ライフサイエンスコーパス: neutrophilic

1 3: COPD predominant, mixed eosinophilic and neutrophilic.

2 s follows: eosinophilic (40%), mixed (6.7%), neutrophilic (5.4%) and paucigranulocytic (47.9%).

3 rbated lung inflammation seen as a result of neutrophilic accumulation is dependent on S100A8/A9 prot

4 Neutrophilic airway diseases, including cystic fibrosis,

5 lead to the different prognosis in these two neutrophilic airway diseases.

6 s adjuvants developed mixed eosinophilic and neutrophilic airway inflammation and airway hyperrespons

7 utophagy in pulmonary CD11c(+) cells induces neutrophilic airway inflammation and hyperreactivity.

8 icrobiome across asthmatic patients, whereas neutrophilic airway inflammation does not.

9 Because azithromycin attenuated neutrophilic airway inflammation in a murine viral bronc

10 ces eosinophilic and endotoxin (LPS)-induced neutrophilic airway inflammation in animal models and he

11 We have found upregulation of neutrophilic airway inflammation in atopic asthmatics ex

12 defense network that may underlie persistent neutrophilic airway inflammation in COPD and modulating

13 Allergen-induced neutrophilic airway inflammation in GSTM1+ asthmatics is

14 The functional GSTM1 genotype promotes neutrophilic airway inflammation in humans with atopic a

15 responses, we defined the effects of ATF3 on neutrophilic airway inflammation in mice.

16 itamin D supplementation on eosinophilic and neutrophilic airway inflammation in patients with nonato

17 ts that neutrophil elastase (NE) released in neutrophilic airway inflammation is a key risk factor fo

18 athogenesis is poorly understood, but excess neutrophilic airway inflammation is seen.

19 ory tract symptoms, airflow obstruction, and neutrophilic airway inflammation were increased in exper

20 Neutrophilic airway inflammation, disease severity, and

21 he implications of IL-17 in the induction of neutrophilic airway inflammation, steroid insensitivity,

22 patients with severe asthma and is linked to neutrophilic airway inflammation, suggesting that these

23 ecular patterns, innate cytokine release and neutrophilic airway inflammation.

24 nfiltrating CD4 T cells express Foxp3 during neutrophilic airway inflammation.

25 Moreover, the iPHIL neutrophilic airway phenotype was shown to be a steroid-

26 ble steroid-resistant character of the iPHIL neutrophilic airway variant suggests underappreciated me

27 roducing iNKT cells were sufficient to drive neutrophilic airways inflammation upon intratracheal ado

28 (-/-) mice, whereas these mice had increased neutrophilic alveolitis and greater lung injury compared

29 gy, despite its moderate reducing effects on neutrophilic and cytokine responses.

30 After allergen exposure, rhinovirus-induced neutrophilic and eosinophilic airway inflammation and hy

31 ired during the allergen challenge phase for neutrophilic and eosinophilic inflammation.

32 emonstrated that Exserohilum provokes robust neutrophilic and granulomatous inflammation capable of t

33 Initial neutrophilic and histiocytic inflammation in affected ti

34 thma, while IL-8 was higher in patients with neutrophilic and mixed asthma (P<.001 for all comparison

35 IL-6 and sIL-6R levels were enriched for the neutrophilic and mixed granulocytic subtypes.

36 69.7-95.0), P=.009] for eosinophilic, mixed, neutrophilic and paucigranulocytic asthma, respectively,

37 7%, respectively) and less frequently in the neutrophilic and paucigranulocytic phenotype (25% and 21

38 abrogated the protection from eosinophilic, neutrophilic, and Th2 pulmonary inflammation seen in Cle

39 ted by leukocyte transmigration, whereas the neutrophilic antimicrobial peptide HNP-1 is noted as a p

40 n the presence of eosinophils in contrast to neutrophilic as well as mixed Th1/Th17/Th2 variant pheno

41 H2/TH17-low asthma had 2 distinct subgroups: neutrophilic asthma (45%) and pauci-inflammatory asthma

42 ), paucigranulocytic asthma (AUC, 92.6%), or neutrophilic asthma (AUC, 91.4%) and healthy control sub

43 atients with sub-optimally controlled severe neutrophilic asthma and in asthma exacerbations.

44 oup of patients with TH2/TH17-low asthma had neutrophilic asthma and increased BAL fluid IL-1alpha, I

45 ike receptor C4 (NLRC4) were associated with neutrophilic asthma and with sputum IL-1beta protein lev

46 Eosinophilic and neutrophilic asthma endotypes are also classified by epi

47 6%), as well as discriminating patients with neutrophilic asthma from those with paucigranulocytic as

48 Sixty percent of the patients with neutrophilic asthma had a pathogenic microorganism in BA

49 t and TH2-predominant asthma, which included neutrophilic asthma in 6% and 0% of patients, respective

50 Our data indicate that neutrophilic asthma in Atg5(-/-) mice is glucocorticoid

51 Neutrophilic asthma in STAT6(-/-) mice was accompanied b

52 Neutrophilic asthma is associated with airway microbiolo

53 n was amplified in bronchial/nasal mucosa of neutrophilic asthma prone to exacerbation, suggesting a

54 A biomarker for neutrophilic asthma was identified.

55 in IL-8 production and likely contributed to neutrophilic asthma.

56 for factors other than IL-17 as targets for neutrophilic asthma.

57 levels in sputum samples from patients with neutrophilic asthma.

58 role for IL-1beta in both acute and chronic neutrophilic asthma.

59 nas taxa relative abundance in patients with neutrophilic asthma.

60 lmonary disease, respiratory infections, and neutrophilic asthma.

61 otential therapeutic target in patients with neutrophilic asthma.

62 function and identify molecular markers for neutrophilic asthma.

63 the pathogenesis of corticosteroid-resistant neutrophilic asthma.

64 ol subjects and was highest in patients with neutrophilic asthma.

65 The overexpression was restricted to neutrophilic asthmatics (sputum neutrophils >/= 76%), wh

66 ell count and % neutrophils were elevated in neutrophilic BOS and RAS compared to stable and non-neut

67 could not detect any differences between non-neutrophilic BOS and stable patients.

68 ulated in RAS compared to stable, whereas in neutrophilic BOS IL-1beta (P<0.001), IL-1Ralpha (P<0.01)

69 hilic BOS and RAS compared to stable and non-neutrophilic BOS patients, whereas also the % eosinophil

70 ble patients, 20 patients suffering from non-neutrophilic BOS, 17 from neutrophilic BOS, and 20 from

71 suffering from non-neutrophilic BOS, 17 from neutrophilic BOS, and 20 from RAS using classic enzyme-l

72 pes (bronchiolitis obliterans syndrome [BOS]-neutrophilic BOS-restrictive allograft syndrome [RAS]) h

73 to interrogate the underlying mechanisms of neutrophilic breach of mucosal barriers.

74 is characterized by epithelial desquamation, neutrophilic bronchiolitis and pneumonia, and obstructiv

75 these proteins even in asthma patients with neutrophilic bronchitis, EPX-based ELISA levels are not

76 Neutrophilic, but not eosinophilic, asthmatics display o

77 more patients in the C4d-positive group had neutrophilic capillaritis (54% vs. 29%, P = .035), there

78 als are expected to decrease the activity of neutrophilic CatC without affecting those of elastase-li

79 increase in Ly6C(hi) monocytic and Gr-1(hi) neutrophilic cells in lymphoid organs and blood.

80 l fluid/blood glucose ratio less than 0.5, a neutrophilic cerebrospinal fluid pleocytosis (> 5 cells/

81 tokines; cluster 2: asthma and COPD overlap, neutrophilic; cluster 3: COPD predominant, mixed eosinop

82 (P = .022) and more dissimilar (P = .005) in neutrophilic compared with eosinophilic participants.

83 Neutrophilic corticosteroid-resistant asthma accounts fo

84 consequence of impaired recruitment of a non-neutrophilic CXCR2 positive leukocyte population.

85 atients presented with neonatal-onset fever, neutrophilic dermatitis/panniculitis, and failure to thr

86 Neutrophilic dermatoses comprise a wide spectrum of infl

87 immunity, providing an animal model of human neutrophilic dermatoses.

88 Pyoderma gangrenosum is an inflammatory neutrophilic dermatosis characterized by painful cutaneo

89 We describe 3 patients with an unusual neutrophilic dermatosis characterized by relapsing episo

90 severe inflammatory syndrome that resembles neutrophilic dermatosis in humans and is characterized b

91 an inflammatory skin disease that resembles neutrophilic dermatosis in humans.

92 mmune-dysregulatory disease chronic atypical neutrophilic dermatosis with lipodystrophy and elevated

93 Sweet syndrome (SS) is a prototypical neutrophilic dermatosis, a class of inflammatory disease

94 Resistin inhibited neutrophilic-differentiated NB4 cell migration and intra

95 ptic shock and acute kidney injury inhibited neutrophilic-differentiated NB4 cell migration.

96 howed elevated resistin levels and inhibited neutrophilic-differentiated NB4 cell migration.

97 th or without severe acute kidney injury) on neutrophilic-differentiated NB4 cells.

98 Neutrophilic differentiation is dependent on CCAAT enhan

99 Sweet syndrome within the broad spectrum of neutrophilic diseases is important for its prompt and pr

100 ould lead to novel therapeutic approaches in neutrophilic diseases, such as cystic fibrosis or rheuma

101 g with CP has a considerable effect on serum neutrophilic enzyme levels, except TIMP-1.

102 as evidenced by salivary and serum levels of neutrophilic enzymes.

103 = 14), paucigranulocytic (n = 60), or mixed neutrophilic-eosinophilic (n = 9) asthma phenotypes.

104 Eosinophilic and mixed neutrophilic/eosinophilic inflammation were more prevale

105 Mice lacking PTX3 have exaggerated neutrophilic/eosinophilic lung inflammation, mucus produ

106 at uric acid crystals became enmeshed in the neutrophilic extracellular traps (NETs) produced from ho

107 ons and noneosinophilic endotypes, including neutrophilic forms of disease.

108 e not associated with asthma, even in severe neutrophilic forms.

109 ongly associated with asthma, even in severe neutrophilic forms.

110 humans, such as Howell-Jolly bodies and low neutrophilic granularity, are commonplace in healthy mic

111 IFN-gamma accumulates in primary neutrophilic granules and is released upon induction of

112 Polymorphonuclear neutrophilic granulocytes (PMN) as cellular components o

113 Neutrophilic granulocytes are the most abundant type of

114 ence that different stimuli induced isolated neutrophilic granulocytes to release microvesicles with

115 psilon)-(carboxymethyl)lysine (CML), VCAM-1, neutrophilic granulocytes, lymphocytes, and macrophages

116 blood vessels and the numbers of infiltrated neutrophilic granulocytes, lymphocytes, and macrophages

117 enhancement of transendothelial migration of neutrophilic granulocytes.

118 ower numbers of infiltrating macrophages and neutrophilic granulocytes.

119 ls in 44 patients with nonatopic asthma with neutrophilic (>/=53%) and/or eosinophilic (>/=3%) airway

120 response in vivo, the first demonstration of neutrophilic IL-1beta production in response to viral lu

121 histologically by the presence of an aseptic neutrophilic infiltrate in the epidermis, dermis, and/or

122 g, elevated CRP level or leukocytosis, and a neutrophilic infiltrate on skin biopsy.

123 ide reduced its expression and the prominent neutrophilic infiltrate, a hallmark of the disease.

124 Predominantly neutrophilic infiltrates are seen in a subset of patient

125 Propofol exposure increased neutrophilic infiltrates into the kidney and enhanced ba

126 racteristically deep dermal and subcutaneous neutrophilic infiltrates without evidence of myeloblasts

127 In vivo, RvD4 (ng/mouse) reduced neutrophilic infiltration (~40%) and enhanced uptake of

128 zation of circulating RANTES decreased liver neutrophilic infiltration and attenuated HCC tumor initi

129 severe liver inflammation; characterized by neutrophilic infiltration and HSC activation with collag

130 Neutrophilic infiltration is a leading contributor to pa

131 Cytometric methods revealed extensive neutrophilic infiltration of oral tissues in GRAKO mice;

132 cigarettes may develop unstable disease and neutrophilic infiltration of the airways, features more

133 n histological patterns of S grafts revealed neutrophilic infiltration surrounding fat accumulation.

134 Furthermore, we observed neutrophilic infiltration was slightly increased in anti

135 ese mice developed severe hepatic steatosis, neutrophilic infiltration, and >10-fold alanine aminotra

136 showed severe epidermal hyperplasia, greater neutrophilic infiltration, and higher expression of Th17

137 ransgenic mice displayed marked decreases in neutrophilic infiltration, tissue damping, and elastance

138 This was associated with increased liver-neutrophilic infiltration, whereas infiltration of lymph

139 aggregation, thrombotic microangiopathy, and neutrophilic infiltration.Sublytic EC injury: Sublethal

140 psy of the erythematous lesions showed dense neutrophilic infiltrations and diagnosis of Sweet's synd

141 Neutrophilic infiltrations were scant.

142 Further, more neutrophilic infiltrations, epithelial damage, and infla

143 s to naive TLR2(-/-) mice was sufficient for neutrophilic inflammation after rhinovirus infection, wh

144 DPH oxidase protects against ALI by limiting neutrophilic inflammation and activating Nrf2, a transcr

145 IL-1 receptor appears to be a marker of neutrophilic inflammation and airflow obstruction in pat

146 1beta responses that drive steroid-resistant neutrophilic inflammation and airway hyperresponsiveness

147 h for prevention and management of excessive neutrophilic inflammation and alveolar barrier dysfuncti

148 a tissue-specific role for AC in regulating neutrophilic inflammation and cytokine production.

149 f host-directed therapies in TBDM, targeting neutrophilic inflammation and diabetic complication path

150 ntagonist anakinra had protective effects on neutrophilic inflammation and emphysema.

151 IL-17A is central for neutrophilic inflammation and has been linked to COPD pa

152 We aimed to investigate the role of neutrophilic inflammation and PAR1 in S pneumoniae-induc

153 PGP elicits an exacerbated neutrophilic inflammation and protease imbalance that fu

154 ective role for HIF-2alpha in persistence of neutrophilic inflammation and provide a platform to diss

155 optosis in vivo, resulting in a reduction in neutrophilic inflammation and reduced tissue injury.

156 Anakinra effectively reduced airway neutrophilic inflammation and resulted in no serious adv

157 NTHi-induced pulmonary neutrophilic inflammation and tumor-associated neutrophi

158 ype) mice demonstrated higher levels of lung neutrophilic inflammation and viral load, but lower leve

159 expected for lower AcPGP levels, markers of neutrophilic inflammation are blunted.

160 IL-17RA and IL-17RC subunits, also promotes neutrophilic inflammation but its effects on vascular ce

161 crophages by 71% and also markedly inhibited neutrophilic inflammation by 80%.

162 Serum amyloid A (SAA) promotes neutrophilic inflammation by its interaction with lung m

163 sponse to pathogenic bacteria, but excessive neutrophilic inflammation can be associated with bystand

164 osa infections coupled with robust, damaging neutrophilic inflammation characterize the chronic lung

165 , indicating an important role for IL-17A in neutrophilic inflammation during cholestasis.

166 4A3 expression is also increased at sites of neutrophilic inflammation in a human model of intraderma

167 ciated with strikingly exaggerated pulmonary neutrophilic inflammation in a nonatopic model of airway

168 , wogonin enhanced resolution of established neutrophilic inflammation in a zebrafish model of steril

169 This review discusses the role of neutrophilic inflammation in angiotropism and pericytic

170 tress may be useful for alleviating damaging neutrophilic inflammation in CF airways.

171 caused by airway mucus obstruction precedes neutrophilic inflammation in Scnn1b-transgenic (Scnn1b-T

172 farinae challenge abrogated eosinophilic and neutrophilic inflammation in the bronchoalveolar lavage

173 ce neutrophil apoptosis in vitro and resolve neutrophilic inflammation in vivo.

174 c epithelial necrosis in the pathogenesis of neutrophilic inflammation independent of bacterial infec

175 Pulmonary neutrophilic inflammation induced by CS is significantly

176 self-resolving and GC-induced resolution of neutrophilic inflammation induced by LPS in mice.

177 poptotic program that promotes resolution of neutrophilic inflammation induced by LPS.

178 rough the lungs, revealed that the IL-17 and neutrophilic inflammation induced by Ym1 limited parasit

179 Neutrophilic inflammation is a common feature of many au

180 Chronic neutrophilic inflammation is a hallmark in the pathogene

181 Neutrophilic inflammation is an important pathologic fea

182 Neutrophilic inflammation is tightly regulated and subse

183 he hypothesis that asthmatic smokers develop neutrophilic inflammation of the airways propagated at l

184 Neutrophilic inflammation often persists for days despit

185 ients with cystic fibrosis (CF), its role in neutrophilic inflammation remains unknown.

186 responses of the airway to HRV, focusing on neutrophilic inflammation that is a potentially unwanted

187 Acute gonorrhea is characterized by neutrophilic inflammation that is insufficient to clear

188 -proline (AcPGP) pathway is a novel means of neutrophilic inflammation that is pathologic in the deve

189 ht a mechanism by which acrolein potentiates neutrophilic inflammation through selective inhibition o

190 We used a mouse model of peritoneal neutrophilic inflammation to determine if Ang-1 could st

191 e data suggest that M. tuberculosis exploits neutrophilic inflammation to preferentially replicate at

192 In contrast to eosinophilic inflammation, neutrophilic inflammation was not promoted by beta2AR si

193 Roles for IL-1beta-induced neutrophilic inflammation were examined using IL-1beta a

194 rom infected mice, but antibody deposits and neutrophilic inflammation were not features of the lesio

195 TH17 cells and the consequent neutrophilic inflammation were poorly sustained by inhal

196 moke developed emphysema with increased PGP, neutrophilic inflammation, and selective inhibition of L

197 owever, its role during S pneumoniae-induced neutrophilic inflammation, and the mechanisms for neutro

198 -associated eosinophilic and Th17-associated neutrophilic inflammation, but the impact of the environ

199 porioides induced a strong TH17 response and neutrophilic inflammation, but very mild airway hyperres

200 ut undergo infection-related weight loss and neutrophilic inflammation, development of anti-pseudomon

201 ntify metabolic activation, indicating local neutrophilic inflammation, in the same regions of intere

202 essed local IL-6 and IL-17A levels, enhanced neutrophilic inflammation, reduced uterine macrophage po

203 most patients with COPD have a predominantly neutrophilic inflammation, some have an increase in eosi

204 roduction of IL-17, which is associated with neutrophilic inflammation, was enhanced.

205 and gammadelta-17 cells, proposed drivers of neutrophilic inflammation, were not strongly associated

206 the LTA4H aminopeptidase activity alleviates neutrophilic inflammation, which contributes to cigarett

207 r 4 (TLR4) to stimulate CXCL-mediated innate neutrophilic inflammation, which in turn facilitates all

208 ytes are key to the successful resolution of neutrophilic inflammation, with dysregulated apoptosis r

209 The cytokine IL-17A is associated with neutrophilic inflammation.

210 lator La-related protein 7 (LaRP7) increased neutrophilic inflammation.

211 haracterization of miRs that are relevant to neutrophilic inflammation.

212 eutrophil migration throughout the course of neutrophilic inflammation.

213 eumoniae, which is associated with excessive neutrophilic inflammation.

214 reduction of both lung IL-1beta release and neutrophilic inflammation.

215 , characterized by natural killer T-cell and neutrophilic inflammation.

216 nt of regional lung metabolism reflective of neutrophilic inflammation.

217 e of TSLPR signaling also led to exaggerated neutrophilic inflammation.

218 learance of pulmonary bacteria and increased neutrophilic inflammation.

219 ite profile of lipid mediators that regulate neutrophilic inflammation.

220 e and was associated with the suppression of neutrophilic inflammation.

221 of asthma in the absence of eosinophilic or neutrophilic inflammation.

222 metic) drive caspase-dependent resolution of neutrophilic inflammation.

223 atients with RA, which likely contributed to neutrophilic inflammation.

224 severe asthma who present with TH17-mediated neutrophilic inflammation.

225 vere asthma often present with TH17-mediated neutrophilic inflammation.

226 CXCR2 antagonists inhibit neutrophilic inflammation; inhibitors of phosphodiestera

227 that are shown to be important mediators of neutrophilic inflammation; selective targeting of their

228 nt a future therapeutic strategy to modulate neutrophilic inflammatory diseases, such as cystic fibro

229 The UV-induced neutrophilic inflammatory response stimulated angiogenes

230 brosis (CF) is characterized by an excessive neutrophilic inflammatory response within the airway as

231 s, a model in which Stx2 induces a primarily neutrophilic inflammatory response.

232 or preventing exacerbation of disease in the neutrophilic inflammatory response.

233 CL8 accumulation correlated to the degree of neutrophilic influx in affected mucosa.

234 mucilaginosus in C57BL/6 mice resulted in a neutrophilic influx with production of proinflammatory c

235 g inflammation, but 6.0 mg/kg of DEP induced neutrophilic influx.

236 The system is dominated by neutrophilic, iron-oxidizing bacteria, including 'marine

237 classification of myeloid disorders, chronic neutrophilic leukaemia (CNL) is recognized as a myelopro

238 mutations in CSF3R (GCSFR) in 60% of chronic neutrophilic leukemia (CNL) and atypical (BCR-ABL-negati

239 Among these cancers are chronic neutrophilic leukemia (CNL) and atypical (BCR-ABL1-negat

240 neoplasms, the pathogenetic basis of chronic neutrophilic leukemia (CNL) has remained elusive.

241 Chronic neutrophilic leukemia (CNL) is a distinct myeloprolifera

242 -proximal mutations seen commonly in chronic neutrophilic leukemia (e.g., T618I), functionally defect

243 a large percentage of patients with chronic neutrophilic leukemia and, more rarely, in other types o

244 h are much more commonly observed in chronic neutrophilic leukemia.

245 , and activating CSF3R mutation with chronic neutrophilic leukemia.

246 This can result in stimulation of both neutrophilic leukocytosis and the release of immature gr

247 ferative neoplasm characterized by sustained neutrophilic leukocytosis, hepatosplenomegaly and bone m

248 garette smoke exposure significantly reduced neutrophilic lung inflammation and production of pro-inf

249 H oxidase-deficient mice developed exuberant neutrophilic lung inflammation and proinflammatory cytok

250 pontaneous airway hyperreactivity and severe neutrophilic lung inflammation in mice.

251 d-glycosyl aesculin significantly suppressed neutrophilic lung inflammation, a hallmark of acute lung

252 In patients with neutrophilic lung inflammation, mature CatC is found in

253 ng inflammation to corticosteroid-refractory neutrophilic manifestation.

254 with the accumulation of cells that express neutrophilic markers Gr-1 and Ly-6G but do not belong to

255 DG172-mediated repression of genes encoding neutrophilic markers in both differentiating wild-type a

256 Mechanistically, pathogenic fungi induce neutrophilic MDSCs through the pattern recognition recep

257 Twenty-five (19%) had neutrophilic meningitis.

258 ignaling into keratinocytes is essential for neutrophilic microabscess formation and contributes to h

259 enotypes have been recognized (eosinophilic, neutrophilic, mixed and paucigranulocytic).

260 andida albicans induces a distinct subset of neutrophilic myeloid-derived suppressor cells (MDSCs), w

261 s an excellent tool for the further study of neutrophilic myeloproliferative neoplasms and implicates

262 nts and classified as eosinophilic (n = 84), neutrophilic (n = 14), paucigranulocytic (n = 60), or mi

263 te that formyl peptide receptor 1 (FPR1) and neutrophilic NADPH oxidase (NOX2) are required for the r

264 trate (predominantly mononuclear vs mixed or neutrophilic; P = .003), presence of eosinophils (presen

265 Sixty percent of neutrophilic patients had a subclinical infection.

266 healthy controls; and 3) effect of the human neutrophilic peptide-1 (HNP-1) on epithelial adhesion mo

267 sed peripheral neutrophilia, which amplified neutrophilic peritoneal inflammation in X-CGD mice.

268 ring airway challenge led to a predominantly neutrophilic phenotype (>15% neutrophils) accompanied by

269 over a 1-year period were observed among the neutrophilic phenotypes.

270 isk over a 1-year period were detected among neutrophilic phenotypes.

271 f alcoholic liver disease (ALD) with intense neutrophilic (polymorphonuclear; PMN) inflammation and h

272 nism that can be entirely managed by CatS in neutrophilic precursor cells.

273 skin and colocalized with IL-36gamma around neutrophilic pustules.

274 Infection-negative neutrophilic RA was associated with an increase in level

275 A subgroup of patients with noninfected neutrophilic RA was associated with systemic inflammatio

276 colony-stimulating factor (G-CSF)-regulated neutrophilic response and prolonged inflammation.

277 This neutrophilic response augmented bacterial clearance and

278 atracheal administration of IL-17 provoked a neutrophilic response in the airways of WT and CF animal

279 s eosinophilic airway inflammation and acute neutrophilic response to inhaled LPS challenge in volunt

280 e of humans, characterized by a dysregulated neutrophilic response to specific bacterial species with

281 C3s, and gammadelta T cells and an increased neutrophilic response without increased inflammation or

282 and STAT6-deficient mice exhibit a primarily neutrophilic response.

283 ase in regulating the amplitude of the early neutrophilic response.

284 1beta) signal that is required for the rapid neutrophilic response.

285 receptor family member Nlrp12 in modulating neutrophilic responses during lethal IAV infection.

286 Additionally, all tissue-based neutrophilic responses to fungal infections necessitate

287 he endosymbiotic bacteria, Wolbachia, induce neutrophilic responses to the human helminth pathogen On

288 ion through the regulation of CXCL1-mediated neutrophilic responses.

289 otomous role for ATF3-mediated regulation of neutrophilic responses: inhibition of neutrophil chemoki

290 asome expression is highest in patients with neutrophilic SA.

291 ntly controlled development of TH17-mediated neutrophilic severe asthma in both acute and chronic HDM

292 odel, it has recently been demonstrated that neutrophilic skin inflammation promotes angiotropism and

293 ated MMP-8, MMP-9, and MPO suggests a common neutrophilic source and provides evidence of neutrophil

294 an alternative approach, we analyzed ex vivo neutrophilic superoxide inhibition in response to beta2

295 and relating genotypes to beta2 AR-mediated neutrophilic superoxide inhibition.

296 d with hematologic malignancies than classic neutrophilic Sweet syndrome.

297 n from eosinophilic T helper cell 2 (TH2) to neutrophilic TH17 polarity.

298 oles to collectively coordinate an effective neutrophilic transepithelial migratory response.

299 We describe 2 patients having neutrophilic urticaria with systemic inflammation (NUSI)

300 is characterized by hemorrhagic necrosis and neutrophilic vasculitis in the graft without preformed,