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1 show that extracts from this strain lack the nicking activity.
2 at can serve as a substrate for Rep-mediated nicking activity.
3 midine junctions greatly reduce or eliminate nicking activity.
4  activity and terminal resolution site (trs) nicking activity.
5  or head-to-tail repeat only supported a DNA nicking activity.
6 th significant strand- and sequence-specific nicking activity.
7 8 is the active form for both the ATPase and nicking activities.
8 r is responsible for both mobility shift and nicking activities.
9 safe harbor" locus revealed similar in vitro nicking activity, a marked reduction of indels character
10 ved amino acid residues of L1 EN abolish its nicking activity and eliminate L1 retrotransposition.
11 previous mutants that effect this processive nicking activity, and the recently published cocrystal s
12 erated at the hairpin structure by a hairpin-nicking activity, as previously detected in somatic cell
13 gent, displaying significant supercoiled DNA-nicking activity at concentrations as low as 1 microM.
14 han the inhibition of human endonuclease III-nicking activity by Ku.
15 e first time, we show Rep protein off-target nicking activity, highlighting the importance of the nic
16 to the H-N-H endonuclease family and possess nicking activity in vitro.
17       ZFNickases show robust strand-specific nicking activity in vitro.
18 ntal evidence, we propose a model where RepC nicking activity is passive and dependent upon the super
19                                          The nicking activity is similar between the double-stranded
20 -modified DNA in Ni2+ buffer even though the nicking activity is sub-optimal compared to the activity
21 four others, increased DNA unwinding and Chi nicking activities of the enzyme.
22 fication method based on the primer-directed nicking activity of a restriction enzyme and the strand
23                                          The nicking activity of AAV-2's Rep68 or Rep78 proteins is e
24 d to a single strand break, Ku inhibited the nicking activity of human endonuclease III as well as th
25                                       The AP nicking activity of the C136R variant was significantly
26                                 The residual nicking activity of the R621H mutant is reduced at least
27                                      The DNA nicking activity of the wild-type enzyme is still activa
28 n sites on dsDNA generates in some cases the nicking activity of these DNA cutters.
29            Furthermore, we show that the DNA nicking activity of TOP1 is a prerequisite for robust eR
30 nt degree of DNA protection against cleaving/nicking activity of various isoschizomeric endonucleases
31 and D252N) retained near wild type levels of nicking activity on abasic (AP) site-containing DNA, G83
32 17N displayed somewhat diminished processive nicking activities relative to that of the wild-type enz
33        Mutation at mcb2 abolished detectable nicking activity, suggesting that binding of this site b
34 s in these composite gel patterns arise from nicking activity that converts replication bubbles to br
35               As an essential feature of its nicking activity, TraI is capable of binding and cleavin
36                                    In short, nicking activity was maximized close to physiological sa
37                                  Glycosylase nicking activities were measured on both thymine glycol-

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