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1 hat the fIJP was purinergic and the sIJP was nitrergic.
2 cating that they were cholinergic as well as nitrergic.
4 of the intrinsic innervation; inhibition of nitrergic and VIPergic electrical field-induced relaxati
5 vival of later-born dopaminergic, GABAergic, nitrergic, and calcitonin gene-related peptide-expressin
9 lexus contained cholinergic, adrenergic, and nitrergic axons and vasoactive intestinal polypeptide-po
12 ion of enteric neurons, with cholinergic and nitrergic components blocked, evoked Ca(2+) transients i
15 alpha7-nAChRs, induced sympathetic-dependent nitrergic dilation of porcine basilar arterial rings.
17 al brainstem region is the primary source of nitrergic fibers in the mouse visual thalamus, similar t
18 ture preparations indicated that most of the nitrergic fibers within cardiac ganglia were extrinsic.
22 trast, basal pyloric hypotension with normal nitrergic inhibition predisposes W/W(v) mice to duodenog
28 evoked prominent cholinergic excitatory and nitrergic inhibitory responses in longitudinal muscles o
31 neurons indicates that: i) DR is a source of nitrergic input to trigeminal structures, and ii) NO and
32 d that the dendritic and axonal processes of nitrergic interneurons already apposed the somata and de
39 onsistent with the idea that cholinergic and nitrergic motor neural inputs are mediated, to a large e
40 motor neurones revealed that cholinergic and nitrergic motor neurones formed close contacts with ICC-
42 e assessed, in addition to colonic and ileal nitrergic myenteric neuron quantifications and motility.
51 ta2-adrenoceptors located on the neighboring nitrergic nerve terminals, resulting in NO release and v
53 Like MPOs, their activity is inhibited by nitrergic nerves and synchronized by excitatory nerves.
58 ut not direct stimulation of parasympathetic nitrergic nerves, caused nitrergic neurogenic dilation.
61 ors of excitatory cholinergic and inhibitory nitrergic neural inputs from the enteric nervous system,
63 IR was coexpressed with both cholinergic and nitrergic neurochemical codes in the myenteric plexus.
65 studying cellular and synaptic mechanisms of nitrergic neuromodulation in a simple pattern-generating
66 to neurons with cholinergic, calretinin, and nitrergic neuronal immunochemical codes in the myenteric
69 establish accurately the localization of the nitrergic neurons and fibers and to assess possible inte
70 rgic subpopulation and decreased the area of nitrergic neurons and vasoactive intestinal peptide (VIP
71 Glutamatergic, GABAergic, cholinergic, and nitrergic neurons are all found in the PCRt and IRt, but
72 ay mediates caspase-3 dependent apoptosis of nitrergic neurons in the MPG following CNI and that ROCK
73 rointestinal motility and reduced numbers of nitrergic neurons in TLR4(Lps-d), TLR4(-/-), and Myd88(-
74 Significant populations of cholinergic and nitrergic neurons overlapped pre-oromotor neurons, but t
76 nly two regions of the mouse brain contained nitrergic neurons that projected to the visual thalamus:
79 trate that nitric oxide synthase-containing (nitrergic) neurons innervating the penis and gastric pyl
80 L-citrulline recycling in the maintenance of nitrergic neurotransmission in the opossum internal anal
83 ine (l-NNA; 100 mum) to block purinergic and nitrergic NMT to characterize non-purinergic, non-nitrer
85 rgic NMT to characterize non-purinergic, non-nitrergic (NNNP) inhibitory NMT and the role of VIP in t
87 f NO-containing fibers with respect to other nitrergic profiles in the somatosensory system; and 3) t
88 onkey LGN receives a significant cholinergic/nitrergic projection from the pedunculopontine tegmentum
91 mportant role for ICC-IM in vagally-mediated nitrergic relaxation in the proximal and distal stomach.
92 We investigated the role of ICCs and SMCs in nitrergic relaxation using mice with cell-specific disru
98 ults provide evidence for the existence of a nitrergic signalling system in molluscan larvae and juve
99 rstitial cells of Cajal (ICCs), might detect nitrergic signals to indirectly regulate smooth muscle t
100 of the canine gastric fundus in response to nitrergic stimulation by NO donors and electrical field
101 uction in [Ca2+]i and tension in response to nitrergic stimulation of the canine gastric fundus does
102 uanylyl cyclase, also blocked the effects of nitrergic stimulation on [Ca2+]i and tension, without af
104 Block of hyperpolarization in response to nitrergic stimulation with tetrapentylammonium chloride
105 he neuronal areas of the general population, nitrergic subpopulation and (VIP) varicosities were redu
106 he neuronal areas of the general population, nitrergic subpopulation and (VIP) varicosities were redu
107 odenum, the HFD increased the density of the nitrergic subpopulation and decreased the area of nitrer
109 m, the density of the general population and nitrergic subpopulation were increased and the neuronal
110 the electrical excitability of MNCs via the nitrergic system and strengthen the importance of HCN ch
111 s a conserved pattern of organization of the nitrergic system from this primitive fish group to amnio
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