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1 e skin barrier defect and premature death of newborn animals.
2                 SC integrity was abnormal in newborn animals.
3 n spiking is observed in young as opposed to newborn animals.
4 ter acute barrier disruption were delayed in newborn animals.
5 s of the stressed mother and siblings on the newborn animals.
6 and from models of brain damage in adult and newborn animals.
7 f gestation and are no longer present in the newborn animals.
8 pha-mediated growth arrest of hepatocytes in newborn animals.
9 hat ultimately produces and delivers milk to newborn animals.
10 ha 1 in the c-ret -/- colon in either E15 or newborn animals.
11 expression of normal respiratory behavior in newborn animals.
12 r V, a band of labeled puncta present in the newborn animals also increased in density until 3 to 4 y
13 ms controlling deficient immune responses in newborn animals are not well understood.
14 s of the loss of these genes were studied in newborn animals because mice lacking Kir2.1 have a cleft
15 ears to be responsible for CD degradation in newborn animals, because serine protease activity is inc
16 esses a transgene (lacZ) in muscle fibers of newborn animals but cannot efficiently penetrate adult m
17  in long dendrites, similar to the case with newborn animals but with f(c)= 20Hz.
18 receptors are expressed in Purkinje cells of newborn animals, but are lost after 2 weeks.
19 r the properties of new neurons generated in newborn animals differ from those added during adulthood
20 a low dose of halofuginone both in adult and newborn animals for 60 d prevented the development of cu
21 RP inhibitor, would reduce brain injury in a newborn animal model.
22 CR1(-/-) mice were normal, in both fetus and newborn animals neutrophil counts were significantly dep
23 om animals aged P4 to P5 and P9 to P11, i.e. newborn animals, showed that the subthreshold membrane i
24 c islet mRNA levels were already apparent in newborn animals, suggesting that loss of Hnf-1alpha func
25 cells were the only lymphocyte population in newborn animals that bound E-selectin chimera.
26        Such plasticity has adaptive value to newborn animals that must fend for themselves from birth
27 l and middle layers was extremely low in the newborn animals, then increased more than 10-fold to adu
28 ition from solely passive decay in vMNs from newborn animals to resonance in young animals coincides
29                 Similarly, when tissues from newborn animals were examined, Hmgi(y) expression was re
30                                       Thirty newborn animals were exposed to 95% to 100% oxygen for 4
31 tion in spider monkeys, immunosuppressed and newborn animals were inoculated.
32                         Topical treatment of newborn animals with a PPARalpha activator increased sec
33              Treatment of muscles taken from newborn animals with c-Kit antibodies blocked postnatal
34 onstrated decreased corneodesmosomes (CD) in newborn animals with decreased expression of desmoglein
35               Intraperitoneal inoculation of newborn animals with murine cytomegalovirus resulted in
36 cessarily derive from the use of abortive or newborn animals with ultrathin hides, but could equally
37 ion by a maternal factor led us to treat the newborn animals with various pregnancy-related hormones

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