ライフサイエンスコーパス: neurosyphilis

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1 d immune impairment may increase the risk of neurosyphilis.

2 um RPR titer helps predict the likelihood of neurosyphilis.

3 isk for neuroinvasion and, thus, at risk for neurosyphilis.

4 Sixty-five subjects (20.1%) had neurosyphilis.

5 he cerebrospinal fluid to exclude associated neurosyphilis.

6 ting spinal fluids of patients with possible neurosyphilis.

7 Six (5%) had late neurosyphilis.

8 L tests, 2 patients were diagnosed as having neurosyphilis.

9 this test in the diagnosis and treatment of neurosyphilis.

10 eatment and may be more likely to experience neurosyphilis.

11 PR) titer > or =1 : 32 increased the odds of neurosyphilis 10.85-fold in human immunodeficiency virus

12 Reports of neurosyphilis and invasion of cerebrospinal fluid by Tre

13 urological signs or symptoms consistent with neurosyphilis, and asymptomatic persons whose serologica

14 None had clinically evident neurosyphilis, and the rate of detection of T. pallidum

15 We tested the hypothesis that neurosyphilis causes cognitive impairment in HIV by ampl

16 of 42 patients infected with type 14d/f had neurosyphilis compared with 10 (24%) of 41 patients infe

17 dition to a reactive CSF-VDRL, the number of neurosyphilis diagnoses would have increased from 47 to

18 (CSF-TPHA) titer of >/=1:640 is specific for neurosyphilis diagnosis.

19 te the epidemiology and clinical spectrum of neurosyphilis in a population with high rates of coexist

20 sults, no evidence was found for undiagnosed neurosyphilis in HIV-infected patients.

21 microL conferred 3.10-fold increased odds of neurosyphilis in HIV-infected subjects.

22 Early syphilis increased the odds of neurosyphilis in univariate but not multivariate analyse

23 cerebrospinal fluid examination to diagnose neurosyphilis is recommended in persons diagnosed with t

24 Neurosyphilis may augment HIV-associated CNS inflammatio

25 Establishing the diagnosis of neurosyphilis may be particularly difficult in human imm

26 biological characteristics of patients with neurosyphilis (NS), and we assessed the diagnostic value

27 ntreponemal (VDRL) titers (median, 1:128) at neurosyphilis presentation.

28 ular syphilis should be treated according to neurosyphilis regimens and should receive cerebrospinal

29 ht (33%) presented with an early symptomatic neurosyphilis syndrome.

30 es were identified more frequently than late neurosyphilis syndromes.

31 and HIV RNA (727 vs. 50 c/mL) were higher in neurosyphilis than in uncomplicated syphilis (P </= .001

32 Neurosyphilis was defined as (i) a reactive CSF Venereal

33 Neurosyphilis was defined as a cerebrospinal fluid (CSF)

34 Neurosyphilis was defined as detection of Treponema pall

35 Neurosyphilis was defined by a newly reactive cerebrospi

36 to the findings from the preantibiotic era, neurosyphilis was identified in young patients most ofte

37 Similar results were obtained when neurosyphilis was more stringently defined as a reactive

38 Neurosyphilis was not related to low MAT scores.

39 cerebrospinal fluid VDRL; 117 patients with neurosyphilis were identified.

40 0 may be useful in identifying patients with neurosyphilis when CSF-VDRL is nonreactive.

41 of the pathogenesis of JHR in patients with neurosyphilis who develop transient neurologic signs.

42 bjects (n=326) with syphilis but no previous neurosyphilis who met 1993 Centers for Disease Control a

43 Ocular syphilis should be treated like neurosyphilis, with daily intravenous or intramuscular p

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