ライフサイエンスコーパス: neutrophil extracellular trap

1 expulsion of their nuclear contents to form neutrophil extracellular traps.

2 bacterial virulence factors and formation of neutrophil extracellular traps.

3 MRSA killing by human neutrophils and within neutrophil extracellular traps.

4 ration of reactive oxygen species (ROS), and neutrophil extracellular traps.

5 ing through phagocytosis, degranulation, and neutrophil extracellular traps.

6 s after surgery, which generated luminal DNA neutrophil extracellular traps.

7 ssociated with proteases, which are known as neutrophil extracellular traps.

8 ess may be intertwined with the formation of neutrophil extracellular traps.

9 ctin-Fc was required to trigger formation of neutrophil extracellular traps.

10 in necrotic tissues and areas that displayed neutrophil extracellular traps.

11 ell line and can also block the formation of neutrophil extracellular traps.

12 ayed a marked impairment in the formation of neutrophil extracellular traps, a bactericidal mechanism

13 e in vivo was revealed with the discovery of neutrophil extracellular traps, a specialized cell death

14 Histones are the major protein components of neutrophil extracellular traps and are known to have cyt

15 of two neutrophil-derived biological agents: neutrophil extracellular traps and ectosomes.

16 t GAS from killing by neutrophils and within neutrophil extracellular traps and neutralizes LL-37 che

17 tion-sensitized' neutrophils, as well as the neutrophil extracellular traps and other products made b

18 elf-DNA (eg, released from dying cells or in neutrophil extracellular traps) and an increased express

19 tional substrate of PAD4, localize H1 within neutrophil extracellular traps, and detect autoantibodie

20 phils to capture exogenous material, extrude neutrophil extracellular traps, and kill bacteria via ca

21 ribe the new players, such as polyphosphate, neutrophil extracellular traps, and microparticles, whic

22 lm formation, decreases bacterial killing by neutrophil extracellular traps, and modulates S. pyogene

23 peptides and reactive oxygen species, escape neutrophil extracellular traps, and promote and accelera

24 ent-mediated lysis, engulfment, formation of neutrophil extracellular traps, and release of antimicro

25 binds to genomic DNA, mitochondrial DNA, and neutrophil extracellular traps, and shuttles them in the

26 is known to interfere with the formation of neutrophil extracellular traps, appears to prolong lysis

27 Neutrophil extracellular traps are networks of DNA and a

28 of type 1 IFN by pDCs, which were induced by neutrophil extracellular traps arising from the endocyto

29 report that an increase in the deployment of neutrophil extracellular traps associated with hyperglyc

30 Cholesterol crystals, neutrophil extracellular traps, atheroprone flow, and lo

31 ericidal activity and enhanced production of neutrophil extracellular traps compared with wild-type n

32 nse self-DNA released from dying cells or in neutrophil extracellular traps complexed to the antimicr

33 rovides a strategy for evasion of the host's neutrophil extracellular traps, digesting the DNA scaffo

34 DNases degrade the chromatin in neutrophil extracellular traps, enabling the bacterium t

35 This defense mechanism is reminiscent of the neutrophil extracellular traps (ETs) recently described

36 e related to their unique ability to release neutrophil extracellular traps even in the absence of pa

37 We previously demonstrated that neutrophil extracellular traps exacerbate pulmonary inju

38 Neutrophil extracellular traps expelled from suicidal ne

39 , length, myeloid cell recruitment, and more neutrophil extracellular trap formation (NETs) in WT com

40 ls, stimulated cytokine release, and induced neutrophil extracellular trap formation and myeloperoxid

41 ndependent mechanism that is associated with neutrophil extracellular trap formation and selective au

42 Neutrophil extracellular trap formation and the expressi

43 s prevented in transgenic mice with impaired neutrophil extracellular trap formation and/or neutrophi

44 which also exhibited an elevated capacity in neutrophil extracellular trap formation at baseline and

45 cient cells showed a significant increase in neutrophil extracellular trap formation but were unable

46 ession by myeloid cells, but did not require neutrophil extracellular trap formation involving peptid

47 enhanced alphaMbeta2 integrin activation and neutrophil extracellular trap formation under inflammato

48 cellular killing and were fully competent in neutrophil extracellular trap formation, a recently iden

49 ines, more severe pulmonary edema, increased neutrophil extracellular trap formation, and elevated co

50 Bacterial phagocytosis, neutrophil extracellular trap formation, and killing wer

51 asing histone-induced neutrophil congestion, neutrophil extracellular trap formation, and thrombosis

52 ectin shedding, oxidative burst, chemotaxis, neutrophil extracellular trap formation, bacterial killi

53 nce of active elastase during the process of neutrophil extracellular trap formation.

54 iated reactive oxygen species production and neutrophil extracellular trap formation.

55 to LPS stimulation, human resistin enhanced neutrophil extracellular trap formation.

56 release of neutrophil elastase--a marker of neutrophil extracellular trap formation.

57 smigration, phagocytosis, degranulation, and neutrophil extracellular trap formation.

58 ence of innate cell activation that included neutrophil extracellular trap generation and elevated su

59 trophil response, and we present evidence of neutrophil extracellular trap generation during experime

60 The discovery of neutrophil extracellular traps has yielded a conceptual

61 Functionally, neutrophil extracellular traps have been shown to induce

62 damage to fungal filaments, suggesting that neutrophil extracellular traps help to protect the epith

63 bind DNA strongly and localize to nuclei and neutrophil extracellular traps in a DNA-dependent manner

64 us on current findings of the involvement of neutrophil extracellular traps in atherogenesis and athe

65 creased expression of Cramp and formation of neutrophil extracellular traps in atherosclerotic arteri

66 +/- 122.4 ng/mL; p </= 0.05) and identified neutrophil extracellular traps in kidney and liver tissu

67 Neutrophil extracellular traps in turn serve as the scaf

68 ype strains, possibly through degradation of neutrophil extracellular traps, innate immune structures

69 ocarditis rat model, we identified layers of neutrophil extracellular traps interconnecting and entra

70 ial responses to its advantage by converting neutrophil extracellular traps into a bacterial weapon a

71 ia is known to counteract histone as well as neutrophil extracellular trap-mediated cytotoxicity agai

72 eparan sulfate proteoglycan(s) is present in neutrophil extracellular traps, modulates histone affini

73 her this causes extracellular DNA (eDNA) and neutrophil extracellular trap (NET) accumulation in the

74 The neutrophil extracellular trap (NET) consists in the extr

75 C1q was recently reported to impede neutrophil extracellular trap (NET) degradation.

76 Recent evidence suggests that enhanced neutrophil extracellular trap (NET) formation activates

77 s binding to immobilized neutrophils induced neutrophil extracellular trap (NET) formation in respons

78 ed a novel role for heme in the induction of neutrophil extracellular trap (NET) formation in SCD.

79 The role of neutrophil extracellular trap (NET) formation in the hos

80 agocytic reactive oxygen species production, neutrophil extracellular trap (NET) formation, and neutr

81 detail which neutrophil functions, including neutrophil extracellular trap (NET) formation, are invol

82 hils, including chemotaxis, phagocytosis and neutrophil extracellular trap (NET) formation.

83 s but prone to immunogenic death, leading to neutrophil extracellular trap (NET) formation.

84 tion, major surface molecule expression, and neutrophil extracellular trap (NET) formation.

85 Severe GN involves local neutrophil extracellular trap (NET) formation.

86 phagocytosis, oxidative burst capacity, and neutrophil extracellular trap (NET) generation (NETosis)

87 release of granule proteins with subsequent neutrophil extracellular trap (NET) release independent

88 bacterial and host components that included neutrophil extracellular trap (NET) structures and that

89 ce factors and allows the bacterium to avoid neutrophil extracellular trap (NET)-mediated killing.

90 (PIT), which is conceptually parallel to the neutrophil extracellular trap (NET).

91 pro-tumorigenic N2 phenotype and unprompted neutrophil extracellular traps (NET) formation.

92 Recently, neutrophil extracellular traps (NET) were implicated in

93 latelet-neutrophil complexes, a signature of neutrophil extracellular traps (NET), in the kidneys of

94 NETosis (neutrophil extracellular trap [NET] generation), a progr

95 The release of neutrophil extracellular traps (NETs [NETosis]), orchest

96 olving apoptosis and cell death by releasing neutrophil extracellular traps (NETs) (NETosis), which w

97 h of infection, neutrophils start to release neutrophil extracellular traps (NETs) against Acinetobac

98 These chromatin traps are termed neutrophil extracellular traps (NETs) and are decorated

99 Eros also contributes to the formation of neutrophil extracellular traps (NETS) and impacts on the

100 ed that Scl-1 mediates bacterial survival in neutrophil extracellular traps (NETs) and protects GAS f

101 Neutrophil extracellular traps (NETs) are an essential c

102 Antimicrobial neutrophil extracellular traps (NETs) are composed of se

103 Neutrophil extracellular traps (NETs) are critical for t

104 Neutrophil extracellular traps (NETs) are DNA structures

105 Neutrophil extracellular traps (NETs) are DNA structures

106 Neutrophil extracellular traps (NETs) are extracellular

107 Neutrophil extracellular traps (NETs) are extracellular

108 Neutrophil extracellular traps (NETs) are found abundant

109 Neutrophil extracellular traps (NETs) are implicated in

110 Neutrophil extracellular traps (NETs) are key players in

111 Neutrophil extracellular traps (NETs) are made of proces

112 Neutrophil extracellular traps (NETs) are part of the in

113 RECENT FINDINGS: Neutrophil extracellular traps (NETs) are released via a

114 Neutrophil extracellular traps (NETs) can be released in

115 The recent discovery of secreted neutrophil extracellular traps (NETs) composed of DNA an

116 Neutrophil extracellular traps (NETs) composed of DNA de

117 Neutrophil extracellular traps (NETs) constitute antimic

118 We hypothesized that neutrophil extracellular traps (NETs) contribute to lung

119 Neutrophil extracellular traps (NETs) extruded from neut

120 Neutrophil extracellular traps (NETs) facilitate the ext

121 Neutrophil extracellular traps (NETs) facilitate the ext

122 We also studied the production of neutrophil extracellular traps (NETs) from single neutro

123 Neutrophil extracellular traps (NETs) have been document

124 Recently, neutrophil extracellular traps (NETs) have been found to

125 Neutrophil extracellular traps (NETs) have been observed

126 Neutrophil extracellular traps (NETs) have been shown to

127 w that these particles induce the release of neutrophil extracellular traps (NETs) in a size-dependen

128 eration of reactive oxygen species (ROS) and neutrophil extracellular traps (NETs) in mouse and human

129 l activation and release of IL-1beta-bearing neutrophil extracellular traps (NETs) in patients with F

130 sensed microbe size and selectively released neutrophil extracellular traps (NETs) in response to lar

131 rom this patient were incapable of producing neutrophil extracellular traps (NETs) in response to ROS

132 t human neutrophils release large amounts of neutrophil extracellular traps (NETs) in the presence of

133 e most recent findings regarding the role of neutrophil extracellular traps (NETs) in thrombosis.

134 ular soluble ICs results in the formation of neutrophil extracellular traps (NETs) in tissues.

135 activated platelets induce the formation of neutrophil extracellular traps (NETs) in transfusion-rel

136 gulf CPPD crystals and form large amounts of neutrophil extracellular traps (NETs) in vitro.

137 oxygen species in the phagosome and release neutrophil extracellular traps (NETs) into their surroun

138 Release of neutrophil extracellular traps (NETs) is a significant a

139 cardiomyocytes and the possible formation of neutrophil extracellular traps (NETs) may result in chro

140 We hypothesized that neutrophil extracellular traps (NETs) mechanistically li

141 HMGB1 facilitates formation of prothrombotic neutrophil extracellular traps (NETs) mediated by RAGE,

142 In this review, we examine the evidence that neutrophil extracellular traps (NETs) play a critical ro

143 There is emerging evidence that neutrophil extracellular traps (NETs) play important rol

144 Neutrophil extracellular traps (NETs) released by PMN co

145 Neutrophil extracellular traps (NETs) represent a novel

146 Neutrophil extracellular traps (NETs) represent an impor

147 Recently, DNA-based neutrophil extracellular traps (NETs) resulting from the

148 Neutrophils cast neutrophil extracellular traps (NETs) to defend the host

149 this study, we analyzed the contribution of neutrophil extracellular traps (NETs) to the mediation o

150 e to microbial invasion, neutrophils release neutrophil extracellular traps (NETs) to trap and kill e

151 Here we report that ANCA induces neutrophil extracellular traps (NETs) via receptor-inter

152 olated polymorhonuclear granulocytes to form neutrophil extracellular traps (NETs) was determined usi

153 Moreover, increased release of neutrophil extracellular traps (NETs) was observed, whic

154 Neutrophil extracellular traps (NETs) were discovered as

155 Neutrophils release neutrophil extracellular traps (NETs) which ensnare path

156 AD)4, exocytosis of chromatin and enzymes as neutrophil extracellular traps (NETs), and death.

157 This study aimed to explore the release of neutrophil extracellular traps (NETs), associated antimi

158 We examined the relationships between CLS neutrophil extracellular traps (NETs), bacterial compone

159 rategies to eliminate pathogens they release neutrophil extracellular traps (NETs), being chromatin f

160 hils partly depends on their ability to form neutrophil extracellular traps (NETs), but the underlyin

161 n of reactive oxygen species, and release of neutrophil extracellular traps (NETs), can result in sev

162 agocytose the yeast and subsequently release neutrophil extracellular traps (NETs), complexes of DNA,

163 Neutrophil extracellular traps (NETs), consisting of nuc

164 named NETosis, characterized by formation of neutrophil extracellular traps (NETs), decondensed chrom

165 The formation of Neutrophil Extracellular Traps (NETs), has been implicat

166 leased into blood, through the generation of neutrophil extracellular traps (NETs), is procoagulant a

167 DNA release associated with the formation of neutrophil extracellular traps (NETs), known as NETosis.

168 ozyme, and lysozyme and PGRP-S colocalize in neutrophil extracellular traps (NETs), suggesting that t

169 n the intestinal lumen, which appeared to be neutrophil extracellular traps (NETs), suggesting that V

170 hly decondensed chromatin structures, termed neutrophil extracellular traps (NETs), that have been im

171 . pertussis lacking ACT induces formation of neutrophil extracellular traps (NETs), whereas wild-type

172 Human and bovine neutrophils release neutrophil extracellular traps (NETs), which are protein

173 have reported that human neutrophils release neutrophil extracellular traps (NETs), which are protein

174 A, histones, and granule proteins to produce neutrophil extracellular traps (NETs), which can trap mi

175 nflammatory stimuli and pathogens, they form neutrophil extracellular traps (NETs), which capture and

176 portant determinants of NTHI survival within neutrophil extracellular traps (NETs), which we have sho

177 Unexpectedly, LukGH promoted the release of neutrophil extracellular traps (NETs), which, in turn, e

178 orm extracellular web-like structures called neutrophil extracellular traps (NETs).

179 s strongly stimulated neutrophils to release neutrophil extracellular traps (NETs).

180 y impairs the ability of neutrophils to form neutrophil extracellular traps (NETs).

181 ild type, including their ability to degrade neutrophil extracellular traps (NETs).

182 ice of DNA and microbicidal enzymes known as neutrophil extracellular traps (NETs).

183 orting its activation through the release of neutrophil extracellular traps (NETs).

184 tangle bacteria, these structures were named neutrophil extracellular traps (NETs).

185 n coated with granule contents, thus forming neutrophil extracellular traps (NETs).

186 ymatic target HNE and DNA, is a component of neutrophil extracellular traps (NETs).

187 hly decondensed chromatin structures, termed neutrophil extracellular traps (NETs).

188 e a significant host component that includes neutrophil extracellular traps (NETs).

189 rial defenses of neonates: inability to form neutrophil extracellular traps (NETs).

190 elease their nuclear material in the form of neutrophil extracellular traps (NETs).

191 ection by releasing chromatin in the form of neutrophil extracellular traps (NETs).

192 extracellular histones, a major component of neutrophil extracellular traps (NETs).

193 in neutrophil recruitment and the release of neutrophil extracellular traps (NETs).

194 nhances bacterial survival after exposure to neutrophil extracellular traps (NETs).

195 timicrobial peptides that are referred to as neutrophil extracellular traps (NETs).

196 e agents to extrude decondensed chromatin as neutrophil extracellular traps (NETs).

197 ol crystals triggered neutrophils to release neutrophil extracellular traps (NETs).

198 polymorphonuclear leukocytes to release DNA [neutrophil extracellular traps (NETs)], thereby immobili

199 Neutrophil extracellular traps (NETs; webs of DNA coated

200 LDGs have heightened capacity to synthesize neutrophils extracellular traps (NETs), which display in

201 ne H3(CitH3) is released into the blood from neutrophil extracellular traps(NETs) in response to seve

202 nts, activation of granular constituents and neutrophil extracellular traps, neutrophils target micro

203 Neutrophil extracellular traps produced in response to a

204 f degranulation, reactive oxygen species and neutrophil extracellular trap production, and endolysoso

205 immune responses, including phagocytosis and neutrophil extracellular trap production.

206 Neutrophil extracellular traps promote and expand vegeta

207 Deimination of linker histones links neutrophil extracellular trap release with autoantibodie

208 y various mechanisms, including formation of neutrophil extracellular traps through a recently descri

209 eria are required to induce the formation of neutrophil extracellular traps through multiple activati

210 he production of reactive oxygen species and neutrophil extracellular traps, two mechanisms utilized

211 a harvested from morning saliva had released neutrophil extracellular traps (undergone NETosis) in vi

212 increase VTE in cancer patients by releasing neutrophil extracellular traps whereas monocytes may exp

213 o the nucleus to initiate DNA extrusion into neutrophil extracellular traps, which bind NE and cathep

214 Induction of DNA neutrophil extracellular traps, which was observed in GB

215 in mutant neutrophils affected formation of neutrophil extracellular traps while not influencing pha