity and neuronal translation, in ASD-affecte

78 hich play an important role in sleep-related neuroplasticity and offline information processing.

79 that play an important role in sleep-related neuroplasticity and offline information processing.

80 ulphate using chondroitinase ABC reactivated neuroplasticity and promoted sensorimotor recovery after

81 ential role for tamalin in ECS-induced adult neuroplasticity and provide new insight into the pathway

82 is a novel therapeutic target for enhancing neuroplasticity and recovery after SCI.

83 Erythropoietin (EPO) increases neuroplasticity and reduces cognitive difficulties in tr

84 data suggest that chronic pain-induced PACAP neuroplasticity and signaling in spinoparabrachioamygdal

85 ry results indicate that tDCS may facilitate neuroplasticity and suggest the potential for refining r

86 may be developed incorporating knowledge of neuroplasticity and the critical ingredients of rehabili

87 However, the linkage between this neuroplasticity and the functional deficits in carpal tu

88 llosal projections, showing interhemispheric neuroplasticity and thus, setting a foundation to develo

89 ulation of CaV2.1 channels to other forms of neuroplasticity and to learning and memory are not known

90 ent of newer and more precise tools to alter neuroplasticity and to treat brain-based disorders.

91 ow and why exercise and energy intake affect neuroplasticity and, conversely, how the brain regulates

92 sease, cerebrovascular deregulation, altered neuroplasticity, and changes in glutamate neurotransmiss

93 ations in neurodevelopment, neurogenesis and neuroplasticity, and is involved in the mechanisms of ac

94 Despite the importance of respiratory system neuroplasticity, and its dependence on estrogen in males

95 enes implicated in cell cycle, neurogenesis, neuroplasticity, and neurosignaling.

96 h, strengthens muscles and bones, stimulates neuroplasticity, and promotes feelings of well-being.

97 discovered and unexpected roles for TLRs in neuroplasticity, and the implications of these findings

98 did not), with enrichment in inflammation-, neuroplasticity- and oxidative stress-related pathways.

99 e cellular mechanisms mediating this form of neuroplasticity are poorly understood.

100 up the perspective of harnessing respiratory neuroplasticity as a therapeutic tool for the management

101 xamining the inter-individual variability in neuroplasticity as modeled by a functional polymorphism

102 c brain regions may be the result of altered neuroplasticity associated with chronic compulsive behav

103 hin-KOR system is a central component of the neuroplasticity associated with negative reinforcement s

104 genous cannabinoid system may be part of the neuroplasticity associated with the development of cocai

105 In other mice, we silenced the neuroplasticity-associated neurotrophin brain-derived ne

106 pathology, thus contributing to maladaptive neuroplasticity at multiple levels.

107 ation and requires time-dependent changes in neuroplasticity at the level of glutamatergic synapses i

108 a and macrophage functions also will enhance neuroplasticity, at and several segments below the injur

109 at enhances endogenous repair and indices of neuroplasticity, at and several segments below the injur

110 Here we develop a neuroplasticity-based computerized cognitive remediation

111 se the hippocampus is a key area involved in neuroplasticity, behavior, and cognition, we hypothesize

112 ddiction is commonly viewed as a disorder of neuroplasticity, but the potential involvement of MeCP2

113 strata pyramidale and radiatum and assessed neuroplasticity by inducing long-term potentiation (LTP)

114 Neuroplasticity can be defined as the ability of the ner

115 Our results indicate that neuroplasticity can manifest itself as differences in co

116 We show that beneficial neuroplasticity can occur alongside decoder adaptation,

117 showed comparable improvement, long-lasting neuroplasticity can only be detected in the tDCS group,

118 ng neural decoding and biofeedback to target neuroplasticity causally links early visual cortical pla

119 sed thalamocortical FC, thus suggesting that neuroplasticity changes are unable to compensate for tis

120 Our findings imply that robust neuroplasticity conferred by musical training is not res

121 Furthermore, we studied whether this neuroplasticity contributed to spinal neuron sensitizati

122 is known to regulate emotional behavior and neuroplasticity, contributes to changes in amygdalar str

123 Furthermore, not all neuroplasticity could be explained by activity-dependent

124 neurogenesis or gliogenesis may help reverse neuroplasticity during abstinence and, thus, may help re

125 ffects of androgen and estrogen treatment on neuroplasticity during photostimulation in male and fema

126 These findings demonstrate acute functional neuroplasticity during stress, with distinct and separab

127 rphism impairs the beneficial behavioral and neuroplasticity effects induced by physical exercise.

128 beneficial effects in spinal cord injury via neuroplasticity enhancement.

129 cy, indicating limited success in modulating neuroplasticity, especially in brains with neural atypic

130 Concomitantly, neurogenesis and neuroplasticity fall significantly.

131 Neuroplasticity following antidepressant drug treatment

132 rol of Arc that could be targeted to harness neuroplasticity for clinical applications.

133 In conclusion, primary somatosensory cortex neuroplasticity for median nerve innervated digits in ca

134 These results highlight the utility of neuroplasticity for real-world neuroprostheses.

135 rdsong and exhibits some of the best-studied neuroplasticity found in the adult brain.

136 facilitation (LTF) is a form of respiratory neuroplasticity frequently induced by acute intermittent

137 iological convergence between stress-related neuroplasticity, functional neurological symptoms and re

138 Models of AIH-induced neuroplasticity have focused on motoneurons; however, mo

139 direct current stimulation to modulate human neuroplasticity have increased in research and clinical

140 direct current stimulation to modulate human neuroplasticity have increased in research and clinical

141 Major advances in the understanding of neuroplasticity have to date yielded few established int

142 essor, has enduring effects on bidirectional neuroplasticity in adulthood, changes in long-term poten

143 pment but continue to play a central role in neuroplasticity in adulthood; and that they work not onl

144 We examined cross-modal neuroplasticity in anatomically defined subregions of He

145 s and depression, and demonstrate a role for neuroplasticity in antidepressant treatment response and

146 oth measures objectively indicate functional neuroplasticity in auditory perceptual learning.SIGNIFIC

147 MRI) has greatly extended the exploration of neuroplasticity in behaving animals and humans.

148 the central role for miRNAs in drug-induced neuroplasticity in brain reward systems that drive the e

149 ncephalography measures are novel markers of neuroplasticity in carpal tunnel syndrome and could be u

150 ibility that the mechanism may also regulate neuroplasticity in circuits other than the olfactory sen

151 in which genetic variation in glutamatergic neuroplasticity in corticolimbic circuitry underlies phe

152 cervical spinal cord to recover respiratory neuroplasticity in disorders of respiratory insufficienc

153 n of synaptic GABA(A) receptors have induced neuroplasticity in dopamine neurons due to this disinhib

154 ntation, pattern recognition capability, and neuroplasticity in each efferent circuit.

155 mprovement of blood flow and facilitation of neuroplasticity in elderly people, might also have a rol

156 ticolimbic regions undergoes cocaine induced neuroplasticity in female rats.

157 Early neuroplasticity in higher visual areas appears to be an

158 specific affect of D2 receptor activation on neuroplasticity in humans, because physiological effects

159 Improved means of assessing neuroplasticity in humans, including biomarkers for pred

160 the amount of D1-like receptor activation on neuroplasticity in humans, we combined sulpiride, a sele

161 Overall, the results suggest that the neuroplasticity in MIo and SIo occurring in parallel ser

162 Hyperalgesic priming, a form of neuroplasticity in nociceptors, is a model of the transi

163 me, is emerging as an important regulator of neuroplasticity in postmitotic neurons.

164 that are believed to result from functional neuroplasticity in prefrontal and auditory cortices.

165 These data show that LTP-like neuroplasticity in prefrontal-accumbens synapses is init

166 The ultimate utility of neuroplasticity in real-world neuroprostheses is thus un

167 their involvement in defective signaling and neuroplasticity in relationship with mood disorders is s

168 Neuroplasticity in response to adverse environmental con

169 ate preterm adolescents had reduced LTD-like neuroplasticity in response to brain stimulation that wa

170 and limbic brain regions, though associative neuroplasticity in sensory structures is increasingly ap

171 athing protocol find expression as long-term neuroplasticity in serial measurements made over 20 days

172 demonstrate the time-course of white matter neuroplasticity in short-term meditation.

173 We also compared neuroplasticity in term-born adolescents with that in te

174 analyses of regional activations, we studied neuroplasticity in terms of differential network connect

175 Neuroplasticity in the amygdala drives pain-related beha

176 ain-related behaviors.SIGNIFICANCE STATEMENT Neuroplasticity in the amygdala has emerged as an import

177 Neuroplasticity in the amygdala plays a key role in emot

178 and genesis of AP can provide insights into neuroplasticity in the auditory system.

179 Neuroplasticity in the cortico-accumbens pathway is regu

180 ECT-induced neuroplasticity in the hippocampus and amygdala relates

181 exerts a nonlinear dose-dependent effect on neuroplasticity in the human motor cortex that differs f

182 following loss of natural reward by causing neuroplasticity in the mesolimbic pathway during the nat

183 Drugs of abuse induce neuroplasticity in the natural reward pathway, specifica

184 increases expression of cellular markers of neuroplasticity in the non-injured S1 compared to TBI ra

185 ion at the wrist by somatotopically distinct neuroplasticity in the primary somatosensory cortex foll

186 o characterized by functional and structural neuroplasticity in the primary somatosensory cortex of t

187 ciplinary findings pertaining to respiratory neuroplasticity in the rat.

188 les provide an innate model for the study of neuroplasticity in the regulation of aggression.

189 lling evidence has demonstrated considerable neuroplasticity in the respiratory control system, few s

190 ared to NH in core and belt ACFs, indicating neuroplasticity in the right hemisphere.

191 t that physical activity could contribute to neuroplasticity in the RVLM.

192 the hypothesis that singing activity induces neuroplasticity in the song control system independent o

193 by TCT in ESZ are associated with structural neuroplasticity in the thalamus.

194 , our findings suggest that learning-related neuroplasticity in the VLO may be necessary for memory r

195 e caused by a focusing effect of nicotine on neuroplasticity in these subjects.

196 TA) are a key target of addictive drugs, and neuroplasticity in this region may underlie some of the

197 We examined motor cortex neuroplasticity in three groups of adolescents who were

198 plore the use of decoder adaptation to shape neuroplasticity in two scenarios relevant for real-world

199 receptors actively regulate cocaine-induced neuroplasticity in vivo, these data support the hypothes

200 ose born deaf can offer unique insights into neuroplasticity, in particular in regions of superior te

201 of several susceptibility genes involved in neuroplasticity, including Bdnf, its receptor TrkB, the

202 nding attenuation of FLX-induced hippocampal neuroplasticity, including decreased hippocampal neuroge

203 Changes related to neuroplasticity, including enhanced adult hippocampal ne

204 genesis and expression of genes critical for neuroplasticity, including WNT pathway components and io

205 The neuroplasticity induced by Pavlovian fear conditioning h

206 finity, concentration level, and the kind of neuroplasticity induced.

207 ficity, concentration level, and the kind of neuroplasticity induced.

208 Neuroplasticity is a fundamental property of the nervous

209 ults suggest that stimulus-specific auditory neuroplasticity is abnormal in schizophrenia.

210 ions, which emerge when experience-dependent neuroplasticity is at its peak.

211 The ability of neuritin to increase neuroplasticity is confirmed in models of learning and m

212 Neuroplasticity is essential for recovery after stroke a

213 Neuroplasticity is essential to prevent clinical worseni

214 Neuroplasticity is limited in maturity, but it is promot

215 These data indicate that PSA-NCAM-mediated neuroplasticity is necessary for antidepressant action;

216 We demonstrated that cholinergic neuroplasticity is necessary for the induction of persis

217 hapes brain development throughout life, but neuroplasticity is variable from one brain system to ano

218 ated in neuroprotection, carcinogenesis, and neuroplasticity, is a Ca(2+)-sensitive and ligand-operat

219 e II (alphaCaMKII), a molecule implicated in neuroplasticity, is a target of CPEB and can also affect

220 Neuroplasticity manifests as normalization of aberrant f

221 Disrupted neuroplasticity may be an important aspect of the neural

222 extinction occurs during DCS administration, neuroplasticity may be enhanced.

223 ibutions of DHT and E2 signaling in songbird neuroplasticity may be regulated by photoperiod and that

224 Neuroplasticity may play a critical role in developing r

225 These data suggest that altered amygdala neuroplasticity may play a role the early dispositional

226 Cocaine-induced neuroplasticity mediated by histone acetylating and deac

227 call induces a limited period of hippocampal neuroplasticity mediated, in part, by S-nitrosylation of

228 Our observations argue that neuroplasticity mediating perceptual learning occurs at

229 To address this issue, we used a neuroplasticity model based on treatment of neuronal cul

230 they may play a role related to the reduced neuroplasticity observed in depression.

231 Neuroplasticity occurs with many variations, in many for

232 rgic inhibition also plays a crucial role in neuroplasticity of adult animals: the balance between ex

233 This study relies on knowledge regarding the neuroplasticity of dual-system components that govern ad

234 It represents a natural example of the neuroplasticity of motion adaptation.

235 Using information from research on the neuroplasticity of selective attention and on the centra

236 We hypothesize that epilepsy-related neuroplasticity of septohippocampal cholinergic neurons

237 lly regulated by environment and underscores neuroplasticity of serotonergic neurons in C. elegans un

238 ave major implications for understanding the neuroplasticity of the brain, which could hypothetically

239 Neuroplasticity of the C. elegans IL2 sensory neurons pr

240 diction may involve progressive drug-induced neuroplasticity of the dorsal striatum.

241 ctory learning, and emphasize the functional neuroplasticity of the GABAergic system as a result of l

242 -shaped effects of D1 receptor activation on neuroplasticity of the motor cortex.

243 The neuroplasticity of the mu-opioid system is of future res

244 stem or 'darkness within.' Understanding the neuroplasticity of the neurocircuitry that comprises the

245 Neuroplasticity of the visual cortex or higher cortical

246 exercise and cognitive challenge to enhance neuroplasticity, our ACM addresses two fundamental quest

247 ic strokes, suggesting impaired capacity for neuroplasticity over this hemisphere as a consequence of

248 They directly regulate inflammation and neuroplasticity pathways and also influence insulin sens

249 lar proteases have emerged as key players in neuroplasticity phenomena.

250 The two major classes of activity-dependent neuroplasticity predict different consequences of activi

251 brain-derived neurotrophic factor (BDNF) in neuroplasticity related to schizophrenia and the recent

252 elationship between connectivity changes and neuroplasticity-related gene expression profiles in the

253 tion epigenetically primes the expression of neuroplasticity-related genes, which is accompanied by h

254 associated with decreased expression of the neuroplasticity-related polysialylated neural cell adhes

255 To advance the translation of neuroplasticity research towards clinical applications,

256 itation (pLTF), a form of spinal respiratory neuroplasticity resulting in increased phrenic nerve mot

257 R-/- mice reverses their deficits in several neuroplasticity signaling pathways and improves their co

258 , decreased dendritic spine density, altered neuroplasticity signaling pathways, and cognitive defici

259 signaling in any form of respiratory system neuroplasticity.SIGNIFICANCE STATEMENT Exposure to acute

260 Conversely, by using Hebbian-like neuroplasticity, silent yet intact circuits enter a hibe

261 Diabetes may impair the capacity for neuroplasticity such that patients experience a slower a

262 ion of genes implicated in the mechanisms of neuroplasticity, such as the NMDA and AMPA subunits and

263 d topographic correspondence between Arc and neuroplasticity suggests Arc may be intrinsic to the neu

264 upon a structural scaffolding with intrinsic neuroplasticity that changes with development, aging, di

265 Long-term potentiation is a form of neuroplasticity that has been recently demonstrated in h

266 The same neuroplasticity that leaves emotional regulation, behavi

267 traint stress causes long-lasting changes in neuroplasticity that likely reflect pathological modific

268 This is interpreted as a failure of adult neuroplasticity that maintains neuropil space.

269 The neuroplasticity that subserves learning and memory is al

270 a suggest a mechanism of aberrant prefrontal neuroplasticity that underlies enhanced propensity for i

271 significantly reduced capacity for cortical neuroplasticity, the key overall mechanism underlying le

272 ognitive training and neuromodulation affect neuroplasticity, their combination could promote greater

273 ic memory and is commonly thought to rely on neuroplasticity to adapt to the ever-changing environmen

274 ation or TrkB signaling in vivo impaired the neuroplasticity to chronic stress and the effects of the

275 mental hypothesis linking decreased amygdala neuroplasticity to early-life dispositional anxiety.

276 us system may interact with aberrant central neuroplasticity to produce the clinical picture.

277 ticofugal system is thought to contribute to neuroplasticity underlying auditory perceptual learning.

278 Neuroplasticity underlying drug-induced sensitization ha

279 Here we demonstrate that the neuroplasticity underlying nociceptor priming requires 7

280 sticity suggests Arc may be intrinsic to the neuroplasticity underlying perceptual learning.

281 Spinal mGluR5 is a key mediator of neuroplasticity underlying persistent pain.

282 ation/ageing identified cardinal examples of neuroplasticity, underlying mechanisms, therapeutic impl

283 The second uses neuroimaging to investigate neuroplasticity, via changes in connectivity, cortical t

284 Nevertheless, hippocampal neuroplasticity was impaired in LV-IRAS-treated rats.

285 The role of neuroplasticity was investigated using fMRI.

286 he molecular and cellular mechanisms of this neuroplasticity, we investigated the effects of olfactor

287 Since inflammation modulates neuroplasticity, we studied the impact of inflammation c

288 After 14 sessions, Arc and neuroplasticity were aligned with target-induced activit

289 Arc expression and physiologically measured neuroplasticity were strong in a high-frequency auditory

290 Neural reuse is a form of neuroplasticity whereby neural elements originally devel

291 t is highly likely that inflammation-induced neuroplasticity, which is not detectable by clinical dia

292 ppocampal neural circuitry provides enhanced neuroplasticity, which plays a crucial role in learning

293 o the long-term molecular changes underlying neuroplasticity, which typically require activation of t

294 e local connectome fingerprint also revealed neuroplasticity within an individual reflected as a decr

295 ered after SCI and characterized DHA-induced neuroplasticity within the adult injured spinal cord.

296 remodels cortical synapses and suggest that neuroplasticity within the healthy oPFC gates the influe

297 target genes that are well-known markers of neuroplasticity within the hippocampus.

298 ted protein Homer2 regulates alcohol-induced neuroplasticity within the nucleus accumbens (NAC), but

299 Activity-dependent and BDNF-dependent neuroplasticity within the OFC coordinate outcome-based

300 localize the site and candidate mechanism of neuroplasticity within upstream regions of this inhibito