ライフサイエンスコーパス: neutrophil infiltration

1 ce of membrane attack complex activation and neutrophil infiltration.

2 interleukin-1beta (IL-1beta), which sustains neutrophil infiltration.

3 dendritic cells and significantly suppressed neutrophil infiltration.

4 cted animals through 24 h, as was peritoneal neutrophil infiltration.

5 tion model, where they caused a reduction in neutrophil infiltration.

6 nd systemically, but was not associated with neutrophil infiltration.

7 s for use in inflammatory diseases involving neutrophil infiltration.

8 mor foci due to tumor development-associated neutrophil infiltration.

9 ntitumor immune response by inducing massive neutrophil infiltration.

10 levels, reduced hepatic necrosis, and lower neutrophil infiltration.

11 e of epithelial erosions, corneal edema, and neutrophil infiltration.

12 n adipose tissue endothelial cells to induce neutrophil infiltration.

13 eroxidase (MPO) assay was used to quantitate neutrophil infiltration.

14 elated with activation of mTOR signaling and neutrophil infiltration.

15 neutrophil [7/4] antigen, indicating reduced neutrophil infiltration.

16 as interleukin-6 and bronchoalveolar lavage neutrophil infiltration.

17 h Th17-induced pathology, nor did it prevent neutrophil infiltration.

18 id TM may be caused by the early increase in neutrophil infiltration.

19 ogical findings of epidermal hyperplasia and neutrophil infiltration.

20 o model of acute lung injury associated with neutrophil infiltration.

21 nt system, mast cell (MC) degranulation, and neutrophil infiltration.

22 ratinocyte-derived cytokine (mouse IL-8) and neutrophil infiltration.

23 se of circulating neutrophils and multiorgan neutrophil infiltration.

24 ty in the lungs, as a result of monocyte and neutrophil infiltration.

25 tomatitis, demonstrating fungal invasion and neutrophil infiltration.

26 , prevented Ing3A-induced hemorrhage but not neutrophil infiltration.

27 sed corneal opacity, neovascularization, and neutrophil infiltration.

28 intestinal inflammation with lymphocyte and neutrophil infiltration.

29 lial wound healing, perhaps due to decreased neutrophil infiltration.

30 tion of astrocytes and microglia, as well as neutrophil infiltration.

31 g challenge requires prior CXCL1/KC-directed neutrophil infiltration.

32 tion, decreased epidermal proliferation, and neutrophil infiltration.

33 s, which did not affect cytokine release and neutrophil infiltration.

34 ered in disease models, in part, by reducing neutrophil infiltration.

35 ere pancreatic injury including necrosis and neutrophil infiltration.

36 red wine significantly reduced post-ischemic neutrophil infiltration.

37 of disease, including AHR and macrophage and neutrophil infiltration.

38 through a mechanism that involved increased neutrophil infiltration.

39 l catheterization, rarely showed evidence of neutrophil infiltration.

40 ammatory disorders that are characterized by neutrophil infiltration.

41 epidermal hyperproliferation, and increased neutrophil infiltration.

42 late phase that is associated with enhanced neutrophil infiltration.

43 ing both proinflammatory cytokine levels and neutrophil infiltration.

44 of increased vascular permeability and early neutrophil infiltration.

45 duced bone marrow granulopoiesis and cardiac neutrophil infiltration 3 days after MI.

46 elayed corneal healing (13.2%) and increased neutrophil infiltration (54.1%) by day 4 in WT mice, whe

47 onal location of the bacterial infection and neutrophil infiltration, a diffuse optical tomography re

48 Unexpectedly, myocardial infarct size and neutrophil infiltration/activity 2 days after AMI were a

49 NPD1 decreased neutrophil infiltration after 2 and 4 days of treatment.

50 IL-22 also played a major role in neutrophil infiltration after imiquimod treatment.

51 talk in the outer medulla and in suppressing neutrophil infiltration after kidney injury.

52 dial capillary fractional area and decreased neutrophil infiltration after MI.

53 lts from airway damage, mucosal dysfunction, neutrophil infiltration, airway coagulopathy with cast f

54 Only the old mice developed airway neutrophil infiltration and a Th17 immune response upon

55 kin inflammation characterized by T cell and neutrophil infiltration and a Th17-biased cytokine respo

56 tubular injury, IgG and C4d deposition, and neutrophil infiltration and activation.

57 ant exhibit delayed mortality, a decrease in neutrophil infiltration and bacterial CNS dissemination.

58 This strain induced higher levels of neutrophil infiltration and caused smaller lesions than

59 Airway neutrophil infiltration and CXCL1 (KC) expression were a

60 This was accompanied by a decrease in neutrophil infiltration and cytokine levels.

61 found that consumption of an HFD resulted in neutrophil infiltration and enhanced MPO expression and

62 rapid corneal epithelial healing, with less neutrophil infiltration and fewer proliferating cells th

63 ng artery ligation model, as well as reduced neutrophil infiltration and fibrosis.

64 ented the onset of meningitis and suppressed neutrophil infiltration and glial cell migration in the

65 inoculum (1 x 10(7) CFU) of BG2 caused less neutrophil infiltration and greater burdens in peritonea

66 re severe renal dysfunction, tubular injury, neutrophil infiltration and greater mortality than nonde

67 wed significantly higher levels of pulmonary neutrophil infiltration and higher amounts of pulmonary

68 nd responded to the infection with extensive neutrophil infiltration and histopathological changes in

69 of either IL-17A or IL-22 reduced T cell and neutrophil infiltration and host defense peptide elabora

70 aneous periodontitis that featured excessive neutrophil infiltration and IL-17 expression; disease wa

71 sive pulmonary inflammation characterized by neutrophil infiltration and IL-17 response with increase

72 NDV-3 induced increases in CD3+ T-cell and neutrophil infiltration and IL-17A, IL-22, and host defe

73 the adult epicardium reduced injury-induced neutrophil infiltration and improved cardiac function.

74 DCs from mice with pancreatitis resulted in neutrophil infiltration and increased levels of systemic

75 trans expression of SsE(M28) in GAS reduced neutrophil infiltration and increased skin invasion in s

76 modeling and IL8 gene expression, leading to neutrophil infiltration and inflammation.

77 -selectin plays an important role in hepatic neutrophil infiltration and injury induced by chronic-bi

78 ive effect on anti-MHC induced IL-8-mediated neutrophil infiltration and innate immune responses that

79 oth acute lung injury groups as evidenced by neutrophil infiltration and levels of cytokines in bronc

80 lpha antibodies also significantly prevented neutrophil infiltration and liver injury.

81 IL-17 secretion then leads to neutrophil infiltration and lung epithelial changes, in

82 These effects were associated with reduced neutrophil infiltration and macrophage accumulation and

83 ly larger infarcts concordant with increased neutrophil infiltration and myocyte apoptosis compared w

84 and exacerbated stroke injury by aggravating neutrophil infiltration and neurodegeneration in vivo.

85 , and apoptotic neutrophils and reduced both neutrophil infiltration and proinflammatory cytokines in

86 lar surface tissues, including elevated Gr1+ neutrophil infiltration and proinflammatory cytokines/ch

87 anti-inflammatory actions, such as limiting neutrophil infiltration and proresolving actions.

88 with a 54% decrease (p < 0.05) in pulmonary neutrophil infiltration and reduced alveolar wall thicke

89 also resolved E. coli infections by limiting neutrophil infiltration and stimulating bacterial phagoc

90 ate the importance of MFG-E8 in ameliorating neutrophil infiltration and suggest MFG-E8 as a novel th

91 Administration of DNase I to mice reduced neutrophil infiltration and tissue damage in the inflame

92 Neutrophil infiltration and upregulation of expression o

93 ibrosis, 0.86 for bilirubinostasis, 0.60 for neutrophil infiltration, and 0.46 for megamitochondria.

94 icant reduction in MMP-9/-3, less peripheral neutrophil infiltration, and a preservation of tight jun

95 less intense cytokine responses, diminished neutrophil infiltration, and accelerated uroepithelial r

96 o and triggered multiple cytokine responses, neutrophil infiltration, and acute inflammatory histopat

97 ED1 monocytes and macrophages, and pulmonary neutrophil infiltration, and also enhanced the accumulat

98 NPCT; reduced lung inflammation and injury, neutrophil infiltration, and bacterial invasion; and imp

99 imaging, clinical scoring, bacterial burden, neutrophil infiltration, and CXCL2 expression.

100 duced kidney injury, apoptosis, monocyte and neutrophil infiltration, and cytokine (IL-6, IL-1beta, a

101 proinflammatory messenger RNA MIP-2, reduced neutrophil infiltration, and decreased apoptosis (caspas

102 ic mice showed reduced ear swelling, reduced neutrophil infiltration, and decreased migration of fluo

103 ed by increased myeloperoxidase activity and neutrophil infiltration, and elevated pulmonary cytokine

104 eduction of microvascular hyperpermeability, neutrophil infiltration, and endothelial adhesion molecu

105 molecule-1 expression, reduced intima/media neutrophil infiltration, and increased DHCR24 and HO-1 m

106 farct, demyelination, P-selectin expression, neutrophil infiltration, and microthrombi formation.

107 ed significantly higher cytokine production, neutrophil infiltration, and more rapid fungal clearance

108 xhibited significantly reduced infarct size, neutrophil infiltration, and myocyte apoptosis compared

109 Infarct size, neutrophil infiltration, and myocyte apoptosis in the le

110 otin-dUTP nick end-labeling)-positive cells, neutrophil infiltration, and proinflammatory mRNAs as we

111 layed significantly attenuated inflammation, neutrophil infiltration, and reduced severity of oviduct

112 ansaminases, histological signs of necrosis, neutrophil infiltration, and serum levels of interleukin

113 opic signs of colon inflammation, macrophage/neutrophil infiltration, and the expression of proinflam

114 an increase in plasma nucleosomes, abundant neutrophil infiltration, and the presence of citrullinat

115 Hepatocyte death is amplified by liver neutrophil infiltration, and the release of necrotic pro

116 and sepsis-induced ALI abolished lung edema, neutrophil infiltration, and tissue damage, thereby reve

117 istones, suppressed intrarenal inflammation, neutrophil infiltration, and tubular cell necrosis and i

118 intercellular adhesion molecule expression, neutrophil infiltration, and vascular leakage.

119 In conclusion, IL-8 production and neutrophil infiltration are increased in a high-glucose

120 vented chronic-binge ethanol-induced hepatic neutrophil infiltration as well as elevation of serum tr

121 fusion injury, and it increased monocyte and neutrophil infiltration, as well as serum and renal cyto

122 by approximately 60% decrease (P < 0.001) in neutrophil infiltration at 24 hours, and macrophages by

123 ctant-1 in the systemic circulation enhanced neutrophil infiltration, BBB permeability, and injury.

124 Increased EAE severity and neutrophil infiltration because of Del-1-deficiency was

125 bone destruction, with significantly reduced neutrophil infiltration but considerably more macrophage

126 ltured endothelial cells while reducing lung neutrophil infiltration by 40% in a mouse model of LPS-i

127 accumulated into the liver, and blockage of neutrophil infiltration by anti-granulocyte receptor 1 d

128 Neutrophil infiltration caused by IL-8 production is a c

129 and induced models of colitis by regulating neutrophil infiltration, colitogenic CD4(+) T cell activ

130 expression of IL-17 transcripts and massive neutrophil infiltration compared to VV inoculation in sa

131 cted STAT1/IL-13 DKO mice also had increased neutrophil infiltration compared with that of RSV-infect

132 Neutrophil infiltration constitutes the first step in wo

133 e dismutase, nitric oxide, heme oxygenase-1, neutrophil infiltration, cysteamine, mucin, hydrogen sul

134 the immune response, including reduction of neutrophil infiltration, decreased T cell cytokine produ

135 ased mortality that correlated with elevated neutrophil infiltration, diminished numbers of mature ol

136 her levels of reactive oxygen species (ROS), neutrophil infiltration, dityrosine and 4-HNE, as well a

137 ed to reveal MFG-E8 roles in regulating lung neutrophil infiltration during ALI.

138 th high HIC index values (0.88-0.96) induced neutrophil infiltration, elevation of pro-inflammatory c

139 ase was associated with increased intestinal neutrophil infiltration, epithelial injury, and permeabi

140 -3 increased hepatocellular damage and local neutrophil infiltration, facilitated local accumulation

141 damage, 4-hydroxynanoneal adduct formation, neutrophil infiltration, fibrosis, and microvascular pru

142 response by releasing IL-1beta and promoting neutrophil infiltration following ICH.

143 le in the disease have long been recognized, neutrophil infiltration has also been assessed in many c

144 nted oxidative stress, NF-kappaB activation, neutrophil infiltration, hepatocyte apoptosis, and liver

145 ysteine also prevented NF-kappaB activation, neutrophil infiltration, hepatocyte apoptosis, and liver

146 Additionally, higher neutrophil infiltration, IL-17 expression, and intestina

147 Bacterial loads, MODS, leukopenia, neutrophil infiltration, immune cell activation, circula

148 e p19 subunit of IL-23, or IL-17A attenuated neutrophil infiltration in acute kidney IRI in mice.

149 n of EC Notch signaling inhibited peritoneal neutrophil infiltration in an ovarian carcinoma mouse mo

150 indices, paw thickness, cartilage damage and neutrophil infiltration in both CIA and CAIA models.

151 e treatment inhibited hepatic macrophage and neutrophil infiltration in CCl4 -induced hepatitis and s

152 As anticipated, postischemic neutrophil infiltration in CD36(-/-) -->CD36(-/-) mice w

153 sion, blood-brain barrier (BBB) leakage, and neutrophil infiltration in damaged white matter.

154 recruited type 2 macrophages, and prevented neutrophil infiltration in diabetic wounded corneas.

155 utic N-acetylcysteine lowers gastric mucosal neutrophil infiltration in H. pylori-infected Le(b)-expr

156 e, neuronal cell death, oedema formation and neutrophil infiltration in H/I mice.

157 appaB activation established a link to favor neutrophil infiltration in inducing liver damage during

158 immunostaining showed a dramatic decrease of neutrophil infiltration in infected lung tissues for lin

159 lepromatous leprosy and is characterized by neutrophil infiltration in lesions, the most overreprese

160 not Munc13-4-deficient mice showed decreased neutrophil infiltration in liver and failed to undergo L

161 cule, chemokine and cytokine expression, and neutrophil infiltration in lung in vivo.

162 ic release of pro-inflammatory cytokines and neutrophil infiltration in lung, liver, and cecum, when

163 rophil recruitment based on the reduction of neutrophil infiltration in mice in which the overall sul

164 train Wakulla induces diffuse hepatitis with neutrophil infiltration in mice with severe combined imm

165 nes by Chlamydia-infected cells and promoted neutrophil infiltration in mouse airways during chlamydi

166 IL-15DeltaE7 also profoundly blocked neutrophil infiltration in SDS- or immiquimod (IMQ)-trea

167 To corroborate these findings in vivo, neutrophil infiltration in self-limited peritonitis was

168 Increased neutrophil infiltration in Splunc1 KO mice was accompani

169 tic target for the restriction of pathogenic neutrophil infiltration in TH17-mediated autoimmune dise

170 ory response characterized by macrophage and neutrophil infiltration in the bladder and kidneys.

171 ed NF-kappaB nuclear staining, and decreased neutrophil infiltration in the cecum.

172 d IL-17-producing CD4(+) T cells, as well as neutrophil infiltration in the cornea.

173 crophages and IL-17A produced in situ drives neutrophil infiltration in the epidermis and dermis of t

174 ke skin model, we show that KO mice had less neutrophil infiltration in the epidermis than controls,

175 ng to either insufficient MDSCs or excessive neutrophil infiltration in the fetomaternal interface ma

176 , UFP ingested mice developed macrophage and neutrophil infiltration in the intestinal villi, accompa

177 le-1, and C-reactive protein, and attenuated neutrophil infiltration in the liver.

178 Anxa2(-/-) mice develop pulmonary edema and neutrophil infiltration in the lung parenchyma in respon

179 However, thrombin did contribute to neutrophil infiltration in the lung, independently of PA

180 APC, more interleukin-6, and show increased neutrophil infiltration in the lungs compared with WT co

181 Histochemical staining revealed that neutrophil infiltration in the lungs occurred in two wav

182 male OVA-specific TH17 cells increased acute neutrophil infiltration in the lungs of OVA-challenged r

183 LMM) hyaluronan (200 kDa) results in greater neutrophil infiltration in the lungs of TLR4(-/-) mice c

184 ation of pro-inflammatory cytokines, reduced neutrophil infiltration in the lungs, and diminished hep

185 k EhMIF had reduced chemokine expression and neutrophil infiltration in the mucosa.

186 sed alveolar bone loss and a lower degree of neutrophil infiltration in the periodontium than vehicle

187 ntent, and can be used as a marker to assess neutrophil infiltration in the tissue.

188 By 48 hours PI, there was significantly more neutrophil infiltration in the vitreous infected with th

189 ction in active glomerular proliferation and neutrophil infiltration in three of five patients, consi

190 In general, enhanced neutrophil infiltration in tissue is due to cell influx;

191 ast cell-derived TNF, as we observed reduced neutrophil infiltration in W(sh)/W(sh) mice reconstitute

192 lation by CL 316,243 promotes adipose tissue neutrophil infiltration in wild type and P-selectin-null

193 ve stress (increased MDA/4-HNE levels), (ii) neutrophil infiltration (increased MPO activity), (iii)

194 To sort out the role of adipose neutrophil infiltration independent of cPLA2alpha induct

195 o WT LTx, with enhanced hepatic necrosis and neutrophil infiltration, indicating a protective role of

196 tor GM6001 reduced the early BBB leakage and neutrophil infiltration, indicating that OPC-derived MMP

197 Ps (NPs-CD11b) into the tumor is mediated by neutrophil infiltration induced by photosensitization (P

198 ed inflammatory cytokines, decreased hepatic neutrophil infiltration, inhibited liver cell apoptosis

199 that anti-TIM-1 Ab treatment decreased local neutrophil infiltration, inhibited sequestration of T ly

200 effectively prevented cPLA2alpha induction, neutrophil infiltration into adipose tissue (likely invo

201 2 receptor and is characterized by extensive neutrophil infiltration into different organs, including

202 Although Foxm1 deficiency did not influence neutrophil infiltration into injured livers, the total n

203 Three days after ligation, neutrophil infiltration into ischemic limbs of AMPKalpha

204 The absence of CIKS completely prevented neutrophil infiltration into joints, bone erosion, and c

205 LPS and BTH also induced neutrophil infiltration into the air pouch, which was no

206 duce either cytokine/chemokine production or neutrophil infiltration into the air pouch.

207 nhibit some aspects of inflammation, such as neutrophil infiltration into the air pouch.

208 tic fibrosis, are characterized by excessive neutrophil infiltration into the airspace.

209 lung inflammation, as evidenced by a reduced neutrophil infiltration into the airways, with diminishe

210 complex, which is characterized by abundant neutrophil infiltration into the alveoli and fibrin depo

211 1(-/-)) exhibited a significant reduction in neutrophil infiltration into the brain after TBI as comp

212 Kinetic studies demonstrate that neutrophil infiltration into the cerebellum and brainste

213 S3(fl/fl) mice is characterized by extensive neutrophil infiltration into the cerebellum and brainste

214 iorates atypical EAE development by reducing neutrophil infiltration into the cerebellum/brainstem.

215 production of proinflammatory cytokines, and neutrophil infiltration into the colon.

216 ed keratinocyte activation and inhibition of neutrophil infiltration into the dermis, demonstrating t

217 s characterized by epithelial extrusions and neutrophil infiltration into the fin.

218 Similarly neutrophil infiltration into the infarct zone was decrea

219 rdination, and working memory, and abrogated neutrophil infiltration into the injured brain.

220 ngth accompanied by prominent macrophage and neutrophil infiltration into the intestinal villi, admin

221 enzymes, focal ischaemic areas and a robust neutrophil infiltration into the liver.

222 -type activated PC and significantly reduced neutrophil infiltration into the lungs of septic mice.

223 mation, lung microvascular permeability, and neutrophil infiltration into the lungs were suppressed i

224 e or 15 mug kg(-1)) reduced zymosan-elicited neutrophil infiltration into the peritoneum 25-50% and s

225 way wall thickness as well as eosinophil and neutrophil infiltration into the respiratory airway.

226 CHS requires prior CXCL1- and CXCL2-mediated neutrophil infiltration into the site.

227 epidermis are associated with macrophage and neutrophil infiltration into the tissues.

228 Preventing Ly6Clo monocyte or Ly6G+ neutrophil infiltration into tumors enhanced inhibition

229 the tumor associated molecules that regulate neutrophil infiltration into tumors may provide new and

230 enous myeloperoxidase infusion revealed that neutrophil infiltration is a prerequisite for myocardial

231 ted with diabetic wounds, while reduction of neutrophil infiltration is associated with enhanced heal

232 Collectively, these data show that decidual neutrophil infiltration is not essential for the inducti

233 tigated the role of VEGF165b in brain edema, neutrophil infiltration, ischemic brain damage, and neur

234 eover, while the absence of IL-1R1 prevented neutrophil infiltration, it did not protect from acantho

235 rrier that results in edema, hemorrhage, and neutrophil infiltration, leading to exacerbated lung inf

236 ion (STAT)6 ameliorated alpha-Galcer-induced neutrophil infiltration, liver injury, and hepatitis.

237 ion including epithelial damage, congestion, neutrophil infiltration, loss of mucin from goblet cells

238 Thus, treatment of cancers associated with neutrophil infiltration may benefit from specific target

239 flammation signature was linked to increased neutrophil infiltration, more cell death and greater par

240 a), neutrophil chemoattractants (MIP-2, KC), neutrophil infiltration (MPO activity), lipid peroxidati

241 Neutrophil infiltration, normally increased by smoke exp

242 se PAR1 is expressed at sites where abundant neutrophil infiltration occurs, we hypothesized that neu

243 TREM-1/3-deficient lungs revealed decreased neutrophil infiltration of the airways.

244 We analyzed neutrophil infiltration of the mouse ileum after allo-HC

245 Neutrophil infiltration of the skin and other organs and

246 tory diseases that all demonstrate excessive neutrophil infiltration of the skin.

247 Neutrophil infiltration of the uteroplacental tissues ha

248 The dermal neutrophil infiltration of the wound, as represented by

249 classical hallmark of acute inflammation is neutrophil infiltration of tissues, a multistep process

250 ssed AHR and failed to dampen macrophage and neutrophil infiltration or inflammatory cytokine product

251 yed-type hypersensitivity (edema, T-cell and neutrophil infiltration, or expression of interleukin-1b

252 y lymph (p < 0.01 at 24 and 48 hr), alveolar neutrophil infiltration (p = 0.04), and pulmonary myelop

253 1 has anti-inflammatory activity by reducing neutrophil infiltration, paw edema and proinflammatory c

254 s are the main cause of ALI, leading to lung neutrophil infiltration, permeability increases, deterio

255 nalyzed for morphological injury, apoptosis, neutrophil infiltration, proinflammatory mRNAs, and TGF-

256 Passive immunization with 2B11 increased neutrophil infiltration, reduced skin invasion, and prot

257 vestigated whether NLRP3 knockdown decreases neutrophil infiltration, reduces brain edema, and improv

258 gulated 10-fold, whereas expression of other neutrophil infiltration-related adhesion molecules (e.g.

259 Neutrophil infiltration remained low in acutely injured

260 Neutrophil infiltration represents the early acute infla

261 on in vivo abrogated alcohol-induced hepatic neutrophil infiltration, resident immune cell activation

262 Enhanced neutrophil infiltration resulted in part from reduced ro

263 to establish a functional link between lung neutrophil infiltration, secretion of chemokines by canc

264 rthermore, platelet depletion abrogated lung neutrophil infiltration, suggesting a sequential partici

265 n induced greater necrotic airway damage and neutrophil infiltration than A2 infection.

266 e using photosensitization rapidly activates neutrophil infiltration that mediates delivery of nanoth

267 ranscription of c-Myb and elevated levels of neutrophil infiltration, thereby alleviating infectious

268 This phenotype correlates with reduced neutrophil infiltration to both the primary tumour and m

269 pathway and production of CXC chemokines and neutrophil infiltration to infected tissues.

270 gulation of nitric oxide and upregulation of neutrophil infiltration to the infarct area.

271 d chemokine levels, as well as a decrease in neutrophil infiltration to the liver.

272 hibitor of IKK-2, resulted in suppression of neutrophil infiltration to the lung tissues and reductio

273 Excessive neutrophil infiltration to the lungs is a hallmark of ac

274 /W(sh) mice, respond to IL-33 treatment with neutrophil infiltration to the peritoneum, whereas other

275 roduction by gammadelta T cells and ILC3 and neutrophil infiltration to the site of infection, were g

276 Neutrophil infiltration to the sites of mucosal Candida

277 This promoted neutrophil infiltration, tumor cell (TC) adhesion to the

278 matory state in endothelial cells, promoting neutrophil infiltration, tumor cell adhesion, and metast

279 The degree of fibrosis, degree of neutrophil infiltration, type of bilirubinostasis, and p

280 reflow injury, promoting repair via limiting neutrophil infiltration, up-regulating Ki67, and Roof pl

281 Reduction of cardiac permeability and neutrophil infiltration was also observed in P2Y4-null m

282 Significant inhibition of neutrophil infiltration was also observed in the WA grou

283 The I/R-evoked neutrophil infiltration was also suppressed in Tg hearts

284 The time course of healing and degree of neutrophil infiltration was evaluated after corneal epit

285 We found that neutrophil infiltration was impaired in IL-1R1(-/-) mice

286 regulated in CSF-1-deficient RT2 tumors, and neutrophil infiltration was increased.

287 nd chemokines was the same as LPS alone, but neutrophil infiltration was inhibited, likely by interru

288 D36(-/-) mice, in which infarcts were small, neutrophil infiltration was large and similar to that of

289 A 100-fold reduction in neutrophil infiltration was observed in the lungs of the

290 Massive neutrophil infiltration was observed in the spleen and l

291 eutrophil recruitment to the brain, and that neutrophil infiltration was required for parenchymal tis

292 okines/chemokines, microglial activation and neutrophil infiltration were evaluated.

293 Apoptosis and neutrophil infiltration were increased in the pancreas o

294 male mice, which had hepatic lymphocyte and neutrophil infiltration, were treated by vancomycin, pol

295 nts, albuminuria, serum urea, and glomerular neutrophil infiltration when compared with WT littermate

296 Mosaic animals also displayed splenic neutrophil infiltration, which was skewed toward the def

297 ase of inflammation-(d1), 15-epi-LXA4 primes neutrophil infiltration with a robust increase of Ccl2 a

298 ous autosomal mutation in a mouse leading to neutrophil infiltration with ulceration in the upper der

299 n challenge requires prior CXCL1/KC-directed neutrophil infiltration within 3-6 h after challenge and

300 This effectively prevented neutrophil infiltration without affecting cPLA2alpha or