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1 negative allosteric modulator at the alpha7 nicotinic acetylcholine receptor.
2 pete with (123)I-5-IA for binding to beta(2)-nicotinic acetylcholine receptor.
3 ges in the gating conformational change of a nicotinic acetylcholine receptor.
4 s mediated by GABAA receptors in addition to nicotinic acetylcholine receptors.
5 sion to overcome imidacloprid binding to the nicotinic acetylcholine receptors.
6 ounds targeting the cannabinoid 1 and alpha7 nicotinic acetylcholine receptors.
7 that act as partial agonists at alpha4beta2-nicotinic acetylcholine receptors.
8 ellate origin that are potent antagonists of nicotinic acetylcholine receptors.
9 o acts as a positive allosteric modulator of nicotinic acetylcholine receptors.
10 a genetic mutation in RIC3; a chaperone for nicotinic acetylcholine receptors.
12 sociated with adult-type mouse neuromuscular nicotinic acetylcholine receptor (AChR) channel gating h
14 is (MG) without detectable antibodies to the nicotinic acetylcholine receptor (AChR) or to muscle-spe
15 le-channel currents recorded from the muscle nicotinic acetylcholine receptor (AChR), we have recentl
20 y Ab-mediated autoimmune responses to muscle nicotinic acetylcholine receptors (AChRs) that impair ne
23 ently used in human therapy (benzimidazoles, nicotinic acetylcholine receptor agonists, macrocyclic l
24 ed transcripts of several targets, including nicotinic acetylcholine receptor alpha 1 and alpha 2 sub
26 d alpha3- and beta4-subunit-containing human nicotinic acetylcholine receptors (alpha3beta4*-nAChRs).
28 novel radiotracer that binds to alpha4beta2 nicotinic acetylcholine receptors (alpha4beta2-nAChRs) a
29 ly, a decrease in the numbers of alpha4beta2 nicotinic acetylcholine receptors (alpha4beta2-nAChRs) i
31 agus nerve regulation of splenic cholinergic nicotinic acetylcholine receptor alpha7 (nAChRa7) signal
32 i-inflammatory cytokines, adenosine, and the nicotinic acetylcholine receptor alpha7 subunit (alpha7n
34 tes lung cancer proliferation via the alpha7-nicotinic acetylcholine receptor (alpha7-nAChR) subtype.
35 SY5Y cells stably overexpressing the alpha-7 nicotinic acetylcholine receptor (alpha7-nAChR), a poten
36 ation in normal and cancer cells, the alpha7 nicotinic acetylcholine receptor (alpha7-nAChR), was mor
37 ent evidence suggests that activating alpha7 nicotinic acetylcholine receptors (alpha7 nAChR) may fac
38 f acetylcholine, which interacts with alpha7 nicotinic acetylcholine receptors (alpha7 nAChR) on immu
39 inding affinities and selectivity for alpha7-nicotinic acetylcholine receptors (alpha7-nAChRs) (Ki =
40 how that calcium-permeable alpha7-containing nicotinic acetylcholine receptors (alpha7-nAChRs), capab
43 unique, distinct, and human-specific alpha7-nicotinic acetylcholine receptor (alpha7nAChR) gene [CHR
46 Here, we show that ILC2s express the alpha7-nicotinic acetylcholine receptor (alpha7nAChR), which is
47 f amyloid-beta(42) (Abeta(42)) by the alpha7-nicotinic acetylcholine receptor (alpha7nAChR), which re
48 septal cholinergic fibers through the alpha7-nicotinic acetylcholine receptor (alpha7nAChR), whose ac
50 y and ischemia through stimulation of alpha7 nicotinic acetylcholine receptors (alpha7nAChR) on monon
53 n the reward circuitry of the brain, alpha-7-nicotinic acetylcholine receptors (alpha7nAChRs) modulat
54 the integration of neural signals and alpha7 nicotinic acetylcholine receptors (alpha7nAChRs) on sple
55 ells in mice that lack the alpha9 subunit of nicotinic acetylcholine receptors (alpha9 KO mice) alter
56 may, by increasing activation of the alpha7-nicotinic acetylcholine receptor, alter the development
57 he extracellular domain of the native alpha7 nicotinic acetylcholine receptor and acetylcholine bindi
58 t cytisine, a partial agonist that binds the nicotinic acetylcholine receptor and is used for smoking
59 RV fibrosis, RV inflammation, and RV alpha-7 nicotinic acetylcholine receptor and muscarinic acetylch
64 endocytosis (neural cell adhesion molecule, nicotinic acetylcholine receptor, and p75 neurotrophin r
65 pproached by sensory nerve fibers expressing nicotinic acetylcholine receptors, and intraurethral app
66 receptor antagonist), and chlorisondamine (a nicotinic acetylcholine receptor antagonist) were applie
67 n a cholinergic input because application of nicotinic acetylcholine receptor antagonists impairs thi
72 howed significantly lower (21%-26%) beta(2)*-nicotinic acetylcholine receptor availability relative t
74 the availability of beta2-subunit-containing nicotinic acetylcholine receptors (beta2*-nAChR) in subj
75 moking partly by binding to beta2-containing nicotinic acetylcholine receptors (beta2*-nAChRs) in the
78 d excitatory transmission was blocked by the nicotinic acetylcholine receptor blocker mecamylamine.
79 MS stem from molecular defects in the muscle nicotinic acetylcholine receptor, but they can also be c
80 ha7nAChR) gene [CHRNA7 (gene-encoding alpha7-nicotinic acetylcholine receptor)] called CHRFAM7A (gene
82 itive allosteric modulators (PAMs) of alpha7 nicotinic acetylcholine receptors can enhance ion channe
83 s16969968 that alters function of the alpha5 nicotinic acetylcholine receptor (CHRNA5) and noncoding
85 1beta release in human and rat monocytes via nicotinic acetylcholine receptors containing subunits al
87 te smoke (SS), or secondhand smoke, promoted nicotinic acetylcholine receptor-dependent exacerbation
88 clothianidin, but not imidacloprid, causes a nicotinic acetylcholine receptor-dependent rapid mitocho
90 ation of antiinflammatory effects via alpha7 nicotinic acetylcholine receptor-expressing splenic macr
91 ters = primary outcome variables) as well as nicotinic acetylcholine receptor expression, muscle mass
92 level of nonsmokers following stimulation of nicotinic acetylcholine receptors (familywise error-corr
93 r organophosphates and methylcarbamates, the nicotinic acetylcholine receptor for neonicotinoids, the
94 s denatured at a higher temperature than the nicotinic acetylcholine receptor from Torpedo californic
95 model, based on the partial structure of the nicotinic acetylcholine receptor from Torpedo marmorata,
96 pamine neurons receive cholinergic input via nicotinic acetylcholine receptors from the Kenyon cells;
97 ent models have demonstrated that the alpha5 nicotinic acetylcholine receptor gene (CHRNA5) is import
100 enes revealed significant association of the nicotinic acetylcholine receptor gene family with both i
102 ling of the joint effect of all 61 SNPs in 7 nicotinic acetylcholine receptor genes revealed signific
103 d the joint associations of 61 tag SNPs in 7 nicotinic acetylcholine receptor genes with subclinical
104 information for each of the family members, nicotinic acetylcholine receptors, glycine receptors, GA
105 interaction between beta-amyloid (Abeta) and nicotinic acetylcholine receptors has been widely studie
107 allosteric modulators (PAMs) of alpha4beta2 nicotinic acetylcholine receptors have the potential to
108 ncluding electron microscopic studies of the nicotinic acetylcholine receptor, high-resolution X-ray
109 studies have found that acute stimulation of nicotinic acetylcholine receptors improves cognition in
111 ore, phosphomimetic Dok-7 mutants aggregated nicotinic acetylcholine receptors in C2C12 myotubes with
112 ular junction, rapsyn, enables clustering of nicotinic acetylcholine receptors in high concentration
113 texts that predict alcohol, and suggest that nicotinic acetylcholine receptors in the NAc are critica
114 based on homology with the Torpedo marmorata nicotinic acetylcholine receptor infer the existence of
116 ression of naturally expressed mouse alpha6*-nicotinic acetylcholine receptors (malpha6*-nAChRs; wher
117 Isoform-specific mechanisms of alpha3beta4*-nicotinic acetylcholine receptor modulation by the proto
118 l impact of hydrogen bonding on human alpha7 nicotinic acetylcholine receptor (nAChR) activation and
119 er cognitive processes that are modulated by nicotinic acetylcholine receptor (nAChR) activation by c
120 ositive allosteric modulator (PAM) of alpha7 nicotinic acetylcholine receptor (nAChR) activation by o
121 linical trials suggest that drugs that alter nicotinic acetylcholine receptor (nAChR) activity can af
122 hether extending plasma levels of the alpha7-nicotinic acetylcholine receptor (nAChR) agonist 3-(2,4-
123 ystemic administration of the partial alpha7 nicotinic acetylcholine receptor (nAChR) agonist SSR 180
125 ation of nicotine or ABT-418, an alpha4beta2 nicotinic acetylcholine receptor (nAChR) agonist, normal
128 CHRNA3-CHRNA5-CHRNB4 cluster coding for the nicotinic acetylcholine receptor (nAChR) alpha3, alpha5
129 imilarly, Abeta exposure increases levels of nicotinic acetylcholine receptor (nAChR) alpha7 subunit
130 Conotoxin GeXIVA inhibits the alpha9alpha10 nicotinic acetylcholine receptor (nAChR) and is analgesi
132 d as binding proteins, the Torpedo marmorata nicotinic acetylcholine receptor (nAChR) and the Lymnaea
141 t promote a desensitized state of the alpha7 nicotinic acetylcholine receptor (nAChR) have been assoc
142 associate with this soluble surrogate of the nicotinic acetylcholine receptor (nAChR) in a cooperativ
147 vity relationships for a new class of alpha7 nicotinic acetylcholine receptor (nAChR) modulators base
149 on clinical trials, comparing varenicline, a nicotinic acetylcholine receptor (nAChR) partial agonist
151 overexpression was via the alpha7 subunit of nicotinic acetylcholine receptor (nAChR) stimulation and
152 y a critical role in the characterization of nicotinic acetylcholine receptor (nAChR) structure and f
154 A is both an antagonist of the alpha9alpha10 nicotinic acetylcholine receptor (nAChR) subtype and an
156 ha-Conotoxin AuIB is a selective alpha3beta4 nicotinic acetylcholine receptor (nAChR) subtype inhibit
159 n mice harboring a null mutant allele of the nicotinic acetylcholine receptor (nAChR) subunit gene al
160 ut and gain-of-function of specific neuronal nicotinic acetylcholine receptor (nAChR) subunit genes.
161 ar domain (ECD) of the human neuronal alpha2 nicotinic acetylcholine receptor (nAChR) subunit in comp
162 d withdrawal are separate processes and that nicotinic acetylcholine receptor (nAChR) upregulation un
164 s for CHRNA7, the gene coding for the alpha7 nicotinic acetylcholine receptor (nAChR), and manifest a
167 s not affect the activity against the alpha7 nicotinic acetylcholine receptor (nAChR), whereas its re
168 Consistently, the rank-order of blockade of nicotinic acetylcholine receptor (nAChR)-stimulated inwa
170 Positive allosteric modulators (PAMs) of nicotinic acetylcholine receptors (nAChR) are important
171 tional regulation and synaptic clustering of nicotinic acetylcholine receptors (nAChR) during neurotr
172 r behavior and, specifically, recruitment of nicotinic acetylcholine receptors (nAChR) in mesencephal
174 ificant associations between variants in the nicotinic acetylcholine receptors (nAChR) subunits and n
179 Inactivation of 42 genes, including the nicotinic acetylcholine receptors nAChRalpha1 and nAChRa
180 ly inhibit human alpha3beta2 and alpha6beta2 nicotinic acetylcholine receptor (nAChRs) and not the cl
181 ial agonist at alpha4beta2* and alpha6beta2* nicotinic acetylcholine receptors (nAChRs) and a full ag
183 compound NS3861 and cytisine are agonists of nicotinic acetylcholine receptors (nAChRs) and both bind
186 The medial habenula (MHb) densely expresses nicotinic acetylcholine receptors (nAChRs) and participa
193 and intensive study over the last 20 years, nicotinic acetylcholine receptors (nAChRs) are still far
204 ubset of cold thermosensors expressed alpha7 nicotinic acetylcholine receptors (nAChRs) but not other
206 ives significant cholinergic innervation and nicotinic acetylcholine receptors (nAChRs) contribute gr
207 ng the most widely co-abused substances, and nicotinic acetylcholine receptors (nAChRs) contribute to
208 ctive compounds that desensitize alpha4beta2 nicotinic acetylcholine receptors (nAChRs) could provide
211 andoxin and shows high binding affinity with nicotinic acetylcholine receptors (nAChRs) expressed on
212 menthol enhances nicotine-induced changes in nicotinic acetylcholine receptors (nAChRs) expressed on
213 anatomical localisation and function of the nicotinic acetylcholine receptors (nAChRs) formed by the
218 ronic nicotine exposure is known to activate nicotinic acetylcholine receptors (nAChRs) in immune cel
219 , we studied cellular and circuit aspects of nicotinic acetylcholine receptors (nAChRs) in mouse STN.
220 The data suggest a complex interaction of nicotinic acetylcholine receptors (nAChRs) in regulating
221 sensitize high-affinity alpha4beta2 neuronal nicotinic acetylcholine receptors (nAChRs) in seconds.
222 X-12 over PhTX-343 for embryonic muscle-type nicotinic acetylcholine receptors (nAChRs) in TE671 cell
223 king results from the binding of nicotine to nicotinic acetylcholine receptors (nAChRs) in the brain,
224 Cigarette smoking leads to upregulation of nicotinic acetylcholine receptors (nAChRs) in the human
225 otine use induces functional upregulation of nicotinic acetylcholine receptors (nAChRs) in the mesoco
226 uced BPD and sought to ascertain the role of nicotinic acetylcholine receptors (nAChRs) in this respo
227 rents, showing that the assembled functional nicotinic acetylcholine receptors (nAChRs) included the
230 ulation of beta2 subunit-containing (beta2*) nicotinic acetylcholine receptors (nAChRs) is implicated
236 actions, but very little is known about how nicotinic acetylcholine receptors (nAChRs) regulate LHb
237 ng the expression and clustering of neuronal nicotinic acetylcholine receptors (nAChRs) remain poorly
239 nduction of this form of LTP needs CP-alpha7 nicotinic acetylcholine receptors (nAChRs) that, like CP
243 and impact of aging on presynaptic neuronal nicotinic acetylcholine receptors (nAChRs) within the ci
245 al homologs of the ligand-binding domains of nicotinic acetylcholine receptors (nAChRs), and they rep
246 tive allosteric modulator (PAM-II) of alpha7 nicotinic acetylcholine receptors (nAChRs), as long as 6
247 erienced by smokers are known to desensitize nicotinic acetylcholine receptors (nAChRs), but the impa
248 rette smoking leads to upregulation of brain nicotinic acetylcholine receptors (nAChRs), including th
249 epends on cholinergic tone and activation of nicotinic acetylcholine receptors (nAChRs), particularly
250 ne is a promising tracer for neuroimaging of nicotinic acetylcholine receptors (nAChRs), subtype alph
251 (DAergic) neurons via high-affinity neuronal nicotinic acetylcholine receptors (nAChRs), the mechanis
252 nists for the alpha(4)beta(2) subtype of the nicotinic acetylcholine receptors (nAChRs), we have synt
269 f nicotine dependence and stimulation of the nicotinic acetylcholine receptor on the ability to inter
270 sitivity and acute effects of stimulation of nicotinic acetylcholine receptors on behavioral and neur
271 survival in animal sepsis models via alpha7 nicotinic acetylcholine receptors on immunocompetent cel
272 t nicotine, acting through alpha7-containing nicotinic acetylcholine receptors on the postsynaptic ne
273 ion of Torpedo electrocyte membranes rich in nicotinic acetylcholine receptors on the surface of micr
274 )] called CHRFAM7A (gene-encoding dup-alpha7-nicotinic acetylcholine receptor) on a locus of chromoso
275 "spiroimidates") and their utility as alpha7 nicotinic acetylcholine receptor partial agonists are de
276 r behaviour in animals through inhibition of nicotinic acetylcholine receptors present in the central
277 age of the cholecystokinin-1 receptor or the nicotinic acetylcholine receptor reversed the protective
278 sphorylation of the ER-resident chaperone of nicotinic acetylcholine receptors, RIC-3, leads to incre
281 Vc1.1 specifically and potently inhibits the nicotinic acetylcholine receptor subtype alpha9alpha10 (
282 ommon variants near the apolipoprotein E and nicotinic acetylcholine receptor subunit alpha 5 genes a
283 regulation of expression and function of the nicotinic acetylcholine receptor subunit cluster of alph
286 hat both common and rare variants of several nicotinic acetylcholine receptor subunits are associated
288 active with ion channels of the ligand-gated nicotinic acetylcholine receptor superfamily (namely alp
290 fic subtype of neuron, expressing non-alpha7 nicotinic acetylcholine receptors, that directly drives
291 resting conformation, and in skeletal muscle nicotinic acetylcholine receptors there is an exponentia
293 amyloid-beta peptide (Abeta) engages alpha7 nicotinic acetylcholine receptors to induce release of a
295 f the anti-inflammatory alpha7-nAChR (alpha7-nicotinic acetylcholine receptor) was similar in young S
297 ed tool to probe the ion channel pore of the nicotinic acetylcholine receptor, which is a prototypica
298 lly, PrP(C)-STI1 engagement activated alpha7 nicotinic acetylcholine receptors, which participated in
299 unique cell types expressing alpha8 subunit nicotinic acetylcholine receptor, while SPO and cOv are
300 CK2: (a) the alpha and beta subunits of the nicotinic acetylcholine receptors with weak interaction,
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