ライフサイエンスコーパス: neuroprostane

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1 nes, with E/D-type prostane rings (E(4)/D(4)-neuroprostanes).

2 enantiomerically pure diastereomer of 13-F4t-neuroprostane.

3 ry definitively identified them as A(4)/J(4)-neuroprostanes.

4 are formed from the dehydration of E(4)/D(4)-neuroprostanes.

5 er of magnitude higher than other classes of neuroprostanes.

6 nitively identified these compounds as D4/E4-neuroprostanes.

7 isoprostane-like compounds, which we term F4-neuroprostanes.

8 vitro increased levels of putative A(4)/J(4)-neuroprostanes 64-fold from 88 +/- 43 to 5463 +/- 2579 n

9 We thus explored whether cyclopentenone neuroprostanes (A(4)/J(4)-neuroprostanes) are formed fro

10 One class of compounds are cyclopentenone neuroprostanes (A(4)/J(4)-NPs), which are highly reactiv

11 Detection of F(4)-neuroprostanes, a neuronal-specific oxidative damage mar

12 F4-neuroprostanes, an index of lipid peroxidation, were une

13 d from docosahexaenoic acid (DHA) are termed neuroprostanes and are more abundant than isoprostanes (

14 tion decreased cerebral isoprostanes but not neuroprostanes and enhanced DHA, but not AA, endoperoxid

15 Neuroprostanes are prostaglandin-like compounds produced

16 her cyclopentenone neuroprostanes (A(4)/J(4)-neuroprostanes) are formed from the dehydration of E(4)/

17 hether D/E-isoprostane-like compounds (D4/E4-neuroprostanes) are formed from the oxidation of docosah

18 Because of the fact that A(4)/J(4)-neuroprostanes contain highly reactive cyclopentenone ri

19 Furthermore, the formation of F4-neuroprostane-containing aminophospholipids might advers

20 Furthermore, F4-neuroprostanes could be detected in normal human cerebro

21 ed from arachidonic acid (AA) oxidation, and neuroprostanes, derived from docosahexaenoic acid (DHA)

22 scribe F(2)-isoprostane (F(2)-IsoP) and F(4)-neuroprostane (F(4)-NP) levels in longitudinally followe

23 ch as the F(2)-isoprostanes (IsoPs) and F(4)-neuroprostanes (F(4)-NeuroPs), may be useful in assessin

24 We then explored the formation of D4/E4-neuroprostanes from a biological source, rat brain synap

25 Indeed, incubation of A(4)/J(4)-neuroprostanes in vitro with excess glutathione resulted

26 A(4)/J(4)-neuroprostanes increased 13-fold, from a basal level of

27 Levels of putative D4/E4-neuroprostanes increased 380-fold after oxidation of doc

28 The formation of neuroprostanes, isoprostane-like compounds formed from o

29 Neuroprostane levels were enhanced in the presence of Sh

30 F4-neuroprostanes may provide a unique marker of oxidative

31 stanes and neuronal stress evaluated by F(4)-neuroprostane measurement.

32 ughout white matter and gray matter, whereas neuroprostanes (NPs) are generated analogously from doco

33 array of IsoP-like compounds that are termed neuroprostanes (NPs).

34 des, neuroketals, in vivo as products of the neuroprostane pathway.

35 pounds (neuroketals) are also formed via the neuroprostane pathway.

36 (NeuroKs) as products of the isoprostane and neuroprostane pathways of free radical-induced lipid per

37 synthetic route to the isoprostanes and the neuroprostanes that could allow ready access to each of

38 that were structurally established to be F4-neuroprostanes using a number of mass spectrometric appr

39 Basal levels of D4/E4-neuroprostanes were 3.8 +/- 0.6 ng/mg of protein and inc

40 F4-neuroprostanes were detected esterified in normal whole

41 oxidation, isoprostane-like compounds termed neuroprostanes, with E/D-type prostane rings (E(4)/D(4)-

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