ライフサイエンスコーパス: nevus

1 were classified as having giant conjunctival nevus.

2 aracteristics of association with dysplastic nevus.

3 culty in differentiating melanoma from an EB nevus.

4 ts were initially diagnosed with benign iris nevus.

5 its clinical similarity to benign choroidal nevus.

6 normal skin and was absent from a congenital nevus.

7 t was willing, a biopsy of the most atypical nevus.

8 sor for cutaneous melanoma is the dysplastic nevus.

9 d a melanoma in situ arising in a dysplastic nevus.

10 , young adult, mid adult, senior adult) were nevus (25%, 36%, 47%, and 47%, respectively), IPE cyst (

11 Of 618 patients with conjunctival nevus, 32 (5%) were classified as having giant conjuncti

12 ase series of 618 patients with conjunctival nevus, 32 of which had giant conjunctival nevus, treated

13 ges, the most common specific diagnoses were nevus (42%), IPE cyst (19%), and melanoma (17%).

14 unctival tumor, the top 5 diagnoses included nevus (492 [61%]), benign reactive lymphoid hyperplasia

15 excisionally biopsied moderately dysplastic nevus 5 years later.

16 , young adult, mid adult, senior adult) were nevus (53%, 57%, 63%, and 63%, respectively) and melanom

17 mmon conjunctival tumors in children include nevus (64%), dermolipoma (5%), lymphangioma (3%) and cap

18 vus, choriocapillaris thinning overlying the nevus (94%), retinal pigment epithelial (RPE) atrophy (4

19 Of those with giant conjunctival nevus, a history of an increase in nevus base or thickne

20 osis nigricans, as an extension of epidermal nevus, after estrogen treatment, and/or in nevoid hyperk

21 were consistent with diagnoses of choroidal nevus, age-related macular degeneration, and multifocal

22 f students (n=274) had at least one new back nevus and 28% (n=103) had at least one nevus that disapp

23 n increased index of suspicion for choroidal nevus and choroidal melanoma in this population.

24 The association between choroidal nevus and earlier start and end to childbearing in preme

25 nt mice may serve as models for white sponge nevus and for understanding the role of K4 in cellular p

26 (congenital hemidysplasia with ichthyosiform nevus and limb defects), an X-linked, male lethal disord

27 ed with either vector was similar to that of nevus and melanoma cells and was independent of exogenou

28 directly test the role of activated BRAF in nevus and melanoma development, we have generated transg

29 RNA was extracted from FFPE sections from 14 nevus and melanoma samples via macrodissection.

30 ssue resources, and summarized the status of nevus and melanoma tissue microarrays, recently develope

31 clinical differential diagnosis included EB nevus and melanoma.

32 cular markers accurately distinguish between nevus and melanoma.

33 s mutations in TEK/Tie-2 in blue rubber bleb nevus and sporadic vascular malformations.

34 nevus, BRAFV600E mutation in the involuting nevus, and no malignant histopathologic characteristics

35 Juvenile xanthogranuloma (JXG) and nevus anemicus (NA) are commonly observed in children wi

36 Nevus anemicus was present on the neck and upper chest i

37 Assessment of the frequency of nevus-associated melanoma and the influence of patient-

38 Most nevus-associated melanomas were found on the trunk (67 [

39 In contrast, nevus-associated melanomas were found significantly less

40 analyses, reported as odds ratios (95% CIs), nevus-associated melanomas were found significantly more

41 ignificant influence on the manifestation of nevus-associated melanomas.

42 melanoma syndrome had a higher frequency of nevus-associated melanomas.

43 of a significantly increased probability of nevus-associated melanomas.

44 ally sized RCM images obtained from the same nevus at follow-up.

45 junctival nevus, a history of an increase in nevus base or thickness was noted in 15 cases (47%) and

46 ry of melanocytic tumors, encompassing Spitz nevus (benign), atypical Spitz tumor (intermediate malig

47 lationship between melanocyte growth stasis, nevus biology, and fibrogenic signaling was further vali

48 actice to comprehensively review melanocytic nevus biology.

49 s were diagnosed, most (72.2%) prior to blue nevus biopsy, including 38.9% in situ and 61.1% with mea

50 nevus showed BRAF wild type in the unchanged nevus, BRAFV600E mutation in the involuting nevus, and n

51 iate small choroidal melanoma from choroidal nevus can be remembered using the mnemonic TFSOM, indica

52 Choroidal nevus can show focal posterior scleral bowing on EDI-OCT

53 ched1 (PTCH1) are associated with basal cell nevus carcinoma syndrome (BCNS or Gorlin syndrome), whic

54 s counts at or above the median in all three nevus categories simultaneously when harboring a pathoge

55 23 of 25 (92%) nevi, predominantly in type C nevus cells and nevic corpuscles.

56 In contrast, MAP-2-positive dermal nevus cells and the invasive cells of primary melanomas

57 It remains intriguing how benign nevus cells can escape oncogene-induced senescence for m

58 However, nevus cells do not fully recapitulate a Schwann cell phe

59 1, and p27 showed that the majority of Spitz nevus cells expressed high levels of p16, with cells of

60 Melanocytic nevus cells in the dermis adopt many morphological featu

61 Melanocytic nevus cells in the dermis and desmoplastic melanomas oft

62 al keratinocytes, the appearance of nests of nevus cells surrounded by collagen fibers, and the struc

63 melanomas but is weaker and less frequent in nevus cells.

64 is case report discusses dynamic dermoscopic nevus changes that occur during dabrafenib therapy and o

65 mplete (35%) choroidal shadowing deep to the nevus, choriocapillaris thinning overlying the nevus (94

66 In children with a suspected Spitz nevus, clinical follow-up was chosen by 49.3% (69 of 140

67 Congenital melanocytic nevus (CMN) is a particular melanocytic in utero prolife

68 Congenital melanocytic nevus (CMN) syndrome is the association of pigmented mel

69 Change in nevus color over time has been noted in 5% of cases and

70 ore employed whole-exome sequencing (WES) in nevus comedonicus (NC), a rare disorder that features co

71 l cysts that bears a striking resemblance to Nevus Comedonicus.

72 elanocytic proliferations composed of both a nevus component with strong BAP1 expression and a large

73 On multivariate analysis, baseline total nevus count (adjusted odds ratio, 9.08; 95% CI, 4.0-23.7

74 s report baseline findings and predictors of nevus count (log total nevi) at the completion of year 1

75 idogen 1 (NID1) gene on 1q42 associated with nevus count (two linked single nucleotide polymorphisms

76 Nevus count and distribution in childhood were correlate

77 ns and patients should not rely on the total nevus count as a sole reason to perform skin examination

78 in diameter, 23 students (20.7%) with total nevus count in the top decile, and 19 students (17.1%) w

79 ation of holidays overseas with an increased nevus count in young white women, which was stronger in

80 e conclude that 66% of the total variance of nevus count is attributable to genetic effects: 7% assoc

81 High nevus count is the strongest risk factor for melanoma, a

82 [HR], 1.21; 95% CI, 1.02-1.44; P = .03), the nevus count of atypical nevi on the buttocks (HR, 14.00;

83 henotype in the 11th grade, defined as total nevus count of the back and 1 randomly selected leg in t

84 onducted a genome-wide association study for nevus count using 297,108 SNPs in 1,524 twins, with vali

85 Total nevus count was associated with skin and hair color and

86 on study single nucleotide polymorphisms for nevus count, at PLA2G6 (P = 1.7 x 10(-49)) and NID1 (P =

87 3 independent patient-related factors (high nevus count, low risk for melanoma, and female sex) and

88 tation carriage and 2-mm, 5-mm, and atypical nevus counts among blood-related members of melanoma fam

89 e interval = 1.75-4.82) as likely to exhibit nevus counts at or above the median in all three nevus c

90 At age 14, median back nevus counts increased by two; 75% of students (n=274) h

91 Atypical nevus counts were 0 (73.3% [n = 415]), 1 to 5 (14.5% [n

92 Total body nevus counts were collected in a cross-sectional study o

93 Nevus counts were, on average, higher in boys (mean = 98

94 ever lived overseas was not associated with nevus counts, but was inversely associated with number o

95 0K) mutations were also associated with high nevus counts.

96 s and are associated with increased atypical nevus counts.

97 79.2, adjusted to age 12 y, p < 0.0001), and nevus densities were higher on sun-exposed sites (92 per

98 Nevus density (ND) increased up to the age of 35 years a

99 Total nevus number (TNN) and total nevus density (TND) were determined.

100 red in hotter countries than in the UK (mean nevus density 41 per m2 in those in the highest quartile

101 We report a correlation between nevus density and sun exposure, particularly that acquir

102 clinical skin examinations was self-reported nevus density at 21 years of age, whereas a family histo

103 Correlations in sex and age adjusted nevus density were higher in MZ pairs (0.94, 95%CI 0.92-

104 e risk prediction model included hair color, nevus density, first-degree family history of melanoma,

105 atric conventional melanoma (CM), congenital nevus-derived melanoma (CNM), and Spitzoid melanoma (SM)

106 23.7; P < .001) and increased variability of nevus dermoscopic pattern (adjusted odds ratio, 4.24; 95

107 ng Ab to IFN-gamma, but not IL-17, inhibited nevus development (p < 0.01).

108 induces benign, growth-arrested melanocytic nevus development, but also drives melanoma formation.

109 y provide both novel and replicated QTLs for nevus development, some of which might overlap with thos

110 anocyte homeostasis, whereas IL-12 supported nevus development.

111 The investigation of nevus distribution by anatomic site has led to interesti

112 Deep penetrating nevus (DPN) is characterized by enlarged, pigmented mela

113 ts: tissue sections of melanoma arising in a nevus; dysplastic nevi; Spitz nevi; and misdiagnosed mel

114 One choroidal nevus each from 30 eyes of 30 patients was included and

115 ficantly associated with history of atypical nevus excision (odds ratio [OR], 2.9; 95% CI, 1.5-5.7);

116 Genes that determine nevus expression may therefore act as low penetrance mel

117 arched eyebrows, hypertelorism, a glabellar nevus flammeus, neonatal feeding difficulties, hypotonia

118 nce that BRAF activation is sufficient for f-nevus formation, that BRAF activation is among the prima

119 ma, and retinal thinning overlying choroidal nevus; fresh subretinal fluid with preservation of photo

120 ion is a robust biomarker for distinguishing nevus from melanoma.

121 t the AMS is genetically determined and that nevus genes are also low penetrance melanoma susceptibil

122 e (</= 5 x 5-mm field-of-view) from the same nevus; good agreement was defined as annotations deviant

123 hase 2 trial in patients with the basal-cell nevus (Gorlin) syndrome indicating that the smoothened i

124 carcinomas, including those with basal-cell nevus (Gorlin) syndrome, need extended treatment.

125 -prone phenotype: 69 students (62.2%) with 1 nevus greater than 5 mm in diameter, 23 students (20.7%)

126 Factors predictive of iris nevus growth to melanoma by multivariable analysis inclu

127 By Kaplan-Meier estimates, iris nevus growth to melanoma occurred in <1%, 3%, 4%, 8%, an

128 h median age 69 years and baseline choroidal nevus higher or lower than 1.5 mm in 51 (14%) and 307 (8

129 s of 318 consecutive patients with choroidal nevus imaged over a 1-year period revealed that 17 cases

130 Of 104 eyes with choroidal nevus imaged with EDI OCT, 51 (49%) displayed image deta

131 alignant melanoma developed within the giant nevus in 1 case after 23 years of observation.

132 The odds of choroidal nevus in postmenopausal women who were overweight and ob

133 ved in the family sample (mean of 1 atypical nevus in those with the allele and 3.5 nevi in those wit

134 ed profound differences in susceptibility to nevus initiation, transformation, tumorigenicity, and me

135 Evolution of conjunctival nevus into malignant melanoma is extremely low (<1%).

136 The giant nevus involved cornea in 11 cases (34%), limbus in 23 (7

137 Spitz nevus is a benign melanocytic neoplasm that can be diffi

138 Spitz nevus is a benign neoplasm of melanocytes that can be di

139 Choroidal nevus is a precursor for uveal melanoma and there are no

140 n of posterior scleral bowing with choroidal nevus is essential to avoid an underestimation of tumor

141 ire fundus, the true prevalence of choroidal nevus is even higher but difficult to accurately estimat

142 Conjunctival nevus is the most common conjunctival tumor in children

143 Conjunctival nevus is the most common tumor of the ocular surface and

144 ng was related to more posterior location of nevus, less/mixed pigmentation, and surrounding halo.

145 histopathologic features characterized by a nevus like silhouette and cytologic composition of large

146 Among US adults, the prevalence of choroidal nevus located within two 45 degrees areas centered on th

147 hickness was not measurable; however, at the nevus margin, the choroid and sclera appeared normal.

148 vivo, depletion of histones correlated with nevus maturation, an established histopathologic paramet

149 By EDI-OCT, the nevus mean thickness was 628 microm (0.63 mm).

150 n classification errors included intradermal nevus misclassified as basal cell carcinoma and nonmelan

151 The melanocytic tumors comprised nevus (n = 1503; 60%), melanocytoma (n = 68; 3%), melano

152 choristoma (n = 2; 5%), gliosis (n = 1; 3%), nevus (n = 1; 3%), hemorrhage (n = 1; 3%), and leiomyoma

153 and group VI, melanoma arising in congenital nevus (n = 6).

154 Total nevus number (TNN) and total nevus density (TND) were de

155 irst report of common variants associated to nevus number and demonstrates association of these varia

156 wed strong evidence for genetic influence on nevus number and density.

157 On the basis of nevus number and type, clinicians can identify a populat

158 io 4.2; confidence interval 1.4-12.5), total nevus number being at least 100 (nevi > or = 2 mm in dia

159 no association between Ala148Thr status and nevus number or history of melanoma, and therefore the r

160 e polymorphism controls the nevus phenotype (nevus number, number atypical nevi or AMS phenotype).

161 phenotypical data were compared with data on nevus numbers in adulthood.

162 res include a well-demarcated hairless fatty nevus on the scalp, benign ocular tumors, and central ne

163 h TSC (56%) had at least 1 connective tissue nevus on the trunk or thighs; of these, 28 of 58 patient

164 c deletion was not observed in a melanocytic nevus or acrochordon from patients with MEN1.

165 ons in the validation sets were diagnosed as nevus or melanoma, and the results were compared with th

166 sociated variants showed no association with nevus or pigmentation phenotypes in a large British case

167 ; and extant findings of at least 1 atypical nevus (OR, 2.2; 95% CI, 1.3-3.9); atypical nevus pattern

168 e intense choroidal shadowing with pigmented nevus (P = 0.046).

169 gender (P = 0.008), extramacular location of nevus (P<0.001), mean distance from foveola more than 3

170 l nevus (OR, 2.2; 95% CI, 1.3-3.9); atypical nevus pattern (>/=20 nevi that were >/=2 mm in diameter)

171 porokeratotic eccrine ostial and dermal duct nevus (PEODDN) tissue but absent in unaffected skin.

172 imulate choroidal melanoma include choroidal nevus, peripheral exudative hemorrhagic chorioretinopath

173 evidence that the polymorphism controls the nevus phenotype (nevus number, number atypical nevi or A

174 nevogenic, although the correlation between nevus phenotype and mutation status is poor.

175 m 179 families of patients with the atypical nevus phenotype and/or a family history of melanoma, and

176 Non-carriers showed an intermediate nevus phenotype between mutation carriers and spouse con

177 f melanocytic nevi so that genes controlling nevus phenotype could be such melanoma susceptibility ge

178 An intermediate nevus phenotype in non-carrier family members suggests t

179 Variation in the nevus phenotype within a population is predominantly gen

180 There was a marginal relationship with nevus phenotype, but this was no longer statistically si

181 hether the XRCC3 polymorphism influences the nevus phenotype.

182 athogenic CDKN2A mutation carriage and other nevus phenotypes including counts of common acquired nev

183 from the diagnostic categories (conjunctival nevus, primary acquired melanosis with mild or no atypia

184 Secondary outcomes included local nevus recurrence and development of primary melanoma at

185 vation group were more likely to demonstrate nevus recurrence than those that were reexcised (3.3% vs

186 Postexcisional biopsy, nevus recurrence was detected in 4 cases (17%), pseudopt

187 Management, nevus recurrence, and nevus transformation into melanoma

188 In an analysis of 1611 cases of iris nevus referred for evaluation at an ocular oncology cent

189 d continued to develop nevi rather than show nevus regression seen in non-carriers and spouse control

190 ocular oncology practice, giant conjunctival nevus represents 5% of conjunctival nevi.

191 f a melanoma in situ arising in a dysplastic nevus revealed a phenotype-genotype paradox that confoun

192 P = .6), and hyporeflective gradation at the nevus-scleral interface (P = .33).

193 dering vessels (P = 1), visualization of the nevus-scleral interface (P = .6), and hyporeflective gra

194 Visualization of the complete nevus-scleral interface was significantly (P = .02) more

195 ons such as keratinocytic epidermal nevi and nevus sebaceous result from somatic mosaicism.

196 The risk of basal cell carcinoma within nevus sebaceus appears to be low, but more conclusive da

197 authors have advocated the prompt removal of nevus sebaceus at a young age due to the risk of maligna

198 the actual incidence of malignant growths in nevus sebaceus is quite rare in young patients.

199 common 'lumps and bumps' seen in childhood: nevus sebaceus, pilomatricoma and juvenile xanthogranulo

200 cuss treatment options for melanocytic nevi, nevus sebaceus, port-wine stains, and hemangiomas.

201 BRAF wild-type results, while the dysplastic nevus showed both BRAF wild-type and BRAF V600E mutation

202 sy findings from 1 unchanged and 1 involuted nevus showed BRAF wild type in the unchanged nevus, BRAF

203 comparison of pigmented versus nonpigmented nevus showed only 1 significant difference of more inten

204 has been noted in 5% of cases and change in nevus size has been documented in 7%.

205 a case of agminated Spitz nevi arising in a nevus spilus and use exome sequencing to identify a clon

206 ults reveal an activating HRAS mutation in a nevus spilus that predisposes to the formation of Spitz

207 taneously, in association with an underlying nevus spilus, or after radiation or chemotherapy.

208 (c.37G-->C) in the Spitz nevi and underlying nevus spilus.

209 a high percentage of melanomas arising in a nevus, Spitz nevi, dysplastic nevi, and misdiagnosed les

210 c patterns may represent distinct biological nevus subsets.

211 py at roughly 7-month intervals as part of a nevus surveillance study.

212 of Ala148Thr as a low penetrance melanoma or nevus susceptibility allele in two separate groups of in

213 8Thr variant is a low penetrance melanoma or nevus susceptibility allele.

214 y influence both melanoma susceptibility and nevus susceptibility.

215 ll carcinoma (aBCC) and basal cell carcinoma nevus syndrome (BCCNS), a rare genetic disorder that gre

216 e sporadic, rare individuals with basal cell nevus syndrome (BCNS) harbor germline defects in PTCH1 a

217 Patients with basal cell nevus syndrome (BCNS) have a greater risk of developing

218 Importance: Patients with basal cell nevus syndrome (BCNS) have a greater risk of developing

219 Basal cell nevus syndrome (BCNS), is a hereditary condition transmi

220 In addition, basal cell nevus syndrome (BCNS), or Gorlin syndrome, which is char

221 ct more than 65% of patients with basal cell nevus syndrome (BCNS).

222 ssor protein that is defective in basal cell nevus syndrome (BCNS).

223 Blue rubber bleb nevus syndrome (Bean syndrome) is a rare, severe disorde

224 cally treated patients with blue rubber bleb nevus syndrome (BRBNS).

225 The coexistence of dysplastic nevus syndrome and a BAP1 germline mutation extends the

226 atocysts are one component of the basal cell nevus syndrome and all patients with odontogenic keratoc

227 atic orbital or periocular BCC or basal cell nevus syndrome and can obviate orbital exenteration in s

228 After exclusion of patients with basal cell nevus syndrome and immunocompromise, 1284 patients (90.0

229 of the gene in patients with the basal cell nevus syndrome and in sporadic basal cell carcinomas.

230 plastic nevi in patients with the dysplastic nevus syndrome and to determine the discriminatory profi

231 rolled trial in patients with the basal-cell nevus syndrome at three clinical centers from September

232 Patients with basal cell nevus syndrome have a high incidence of multiple basal c

233 Research Institute and Basal Cell Carcinoma Nevus Syndrome Life Support Network.

234 cated in eight exons in 13 of the basal cell nevus syndrome patients and in three of the tumors.

235 ymorphism analysis of DNA from 86 basal cell nevus syndrome probands, 26 sporadic basal cell carcinom

236 w-up time, immunosuppression, and basal cell nevus syndrome status.

237 P < .001), accounting for age and basal cell nevus syndrome status.

238 atic orbital or periocular BCC or basal cell nevus syndrome treated with the Hedgehog pathway inhibit

239 r patients such as those with the basal cell nevus syndrome who are destined to develop large numbers

240 ed patients aged 35-75 years with basal-cell nevus syndrome with at least ten surgically eligible bas

241 found in human patients with the basal cell nevus syndrome, a disease causing developmental defects

242 gene (PTCH) in patients with the basal-cell nevus syndrome, a hereditary disease characterized by mu

243 patient with multiple melanomas, dysplastic nevus syndrome, and an inactivating germline BAP1 mutati

244 nevus syndrome, familial melanoma/dysplastic nevus syndrome, and xeroderma pigmentosum.

245 the skin develop many features of basal cell nevus syndrome, demonstrating that SHH is sufficient to

246 es of three hereditary syndromes: basal cell nevus syndrome, familial melanoma/dysplastic nevus syndr

247 anoma, dysplastic nevi, Spitz nevi, atypical nevus syndrome, family history of melanoma only, and oth

248 nt was stratified by diagnosis of basal-cell nevus syndrome, geographical region, and immunosuppressi

249 history of invasive melanoma and dysplastic nevus syndrome, history of invasive melanoma and at leas

250 o was initially seen in 2003 with dysplastic nevus syndrome, multiple atypical melanocytic proliferat

251 carcinomas, including those with basal-cell nevus syndrome, who had one or more histopathologically

252 basal cell carcinomas, and seven basal cell nevus syndrome-associated basal cell carcinomas.

253 e cutaneous melanomas and classic dysplastic nevus syndrome.

254 hy, neurofibromatosis type 2, and basal cell nevus syndrome.

255 l carcinomas in patients with the basal-cell nevus syndrome.

256 ional inactivation of PTCH in the basal cell nevus syndrome.

257 utational inactivation of PTCH in basal-cell nevus syndrome.

258 n 15 of 17 individuals with blue rubber bleb nevus syndrome.

259 carcinoma growth in patients with basal-cell nevus syndrome.

260 ma tumour burden in patients with basal-cell nevus syndrome.

261 other features characteristic of basal cell nevus syndrome.

262 s had locally advanced BCC; 2 had basal cell nevus syndrome.

263 r for moderately dysplastic nevi or atypical nevus syndrome; biannually for up to 3 years, then annua

264 d more than 4 times higher odds of choroidal nevus than those who first gave birth after age 35 years

265 had nearly 5 times higher odds of choroidal nevus than those who gave birth to their last child afte

266 back nevus and 28% (n=103) had at least one nevus that disappeared.

267 he contribution of the ugly duckling sign (a nevus that is obviously different from the others in a g

268 umans lead to a disorder called white sponge nevus, the K4-deficient mice may serve as models for whi

269 Underlying the nevus, the scleral thickness was not measurable; however

270 Of the 51 suitable cases, mean nevus thickness was 685 mum (median, 628 mum; range, 184

271 TPMs acquired at the transition from benign nevus to malignant melanoma do not support telomere main

272 Growth of iris nevus to melanoma was confirmed in 2% of eyes (n = 27) o

273 iate stage in the step-wise progression from nevus to melanoma.

274 a general biomarker for the transition from nevus to melanoma.

275 Management, nevus recurrence, and nevus transformation into melanoma.

276 al nevus, 32 of which had giant conjunctival nevus, treated at an ocular oncology service between Jul

277 e patient refused to undergo excision of the nevus until we made excision conditional for continued l

278 cept, human skin measurements on melanocytic nevus, vitiligo, and venous occlusion conditions were pe

279 The weighted prevalence of choroidal nevus was 4.5%.

280 The prevalence of choroidal nevus was 4.7% overall and increased with age (4.7%, 3.1

281 The mean age at referral for iris nevus was 51 years (median, 54; range, <1-94 years).

282 One clinically dysplastic nevus was associated with a 2-fold risk (95% confidence

283 Choroidal nevus was associated with hypertension (odds ratio [OR],

284 The nevus was classified as pigmented (n = 3; 18%), nonpigme

285 The nevus was clinically estimated to be of 4.91-mm basal di

286 The thickness of choroidal nevus was measured by combining Heidelberg's autosegment

287 Half of each nevus was protected by either a physical barrier or a su

288 whites, the presence of at least 1 perianal nevus was significantly associated with history of atypi

289 Melanoma, compared with nevus, was associated with older children (aged >10-15 y

290 ting for age and race, the odds of choroidal nevus were 10-fold higher in whites than in blacks, 5-fo

291 logic differences between the halves of each nevus were demonstrable even when in vivo examination de

292 re solar elastosis, and lack of a coexisting nevus were independently associated with amelanotic mela

293 l records of 12 patients with divided eyelid nevus were reviewed with attention to presenting feature

294 Eight patients with divided nevus were treated with a staged surgical approach: 5 pr

295 105N-T1106P is recurrent in blue rubber bleb nevus, whereas Y897C-R915C is recurrent in sporadically

296 nin pathway change the phenotype of a common nevus with BRAF mutation into that of DPN, with increase

297 Imaging of choroidal nevus with EDI OCT enables precise measurement of tumor

298 ant melanoma that developed on a preexisting nevus within a tattoo during and between the phases of l

299 White sponge nevus (WSN) is an autosomal-dominantly inherited form of

300 mutated in human patients with white sponge nevus (WSN).