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1 show that extracts from this strain lack the nicking activity.
2 at can serve as a substrate for Rep-mediated nicking activity.
3 midine junctions greatly reduce or eliminate nicking activity.
4 activity and terminal resolution site (trs) nicking activity.
5 or head-to-tail repeat only supported a DNA nicking activity.
6 th significant strand- and sequence-specific nicking activity.
7 8 is the active form for both the ATPase and nicking activities.
8 r is responsible for both mobility shift and nicking activities.
9 safe harbor" locus revealed similar in vitro nicking activity, a marked reduction of indels character
10 ved amino acid residues of L1 EN abolish its nicking activity and eliminate L1 retrotransposition.
11 previous mutants that effect this processive nicking activity, and the recently published cocrystal s
12 erated at the hairpin structure by a hairpin-nicking activity, as previously detected in somatic cell
13 gent, displaying significant supercoiled DNA-nicking activity at concentrations as low as 1 microM.
15 e first time, we show Rep protein off-target nicking activity, highlighting the importance of the nic
18 ntal evidence, we propose a model where RepC nicking activity is passive and dependent upon the super
20 -modified DNA in Ni2+ buffer even though the nicking activity is sub-optimal compared to the activity
22 fication method based on the primer-directed nicking activity of a restriction enzyme and the strand
24 d to a single strand break, Ku inhibited the nicking activity of human endonuclease III as well as th
30 nt degree of DNA protection against cleaving/nicking activity of various isoschizomeric endonucleases
31 and D252N) retained near wild type levels of nicking activity on abasic (AP) site-containing DNA, G83
32 17N displayed somewhat diminished processive nicking activities relative to that of the wild-type enz
34 s in these composite gel patterns arise from nicking activity that converts replication bubbles to br
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