ライフサイエンスコーパス: nigra

1 s accumbens, globus pallidus, and substantia nigra).

2 midbrain (ventral tegmental area/substantia nigra).

3 ss of dopaminergic neurons of the substantia nigra.

4 healthy controls, but not in the substantia nigra.

5 opaminergic neuronal count in the substantia nigra.

6 ss of dopaminergic neurons in the substantia nigra.

7 vity between ventral striatum and substantia nigra.

8 the dopaminergic neurons of human substantia nigra.

9 oning dopaminergic neurons of the substantia nigra.

10 y have elevated free-water in the substantia nigra.

11 th of dopaminergic neurons in the substantia nigra.

12 ed in dopaminergic cells from the substantia nigra.

13 sm had elevated free-water in the substantia nigra.

14 eurturin delivered to putamen and substantia nigra.

15 port of neurturin from putamen to substantia nigra.

16 esidual dopaminergic cells in the substantia nigra.

17 sing human alpha-synuclein in the substantia nigra.

18 nvasive progression marker of the substantia nigra.

19 ss of dopaminergic neurons in the substantia nigra.

20 ions of interest in the posterior substantia nigra.

21 halamic reticular nucleus and the substantia nigra.

22 rrents in dopamine neurons in the substantia nigra.

23 ), globus pallidus, striatum, and substantia nigra.

24 ss of dopaminergic neurons in the substantia nigra.

25 ss of dopaminergic neurons in the substantia nigra.

26 roxylase-immunostained neurons in substantia nigra.

27 on of dopaminergic neurons in the substantia nigra.

28 ss of dopaminergic neurons in the substantia nigra.

29 e SVPE signal was detected in the substantia nigra.

30 roxylase (TH)-positive neurons of substantia nigra.

31 gions of the ventral pallidum and substantia nigra.

32 ss of dopaminergic neurons in the substantia nigra.

33 dopaminergic (DA) neurons in the substantia nigra.

34 ecrease in entopeduncular nucleus/substantia nigra.

35 lamus (0.26% increase, P < .001), substantia nigra (0.25% increase, P = .01), red nucleus (0.25% incr

36 rend in BP(ND) was present in the substantia nigra (-14% +/- 15%).

37 ium vulgare, 19 from C. palustre, 15 from C. nigra, 17 from C. scabiosa, 14 from Sonchus asper, 17 fr

38 rin injected bilaterally into the substantia nigra (2.0 x 10(11) vector genomes) and putamen (1.0 x 1

39 S. nigra and (JAxNI)xCER flower extracts were characterized

40 rvival of dopaminergic neurons in substantia nigra and an increased number of microglia expressing ma

41 of isoprene emission from leaves of Populus nigra and hybrid aspen (Populus tremula x P. tremuloides

42 drove continuous T cell infiltration to the nigra and incessant glial inflammation.

43 catecholaminergic neurons of the substantia nigra and locus coeruleus, which are implicated in neuro

44 Depigmentation of the substantia nigra and other brainstem nuclei was present.

45 ty of dopaminergic neurons in the substantia nigra and reduced dopaminergic fibres in the striatum.

46 o PDE10A in striatoentopeduncular/substantia nigra and striatopallidal pathways might tightly interac

47 ectly by two-photon microscopy in substantia nigra and striatum brain slices.

48 and temporal cortices, striatum, substantia nigra and subthalamic nucleus were assessed.

49 pression in dopaminergic neurons of human PD nigra and that APP(-/-) mice develop iron-dependent nigr

50 ss of dopaminergic neurons in the substantia nigra and the coincidental appearance of Lewy bodies con

51 ss of dopaminergic neurons in the substantia nigra and the formation of Lewy body inclusions containi

52 minergic (DAergic) neurons in the substantia nigra and the gradual depletion of dopamine (DA).

53 dopaminergic deficiencies in the substantia nigra and the premotor and motor cortices, and with stat

54 ss of dopaminergic neurons in the substantia nigra and the presence of intraneuronal inclusions consi

55 ections to entopeduncular nucleus/substantia nigra and to external globus pallidus.

56 s effect was not reflected in the substantia nigra and ventral tegmental area (SN/VTA), medial tempor

57 midbrain region, encompassing the substantia nigra and ventral tegmental area, in 18 daily smokers (7

58 95% CI, 1.40-4.99; Lewy bodies in substantia nigra and/or locus ceruleus in ACT: RR for TBI with LOC

59 droxylase-positive neurons in the substantia nigra, and attenuated the decrease of striatal dopamine

60 rtical areas via globus pallidus, substantia nigra, and thalamus.

61 gic neurons of the basal ganglia, substantia nigra, and ventral tegmental area (VTA) where they regul

62 The striatum as well as substantia nigra appeared normal and no loss of dopamine expressing

63 iron and nitric oxide (NO) in the substantia nigra are associated with Parkinson's disease (PD) patho

64 vitro study confirmed that the fruits of S. nigra are capable of protecting colonic cells against th

65 idate free water in the posterior substantia nigra as a progression marker in Parkinson's disease, an

66 ration of CD4(+) and CD8(+) T cells into the nigra, attenuated nigral expression of proinflammatory m

67 S. nigra berries contained highest levels of total organic

68 turin delivery to the putamen and substantia nigra bilaterally in PD was not superior to sham surgery

69 Beyond the substantia nigra, both multiple system atrophy and progressive supr

70 e contacts included the thalamus, substantia nigra, brainstem and superior frontal gyrus.

71 ase-associated regions (striatum, substantia nigra), but also within anterior cingulate cortex, amygd

72 alpha-synuclein in the adult rat substantia nigra by adeno-associated virus-mediated delivery of a s

73 wy bodies and neurites and severe substantia nigra cell loss.

74 hains and markedly reduced in the substantia nigra compared with the neocortex.

75 o brain regions, the amygdala and substantia-nigra, compared to controls.

76 presumed disease epicenter in the substantia nigra, compatible with a trans-neuronal spread of the di

77 ht mulberry clones from Morus alba and Morus nigra cultivated in Spain.

78 e loss of dopamine neurons in the substantia nigra, decreased striatal dopamine levels, and consequen

79 Poplars (Populus deltoides x nigra, DN34), a model plant with complete genomic sequen

80 Furthermore, we detected elevated substantia nigra dopamine messenger RNA levels of NCS-1 (but not Ca

81 Substantia nigra dopamine neurons are involved in behavioral proces

82 Substantia nigra dopamine neurons fire tonically resulting in actio

83 ltiphoton microscopy to show that substantia nigra dopamine neurons, which are sensitive to stress in

84 sfunction have been implicated in substantia nigra dopaminergic neurodegeneration in Parkinson's dise

85 nduces the marked degeneration of substantia nigra dopaminergic neurons and axonal pathology, a cardi

86 inhibition.SIGNIFICANCE STATEMENT Substantia nigra dopaminergic neurons can be divided into two popul

87 n aggregation and degeneration of substantia nigra dopaminergic neurons.

88 nd may provide a means to inhibit substantia nigra dopaminergic neurons.

89 ifferent lectins such as those from Sambucus nigra (elder berry), Arachis hypogaea (peanut), Ulex eur

90 key brain regions, including the substantia nigra, entopeduncular nucleus, and nucleus accumbens she

91 c liposomes containing black mulberry (Morus nigra) extract (BME) were prepared by high pressure homo

92 elevated free-water levels in the substantia nigra for patients with Parkinson's disease compared wit

93 cal analysis of human post-mortem substantia nigra from Parkinson's disease suggests that this endoge

94 The highest TPC has been determined in S. nigra fruits and flowers.

95 Microwave assisted extraction (MAE) of Morus nigra fruits was optimized in order to elicit process pa

96 nced technique to obtain extracts from Morus nigra fruits with higher bioactive content and activity.

97 ition of white (Morus alba) and black (Morus nigra) fruits grown in Spain, in 2013 and 2014.

98 Elevated CO2 affected B. oleracea but not B. nigra glucosinolates; responses to soil fertility and da

99 ar in free water in the posterior substantia nigra in a large cohort of de novo patients with Parkins

100 the D-loop region is found in the substantia nigra in Parkinson disease (n = 10) with respect to cont

101 in other brain regions beyond the substantia nigra in Parkinson's disease, multiple system atrophy, a

102 OS)-positive myeloid cells in the substantia nigra in response to alpha-synuclein overexpression or L

103 , overexpression of necdin in the substantia nigra in vivo of adult mice protects dopaminergic neuron

104 free water level in the posterior substantia nigra increased over 1 year in de novo Parkinson's disea

105 66017 when microinjected into the substantia nigra, infralimbic cortex, BLA, and CeA.

106 ses and two anthocyanidin reductases from P. nigra involved in catalyzing the last steps of flavan-3-

107 that free water in the posterior substantia nigra is a valid, progression imaging marker of Parkinso

108 Free-water in the posterior substantia nigra is elevated in Parkinson's disease, increases with

109 depth characterization from human substantia nigra is necessary.

110 Morus nigra L. is a beneficial food due to rich phenolic compo

111 Elderberry (EDB) Sambucus nigra L. is one of the oldest medicinal plants which is

112 prenoids profile from elderflowers (Sambucus nigra L.) was established for two cultivars by multidime

113 f Aleuria Aurantia Lectin (AAL) and Sambucus Nigra Lectin (SNA).

114 od and treadmill tests, caused by substantia nigra lesioning in mice.

115 = 13), globus pallidus (n = 13), substantia nigra (n = 13), posterior thalamus (n = 12), red nucleus

116 ltured neurons in vitro, in mouse substantia nigra neurons in vivo and in human fibroblasts from an i

117 Similarly, TRPC1(-/-) substantia nigra neurons showed increased L-type Ca(2+) currents, d

118 dopaminergic (DA) neurons in the substantia nigra, non-motor symptoms including anxiety, cognitive d

119 Direct measures of DNAm in the substantia nigra of 39 cases and 13 control samples were used to in

120 the burden of alpha-syn in DA neurons in the nigra of A53T transgenic (A53T-Tg) mouse.

121 n of human alpha-synuclein in the substantia nigra of aged (18 to 21-month-old) L444P Gba1 mice.

122 of macaque monkeys, close to the substantia nigra of both sides.

123 ites in the entorhinal cortex and substantia nigra of control human postmortem brains, using the 454

124 and mutant alpha-synuclein in the substantia nigra of mice demonstrated that blocking alpha-synuclein

125 ed in dopaminergic neurons of the substantia nigra of Parkinson's disease (PD) patients and decreased

126 to be increased in the posterior substantia nigra of Parkinson's disease compared with controls at a

127 ses that free-water levels in the substantia nigra of Parkinson's disease increase following 1 year o

128 ve provided mixed findings in the substantia nigra of Parkinson's disease, but recent work using a bi

129 sed in the anterior and posterior substantia nigra of Parkinson's disease, multiple system atrophy, a

130 ally over 1 year in the posterior substantia nigra of Parkinson's disease.

131 loss of pigmented neurons in the substantia nigra of parkinsonian patients.

132 s were increased in the posterior substantia nigra of patients with Parkinson's disease compared with

133 regated synuclein deposits in the substantia nigra of patients with Parkinson's disease.

134 significantly upregulated in the substantia nigra of patients with Parkinson's disease.

135 ctivity have been observed in the substantia nigra of PD patients, and loss of Parkin results in the

136 er a (RGMa) is upregulated in the substantia nigra of PD patients.

137 droxylase-positive neurons in the substantia nigra of PLP-SYN mice.

138 When injected into the substantia nigra of rat brains, DOPAL causes the loss of dopaminerg

139 ical diseases are enhanced in the substantia nigra of rats with alpha-SYN overexpression, and inhibit

140 ynuclein toxicity in vivo, in the substantia nigra of rats.

141 cortex, hippocampus, stratum and substantia nigra of the nGD mice.

142 melanin-containing neurons in the substantia nigra (off-target binding).

143 regions of involvement (striatum, substantia nigra, olivary and pontine nuclei, hippocampus, forebrai

144 a swallow-tail appearance in the substantia nigra on high-resolution SWI, representing nigrosome-1,

145 very of human alpha-SYN into their sustantia nigra or by treatment with l-DOPA, suggesting that alpha

146 ressed in striatoentopeducuncular/substantia nigra or striatopallidal pathways, respectively.

147 es constitutively emitting isoprene (Populus nigra) or monoterpenes (Quercus ilex), or that do not em

148 Parkinson's disease (PD), in the substantia nigra par compacta (SNpc) of the brain in a PD mouse mod

149 d dopaminergic neurons in the rat substantia nigra pars compact, increases the recruitment of endogen

150 Dopamine neurons from the substantia nigra pars compacta (SNc) and ventral tegmental area (VT

151 ventral tegmental area (VTA) and substantia nigra pars compacta (SNC) contain dopamine neurons inter

152 Substantia nigra pars compacta (SNc) dopamine neurons and their tar

153 ior studies have established that substantia nigra pars compacta (SNc) dopamine neurons are a key nod

154 jor factor underlying the loss of substantia nigra pars compacta (SNc) dopaminergic neurons in Parkin

155 Burst spiking in substantia nigra pars compacta (SNc) dopaminergic neurons is a key

156 vior that depend on the firing of substantia nigra pars compacta (SNc) dopaminergic neurons, we ident

157 egion homologous to the mammalian substantia nigra pars compacta (SNc) evokes increasing activation o

158 dopaminergic (DA) neurons of the substantia nigra pars compacta (SNc) govern movements requires a de

159 le of this receptor in regulating substantia nigra pars compacta (SNc) neuron physiology in both mice

160 unctional heterogeneity among the substantia nigra pars compacta (SNc) neurons.

161 o what extent dopamine neurons in substantia nigra pars compacta (SNc) play such roles.

162 The substantia nigra pars compacta (SNc) projects specifically into the

163 gic (mdDA) neurons, including the substantia nigra pars compacta (SNc) subpopulation that preferentia

164 of dopamine neuron in the monkey substantia nigra pars compacta (SNc) that retains past learned rewa

165 e nigrostriatal pathway, from the substantia nigra pars compacta (SNc) to the dorsal striatum, and on

166 Most dopaminergic neurons in the substantia nigra pars compacta (SNc), but not in ventral tegmental

167 t has been suggested that, in the substantia nigra pars compacta (SNc), the pacemaking relies more on

168 nd project to dopamine neurons in substantia nigra pars compacta (SNc), whereas matrix neurons receiv

169 ty of dopamine neurons within the substantia nigra pars compacta (SNc).

170 on of dopaminergic neurons in the substantia nigra pars compacta (SNc).

171 ly in dopaminergic neurons of the substantia nigra pars compacta (SNc).

172 ventral tegmental area (VTA) and substantia nigra pars compacta (SNc).

173 es of dopamine neurons within the substantia nigra pars compacta (SNc).

174 tects dopaminergic neurons of the substantia nigra pars compacta (SNpc) against 6-OHDA and MPTP.

175 dopaminergic (DA) neurons in the substantia nigra pars compacta (SNpc) and of noradrenergic neurons

176 ons onto these neurons in the rat substantia nigra pars compacta (SNpc) and ventral tegmental area (V

177 y loss of dopamine neurons in the substantia nigra pars compacta (SNpc) and widespread aggregates of

178 higher centers, compromising the substantia nigra pars compacta (SNpc) and, later, the cerebral cort

179 unoreactive (THir) neurons in the substantia nigra pars compacta (SNpc) compared with saline treatmen

180 function/degeneration of midbrain substantia nigra pars compacta (SNpc) dopaminergic (DA) neurons con

181 ed in dopaminergic neurons of the substantia nigra pars compacta (SNpc) of human PD patients.

182 ergic (DA) neurons at the ventral substantia nigra pars compacta (SNpc) preferentially degenerate in

183 t loss of dopaminergic neurons in substantia nigra pars compacta (SNpc), and there was no loss of dop

184 rogressive degeneration of DNs in substantia nigra pars compacta (SNpc), decreased striatal dopamine

185 signal intensity loss within the substantia nigra pars compacta (SNpc), locus coeruleus, and ventral

186 lume of dopaminergic cells in the substantia nigra pars compacta (SNpc), ventral tegmental area (VTA)

187 linergic terminals in the ventral substantia nigra pars compacta (vSNc) or to the ventral tegmental a

188 s of dopaminergic (DA) neurons in substantia nigra pars compacta and age-dependent L-DOPA-sensitive m

189 of dopaminergic neurons from the substantia nigra pars compacta and noradrenergic neurons from the l

190 cell trafficking, in vivo in the substantia nigra pars compacta and the serum of 1-methyl-4-phenyl-1

191 c dopamine neurons located in the substantia nigra pars compacta and the ventral tegmental area, whic

192 dopaminergic (DA) neurons in the substantia nigra pars compacta and ventral tegmental area regulate

193 Dopamine neurons in the substantia nigra pars compacta and ventral tegmental area regulate

194 uced dopamine terminal damage and substantia nigra pars compacta cell loss.

195 in vivo recordings of identified substantia nigra pars compacta dopamine neurons in R1441C LRRK2 tra

196 ity and thereby properly maintain substantia nigra pars compacta dopaminergic neurons and their inner

197 Studying mouse substantia nigra pars compacta dopaminergic neurons both in brain s

198 ive degeneration of DA neurons in substantia nigra pars compacta is a key neuropathological feature i

199 on of dopaminergic neurons in the substantia nigra pars compacta is the primary cause for motor sympt

200 type B (trkB) receptor occurs in substantia nigra pars compacta neurons and is required for neuropro

201 vulnerable dopaminergic (DAergic) substantia nigra pars compacta neurons, only select downregulation

202 xin were also up-regulated in the substantia nigra pars compacta of post-mortem PD brains as compared

203 on of dopaminergic neurons in the substantia nigra pars compacta portion of the brain.

204 pigmented nucleus and dorsomedial substantia nigra pars compacta) mesodiencephalic dopaminergic neuro

205 ration of dopamine neurons of the substantia nigra pars compacta, a deficit in dopamine neurotransmis

206 nd in dopaminergic neurons of the substantia nigra pars compacta, a susceptible brain region in PD.

207 l protein accumulation within the substantia nigra pars compacta, suggesting that nigrostriatal dopam

208 ous subgroups of neurons, such as substantia nigra pars compacta, ventral tegmental area and retrorub

209 s and dopaminergic neurons of the substantia nigra pars compacta.

210 l loss of dopamine neurons in the substantia nigra pars compacta.

211 in the ventral tegmental area and substantia nigra pars compacta.

212 d dopamine neuron activity in the substantia nigra pars compacta.

213 ntials in dopamine neurons of the substantia nigra pars compacta.

214 vated dopaminergic neurons in the substantia nigra pars compacta.

215 iated dopamine neurons from mouse substantia nigra pars compacta.

216 in the dorsal GL or in the right substantia nigra pars compacta.

217 not in ventral tegmental area or substantia nigra pars lateralis, consistently represented the onset

218 caudal-dorsal-lateral part of the substantia nigra pars reticulata (cdlSNr), directly or indirectly t

219 Hz range in LFPs recorded in the substantia nigra pars reticulata (SNpr) and motor cortex (MCx) in t

220 e pedunculopontine nucleus to the substantia nigra pars reticulata (SNr) act on muscarinic acetylchol

221 of the basal ganglia through the substantia nigra pars reticulata (SNr) controls active avoidance.

222 g, we measured BG output from the substantia nigra pars reticulata (SNr) in mice while monitoring the

223 y active GABAergic neurons of the substantia nigra pars reticulata (SNr), a major output of the basal

224 reach their midbrain target, the substantia nigra pars reticulata (SNr), at E14 in the mouse with a

225 We recorded from the substantia nigra pars reticulata (SNR), the major basal ganglia out

226 ipsilateral and the contralateral substantia nigra pars reticulata (SNr).

227 eurons in the dorsal striatum and substantia nigra pars reticulata by activating TRPM2 channels.

228 asured in the globus pallidus and substantia nigra pars reticulata is caused by abnormal striatal act

229 nstant the average firing rate of substantia nigra pars reticulata reduces the incidence of seizures.

230 ergic output projections from the substantia nigra pars reticulata to the deep layers of the superior

231 We found that activity in the substantia nigra pars reticulata, a basal ganglia output, predictab

232 nervated GABAergic neurons in the substantia nigra pars reticulata.

233 ulus deltoides with gamma-irradiated Populus nigra pollen to produce >500 F1 seedlings containing dos

234 baseline free-water levels in the substantia nigra predict the change in bradykinesia following 1 yea

235 se in free water in the posterior substantia nigra predicts subsequent long-term progression on the H

236 ections to entopeduncular nucleus/substantia nigra, preferentially expressing D1 receptors that stimu

237 o develop non-invasive markers of substantia nigra progression in Parkinson's disease.

238 Substantia nigra, putamen, and cortical p11 protein levels were ass

239 s seen in the posterior thalamus, substantia nigra, red nucleus, cerebellar peduncle, colliculi, dent

240 nalyzed MTA1 and TH levels in the substantia nigra region of a large cohort of human brain tissue sam

241 tivity of adult DA neurons in the substantia nigra region.

242 of neuromelanin granules in human substantia nigra required high tissue amounts.

243 (MSNs) directly projecting to the substantia nigra reticulata (SNr) lose tonic presynaptic inhibition

244 nally opposes DOP transmission in substantia nigra reticulata and that NOP receptor antagonists might

245 three different elderberry species (Sambucus nigra, Sambucus cerulea, Sambucus javanica) and seven in

246 ta, Euterpe oleracea, Malusxdomestica, Morus nigra, Sambucus nigra, Vaccinium macrocarpon, Vaccinium

247 stochemistry analysis for MTA1 in substantia nigra sections revealed that 74.1% of the samples had a

248 nstrate that the VTA, but not the substantia nigra, sends dense intra- and interhemispheric projectio

249 on of dopaminergic neurons in the substantia nigra (SN) and affected the integrity of the nigrostriat

250 uding dopaminergic neurons of the substantia nigra (SN) and cholinergic neurons of the dorsal motor n

251 inergic (DA) neuronal loss in the substantia nigra (SN) and ventral tegmental area (VTA), supporting

252 The substantia nigra (SN) consists of GABAergic neurons of the pars ret

253 uction leading to degeneration of substantia nigra (SN) dopamine (DA) neurons, mimicking idiopathic P

254 DBS provides neuroprotection for substantia nigra (SN) dopamine neurons and increases BDNF in the ni

255 tenuated the MPTP-induced loss of substantia nigra (SN) dopamine neurons and striatal dopamine turnov

256 ogically by the selective loss of substantia nigra (SN) dopaminergic (DAergic) neurons.

257 ventral tegmental area (VTA) and substantia nigra (SN) has been examined at multiple levels.

258 The dopaminergic substantia nigra (SN) is implicated in the drive to explore novel s

259 f highly vulnerable dopamine (DA) substantia nigra (SN) neurons.

260 Dopaminergic neurons of the substantia nigra (SN) play a vital role in everyday tasks, such as

261 DA neurons originating in the substantia nigra (SN) projecting to the dorsal striatum (DS) are tr

262 The substantia nigra (SN) provides the largest dopaminergic input to th

263 y of dopamine (DA) neurons in the substantia nigra (SN) to neurodegenerative stressors causes Parkins

264 ctional organization of the human substantia nigra (SN) using diffusion and functional MRI data from

265 udate, putamen, ventral striatum, substantia nigra (SN), and cerebellum were manually drawn on coregi

266 pha-syn fibril seeds into the rat substantia nigra (SN), in combination with adenoassociated virus (A

267 mental area (VTA), but not in the substantia nigra (SN), of D2R-OE mice.

268 : the dentate nucleus (DN), pons, substantia nigra (SN), pulvinar thalami, and globus pallidus (GP).

269 dopaminergic (DA) neurons in the substantia nigra (SN), which may contribute to their selective dege

270 ere injected unilaterally, in the substantia nigra (SN), with AAV1/2-A53T-aSyn or control vector.

271 l area (VTA) and the other in the substantia nigra (SN).

272 sporter (DAT) in the striatum and substantia nigra (SN).

273 lpha-synuclein (alpha-syn) in the substantia-nigra (SN).

274 3 ng/mL of glycosylated fPSA using Sambucus nigra (SNA) lectin, both within the relevant clinical ra

275 ncentrations were assessed in the substantia nigra (SNc), dentate and caudate nucleus, red nucleus, p

276 with increased iron levels in the substantia nigra (SNc).

277 us through different parts of the substantia nigra so that the animal looks preferentially at high-va

278 We show in Arabidopsis thaliana and Brassica nigra that localized FR enrichment at the lamina tip ind

279 to the ventral tegmental area and substantia nigra; the dopamine systems themselves; glutamatergic af

280 reasing TGF-beta signaling in the substantia nigra through adeno-associated virus expressing a consti

281 ilar to those isolated from human substantia nigra tissues.

282 e loss of dopamine neurons in the substantia nigra together with the presence of Lewy bodies and Lewy

283 (mood, anxiety), and inferior STN/substantia nigra (UPDRS tremor, working memory).

284 acea, Malusxdomestica, Morus nigra, Sambucus nigra, Vaccinium macrocarpon, Vaccinium myrtillus, Vitis

285 ntrast to dopamine neurons in the substantia nigra, vagal motoneurons do not enhance their excitabili

286 mber of CARTp-ir terminals in the substantia nigra, ventral tegmental area, periaqueductal gray, para

287 r binding in D3-rich regions: the substantia nigra/ventral tegmental area (SN/VTA) (+20%; p=0.02), ve

288 ding, BOLD response slopes in the Substantia Nigra/Ventral Tegmental Area (SN/VTA) and ventral striat

289 pends on interactions between the substantia nigra/ventral tegmental area complex (SN/VTA) and the hi

290 and the mean total (18)F-AV-1451 substantia nigra volume of distribution was decreased by 33% after

291 o value in ventral tegmental area/substantia nigra (VTA/SN) shows context-sensitivity, an effect enha

292 rosine hydroxylase neurons in the substantia nigra was determined by stereological tests after MPTP i

293 Neuronal loss of the substantia nigra was either absent or very mild in the preclinical

294 that free water in the posterior substantia nigra was elevated in Parkinson's disease compared to co

295 The bilateral substantia nigra was evaluated by two neuroradiologists for the pre

296 ough marked infiltration of T cells into the nigra was found on 1 d of MPTP insult, T cell infiltrati

297 his study, poplar trees (Populus deltoides x nigra) were exposed hydroponically to 50-nM CdSe/CdZnS Q

298 ity of flavan-3-ols in black poplar (Populus nigra), which include both monomers, such as catechin, a

299 investigate the stress responses of Brassica nigra (wild black mustard) exposed consecutively to O3 a

300 opolysaccharide injections in the substantia nigra, with a marked increase in the recruitment of CD68