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1 e phosphate to form R-[4-xH]-labeled reduced nicotinamide adenine dinucleotide phosphate.
2 ratios of the reduced and oxidized forms of nicotinamide adenine dinucleotide phosphate.
3 patches in the presence of 0.5-1 mM reduced nicotinamide adenine dinucleotide phosphate.
4 y nicotinamide adenine dinucleotide, but not nicotinamide adenine dinucleotide phosphate.
5 ADPH, by the negatively charged hydride from nicotinamide adenine dinucleotide phosphate.
6 nd absence of cofactors (the reduced form of nicotinamide adenine dinucleotide phosphate and S-adenos
7 OS1 protein used the substrates arginine and nicotinamide adenine dinucleotide phosphate and was acti
8 which match the previously described reduced nicotinamide-adenine dinucleotide phosphate and superoxi
9 ing the cofactors adenosine triphosphate and nicotinamide adenine dinucleotide phosphate, and have be
10 derivatives in complex with PfIspC, reduced nicotinamide adenine dinucleotide phosphate, and Mg(2+)
11 ion, depleting and oxidizing glutathione and nicotinamide adenine dinucleotide phosphate, and signifi
12 s the reduction of glucose to sorbitol using nicotinamide adenine dinucleotide phosphate as an essent
13 to synthesize cholesterol and obtain reduced nicotinamide adenine dinucleotide phosphate by the malic
14 ococcus furiosus uses electrons from reduced nicotinamide adenine dinucleotide phosphate, by way of r
15 on and activation of cytochrome P450 and the nicotinamide adenine dinucleotide phosphate complex were
16 radicals produced by cytochrome P450 and the nicotinamide adenine dinucleotide phosphate complexes co
17 f the chemotherapeutic drugs (DT-diaphorase, nicotinamide adenine dinucleotide phosphate:cytochrome P
20 ide polymorphisms (SNPs) in the cytoplasmic, nicotinamide adenine dinucleotide phosphate-dependent, t
21 ltransferase (ChAT) immunocytochemistry, and nicotinamide adenine dinucleotide phosphate diaphorase (
22 n significantly and preferentially increased nicotinamide adenine dinucleotide phosphate diaphorase (
25 , P15, and P21 and adults were examined with nicotinamide adenine dinucleotide phosphate diaphorase (
26 ntibody directed against nNOS and stained by nicotinamide adenine dinucleotide phosphate diaphorase (
27 d mollusc Clione limacina were studied using nicotinamide adenine dinucleotide phosphate diaphorase (
28 complished this by characterizing regions of nicotinamide adenine dinucleotide phosphate diaphorase (
29 mmunocytochemically, and colocalization with nicotinamide adenine dinucleotide phosphate diaphorase (
30 al polypeptide (VIP)-immunoreactivities, and nicotinamide adenine dinucleotide phosphate diaphorase (
31 (Arg) and histochemical staining for reduced nicotinamide adenine dinucleotide phosphate diaphorase (
32 istochemical method for demonstration of the nicotinamide adenine dinucleotide phosphate diaphorase (
34 ion of endothelial nitric oxide synthase and nicotinamide adenine dinucleotide phosphate diaphorase r
35 s analyzed by Northern analysis, and reduced nicotinamide adenine dinucleotide phosphate diaphorase w
36 gnificantly decreased the number of NADPH-d (nicotinamide adenine dinucleotide phosphate diaphorase)
37 projection cells contained serotonin but not nicotinamide adenine dinucleotide phosphate diaphorase.
38 these two populations of neurons using Beta-nicotinamide adenine dinucleotide phosphate-diaphorase (
39 this study, we used two different techniques-nicotinamide adenine dinucleotide phosphate-diaphorase (
40 unocytochemistry, in situ hybridization, and nicotinamide adenine dinucleotide phosphate-diaphorase (
41 yltransferase (ChAT) immunohistochemical and nicotinamide adenine dinucleotide phosphate-diaphorase (
43 e performed for acetylcholinesterase (AChE), nicotinamide adenine dinucleotide phosphate-diaphorase (
45 S with a monoclonal antibody as well as beta-nicotinamide adenine dinucleotide phosphate-diaphorase h
46 ase activity measured by L-citrulline assay, nicotinamide adenine dinucleotide phosphate-diaphorase s
47 ements of the FAD and NADPH (reduced form of nicotinamide adenine dinucleotide phosphate) domains.
48 icotinamide adenine dinucleotide phosphate / nicotinamide adenine dinucleotide phosphate , flavin ade
49 NADPH/NADP(+) (the reduced form of NADP(+)/nicotinamide adenine dinucleotide phosphate) homeostasis
50 production in guard cells, generated by the nicotinamide adenine dinucleotide phosphate hydrogen (NA
51 ependent protein kinase II), which decreases nicotinamide adenine dinucleotide phosphate hydrogen syn
52 bound enzyme-substrate complexes and reduced nicotinamide adenine dinucleotide phosphate induced by t
53 produce peroxide, cyclic ADP-ribose (cADPR), nicotinamide adenine dinucleotide phosphate (NAADP) and
54 cleus and that this source of ROS is reduced nicotinamide adenine dinucleotide phosphate (NAD(P)H) ox
55 nhibitor, diphenyleneiodonium (DPI), reduced nicotinamide adenine dinucleotide phosphate (NAD(P)H) ox
56 translocation of the cytosolic components of nicotinamide adenine dinucleotide phosphate (NAD(P)H)-ox
57 rtas of wild-type mice or mice deficient for nicotinamide adenine dinucleotide phosphate [NAD(P)H] ox
58 olism via endogenous fluorescence of reduced nicotinamide adenine dinucleotide (phosphate) (NAD(P)H)
60 e adenine dinucleotide (NAD(+) and NADH) and nicotinamide adenine dinucleotide phosphate (NADP(+) and
61 (+) and NADH), oxidized and reduced forms of nicotinamide adenine dinucleotide phosphate (NADP(+) and
62 drofolate reductase (DHFR) with its cofactor nicotinamide adenine dinucleotide phosphate (NADP(+)) ha
63 glucose-6-phosphate dehydrogenase (G6PD) and nicotinamide adenine dinucleotide phosphate (NADP(+)); t
65 cificity for all-trans retinol and preferred nicotinamide adenine dinucleotide phosphate (NADP) as th
66 hylogenetic analysis reveals that the use of nicotinamide adenine dinucleotide phosphate (NADP) by pr
67 as a coenzyme, they can be engineered to use nicotinamide adenine dinucleotide phosphate (NADP) inste
69 emical and in vivo experiments suggests that nicotinamide adenine dinucleotide phosphate (NADP) plays
70 tions across the enzyme network that reduces nicotinamide adenine dinucleotide phosphate (NADP) to NA
71 m nicotinamide adenine dinucleotide (NAD) to nicotinamide adenine dinucleotide phosphate (NADP), alwa
72 ing Tris, Triton X-100, glucose-6-phosphate, nicotinamide adenine dinucleotide phosphate (NADP), phen
73 Nicotinamide adenine dinucleotide (NAD) and nicotinamide adenine dinucleotide phosphate (NADP), whic
74 yzes the fourth reaction in the pathway, the nicotinamide adenine dinucleotide phosphate- (NADP-) dep
75 n-dependent, reductive activation of sorghum nicotinamide adenine dinucleotide phosphate- (NADP-) dep
76 oduction of reactive oxygen species (ROS) by nicotinamide adenine dinucleotide phosphate (NADPH) 2 (N
77 e) into chlorophyllide (Chlide) with reduced nicotinamide adenine dinucleotide phosphate (NADPH) as a
78 dehyde as substrates and the reduced form of nicotinamide adenine dinucleotide phosphate (NADPH) as c
80 tinamide adenine dinucleotide (NADH) or beta-nicotinamide adenine dinucleotide phosphate (NADPH) cofa
81 ed, as were interneurones containing reduced nicotinamide adenine dinucleotide phosphate (NADPH) diap
82 s both flavin adenine dinucleotide (FAD) and nicotinamide adenine dinucleotide phosphate (NADPH) for
83 ophosphate pathway (HMP), the sole source of nicotinamide adenine dinucleotide phosphate (NADPH) in e
84 to support the synthesis and regeneration of nicotinamide adenine dinucleotide phosphate (NADPH) in p
85 dinucleotide (FAD) in the enzyme by reduced nicotinamide adenine dinucleotide phosphate (NADPH) in r
87 each strain when cellular demand for reduced nicotinamide adenine dinucleotide phosphate (NADPH) is g
88 cofactor is even less stable (i.e., reduced nicotinamide adenine dinucleotide phosphate (NADPH) is m
89 t role through preservation of intracellular nicotinamide adenine dinucleotide phosphate (NADPH) leve
93 logic stretch rapidly activates reduced-form nicotinamide adenine dinucleotide phosphate (NADPH) oxid
95 However, gp91(phox) and p22(phox) reduced nicotinamide adenine dinucleotide phosphate (NADPH) oxid
96 blast function, activating integrin-mediated nicotinamide adenine dinucleotide phosphate (NADPH) oxid
97 '-triphosphate (ATP) production, and reduced nicotinamide adenine dinucleotide phosphate (NADPH) oxid
99 lomatous disease (CGD), defective phagocytic nicotinamide adenine dinucleotide phosphate (NADPH) oxid
100 initiated by the respiratory burst, in which nicotinamide adenine dinucleotide phosphate (NADPH) oxid
101 8-mediated ROS was generated through reduced nicotinamide adenine dinucleotide phosphate (NADPH) oxid
102 form via a unique death pathway signaled by nicotinamide adenine dinucleotide phosphate (NADPH) oxid
103 ivation of phosphoinositide-3 (PI-3) kinase, nicotinamide adenine dinucleotide phosphate (NADPH) oxid
105 ession and activity of the calcium-dependent nicotinamide adenine dinucleotide phosphate (NADPH) oxid
107 evaluated superoxide production by neuronal nicotinamide adenine dinucleotide phosphate (NADPH) oxid
108 he hypotheses that UVA-induced activation of nicotinamide adenine dinucleotide phosphate (NADPH) oxid
109 xide due to activation in neurons of reduced nicotinamide adenine dinucleotide phosphate (NADPH) oxid
110 at reactive oxygen species (ROS) produced by nicotinamide adenine dinucleotide phosphate (NADPH) oxid
111 reactive oxygen species (ROS) by the reduced nicotinamide adenine dinucleotide phosphate (NADPH) oxid
112 creasing superoxide production from vascular nicotinamide adenine dinucleotide phosphate (NADPH) oxid
113 Superoxide produced by the phagocyte reduced nicotinamide adenine dinucleotide phosphate (NADPH) oxid
114 2O2 derived from the interaction of platelet nicotinamide adenine dinucleotide phosphate (NADPH) oxid
117 cells expressed a functional phagocyte-type nicotinamide adenine dinucleotide phosphate (NADPH) oxid
118 eases in renal mRNA for superoxide-producing nicotinamide adenine dinucleotide phosphate (NADPH) oxid
119 g reactive oxygen species (ROS) from reduced nicotinamide adenine dinucleotide phosphate (NADPH) oxid
120 A novel isoform of the NOX-2 subunit of nicotinamide adenine dinucleotide phosphate (NADPH) oxid
121 nt pathway that negatively regulates reduced nicotinamide adenine dinucleotide phosphate (NADPH) oxid
122 ne triphosphatase (GTPase)-regulated reduced nicotinamide adenine dinucleotide phosphate (NADPH) oxid
123 chemotaxis, and superoxide production by the nicotinamide adenine dinucleotide phosphate (NADPH) oxid
124 chloride-sensitive calcium-dependent reduced nicotinamide adenine dinucleotide phosphate (NADPH) oxid
125 sence of the phagocyte superoxide-generating nicotinamide adenine dinucleotide phosphate (NADPH) oxid
126 used by defects in the superoxide-generating nicotinamide adenine dinucleotide phosphate (NADPH) oxid
127 munohistochemical localization of neutrophil nicotinamide adenine dinucleotide phosphate (NADPH) oxid
128 when it was added before assembly of reduced nicotinamide adenine dinucleotide phosphate (NADPH) oxid
129 iency of the superoxide-generating phagocyte nicotinamide adenine dinucleotide phosphate (NADPH) oxid
130 (PMNs) requires recruitment of a functional nicotinamide adenine dinucleotide phosphate (NADPH) oxid
131 of the superoxide (O-2) generating phagocyte nicotinamide adenine dinucleotide phosphate (NADPH) oxid
132 artially to restore gp91-phox deficiency and nicotinamide adenine dinucleotide phosphate (NADPH) oxid
133 neutrophils exhibited reduced (hydrogenated) nicotinamide adenine dinucleotide phosphate (NADPH) oxid
134 this study, we show that antibiotics rescue nicotinamide adenine dinucleotide phosphate (NADPH) oxid
135 ure to LPS led to up-regulated expression of nicotinamide adenine dinucleotide phosphate (NADPH) oxid
137 sing intravital microscopy with mice lacking nicotinamide adenine dinucleotide phosphate (NADPH) oxid
138 ation of p47(phox) (an organizer subunit for nicotinamide adenine dinucleotide phosphate (NADPH) oxid
139 ng protein 9 (CARD9), but was independent of nicotinamide adenine dinucleotide phosphate (NADPH) oxid
140 because of defective activation of phagocyte nicotinamide adenine dinucleotide phosphate (NADPH) oxid
141 s in the genes that encode components of the nicotinamide adenine dinucleotide phosphate (NADPH) oxid
143 causing hemolytic anemia linked to impaired nicotinamide adenine dinucleotide phosphate (NADPH) prod
144 s, G6PD is upregulated and generates reduced nicotinamide adenine dinucleotide phosphate (NADPH) that
145 ediate that transfers electrons from reduced nicotinamide adenine dinucleotide phosphate (NADPH) to a
146 cofactor specificity of oxidoreductases from nicotinamide adenine dinucleotide phosphate (NADPH) to n
147 l mechanism with ordered addition of reduced nicotinamide adenine dinucleotide phosphate (NADPH) to t
149 the enzyme with acyl-coenzyme A and reduced nicotinamide adenine dinucleotide phosphate (NADPH), a p
150 -N delta-hemiphthaloyl-L-ornithine], reduced nicotinamide adenine dinucleotide phosphate (NADPH), and
151 tosynthesis is the primary source of reduced nicotinamide adenine dinucleotide phosphate (NADPH), whi
152 C and IDH2 R172K proteins catalyze the novel nicotinamide adenine dinucleotide phosphate (NADPH)-depe
153 present study the relationship among reduced nicotinamide adenine dinucleotide phosphate (NADPH)-depe
154 operoxidase (LPO) and myeloperoxidase (MPO), nicotinamide adenine dinucleotide phosphate (NADPH)-depe
155 nvestigated by nNOS immunohistochemistry and nicotinamide adenine dinucleotide phosphate (NADPH)-diap
156 , tyrosine hydroxylase, or the histochemical nicotinamide adenine dinucleotide phosphate (NADPH)-diap
158 identified in spiral ganglion cells by using nicotinamide adenine dinucleotide phosphate (NADPH)-diap
159 t stimulation by double labeling for reduced nicotinamide adenine dinucleotide phosphate (NADPH)-diap
160 tudies of the retrograde tracer with reduced nicotinamide adenine dinucleotide phosphate (NADPH)-diap
161 vealed age-related changes in the density of nicotinamide adenine dinucleotide phosphate (NADPH)-diap
162 reatment with RAGE ligands rapidly activated nicotinamide adenine dinucleotide phosphate (NADPH)-oxid
163 e superoxide-generating subunit of phagocyte nicotinamide adenine dinucleotide phosphate (NADPH)-oxid
164 es have indicated roles for Fas/CD95 and the nicotinamide adenine dinucleotide phosphate (NADPH)-oxid
165 a newly isolated cytosolic component of the nicotinamide adenine dinucleotide phosphate (NADPH)-oxid
166 , which exhibited decreased steatohepatitis, nicotinamide adenine dinucleotide phosphate (NADPH)-oxid
167 y immunodeficiency caused by a defect in the nicotinamide adenine dinucleotide phosphate (NADPH)-oxid
168 and is functionally analogous to microsomal nicotinamide adenine dinucleotide phosphate (NADPH)-P450
169 ministered to block superoxide production by nicotinamide adenine dinucleotide phosphate (NADPH).
170 strate dihydrofolate (DHF) and its cofactor, nicotinamide adenine dinucleotide phosphate (NADPH).
171 nce at 340 nm, corresponding to oxidation of nicotinamide adenine dinucleotide phosphate (NADPH).
172 termediate, using two equivalents of reduced nicotinamide adenine dinucleotide phosphate (NADPH).
173 ydrofolate reductase (DHFR) and the coenzyme nicotinamide adenine dinucleotide phosphate (NADPH); the
174 n agreement with Prelog's rule, in which the nicotinamide-adenine dinucleotide phosphate (NADPH) cofa
176 synthase [iNOS], superoxide dismutase [SOD], nicotinamide adenine dinucleotide phosphate [NADPH] oxid
179 tase catalyzes the isomerization and reduced nicotinamide adenine dinucleotide phosphate- (NADPH-) de
180 for G6PD enzyme activity, cellular oxidized nicotinamide adenine dinucleotide phosphate/NADPH levels
181 MP], nicotinamide adenine dinucleotide /NAD, nicotinamide adenine dinucleotide phosphate / nicotinami
183 his, as well as the observation that reduced nicotinamide adenine dinucleotide phosphate oxidase (NAD
184 To investigate the potential role of reduced nicotinamide adenine dinucleotide phosphate oxidase (NAD
187 Reactive oxygen species (ROS) generated by nicotinamide adenine dinucleotide phosphate oxidase (NOX
188 ve damage, and membrane translocation of the nicotinamide adenine dinucleotide phosphate oxidase (NOX
189 ivity for macrophages and myofibroblasts and nicotinamide adenine dinucleotide phosphate oxidase (NOX
192 uced blood pressure, whereas the transfer of nicotinamide adenine dinucleotide phosphate oxidase 2-de
194 on with a reduction in the small increase in nicotinamide adenine dinucleotide phosphate oxidase acti
196 x and inhibition of Rac1-mediated attenuated nicotinamide adenine dinucleotide phosphate oxidase acti
198 Furthermore, direct activation of reduced nicotinamide adenine dinucleotide phosphate oxidase acti
199 at adiponectin directly decreases myocardial nicotinamide adenine dinucleotide phosphate oxidase acti
200 rated that adiponectin suppresses myocardial nicotinamide adenine dinucleotide phosphate oxidase acti
201 nulomatous disease (CGD) is due to defective nicotinamide adenine dinucleotide phosphate oxidase acti
202 the signal involves plasma membrane reduced nicotinamide adenine dinucleotide phosphate oxidase acti
203 p47(phox) subunit beyond and independent of nicotinamide adenine dinucleotide phosphate oxidase acti
204 euron death through activation of microglial nicotinamide adenine dinucleotide phosphate oxidase and
205 fragmentation via Ab activation of platelet nicotinamide adenine dinucleotide phosphate oxidase and
206 s (proinflammatory cytokine expression, beta-nicotinamide adenine dinucleotide phosphate oxidase and
207 neuroinflammation by selectively inhibiting nicotinamide adenine dinucleotide phosphate oxidase and
208 the generation of reactive oxygen species by nicotinamide adenine dinucleotide phosphate oxidase and
209 blocked agonist-initiated association of the nicotinamide adenine dinucleotide phosphate oxidase comp
210 is unable to completely rescue migration and nicotinamide adenine dinucleotide phosphate oxidase defi
211 airment through neurohormonal activation of (nicotinamide adenine dinucleotide phosphate oxidase depe
213 oxidative stress, including up-regulation of nicotinamide adenine dinucleotide phosphate oxidase in a
214 nduced reactive oxygen species (ROS) through nicotinamide adenine dinucleotide phosphate oxidase in l
215 breathing control with administration of the nicotinamide adenine dinucleotide phosphate oxidase inhi
217 ing the efficacy of an ultra-low dose of the nicotinamide adenine dinucleotide phosphate oxidase inhi
218 ), TEMPOL (a general antioxidant), apocynin (nicotinamide adenine dinucleotide phosphate oxidase inhi
220 tion of reactive oxygen species (ROS) by the nicotinamide adenine dinucleotide phosphate oxidase is a
221 either poly(ADP-ribose) polymerase or of the nicotinamide adenine dinucleotide phosphate oxidase prev
223 Mice lacking the gp91 protein of reduced nicotinamide adenine dinucleotide phosphate oxidase show
225 ed baroreflex sensitivity is associated with nicotinamide adenine dinucleotide phosphate oxidase subu
227 xpression in P47 and Rac-1 expression of two nicotinamide adenine dinucleotide phosphate oxidase subu
228 own experimentally to activate transmembrane nicotinamide adenine dinucleotide phosphate oxidase type
229 th P67 dominant negative mice to inhibit the nicotinamide adenine dinucleotide phosphate oxidase were
233 lained by the absence of the gp91 subunit of nicotinamide adenine dinucleotide phosphate oxidase, as
234 show that the oxidative burst, catalyzed by nicotinamide adenine dinucleotide phosphate oxidase, can
236 to beta-amyloid peptides activates the glial nicotinamide adenine dinucleotide phosphate oxidase, fol
237 cleotide phosphate oxidase 4 (Nox4), a major nicotinamide adenine dinucleotide phosphate oxidase, med
238 drial electron transport chain reactions and nicotinamide adenine dinucleotide phosphate oxidase, or
239 uncoupled endothelial nitric oxide synthase, nicotinamide adenine dinucleotide phosphate oxidase, xan
241 e persistence of tumor hemorrhage in C3- and nicotinamide adenine dinucleotide phosphate oxidase-defi
242 rophil oxidative burst or in human or murine nicotinamide adenine dinucleotide phosphate oxidase-defi
244 Furthermore, although it was shown that nicotinamide adenine dinucleotide phosphate oxidase-deri
245 oding adiponectin) led to reduced myocardial nicotinamide adenine dinucleotide phosphate oxidase-deri
246 tion by restoring renal blood flow, reducing nicotinamide adenine dinucleotide phosphate oxidase-deri
248 oxidative stress generated by the astrocytic nicotinamide adenine dinucleotide phosphate oxidase.
249 d mesencephalic neuron-glia cultures through nicotinamide adenine dinucleotide phosphate oxidase.
250 2 is important for the assembly of phagocyte nicotinamide adenine dinucleotide phosphate oxidase.
251 regulated by growth factors and Nox4 reduced nicotinamide adenine dinucleotide phosphate oxidase.
253 dant production through endothelial NAD(P)H (nicotinamide adenine dinucleotide phosphate) oxidase bec
254 vascular wall include NADPH (reduced form of nicotinamide adenine dinucleotide phosphate) oxidase, xa
255 ial susceptibility to NADPH (reduced form of nicotinamide adenine dinucleotide phosphate) oxidase-dep
256 enchymal stem/stromal cell therapy decreased nicotinamide adenine dinucleotide phosphate-oxidase 2 an
257 which was accompanied by down-regulation of nicotinamide adenine dinucleotide phosphate-oxidase and
258 reperfusion injury by inhibition of iNOS and nicotinamide adenine dinucleotide phosphate-oxidase prot
259 dues 297 to 331 of the 47-kDa subunit of the nicotinamide-adenine dinucleotide phosphate-oxidase comp
260 s attributable to constitutive activation of nicotinamide adenine dinucleotide phosphate oxidases (NO
261 activation, implicating a crosstalk between nicotinamide adenine dinucleotide phosphate oxidases and
262 downstream effectors such as Rho kinase and nicotinamide adenine dinucleotide phosphate oxidases are
263 Dual oxidases (DUOX) are conserved reduced nicotinamide adenine dinucleotide phosphate oxidases tha
265 omic DNA was extracted and genotyped for the nicotinamide adenine dinucleotide phosphate p22 phagocyt
266 terleukin-10, protein expressions of reduced nicotinamide-adenine dinucleotide phosphate:quinone oxid
268 wide variety of agents through activation of nicotinamide adenine dinucleotide phosphate reduced (NAD
269 tiation of these cells was evaluated by beta-nicotinamide adenine dinucleotide phosphate reduced diap
270 ion, lipid peroxidation, H(2)O(2), O(2).(-), nicotinamide adenine dinucleotide phosphate reduced form
271 nship between two membrane transporters, the Nicotinamide adenine dinucleotide phosphate reduced form
272 tive was to investigate the possible role of nicotinamide adenine dinucleotide phosphate reduced form
274 tion of Fas at cysteine 294 independently of nicotinamide adenine dinucleotide phosphate reduced oxid
276 e hypothesized that AGEs induce TACE through nicotinamide adenine dinucleotide phosphate reduced oxid
277 cetylcholinesterase (AchE), whereas cofactor nicotinamide adenine dinucleotide phosphate (reduced for
278 O2-) production by impairing the assembly of nicotinamide adenine dinucleotide phosphate (reduced for
279 . gambiae cDNAs encoding cytochrome b(5) and nicotinamide adenine dinucleotide phosphate (reduced for
281 ons in any of 4 genes encoding components of nicotinamide adenine dinucleotide phosphate (reduced for
282 a significant multilocus effect between the nicotinamide adenine dinucleotide (phosphate) reduced:qu
284 relation exists between decreased binding of nicotinamide adenine dinucleotide phosphate, reduced (NA
285 osynthetic electron transport in the form of nicotinamide adenine dinucleotide phosphate, reduced are
286 ) to GSH in the presence of beta-NADPH (beta-nicotinamide adenine dinucleotide phosphate, reduced for
287 PI3K-dependent activation of Rac1, and that nicotinamide adenine dinucleotide phosphate, reduced for
289 pha and Cdelta by use of 4-(2)H-labeled beta-nicotinamide adenine dinucleotide phosphate, reduced for
292 nockout, PBL13 is able to associate with the nicotinamide adenine dinucleotide phosphate, reduced oxi
294 forms of nicotinamide adenine nucleotide and nicotinamide adenine dinucleotide phosphate, respectivel
296 roid dehydrogenase type 1 depends on reduced nicotinamide adenine dinucleotide phosphate synthesized
297 all nucleotides and coenzymes including CoA, nicotinamide adenine dinucleotide phosphate, thiamine ph
298 beled isopropanol to the re face of oxidized nicotinamide adenine dinucleotide phosphate to form R-[4
299 enzyme that mediates electron transfer from nicotinamide adenine dinucleotide phosphate to molecular
300 was modified (i) after the adenine moiety of nicotinamide adenine dinucleotide phosphate was changed
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