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1  2-SMe-ADP(alpha-S), 7A, as a most promising neuroprotectant.
2   Hence, we propose 7A as a highly promising neuroprotectant.
3 sodium channels, was initially proposed as a neuroprotectant.
4   Hence, we propose 7a as a highly promising neuroprotectant.
5 llin (Cryab), an endogenous immunomodulatory neuroprotectant.
6  have potential as an effective and nontoxic neuroprotectant.
7 CS1 and as a consequence is able to act as a neuroprotectant.
8 ighlight the potential of this compound as a neuroprotectant.
9 ariety of antioxidants have been examined as neuroprotectants.
10 opted recently to investigate four candidate neuroprotectants.
11  17beta-estradiol and its analogs are potent neuroprotectants.
12 quinazoline scaffolds that themselves act as neuroprotectants.
13 K-1 is a target for general anaesthetics and neuroprotectants.
14 licity-properties favoring their efficacy as neuroprotectants.
15 therapeutic potential as anticonvulsants and neuroprotectants.
16          Here, we developed nucleotide-based neuroprotectants acting dually as antioxidants and P2Y-R
17  oxidase and elucidated the role of CK2 as a neuroprotectant after oxidative insults to the brain.
18 s less potent or altogether ineffective as a neuroprotectant after sham surgery compared to OVX or af
19                          GPT is an effective neuroprotectant against glutamate excitotoxicity.
20 this study supports IV Ca/Mg as an effective neuroprotectant against oxaliplatin-induced cumulative s
21 xifen (TMX), has been shown to function as a neuroprotectant against the nigrostriatal dopaminergic (
22 ng interest in the functions of estrogens as neuroprotectants against neurodegenerative diseases incl
23 ty and that DMF could serve as an adjunctive neuroprotectant and HIV disease modifier in ART-treated
24 rials for multiple sclerosis, as a potential neuroprotectant and HIV disease-modifying agent.
25  endogenous levels of estrogen, a well-known neuroprotectant and immunomodulator.
26 es demonstrated that exogenous EPO acts as a neuroprotectant and regulates neurogenesis.
27 t QXs 12a and 12e are also active in vivo as neuroprotectants and also have antinociceptive activity
28 in the development of future highly specific neuroprotectants and cardioprotectants.
29 pendent on the impact of the insult and that neuroprotectants are more likely to benefit a patient in
30 r (H3R) were obtained by optimization of the neuroprotectant ASS234 by incorporating generally accept
31                         Estrogens are potent neuroprotectants both in vitro and in vivo.
32 rophic factor (BDNF) is a well-known retinal neuroprotectant, but its effectiveness is limited: highe
33                         Insulin may act as a neuroprotectant by increasing extracellular GABA resulti
34 ccumbens; and (4) estrogen may function as a neuroprotectant by reducing the uptake of neurotoxin int
35               We conclude that if a putative neuroprotectant can be administered in such a way that i
36         The lack of availability of specific neuroprotectant compounds in the United States and the l
37                   Protein S is a significant neuroprotectant during ischemic brain injury with direct
38 e, the authors tested the effects of sex and neuroprotectant exposure on the behavioral consequences
39                                     The best neuroprotectant for acute ischaemic stroke would always
40 that cobalamin may function as an endogenous neuroprotectant for RGCs through a superoxide-associated
41 te antagonist under clinical evaluation as a neuroprotectant for stroke and head trauma.
42 , suggesting that IGF-1 may have a role as a neuroprotectant for surviving neurons and signal for loc
43 hesized that SIRT1 might serve as a critical neuroprotectant for wake neurons in young animals but th
44            SIRT1 is a critical age-sensitive neuroprotectant for wake neurons, and its deficiency res
45 need for novel, biocompatible, and effective neuroprotectants for the treatment of neurodegenerative
46                                          The neuroprotectant fructose-1,6-bisphosphate (FBP) preserve
47                   Unfortunately, none of the neuroprotectants has been proved effective in clinical t
48 end any specific agent for clinical use as a neuroprotectant, however.
49  gas xenon has been shown to be an effective neuroprotectant in a variety of in vitro and in vivo mod
50 an NMDA antagonist, thereby functioning as a neuroprotectant in a wide range of pathological states.
51 ines the studies indicating that estrogen is neuroprotectant in animal models and explores potential
52  efficiently transduce a biologically active neuroprotectant in experimental cerebral ischemia.
53                   Estrogen is an established neuroprotectant in experimental stroke.
54 gest that EPO may be a potential therapeutic neuroprotectant in glaucoma.
55 e pharmacotherapeutic potential of PEDF as a neuroprotectant in human motor neuron degeneration.
56 K3A-APC has advanced to clinical trials as a neuroprotectant in ischemic stroke.
57 unction, methylene blue (MB) is an effective neuroprotectant in many neurological disorders (e.g., Pa
58      The data show that BDNF is an effective neuroprotectant in primate-sized eyes after optic nerve
59 se Mn-SOD gene and plays a crucial role as a neuroprotectant in regulating levels of reactive oxygen
60 ed protein C has been shown to be a powerful neuroprotectant in stressed neurons and in hypoxic brain
61 These data indicate that PQQ may be a useful neuroprotectant in stroke therapy.
62 h PEDF as both a metastatic suppressor and a neuroprotectant in the brain, highlighting its role as a
63 has recently been recognized as an important neuroprotectant in the central nervous system.
64 trophic factor (BDNF), a particularly potent neuroprotectant in the mammalian eye and the basis of ou
65  data show that tPA can act as an endogenous neuroprotectant in the murine hippocampus.
66 pothesis that 17beta-estradiol (betaE2) is a neuroprotectant in the retina, using two experimental ap
67            IL-6 may serve as a photoreceptor neuroprotectant in the setting of retinal-RPE separation
68 e inert gases helium and xenon are effective neuroprotectants in a model for traumatic brain injury,
69 ded that the lack of efficacy of a number of neuroprotectants in clinical trials may well be a conseq
70 valuation of complement inhibitors and other neuroprotectants intended for ocular use.
71 reatment with elevated concentrations of the neuroprotectants KCl or cAMP at the time of deprivation
72 Cryab is an endogenous anti-inflammatory and neuroprotectant molecule produced after stroke, whose be
73 wever, while adenosine acts as an endogenous neuroprotectant, NO is believed to be the effector of gl
74 eroid hormone estrogen (E) can function as a neuroprotectant of nigrostriatal dopaminergic (NSDA) neu
75 ard to estradiol's capacity to function as a neuroprotectant of the nigrostriatal dopaminergic system
76  corpus striatum, and in this way serve as a neuroprotectant of the nigrostriatal dopaminergic system
77 he effects of mild hypothermia, an effective neuroprotectant, on fluid shifts, cerebral perfusion and
78 nt strategies involving fibrates, connexins, neuroprotectants, photobiomodulation, and anti-inflammat
79                            Here, we used the neuroprotectant riluzole as a template for the design of
80 nsufficient data on the use of the potential neuroprotectant selegiline and patients on pramipexole i
81 ts identify Cdc25A as a potential target for neuroprotectant strategy for the treatment of delayed is
82             Efforts to identify a successful neuroprotectant strategy would have a major impact on im
83 ated fatty acids (such as arachidonic acid), neuroprotectants (such as riluzole) and volatile and gas
84 ecause of decreased availability of IGF-1, a neuroprotectant that decreases with advancing age and is
85                    The only stroke trial for neuroprotectants that showed benefit to patients targete
86 l cells (ECs) in the brain, functioning as a neuroprotectant through the activation of the neurotroph
87  indicates that estrogens could be used as a neuroprotectant to prolong the therapeutic window of thr
88 e, suggesting that NGB acts as an endogenous neuroprotectant to reduce oxidative stress and improve m
89                              For the SAINT I neuroprotectant trial, considered positive by "shift" mR
90 h a view to identify novel and biocompatible neuroprotectants, we designed nucleoside 5'-thiophosphat
91 ures of both helium and xenon were effective neuroprotectants when applied in addition to 1 atm of ai
92 ovide future opportunities for generation of neuroprotectants with high specificity.
93  (chlorpromazine & promethazine) as additive neuroprotectants, with the aim of augmenting its efficac
94 e of the bases for TMX to function as a NSDA neuroprotectant within male mice.
95                    Here, we designed a novel neuroprotectant (ZL006) loaded dual targeted nanocarrier

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