ライフサイエンスコーパス: neurotransmitter receptor

1 on the activation dynamics of this important neurotransmitter receptor.

2 sm of regulation for this important class of neurotransmitter receptor.

3 ding proteins and the rat GluR2 AMPA subtype neurotransmitter receptor.

4 to contain a binding domain for a different neurotransmitter receptor.

5 ve zone apposed to a postsynaptic cluster of neurotransmitter receptors.

6 cytosis model for the dendritic targeting of neurotransmitter receptors.

7 ease sites opposite clusters of postsynaptic neurotransmitter receptors.

8 ls are important determinants of function in neurotransmitter receptors.

9 roteins essential for synaptic clustering of neurotransmitter receptors.

10 of which are known to inhibit endocytosis of neurotransmitter receptors.

11 interact with ion channels and cytokine and neurotransmitter receptors.

12 macological conditions that block inhibitory neurotransmitter receptors.

13 closer to the photoreceptors than the other neurotransmitter receptors.

14 terconnections and dominant actions for fast neurotransmitter receptors.

15 n be further activated via G-protein-coupled neurotransmitter receptors.

16 ling of synaptic vesicles and trafficking of neurotransmitter receptors.

17 investigations of the mechanism of action of neurotransmitter receptors.

18 synaptic vesicular release with postsynaptic neurotransmitter receptors.

19 ich thus acquired functional Torpedo and rat neurotransmitter receptors.

20 nal growth and differentiation, and modulate neurotransmitter receptors.

21 of synaptic strength involve the turnover of neurotransmitter receptors.

22 aling complexes around specific postsynaptic neurotransmitter receptors.

23 c terminal and the activation of presynaptic neurotransmitter receptors.

24 important proteins, such as ion channels and neurotransmitter receptors.

25 nd localization of specific ion channels and neurotransmitter receptors.

26 ction pathway through a variety of monoamine neurotransmitter receptors.

27 d active zones are aligned with postsynaptic neurotransmitter receptors.

28 ase associated with these widely distributed neurotransmitter receptors.

29 release and the number or responsiveness of neurotransmitter receptors.

30 vergence site for mAChR activation and other neurotransmitter receptors.

31 nt enhancement of responses mediated through neurotransmitter receptors.

32 ugh high-affinity interactions with specific neurotransmitter receptors.

33 sing the efficacy of toxins directed against neurotransmitter receptors.

34 tes the activity of various ion channels and neurotransmitter receptors.

35 is precisely juxtaposed to the postsynaptic neurotransmitter receptors.

36 cally released transmitters and postsynaptic neurotransmitter receptors.

37 and postsynaptic specialization and recruit neurotransmitter receptors.

38 loop ligand-gated ion channel superfamily of neurotransmitter receptors.

39 ze neural function through the modulation of neurotransmitter receptor abundance, ion channel density

40 by the regulation of ion channel expression, neurotransmitter receptor abundance, or modulation of pr

41 junction led to an influential model of how neurotransmitter receptors accumulate in the postsynapti

42 ar Ca2+ stores and Ca2+-permeable ionotropic neurotransmitter receptors, activate SK channels.

43 kinetic investigations of the membrane-bound neurotransmitter receptor activated by glycine.

44 o address this challenge and monitor in situ neurotransmitter receptor activation.

45 Positive and negative regulation of neurotransmitter receptor aggregation on the postsynapti

46 ntraventricular injection of many individual neurotransmitter receptor agonists have been well docume

47 tter biosynthetic enzymes, transporters, and neurotransmitter receptors, allows functional characteri

48 elays the appearance of neuronal inclusions, neurotransmitter receptor alterations and onset of sympt

49 a(2+) entry takes place through postsynaptic neurotransmitter receptors, although postsynaptic calciu

50 reases the functional plasticity of this key neurotransmitter receptor and is thought to contribute t

51 Several representative examples of neurotransmitter receptors and ABC transporters with the

52 Palmitoylation of neurotransmitter receptors and associated scaffold prote

53 ized multiprotein structures associated with neurotransmitter receptors and cell-adhesion proteins fu

54 n shown to regulate the localization of both neurotransmitter receptors and downstream signaling mach

55 rsely, Foxo1 increases expression of several neurotransmitter receptors and fails to regulate target

56 n deletions decreased the synaptic levels of neurotransmitter receptors and had no effect on presynap

57 nd interrogate the relative contributions of neurotransmitter receptors and intracellular signaling c

58 Membrane proteins identified included neurotransmitter receptors and ion channels implicated i

59 gulation of synaptic phosphoproteins such as neurotransmitter receptors and ion channels implicated i

60 alization, clustering, and immobilization of neurotransmitter receptors and ion channels play importa

61 Q3/Q5 and was selective over a wide range of neurotransmitter receptors and ion channels such as volt

62 ) receptor, good selectivities against other neurotransmitter receptors and ion channels, acceptable

63 Numerous neurotransmitter receptors and ion channels, including g

64 LN(v)s, we blocked or activated a variety of neurotransmitter receptors and measured effects on netwo

65 uding neurotransmitter synthesizing enzymes, neurotransmitter receptors and neuropeptides, we show th

66 also regulating the expression of ionotropic neurotransmitter receptors and putative stretch receptor

67 e the expression of numerous genes including neurotransmitter receptors and scaffolding proteins.

68 te to spine-specific compartmentalization of neurotransmitter receptors and signaling molecules and t

69 N-methyl-D-aspartate (NMDA) neurotransmitter receptors and the postsynaptic density-

70 IRKs) provide a common link between numerous neurotransmitter receptors and the regulation of synapti

71 ends on the maintenance of a high density of neurotransmitter receptors and their associated scaffold

72 ng technique that can be used to investigate neurotransmitter receptors and transporters directly by

73 vities displayed by methaqualone at numerous neurotransmitter receptors and transporters in an elabor

74 ighly selective for nAChRs over the other 45 neurotransmitter receptors and transporters tested.

75 characterizing alterations in the levels of neurotransmitter receptors and transporters that are ass

76 w also that these postmortem membranes carry neurotransmitter receptors and voltage-operated channels

77 ion levels of several transcription factors, neurotransmitter receptors, and intracellular signaling

78 pregnanolone, modulate ionotropic amino acid neurotransmitter receptors, and may function as endogeno

79 tiple exons in transcripts for ion channels, neurotransmitter receptors, and other synaptic proteins.

80 nse to local activation of growth factor and neurotransmitter receptors, and preferentially localize

81 derived neurons expressed neurotransmitters, neurotransmitter receptors, and synaptic proteins in vit

82 nd inverse agonists, dimerization with other neurotransmitter receptors, and the main presynaptic and

83 ion, activity is induced and modulated using neurotransmitter receptor antagonists and is measured us

84 The application of neurotransmitter receptor antagonists and putative gap j

85 d on the proper assembly of the postsynaptic neurotransmitter receptor apparatus.

86 ostsynaptic membrane of excitatory synapses, neurotransmitter receptors are attached to large protein

87 Neurotransmitter receptors are central to communication

88 Calcium-permeable neurotransmitter receptors are concentrated into structu

89 These neurotransmitter receptors are expressed on both presyna

90 Assembly and trafficking of neurotransmitter receptors are processes contingent upon

91 either intracellular signaling molecules or neurotransmitter receptors are required for barrel forma

92 ptors (GABAAR), the brain's major inhibitory neurotransmitter receptors, are the targets for many gen

93 from mature neuronal membranes that contain neurotransmitter receptors as a sensitive detector, we f

94 Using CHO cells expressing high-affinity neurotransmitter receptors as biosensors, we show that g

95 , antioxidant or detoxification enzymes, and neurotransmitter receptors, as well as structural and ho

96 euron that helps to concentrate and organize neurotransmitter receptors at a chemical synapse.

97 n applying these analytical tools to glycine neurotransmitter receptors at inhibitory synapses, we fi

98 Aggregation of neurotransmitter receptors at pre- and postsynaptic stru

99 The surface density of neurotransmitter receptors at synapses is a key determin

100 The regulated trafficking of neurotransmitter receptors at synapses is critical for s

101 ission is ensured by a high concentration of neurotransmitter receptors at the postsynaptic membrane.

102 strength can occur by altering the amount of neurotransmitter receptors at the synapse or by altering

103 oride channels, synapse-associated proteins, neurotransmitter receptors, axon and dendrite pathfinder

104 ystem in which to analyse the segregation of neurotransmitter receptors, because muscle cells receive

105 proteins, vesicular trafficking proteins and neurotransmitter receptors becomes apparent.

106 id(A) (GABA(A)) receptor, a major inhibitory neurotransmitter receptor, belongs to a family of membra

107 multielectrode array, both with and without neurotransmitter receptor blockers or allosteric modulat

108 ithin transmembrane 4-alpha-helix bundles of neurotransmitter receptors, but confirmation of binding

109 ferential sorting, delivery and retention of neurotransmitter receptors, but the mechanisms underlyin

110 ndrocyte precursor cells (OPCs) express most neurotransmitter receptors, but their function remains u

111 ilepsy-linked gamma-aminobutyric acid (GABA) neurotransmitter receptor by phenobarbital is presented.

112 Acid-sensing ion channels (ASICs) act as neurotransmitter receptors by responding to synaptic cle

113 Activation of certain neurotransmitter receptors can regulate Abeta metabolism

114 s, suggests that NMDA channels, unlike other neurotransmitter receptors, cannot open unless all bindi

115 ns acted in the synapse (34 of 40, including neurotransmitter receptors, cation channels, adhesion an

116 s mitochondria, synaptic vesicle precursors, neurotransmitter receptors, cell signaling and adhesion

117 s a mosaic of specialized microdomains where neurotransmitter receptors cluster in register with the

118 ynaptic organization by their involvement in neurotransmitter receptor clustering and signaling compl

119 However, the mechanisms directing neurotransmitter-receptor clustering and maintenance are

120 sites, in precise apposition to postsynaptic neurotransmitter receptor clusters; however, the molecul

121 Variations in synapse morphology, neurotransmitter receptor composition, and receptor dist

122 Diffuse extrasynaptic neurotransmitter receptors constitute an abundant pool o

123 acological dissection approach to identify a neurotransmitter receptor defect in Egr3(-/-) mice that

124 was used to determine whether alterations in neurotransmitter receptor densities occurred before over

125 us and a shift of the balance of hippocampal neurotransmitter receptor densities toward inhibition (p

126 on the mechanisms that maintain and regulate neurotransmitter receptor density at postsynaptic sites.

127 Neurotransmitter receptor density is a major variable in

128 tent with the hypothesis that alterations in neurotransmitter receptor density precede cell loss and

129 we find that none of the previously reported neurotransmitter receptors detected by antibodies alone

130 esult may reflect greater PTK recruitment by neurotransmitter receptors, distinct from the NMDA-R, th

131 Therefore, neurotransmitter receptor dynamism associated with rapid

132 agents act on voltage-gated ion channels and neurotransmitter receptors, enabling control of neuronal

133 Expression of neurotransmitter receptors encoded by the nicotinic acet

134 ia, including defects in neuronal migration, neurotransmitter receptor expression and myelination.

135 ural fate determination and specification of neurotransmitter receptor expression.

136 itiation and maintenance of the postsynaptic neurotransmitter-receptor field are important steps duri

137 he membrane-spanning domains of the paradigm neurotransmitter receptor for acetylcholine (AChR) displ

138 ression of a gain-of-function variant of the neurotransmitter receptor for glycine (GlyR) that is fou

139 he mechanisms underlying the activity of the neurotransmitter receptors for glutamate.

140 late to investigate the gating of eukaryotic neurotransmitter receptors, for which intermediate state

141 tromer in supporting fast, local delivery of neurotransmitter receptors from endosomes to the dendrit

142 own previously that cell membranes, carrying neurotransmitter receptors from the human postmortem bra

143 Neurotransmitter receptor function can be influenced by

144 sophila to human, inhibition of postsynaptic neurotransmitter receptor function causes a homeostatic

145 An unresolved problem in understanding neurotransmitter receptor function concerns the mechanis

146 rom fly to human, a decrease in postsynaptic neurotransmitter receptor function elicits a homeostatic

147 studies of the Cys loop family of ionotropic neurotransmitter receptors (GABA, nACh, 5-HT(3), and gly

148 Honokiol (HNK), a phenolic neurotransmitter receptor (gamma-aminobutyric acid type

149 late the transcription of a widely expressed neurotransmitter receptor gene by binding a common eleme

150 Interestingly, KLF11 regulates neurotransmitter receptor gene expression in differentia

151 l(1)sc and controls expression of a peptide neurotransmitter receptor gene.

152 data demonstrate comprehensive screening of neurotransmitter receptor genes in a controlled neuronal

153 we detected differential methylation in two neurotransmitter receptor genes, the gamma-Aminobutyric

154 eptide receptor genes, and 31 small-molecule neurotransmitter receptor genes.

155 arch investigating individual differences in neurotransmitter receptor genotypes has highlighted the

156 ing the cell adhesion molecule L1/NgCAM, the neurotransmitter receptor GluA2, and beta-APP.

157 ones signal by stimulating G protein-coupled neurotransmitter receptors (GPCRs), which activate G pro

158 This neurotransmitter receptor has been shown previously to b

159 ancement of asynchronous EPSCs by a specific neurotransmitter receptor has not been reported previous

160 mission in the central nervous system, these neurotransmitter receptors have been shown to influence

161 on the trafficking of potassium channels and neurotransmitter receptors have revealed unexpected comp

162 ivation reaction predict that, after binding neurotransmitter, receptors hesitate for approximately 4

163 ermore, recent studies demonstrate that some neurotransmitter receptor heteromers can exert an effect

164 Neurotransmitter receptor heteromers can function as pro

165 The existence of neurotransmitter receptor heteromers is becoming broadly

166 ndamental properties of these key inhibitory neurotransmitter receptors; however, the ratio of alpha1

167 , including (i) acetylcholine receptors, the neurotransmitter receptor, (ii) muscle-specific kinase (

168 annels (ASICs), a small family of excitatory neurotransmitter receptors implicated in pain and neuroi

169 c, titratable Arg analog, canavanine, into a neurotransmitter receptor in a living cell, utilizing a

170 et al. document pro-survival functions of a neurotransmitter receptor in glioma progenitor cells, wi

171 AMPA receptor (AMPA-R) is a major excitatory neurotransmitter receptor in the brain.

172 coding for subunits of the major inhibitory neurotransmitter receptor in the central nervous system,

173 GABA(B)R2 subunits suggests a novel role for neurotransmitter receptors in controlling gene expressio

174 e GABA(A) receptors are the major inhibitory neurotransmitter receptors in mammalian brain.

175 on, and regulate the selective expression of neurotransmitter receptors in neurons and at the neuromu

176 The spatiotemporal organization of neurotransmitter receptors in postsynaptic membranes is

177 quired for the physiological organization of neurotransmitter receptors in postsynaptic specializatio

178 gating and channel properties of ionotropic neurotransmitter receptors in some hereditary epilepsies

179 , and it is highly desirable to know whether neurotransmitter receptors in such tissues are still fun

180 Psychotropic agents act on a variety of neurotransmitter receptors in the brain-gut regulatory p

181 ate receptors (AMPARs), the major excitatory neurotransmitter receptors in the brain.

182 AMPA receptors are the major excitatory neurotransmitter receptors in the central nervous system

183 ation regulates key functional properties of neurotransmitter receptors in the central nervous system

184 nted research on GABA(A)Rs, a major class of neurotransmitter receptors in the central nervous system

185 tors (NMDARs) are the predominant excitatory neurotransmitter receptors in the mammalian brain.

186 cid (AMPA) receptors are the main excitatory neurotransmitter receptors in the mammalian central nerv

187 ep in synapse formation is the clustering of neurotransmitter receptors in the postsynaptic membrane,

188 of synapse development is the clustering of neurotransmitter receptors in the postsynaptic membrane.

189 cause they are the most prevalent excitatory neurotransmitter receptors in the vertebrate central ner

190 receptors are the most prevalent excitatory neurotransmitter receptors in the vertebrate central ner

191 stems where they can be modulated by several neurotransmitter receptors including histamine H(1) rece

192 ies, and broad screening toward other common neurotransmitter receptors indicated that compound 43 is

193 ity of CTZ to interact with various types of neurotransmitter receptors indicates that the drug has m

194 Deregulated neuroendocrine and neurotransmitter receptor interactions were observed in

195 ral nervous system by coupling extracellular neurotransmitter-receptor interactions to ion channel co

196 The delivery of neurotransmitter receptors into synapses is essential fo

197 The delivery of neurotransmitter receptors into the synaptic membrane is

198 ting neuronal response through regulation of neurotransmitter receptor intraneuronal fate, we hypothe

199 tion of complex sugars to adhesion proteins, neurotransmitter receptors, ion channels and secreted tr

200 10 microM) over other P2 receptors and other neurotransmitter receptors, ion channels, and enzymes.

201 rs), rhodopsin, G-protein-coupled receptors, neurotransmitter receptors, ion channels, and so on from

202 ociated with the induction of genes encoding neurotransmitter receptors, ion channels, growth factors

203 The subunit composition of postsynaptic neurotransmitter receptors is a key determinant of synap

204 Endocytic trafficking of neurotransmitter receptors is critical to neuronal signa

205 Regulation of cell surface expression of neurotransmitter receptors is crucial for determining sy

206 Synaptic clustering of neurotransmitter receptors is crucial for efficient sign

207 Regulation of synaptic neurotransmitter receptors is currently thought to be a

208 Elucidating subunit stoichiometry of neurotransmitter receptors is preferably carried out in

209 Heterogeneity in the composition of neurotransmitter receptors is thought to provide functio

210 ticity in which perturbation to postsynaptic neurotransmitter receptors leads to a retrograde enhance

211 Anatomical visualization of neurotransmitter receptor localization is facilitated by

212 Candidate genes include several neurotransmitter receptor loci, particularly monoamine r

213 ogic phenotypes that arise from mutations in neurotransmitter receptors (lurcher mice) and ion channe

214 Altered levels of neurotransmitter receptors may disrupt neuronal function

215 ed in memory storage or retrieval, and which neurotransmitter receptor mechanisms serve its function.

216 Ionotropic neurotransmitter receptors mediate fast synaptic transmi

217 Ionotropic neurotransmitter receptors mediate fast synaptic transmi

218 Ionotropic neurotransmitter receptors mediate rapid synaptic transm

219 fic proteins, generate action potentials and neurotransmitter receptor-mediated currents.

220 esynaptic patterns to postsynaptic cells via neurotransmitter receptor-mediated intracellular signals

221 rmones in rat and human, respectively, alter neurotransmitter receptor-mediated responses in the brai

222 ate receptors are the predominant excitatory neurotransmitter receptors mediating fast synaptic trans

223 found that a wide variety of neuromodulatory neurotransmitter receptors (metabotropic glutamate recep

224 Tonic activation of metabotropic neurotransmitter receptors (mGluRs, alpha1 adrenergic re

225 Neurotransmitter receptors need to be positioned in high

226 organize a multitude of receptors including neurotransmitter receptors (NMDA and AMPA receptors), si

227 now possible to determine the properties of neurotransmitter receptors of normal and diseased human

228 very little is known about the properties of neurotransmitter receptors of the AD human brain.

229 Glutamate recognition by neurotransmitter receptors often relies on Arg residues

230 rovide a new explanation for the reliance of neurotransmitter receptors on Arg side chains and highli

231 we review the evidence for the expression of neurotransmitter receptors on microglia and the conseque

232 Neurotransmitter receptors on postsynaptic cells change

233 f synaptic connections requires alignment of neurotransmitter receptors on postsynaptic dendrites opp

234 (RGCs) is unaffected by blockade of specific neurotransmitter receptors or global activity.

235 xual behavior is by increasing production of neurotransmitter receptors or of enzymes that regulate n

236 They contain neurotransmitter receptors, organelles, and signaling sy

237 y also indicate that protons and ASICs are a neurotransmitter/receptor pair critical for amygdala-dep

238 euronal synaptic integration and appropriate neurotransmitter/receptor phenotype.

239 Neurotransmitter receptors previously implicated in C. e

240 Heteromerization of neurotransmitter receptors produces functional entities

241 and temporal stages of AD, thereby altering neurotransmitter receptor properties, disrupting membran

242 receptor stimulation, an alphav integrin/PLP/neurotransmitter receptor protein complex forms that red

243 Neurotransmitter receptor recruitment at postsynaptic sp

244 hesis that all iGluRs, and potentially other neurotransmitter receptors, rely on the cooperative bind

245 the selected binding of antipsychotics to 14 neurotransmitter receptors revealed only dopamine type 3

246 ion of a presynaptic nerve terminal with the neurotransmitter receptor-rich postsynaptic apparatus.

247 rge-scale gene expression changes, including neurotransmitter receptors, signal transduction cascades

248 We hypothesized that the mER and neurotransmitter receptor signaling pathways converge to

249 ith the postsynaptic membrane that organizes neurotransmitter receptors, signaling pathways, and regu

250 nce for three possible triggers: activity of neurotransmitter receptors, signaling through adhesion p

251 alpha, encoded by Gnas, mediates hormone and neurotransmitter receptor-stimulated cAMP generation.

252 Clustering of a neurotransmitter receptor subunit in the muscle at the n

253 plicing of pre-mRNAs encoding the inhibitory neurotransmitter receptor subunits GABA(A)Rgamma2 and Gl

254 ebalancing ion channel expression, modifying neurotransmitter receptor surface expression and traffic

255 derstanding this pharmacologically important neurotransmitter receptor system.

256 nervous system (CNS) functions by modulating neurotransmitter receptor systems in the brain.

257 eceptors to modulate "cross-talk" with other neurotransmitter receptor systems, it is notable that ab

258 t from the TGFbeta receptor family and known neurotransmitter receptor targets of antipsychotics.

259 Neurotransmitter receptors that are activated by various

260 of mGluR1 and mGluR5) are G-protein-coupled neurotransmitter receptors that are found in the perisyn

261 ype Ca(2+) channels is mediated primarily by neurotransmitter receptors that couple to pertussis toxi

262 iation is the array of neurotransmitters and neurotransmitter receptors that each neuron possesses.

263 Neurotransmitter receptors that inhibit the release of o

264 be gained by bringing control to endogenous neurotransmitter receptors that mediate synaptic transmi

265 lation of the B cell receptor (BCR) and/or a neurotransmitter receptor, the beta(2)-adrenergic recept

266 2 subunit of a human gamma-aminobutyric acid neurotransmitter receptor, the GABA(A) receptor, is link

267 was the chemical identification of the first neurotransmitter receptor, the nicotinic acetylcholine r

268 transcription and postsynaptic targeting of neurotransmitter receptors through distinct molecular fu

269 mediated through the G proteins that couple neurotransmitter receptors to adenylyl cyclase showed a

270 Here we build on previous studies of neurotransmitter receptors to analyze the modes of attac

271 ing molecules, other adhesion molecules, and neurotransmitter receptors to bring together the key com

272 Synaptogenesis requires recruitment of neurotransmitter receptors to developing postsynaptic sp

273 Precise targeting of neurotransmitter receptors to different neuronal compart

274 smission under basal conditions by targeting neurotransmitter receptors to synapses.

275 r MALS proteins in regulating recruitment of neurotransmitter receptors to the PSD.

276 tagamma combinations are capable of coupling neurotransmitter receptors to VD inhibition of N-type Ca

277 nteractions, second messenger signaling, and neurotransmitter receptor trafficking and function are a

278 at regulates synapse function by controlling neurotransmitter receptor trafficking and homeostatic sy

279 Neurotransmitter receptor trafficking during synaptic pl

280 Neurotransmitter receptor trafficking is fundamentally i

281 b (GT1b:GM1 > 4; p < 10(-4)), three regulate neurotransmitter receptor trafficking: Thorase (ATPase f

282 synaptic transmission span the modulation of neurotransmitter receptors, transporters, activation of

283 communicate with postsynaptic partners, the neurotransmitter receptor type used to receive input fro

284 th ligands for a variety of G-protein-linked neurotransmitter receptor types that have been associate

285 neuron the correct targeting of the diverse neurotransmitter receptor types to discrete synaptic reg

286 Assembly of ionotropic neurotransmitter receptors typified by acetylcholine rec

287 t, to a decrease in synaptic distribution of neurotransmitter receptors upon deletion of neuroligins.

288 ange of potential cargoes - including mRNAs, neurotransmitter receptors, vesicles and mitochondria -

289 t-ACPD, suggesting that the distribution of neurotransmitter receptors was not responsible for the r

290 reciate the function and regulation of these neurotransmitter receptors, we must understand their int

291 dies and broad screening toward other common neurotransmitter receptors were also carried out to furt

292 ediate uncoupling and endocytosis of certain neurotransmitter receptors, which are activated in a rev

293 various neural precursors express functional neurotransmitter receptors, which include G protein-coup

294 Oligodendrocytes are endowed with neurotransmitter receptors whose expression levels and p

295 eceptor ion channels (iGluRs) are excitatory neurotransmitter receptors with a unique molecular archi

296 g in the nervous system requires matching of neurotransmitter receptors with cognate neurotransmitter

297 The spatial coordination of neurotransmitter receptors with other postsynaptic signa

298 Our results show how neurotransmitter receptors with similar structures and g

299 as shallow energy traps (~3 kBT) for glycine neurotransmitter receptors, with a depth modulated by th

300 complexity and cross-talk between different neurotransmitter receptors within the same synapse or ac