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1 ) was synthesized for potential use in boron neutron capture therapy.
2 ntially could be used as delivery agents for neutron capture therapy.
3 boronated EGF to EGFR (+) gliomas for boron neutron capture therapy.
4 investigated as a potential agent for boron neutron capture therapy.
5 delivery and subsequent application in boron neutron capture therapy.
6 PA, and, subsequently, the efficacy of boron neutron capture therapy.
7 sting templates as carriers of 10B for boron neutron capture therapy.
8 , inhibition of hydrolytic enzymes and boron neutron capture therapy.
9 potential in traditional areas such as Boron Neutron Capture Therapy agents, but also as pharmacophor
11 cleotides are potential candidates for boron neutron capture therapy, antisense technology, and as to
12 ufficient amounts of 10B for effective boron neutron capture therapy ( approximately 10(8)-10(9) 10B
17 -yl] thymidine, designated N5-2OH, for boron neutron capture therapy (BNCT) of brain tumors using the
22 e 2 was also tested as a candidate for boron neutron capture therapy, but showed poor tumor retention
23 exceptional challenge, because viable boron neutron-capture therapy by this method will require the
24 s to determine whether the efficacy of boron neutron capture therapy could be enhanced by means of in
27 ylalanine has been applied in clinical boron neutron capture therapy for the treatment of high-grade
28 linked to epidermal growth factor (EGF) for neutron capture therapy in rats bearing a syngeneic epid
33 tein-based targeting strategies to the boron neutron-capture therapy of cancer poses an exceptional c
34 n these favorable in vitro and in vivo data, neutron capture therapy studies will be initiated using
36 on cluster system, previously used for boron neutron capture therapy, was modified by the addition of
37 boronated EGF as a delivery agent for boron neutron capture therapy, which is based on the capture r
38 o a novel class of boron delivery agents for neutron capture therapy, which was designated 3-carboran
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