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1 arkers (such as kidney injury molecule-1 and neutrophil gelatinase-associated lipocalin).
2 OB-4 and keratin 20), and immune modulation (neutrophil-gelatinase-associated lipocalin).
3 ry excretion of kidney injury molecule-1 and neutrophil gelatinase-associated lipocalin.
4 acid inhibited induction of c-fos, junb, and neutrophil gelatinase-associated lipocalin.
5 group (P = 0.0004), and was noninferior for neutrophil gelatinase-associated lipocalin [14.7 mug/L (
6 luble Tek/Tie2 receptor and in urinary MMP-9/neutrophil gelatinase-associated lipocalin activity afte
7 s no difference in concentrations of urinary neutrophil gelatinase-associated lipocalin after the 2 i
8 ant interaction between the concentration of neutrophil gelatinase-associated lipocalin, an establish
9 ression determined the prognostic utility of neutrophil gelatinase-associated lipocalin and creatinin
10 retion associated with AKI severity, and for neutrophil gelatinase-associated lipocalin and cystatin
15 rmine the prognostic utility of both urinary neutrophil gelatinase-associated lipocalin and varying c
16 jury biomarkers kidney injury molecule-1 and neutrophil gelatinase-associated lipocalin, and doses>/=
19 nt levels of high-mobility group protein B1, neutrophil gelatinase-associated lipocalin, and S100B we
20 cular cell adhesion molecule, periostin, and neutrophil gelatinase-associated lipocalin; and a two-fo
24 ure for serum creatinine, interleukin-6, and neutrophil gelatinase-associated lipocalin concentration
25 of known markers of renal injury, including neutrophil gelatinase-associated lipocalin, CXCL1, and C
26 ) kidney injury proteins (osteopontin [OPN], neutrophil gelatinase-associated lipocalin, cystatin C,
27 urinary levels of the iron acceptor proteins neutrophil gelatinase-associated lipocalin, hemopexin, a
28 serum levels of novel biomarkers, including neutrophil gelatinase-associated lipocalin, high-mobilit
30 sured the following five urinary biomarkers: neutrophil gelatinase-associated lipocalin, IL-18, kidne
31 al AKI, the highest tertiles of peak urinary neutrophil gelatinase-associated lipocalin, IL-18, KIM-1
32 valuation of novel urinary biomarkers (e.g., neutrophil gelatinase-associated lipocalin) in this pati
33 lipocalin and cystatin C) and a urine panel (neutrophil gelatinase-associated lipocalin, interleukin
35 IMP-1 in a ternary complex with progel B and neutrophil gelatinase-associated lipocalin is also susce
36 mporal change markers of renal injury (urine neutrophil gelatinase-associated lipocalin, kidney injur
37 6 hours after surgery) urine interleukin 18, neutrophil gelatinase-associated lipocalin, kidney injur
38 cohort study, 5 urinary biomarkers (urinary neutrophil gelatinase-associated lipocalin, kidney injur
39 , gamma-glutamyl transpeptidase, cystatin C, neutrophil gelatinase-associated lipocalin, kidney injur
40 put <0.5 mL/kg/h for >/=12 hours) and plasma neutrophil gelatinase-associated lipocalin level higher
44 kedly regulated the expression of NGAL/LCN2 (neutrophil gelatinase-associated lipocalin/lipocalin 2),
46 al perfusion and blood flow, whereas GFR and neutrophil gelatinase-associated lipocalin, monocyte che
47 n addition, we measured renal vein levels of neutrophil gelatinase-associated lipocalin, monocyte che
48 Neutrophil gelatinase-associated lipocalin, myeloperoxid
52 ns, the overall structure is most similar to neutrophil gelatinase associated lipocalin (NGAL), a pro
53 e investigated whether cystatin C (CysC) and neutrophil gelatinase-associated lipocalin (NGAL) can pr
55 label-free electrochemical immunosensor for neutrophil gelatinase-associated lipocalin (NGAL) detect
57 dy was to investigate the prognostic role of neutrophil gelatinase-associated lipocalin (NGAL) in a l
64 rochemical immunosensor for the detection of neutrophil gelatinase-associated lipocalin (NGAL) is dev
66 udy was to determine the association between neutrophil gelatinase-associated lipocalin (NGAL) levels
68 metalloproteinase (MMP)-3, MMP-8, MMP-9, and neutrophil gelatinase-associated lipocalin (NGAL) were f
69 bronectin, interleukin-8 (IL-8), biotin, and neutrophil gelatinase-associated lipocalin (NGAL) were t
70 tly, the secreted protein 24p3 (lipocalin-2, neutrophil gelatinase-associated lipocalin (NGAL)), whic
71 ntrasted the results with those obtained for neutrophil gelatinase-associated lipocalin (NGAL), a com
73 t HGF stimulates epithelial cells to express neutrophil gelatinase-associated lipocalin (Ngal), a mem
74 med to assess the prognostic value of plasma neutrophil gelatinase-associated lipocalin (NGAL), a nov
76 ti-DNA antibodies induced high expression of neutrophil gelatinase-associated lipocalin (NGAL), an ir
77 (AAG), transferrin (TF), ceruloplasmin (CP), neutrophil gelatinase-associated lipocalin (NGAL), and m
78 ized genes was further characterized, namely neutrophil gelatinase-associated lipocalin (NGAL), becau
79 dney transplant patients to evaluate urinary neutrophil gelatinase-associated lipocalin (NGAL), IL-18
82 ured urinary concentrations of microalbumin, neutrophil gelatinase-associated lipocalin (NGAL), kidne
83 s compared with urinary interleukin (IL)-18, neutrophil gelatinase-associated lipocalin (NGAL), kidne
85 ing of these novel AKI biomarkers, including neutrophil gelatinase-associated lipocalin (NGAL), kidne
86 postoperative measures of urine IL-18, urine neutrophil gelatinase-associated lipocalin (NGAL), or pl
87 postoperative measures of urine IL-18, urine neutrophil gelatinase-associated lipocalin (NGAL), or pl
88 oparticle-based immunoassay for detection of neutrophil gelatinase-associated lipocalin (NGAL), which
93 eatinine ratio (ACR), and urinary and plasma neutrophil gelatinase-associated lipocalin (NGAL); each
95 dentified as a neutrophil granule component, neutrophil gelatinase-associated lipocalin (NGAL; also c
97 and in combination) of 4 urinary biomarkers (neutrophil gelatinase-associated lipocalin [NGAL], inter
98 damage (kidney injury molecule-1, KIM-1 and neutrophil gelatinase-associated lipocalin, NGAL), kidne
100 siderocalin (SCN; also known as lipocalin 2, neutrophil gelatinase-associated lipocalin/NGAL, or 24p3
101 Primary outcome was noninferiority for urine neutrophil gelatinase-associated lipocalin on the day af
102 siderocalin (SCN; also known as lipocalin-2, neutrophil gelatinase-associated lipocalin, or 24p3) int
103 rkers tested, the optimal 3-marker panel was neutrophil gelatinase-associated lipocalin, protein C, a
105 ll as pimelylcarnitine linked with levels of neutrophil gelatinase-associated lipocalin representing
106 the mouse and human homologues 24p3/lcn2 and neutrophil gelatinase-associated lipocalin, show great f
107 redox-active iron, interleukin (IL)-18, and neutrophil gelatinase-associated lipocalin to predict PN
108 nding protein, redox-active iron, IL-18, and neutrophil gelatinase-associated lipocalin to predict vi
109 on of apoptotic pathways, and no increase in neutrophil gelatinase-associated lipocalin to suggest tu
111 observed glycoproteins (e.g., cadherin 5 and neutrophil gelatinase-associated lipocalin) typically ci
112 tudies evaluating the cut-off level of urine neutrophil gelatinase-associated lipocalin (uNGAL) for d
115 ysC]) with a tubular damage biomarker (urine neutrophil gelatinase-associated lipocalin [uNGAL]), for
117 were elevated in intrinsic AKI, but urinary neutrophil gelatinase-associated lipocalin was most usef
119 e presence of recombinant wild-type sequence neutrophil gelatinase-associated lipocalin, wild-type cy
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