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1 r to that of other (11)C-labeled ligands for neuroreceptors.
2  to image and measure radioligand binding to neuroreceptors.
3  have demonstrated widespread structural and neuroreceptor abnormalities beyond the region of MCD tha
4 PET/fMRI enables characterization of dynamic neuroreceptor adaptations in vivo, and may offer a first
5 own to modulate the activity of a variety of neuroreceptors and ion channels.
6 discovery efforts targeting ion channels and neuroreceptors and mechanistic insights for the complex
7 luminating subtle structural distinctions in neuroreceptors and related integral membrane proteins.
8  activated by an immunoreceptor (CD86) and a neuroreceptor (beta2AR) converge to regulate the IgG1 re
9 re concurrent declines in caudate/putamen D2 neuroreceptor binding and tissue volume.
10  cerebral blood flow, glucose metabolism and neuroreceptor binding are influenced by partial-volume a
11 etry (AMS) allowed the use of [(14)C]TETS in neuroreceptor binding studies with rat brain membranes i
12  noninvasive measures of neurophysiology and neuroreceptor binding, are powerful and sensitive tools
13                          However, subsequent neuroreceptor changes including monoamine deficits compl
14 g was observed with 10 nM GE-179 at 60 other neuroreceptors, channels, or transporters.
15 ted two and three UAAs simultaneously into a neuroreceptor expressed in vivo.
16 he elucidation of the broad diversity of the neuroreceptor families.
17                                              Neuroreceptor imaging in the nonhuman primate (NHP) is v
18 f kinetic analysis of radioligand binding to neuroreceptors in brain in vivo, here applied to noradre
19 ytryptamine-2 or 5-HT2A) and dopamine-2 (D2) neuroreceptors in subjects being treated with typical or
20 -gated ion channels that are the predominant neuroreceptors in the mammalian brain.
21 it is essential for clustering of inhibitory neuroreceptors in the postsynaptic membrane.
22                 The brain's major inhibitory neuroreceptor is the ligand-gated ion channel gamma-amin
23  inferred that the approach to deliver small neuroreceptor ligands across the BBB by transport peptid
24 d not appear to bind significantly to common neuroreceptors or transporter sites.
25 ronal activation using Fos and neurochemical/neuroreceptor profiles on brain areas involved in these
26 e introduced and commonly used PET and SPECT neuroreceptor quantification models are discussed.
27                Positron emission tomographic neuroreceptor studies are needed to determine whether po
28 e A receptor (GABAA-R) is a major inhibitory neuroreceptor that is activated by the binding of GABA.
29 ludes the sinus node or AV node or important neuroreceptors; whether many small arteries are occluded

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