1 (RFA) to create a quantifiable prothrombotic
nidus.
2 s, it is concluded that AEF serves as both a
nidus and a template for amyloid formation.
3 alprotectin is not released into the abscess
nidus and does not limit Mn in this organ.
4 no cortical coverage was present between the
nidus and neural structure in danger.
5 e index of 2.5 for arterial feeders, 3.0 for
nidus,
and 3.0 for venous drainage.
6 y, dimensions, and temporal stability of the
nidus are determined largely by pathogen natural history
7 age of the calcification area over the total
nidus area).
8 id osteoma in relation to the native cortex,
nidus area, nidus calcification area and attenuation at
9 duration and other variables, including the
nidus area.
10 Dense and plexiform structure of AVM
nidus as well as a low number of supplying vessels and a
11 n relation to the native cortex, nidus area,
nidus calcification area and attenuation at CT, and nidu
12 This
nidus can then mineralize if the balance of pro-minerali
13 Unfavorable factors include
nidus drainage into multiple compartments as well as mul
14 esis by altering the destiny of CEP55 from a
nidus for abscission to an integral component of the int
15 as the potential to function physically as a
nidus for appositional new bone growth in alveolar socke
16 onses to pathogenic organisms can act as the
nidus for autoimmunity.
17 Contaminated vascular devices can act as a
nidus for bloodstream infection and systemic pathogen di
18 proteinase-2, which converts exosomes into a
nidus for calcification.
19 all intestine, liver, or spleen, providing a
nidus for future viral strain recombination.
20 umorigenesis and that aneuploidy itself is a
nidus for genomic instability.
21 ular matrix of target organs and acting as a
nidus for in situ immune complex formation.
22 A thrombus is a
nidus for infection, while infection increases the risk
23 dard permanent electronic hardware acts as a
nidus for infection: bacteria form biofilms along percut
24 icles have previously been identified as the
nidus for mineral nucleation.
25 smooth muscle cells (VSMCs), form the first
nidus for mineralization and fetuin-A, a potent circulat
26 N-type Ca(2+) channels (CaV2.2) are a
nidus for neurotransmitter release and nociceptive trans
27 low numbers of cryptococci that can become a
nidus for re-activation disease during immunodeficient s
28 rcation periodontal breakdown may serve as a
nidus for recolonization and disease recurrence or for t
29 or adjuvant chemotherapy, and providing the
nidus for recurrent prostate cancer.
30 Thus, although the ChEL domain provides a
nidus for Tg dimerization, interactions of upstream Tg r
31 Rab proteins form a
nidus for the assembly of multiprotein complexes on dist
32 table protective environment and acting as a
nidus for the dissemination of large numbers of microorg
33 rmed stem cells represent a potential common
nidus for the primary cancer and the recurrent cancer th
34 aggregates readily and appears to provide a
nidus for the subsequent aggregation of A beta 40, resul
35 These foci are a
nidus for vigorous actin polymerization, generating long
36 tion of A beta (template-independent initial
nidus formation) and deposition of A beta (template-depe
37 nd was greater in patients with a larger AVM
nidus (
hazard ratio 1.3, 95% CI 1.1-1.7).
38 alcification area and attenuation at CT, and
nidus mineralization ratio (percentage of the calcificat
39 The patients' age and
nidus mineralization ratio increased significantly with
40 The
nidus mineralization ratio of osteoid osteomas increases
41 steoid osteomas demonstrate a lower ratio of
nidus mineralization.
42 This
nidus of amyloid fibrils then allows the progressive acc
43 d of the pancreas has become apparent as the
nidus of chronic inflammation.
44 herence and decreasing its ability to form a
nidus of infection in the skin.
45 abscesses have been shown to be an important
nidus of infection.
46 ions and are formed, in part, to contain the
nidus of infection.
47 central memory responses that can clear the
nidus of initial virus-infected cells at mucosal surface
48 est that endothelial cells may be an in vivo
nidus of mammalian infection.
49 , the presence of folded cells, and abundant
nidus of polymerization under the membrane.
50 ed only by identification and removal of the
nidus of sequestration.
51 that would entrap lipoproteins, forming the
nidus of the atherosclerotic plaque.
52 ave been present on the platelet core at the
nidus of the injury, bound proteins were not evident on
53 The spatially defined focus, or
nidus,
of transmission may be characterized by vegetatio
54 A subsequent DSA depicted a medium-sized
nidus,
receiving blood supply from multiple origins but
55 lood vessel injury and acting as coagulation
nidus sites.
56 0.85 for arterial feeders, kappa of 1.00 for
nidus size, and kappa of 0.82 for venous drainage.
57 aracterization consisted of arterial feeder,
nidus size, and venous drainage type identification comp
58 Except
nidus structure and AVM size, all single parameters form
59 The regulatory
nidus that links these systems, MpeR, is expressed exclu
60 , MR imaging depicted an absence of residual
nidus vascularity in 32 (76%) of the 42 patients who wer
61 However,
nidus vascularization still persisted after treatment in
62 es of osteoid osteoma (edema, hyperemia, and
nidus vascularization) were considered at baseline and a
63 Volume and attenuation of the
nidus were measured.
64 us antigens that can trigger an inflammatory
nidus within the arterial wall.