ライフサイエンスコーパス: neurosensory

1 ntional and unconventional, are critical for neurosensory activities.

2 Neurosensory and behavioural disruptions are some of the

3 Neurosensory and neurologic dysfunctions were assessed a

4 ory epithelium (OE) to regenerate fully both neurosensory and nonneuronal cell types after severe epi

5 g the otic vesicle; the latter gives rise to neurosensory and nonsensory elements of the adult membra

6 pothyroidism, osteoporosis, cardiopulmonary, neurosensory, and neuromotor impairments.

7 ndocrine, gastrointestinal, musculoskeletal, neurosensory, and neuromotor impairments.

8 : neuromotor (nonambulatory cerebral palsy), neurosensory (blindness, deafness, or need for visual/he

9 ndings establish and characterize a distinct neurosensory cell signaling pathway that determines the

10 al hearing outcome and the long-term fate of neurosensory cells in the cochlea, i.e., hair cells and

11 human cell line 293 and also in vivo, within neurosensory cells of guinea pig eye.

12 c acoel, Symsagittifera roscoffensis, and in neurosensory cells of the jellyfish Clytia hemisphaerica

13 in neurons of the circumventricular organs, neurosensory cells responsive to systemic osmotic pressu

14 NEBs), innervated clusters of neuroendocrine/neurosensory cells within the bronchial epithelium, reve

15 ly expressed in differentiating nematocytes (neurosensory cells) and in statocytes (ciliated mechanos

16 The channel also occurs in other neurosensory cells, including inner-ear hair cells, sens

17 eveal in detail the spatial loss of cochlear neurosensory cells, providing new insights into the path

18 nia (22%), infection (13%), stomatitis (9%), neurosensory changes (7%), myalgia (7%), and diarrhea (7

19 Nonhematologic toxicity included neurosensory changes in 21% of patients (severe in 3%) a

20 ssociated with BCVA gains, some IRC-mediated neurosensory damage remained permanent.

21 the type of autosomal recessive nonsyndromic neurosensory deafness known as "DFNB1." Studies indicate

22 tients consist of hypopigmentation, cochlear neurosensory deafness, and enteric aganglionosis.

23 Recognition of these early neurosensory defects would enable a better understanding

24 val between the procedure that resulted in a neurosensory deficiency and the LC, qualifications of th

25 ng the surgical procedure that resulted in a neurosensory deficiency in 73 LCs (79.3%), and the DI wa

26 LCs for DIs that result in a neurosensory deficiency pose a legal risk to the practit

27 and analyzes a large series of patients with neurosensory deficiency related to the placement of dent

28 aling), and treatment after the diagnosis of neurosensory deficiency were recorded and analyzed.

29 (AS), a progressive disease characterized by neurosensory deficits and by metabolic defects including

30 Alms1-disrupted mice, which recapitulate the neurosensory deficits of human Alstrom Syndrome, cochlea

31 hyperinsulinemia, chronic hyperglycemia and neurosensory deficits.

32 lstrom syndrome, a disorder characterised by neurosensory degeneration, metabolic defects and cardiom

33 l for inner ear neurosensory development and neurosensory-dependent morphogenesis.

34 nal pigment epithelium detachment (PED), and neurosensory detachment (NSD).

35 The assessment of neurosensory detachment as well as other ultrastructural

36 kness, subretinal fluid volume and height of neurosensory detachment before and after treatment with

37 Patients with alternative etiologies for neurosensory detachment or pigment epitheliopathy were e

38 al coherence tomography (OCT) showed macular neurosensory detachment with central highly reflective s

39 to have developed at the margins of chronic neurosensory detachment.

40 nvascularized, serous PEDs with an overlying neurosensory detachment.

41 trates that miRNAs are crucial for inner ear neurosensory development and neurosensory-dependent morp

42 of Sox2, we explored the function of Sox2 in neurosensory development in a model with limited cell ty

43 The role of Sox2 in neurosensory development is not yet fully understood.

44 onstrated a critical role for this ligand in neurosensory development of the vertebrate inner ear, an

45 nd changes the current paradigm of inner ear neurosensory development.

46 egies for managing children with physical or neurosensory disability.

47 r proteins, two of which have been linked to neurosensory disease phenotypes.

48 enes for hereditary and/or sporadic forms of neurosensory disorders in humans.

49 ous disease susceptibility, blood disorders, neurosensory disorders, drug addiction and toxicity.

50 seases, atherosclerosis, blood disorders and neurosensory disorders.

51 ers, tub and tulp1, have been shown to cause neurosensory disorders.

52 y sinus or nasal fossa, sinus lift sequelae, neurosensory disturbances, injuries to adjacent teeth, t

53 hatic duct outgrowth, and in the prospective neurosensory domain of the otic epithelium as morphogene

54 luorescence imaging can reveal the extent of neurosensory dysfunction in gyrate atrophy patients.

55 These neurosensory elements are innervated by a sound-activate

56 teins at the basal and apical aspects of the neurosensory epithelia suggests the existence of regulat

57 he aminoglycoside gentamicin, the vestibular neurosensory epithelia undergo degeneration and then lim

58 We demonstrate Cdh23 expression in the neurosensory epithelium and show that during early hair-

59 tion task following surgical ablation of the neurosensory epithelium in one labyrinth.

60 ing neuroblasts, increased cell death in the neurosensory epithelium, and significantly reduced the C

61 SERPINB6A is present in the neurosensory epithelium, lateral wall, and spiral limbus

62 ntal canal morphogenesis and another set for neurosensory formation of the horizontal crista and asso

63 ontribution of the channel in either cell to neurosensory function remains to be elucidated.

64 Congenital hearing loss is the most common neurosensory handicap in neonates.

65 ch are critical for the survival of auditory neurosensory HCs.

66 liferate, and HC death leads to irreversible neurosensory hearing loss and balance impairment.

67 odel for the study of conductive rather than neurosensory hearing loss that has direct relevance to h

68 Neurosensory hearing loss, ataxia, spastic paraparesis,

69 by mice also suffer retinal degeneration and neurosensory hearing loss.

70 s as retinitis pigmentosa (RP) and bilateral neurosensory hearing loss.

71 (280 [58.7%]) did not have increased risk of neurosensory impairment (risk difference [RD], 0.01; 95%

72 an increased risk of the primary outcomes of neurosensory impairment (risk ratio, 0.95; 95% confidenc

73 Serious respiratory morbidity, neurosensory impairment at 18 to 21 months of age, and a

74 t associated with increased risk of combined neurosensory impairment at 4.5 years but was associated

75 rious respiratory morbidity, 257 infants had neurosensory impairment, and 12 infants died after disch

76 to isolate cognitive outcomes from motor and neurosensory impairment, and the strategy for dealing wi

77 The primary outcome was neurosensory impairment, defined as poor performance in

78 tcome was survival without cerebral palsy or neurosensory impairment, or a Bayley III developmental s

79 For serious neurosensory impairment, the AOR and AUC at 40 weeks' PM

80 Seventeen percent of patients developed neurosensory impairment.

81 were restricted to the participants without neurosensory impairment.

82 They had higher rates of neurosensory impairments (10 percent vs. <1 percent, P<0

83 igher proportion of premature adults without neurosensory impairments identified themselves as nonhet

84 After exclusion of individuals with neurosensory impairments, differences in employment, soc

85 ts pliable, easily mobilized skin, preserves neurosensory innervation, and facilitates early hand mob

86 nt, schooling position, and variation in the neurosensory lateral line.

87 aining clones, we found evidence of a shared neurosensory lineage in the middle ear.

88 Here, we provide the neurosensory lineage reconstruction of a complex sensory

89 see these patients to be well versed in the neurosensory manifestations so that appropriate diagnosi

90 n acid nociception, but its possible role in neurosensory mechanotransduction is disputed.

91 e techniques provide an opportunity to probe neurosensory mechanotransduction with a defined substrat

92 it also displayed a good ability to predict neurosensory morbidity at 18 to 21 months.

93 e, and a composite outcome of respiratory or neurosensory morbidity or death after discharge.

94 In response to hypoxic challenge neurosensory odontoblasts express hypoxia-inducible fact

95 controlled microcirculation of craniofacial neurosensory organs is an essential evolutionary adaptat

96 in hair cell- and supporting cell-containing neurosensory organs is conserved in the zebrafish, in wh

97 ld influence the development and function in neurosensory organs, and contribute to functional altera

98 in the development and function of ciliated neurosensory organs.

99 se in the CSF, the tonotopic distribution of neurosensory pathologies in the cochlea, and the long-te

100 nces in the vulnerability of biochemical and neurosensory pathways of the visual signal transduction

101 The relationship between the neurosensory photoreceptors and the adjacent retinal pig

102 ells became concentrated to generate a large neurosensory precursor population.

103 of hair cells, possibly derived from common neurosensory precursors.

104 achable part of the brain' for investigating neurosensory processes.

105 inal cord drives pathological alterations in neurosensory processing and shapes functional outcome ea

106 ATP is involved in neurosensory processing, including nociceptive transduct

107 nt cell types arise from a common sensory or neurosensory progenitor, although little is known about

108 neration of the two cell types from a common neurosensory progenitor.

109 Here, we identified a population of common neurosensory progenitors in the zebrafish inner ear and

110 ed neurotoxicity was low grade and primarily neurosensory rather than neuromotor.

111 The ability of neurosensory release of serotonin to control cellular st

112 link between eosinophil-mediated events and neurosensory responses following exposure to some contac

113 modal reorganization is less detrimental for neurosensory restoration than previously thought.

114 nical tool for an individualized approach to neurosensory restoration with cochlear implants.

115 l implants) has led to remarkable success in neurosensory restoration, particularly in the auditory s

116 s (440 femtomoles/mg protein), lowest in the neurosensory retina (14 femtomoles/mg protein), and inte

117 ght damage-induced transcript changes within neurosensory retina (NSR) and isolated retinal pigment e

118 tinal peptide (VIP), adult rat RPE cells, or neurosensory retina (NSR) for 5 days.

119 rsus age-matched controls in RPE/choroid and neurosensory retina (NSR), which corresponded to hyperme

120 This study suggests that rat neurosensory retina (R28) cells are more sensitive than

121 edly upregulated (>20-fold, P < 0.01) in the neurosensory retina 30 minutes postoperatively and maint

122 eneralized recovery with preservation of the neurosensory retina 7 weeks after PDT.

123 etinal laser photocoagulation can damage the neurosensory retina and cause iatrogenic visual impairme

124 The neurosensory retina and retinal pigment epithelium (RPE)

125 roborated ERalpha staining of a young female neurosensory retina and RPE.

126 Destruction of neurosensory retina and visual pathways after accidental

127 ented epithelium-choriocapillaris, iris, and neurosensory retina are predominately of the alpha2A sub

128 s characterized by the schitic separation of neurosensory retina between outer plexiform and outer nu

129 gical level, a reduction in thickness of the neurosensory retina due to shortening of the rod outer a

130 g the proteome of the macular and peripheral neurosensory retina during four progressive stages of AM

131 in the macular and peripheral regions of the neurosensory retina from donors at different stages of A

132 EAMs can transduce and rescue cells from the neurosensory retina in vivo.

133 For example, the architecture of the neurosensory retina is a highly organized structure with

134 isual cycle for cone photopigment within the neurosensory retina may contribute to their favorable co

135 schitic or cavitated lamellar separation of neurosensory retina on spectral-domain optical coherence

136 C3 (>5-fold) and CFB (>30-fold) genes in the neurosensory retina was also significantly upregulated (

137 The neurosensory retina was removed from one globe of the pa

138 mRNA and protein were present in the RPE and neurosensory retina whereas the Wilson mRNA and protein

139 epithelium (RPE) in the treated area, intact neurosensory retina, and reperfusion of the choriocapill

140 cataract, minute crystalline deposits in the neurosensory retina, and retinal detachment.

141 eal contour, separation of the layers of the neurosensory retina, and the absence of full-thickness m

142 Both IP-10 and eotaxin were expressed in the neurosensory retina, but there was no detectable differe

143 tent in retinal pigment epithelium (RPE) and neurosensory retina, including a 95% reduction in retiny

144 s thickness/volume measurements of ICS, ONL, neurosensory retina, pigment epithelial detachments (PED

145 racterized by an elevated lesion beneath the neurosensory retina, resembling an egg yolk.

146 c membranes on the epiretinal surface of the neurosensory retina, resulting in a traction retinal det

147 nd colobomatouslike excavation involving the neurosensory retina, retinal pigment epithelium, and cho

148 Thickness and volume were calculated for neurosensory retina, subretinal fluid (SRF), subretinal

149 delineated by these boundaries included the neurosensory retina, subretinal fluid, subretinal tissue

150 isease of the retinal pigment epithelium and neurosensory retina, we conducted a genomewide scan in 3

151 ented epithelium-choriocapillaris, iris, and neurosensory retina.

152 ar region of the retina and splitting of the neurosensory retina.

153 induced by secondary formation of IRC in the neurosensory retina.

154 etween the posterior surface of the lens and neurosensory retina.

155 phy, with minimal pigment migration into the neurosensory retina.

156 l pigment epithelial cells (ARPE-19) and rat neurosensory retinal cells (R28) were grown in tissue cu

157 Diabetic macular edema (DME) with neurosensory retinal detachment (NSD) remains an importa

158 led clinical findings of bilateral subfoveal neurosensory retinal detachment associated with MEK inhi

159 pt the ophthalmologist to consider subfoveal neurosensory retinal detachment.

160 MEK inhibitors developed bilateral subfoveal neurosensory retinal detachment.

161 We report on a series of bilateral subfoveal neurosensory retinal detachments in patients with metast

162 Although neurosensory retinal edema and SRF showed an early reduc

163 Overall neurosensory retinal thickening in eyes with AMD versus

164 The neurosensory sequelae can be difficult to diagnose with

165 city, cerebrovascular injury, neurologic and neurosensory sequelae, and subsequent neoplasms.

166 t risk for both early and late neurologic or neurosensory sequelae.

167 traumatic brain injury with attention to the neurosensory sequelae.

168 evidence for such a response in an elemental neurosensory structure, human dental pulp, following chr

169 igm for understanding angiogenic capacity of neurosensory structures and aberrations of this response

170 Despite extreme plasticity of neurosensory structures, the capacity to reconcile barri

171 This direct effect of the neurosensory system on keratinocyte nerve growth factor

172 hared evolutionary homology of teeth and the neurosensory system, and the archival nature of dentine

173 training reduces the impaired performance on neurosensory tests of tactile function that is commonly

174 rrhea, vomiting, and mucositis), and grade 3 neurosensory toxicity 3.9%.

175 Because of the unusual neurosensory toxicity of oxaliplatin, detailed neurologi

176 3 stomatitis were less frequent, and severe neurosensory toxicity was more frequent in those who rec

177 Grade 3 neurosensory toxicity was noted in 8.2% of patients rece

178 increased hematologic toxicity and decreased neurosensory toxicity.