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1 hat the fIJP was purinergic and the sIJP was nitrergic.
2 cating that they were cholinergic as well as nitrergic.
3                                              Nitrergic and alpha2-adrenergic modulation of gastric se
4  of the intrinsic innervation; inhibition of nitrergic and VIPergic electrical field-induced relaxati
5 vival of later-born dopaminergic, GABAergic, nitrergic, and calcitonin gene-related peptide-expressin
6 a were contacted by cholinergic, adrenergic, nitrergic, and VIP-positive terminals.
7  immunoreactivity, indicating that they were nitrergic as well.
8  immunoreactivity, indicating that they were nitrergic as well.
9 lexus contained cholinergic, adrenergic, and nitrergic axons and vasoactive intestinal polypeptide-po
10                         In the second phase, nitrergic cell bodies in the ganglia are lost via apopto
11                                          The nitrergic component is characterized by membrane potenti
12 ion of enteric neurons, with cholinergic and nitrergic components blocked, evoked Ca(2+) transients i
13           This is the first demonstration of nitrergic degeneration in diabetic cerebral arteries, wh
14                         In the second phase, nitrergic degeneration takes place in the cell bodies in
15 alpha7-nAChRs, induced sympathetic-dependent nitrergic dilation of porcine basilar arterial rings.
16  Thus, hyperpolarization is not required for nitrergic effects on [Ca2+]i and tension.
17 al brainstem region is the primary source of nitrergic fibers in the mouse visual thalamus, similar t
18 ture preparations indicated that most of the nitrergic fibers within cardiac ganglia were extrinsic.
19  in the ganglia, leading to complete loss of nitrergic function.
20  purinergic component was reduced, while the nitrergic IJP was slightly increased.
21 ance in smooth muscles and may contribute to nitrergic inhibition of gastrointestinal muscles.
22 trast, basal pyloric hypotension with normal nitrergic inhibition predisposes W/W(v) mice to duodenog
23                      Slow wave amplitude and nitrergic inhibitory junction potentials were reduced wh
24 it TREK-1 channels, native SDK channels, and nitrergic inhibitory junction potentials.
25 ortance of ICC in cholinergic excitatory and nitrergic inhibitory motor neurotransmission.
26 P developed, both cholinergic excitatory and nitrergic inhibitory neural responses were intact.
27 ial cells of Cajal (ICC) act as mediators in nitrergic inhibitory neurotransmission.
28  evoked prominent cholinergic excitatory and nitrergic inhibitory responses in longitudinal muscles o
29               In this study, we examined the nitrergic innervation to the adult mouse visual thalamus
30 -d) histochemistry to identify the source of nitrergic innervation.
31 neurons indicates that: i) DR is a source of nitrergic input to trigeminal structures, and ii) NO and
32 d that the dendritic and axonal processes of nitrergic interneurons already apposed the somata and de
33             However, it is not known whether nitrergic interneurons are in this position early in lif
34                                 In contrast, nitrergic interneurons developed more rapidly.
35                     The results suggest that nitrergic interneurons play a role in refining the corti
36                Nitric oxide (NO) containing (nitrergic) interneurons are well-positioned to convey th
37 duced by l-NAME administration, suggesting a nitrergic mechanism of gastroinhibition.
38 opment of acute inflammatory hyperaemia, but nitrergic mechanisms may supervene subsequently.
39 onsistent with the idea that cholinergic and nitrergic motor neural inputs are mediated, to a large e
40 motor neurones revealed that cholinergic and nitrergic motor neurones formed close contacts with ICC-
41                  These disorders precede the nitrergic myenteric cell loss observed in the proximal c
42 e assessed, in addition to colonic and ileal nitrergic myenteric neuron quantifications and motility.
43  disorders inducing constipation and loss of nitrergic myenteric neurons in the proximal colon.
44                DOReGFP was present mostly in nitrergic myenteric neurons of colon.
45                               The numbers of nitrergic myenteric neurons were reduced in the proximal
46 ess the impact of these factors on autonomic nitrergic nerve and endothelial function.
47                          In the first phase, nitrergic nerve fibers lose some of their neuronal nitri
48             In the first phase, perivascular nitrergic nerve fibers remain intact while they lose the
49 ponsiveness and greatly reduces responses to nitrergic nerve stimulation.
50       NO, produced by NOS-1 in pericytes and nitrergic nerve terminals and by NOS-3 in vascular endot
51 ta2-adrenoceptors located on the neighboring nitrergic nerve terminals, resulting in NO release and v
52                                              Nitrergic nerves and interstitial cells of Cajal (ICC) h
53    Like MPOs, their activity is inhibited by nitrergic nerves and synchronized by excitatory nerves.
54              Here, we show that perivascular nitrergic nerves around the cerebral arteries degenerate
55           However, the effect of diabetes on nitrergic nerves is unknown.
56                    It has been proposed that nitrergic nerves mediate lower esophageal sphincter (LES
57                              Cholinergic and nitrergic nerves were also present in W/W(V) mice, but f
58 ut not direct stimulation of parasympathetic nitrergic nerves, caused nitrergic neurogenic dilation.
59 ng nitric oxide (NO); hence, they are called nitrergic nerves.
60 ould occur via nonadrenergic, noncholinergic nitrergic neural and/or endocrine pathways.
61 ors of excitatory cholinergic and inhibitory nitrergic neural inputs from the enteric nervous system,
62 used loss of ICC and loss of cholinergic and nitrergic neural responses.
63 IR was coexpressed with both cholinergic and nitrergic neurochemical codes in the myenteric plexus.
64  of parasympathetic nitrergic nerves, caused nitrergic neurogenic dilation.
65 studying cellular and synaptic mechanisms of nitrergic neuromodulation in a simple pattern-generating
66 to neurons with cholinergic, calretinin, and nitrergic neuronal immunochemical codes in the myenteric
67 h in the ganglia, which is selective for the nitrergic neurones.
68                          The analysis of the nitrergic neurons (with the enzyme nitric oxide synthase
69 establish accurately the localization of the nitrergic neurons and fibers and to assess possible inte
70 rgic subpopulation and decreased the area of nitrergic neurons and vasoactive intestinal peptide (VIP
71   Glutamatergic, GABAergic, cholinergic, and nitrergic neurons are all found in the PCRt and IRt, but
72 ay mediates caspase-3 dependent apoptosis of nitrergic neurons in the MPG following CNI and that ROCK
73 rointestinal motility and reduced numbers of nitrergic neurons in TLR4(Lps-d), TLR4(-/-), and Myd88(-
74   Significant populations of cholinergic and nitrergic neurons overlapped pre-oromotor neurons, but t
75                   Each of these identifiable nitrergic neurons projects into the ipsilateral pedal ga
76 nly two regions of the mouse brain contained nitrergic neurons that projected to the visual thalamus:
77            In contrast, less than 10% of the nitrergic neurons were activated after TNF exposure.
78  did not act directly on the parasympathetic nitrergic neurons.
79 trate that nitric oxide synthase-containing (nitrergic) neurons innervating the penis and gastric pyl
80 L-citrulline recycling in the maintenance of nitrergic neurotransmission in the opossum internal anal
81           The data show that cholinergic and nitrergic neurotransmission only occurs in the gastric l
82 suggesting that ICC-IM do not play a role in nitrergic neurotransmission.
83 ine (l-NNA; 100 mum) to block purinergic and nitrergic NMT to characterize non-purinergic, non-nitrer
84 timuli (4 s) were mediated by purinergic and nitrergic NMT.
85 rgic NMT to characterize non-purinergic, non-nitrergic (NNNP) inhibitory NMT and the role of VIP in t
86                           Dysfunction of the nitrergic pathway has been shown to cause LES hypertensi
87 f NO-containing fibers with respect to other nitrergic profiles in the somatosensory system; and 3) t
88 onkey LGN receives a significant cholinergic/nitrergic projection from the pedunculopontine tegmentum
89                   In nNOS(-/-) mice, loss of nitrergic pyloric inhibition leads to gastric stasis and
90                             In regard to the nitrergic regulation of the circulation, the fetus is cl
91 mportant role for ICC-IM in vagally-mediated nitrergic relaxation in the proximal and distal stomach.
92 We investigated the role of ICCs and SMCs in nitrergic relaxation using mice with cell-specific disru
93                                     However, nitrergic relaxation was not observed in SM/ICC-GCKO mic
94 atus may be the primary mechanisms mediating nitrergic responses in these muscles.
95 NOS(+) neurons and subsequent restoration of nitrergic responses.
96 g in Aglantha, possibly by means of putative nitrergic sensory neurons in its tentacles.
97 from the combination of SMCs and ICCs blocks nitrergic signaling.
98 ults provide evidence for the existence of a nitrergic signalling system in molluscan larvae and juve
99 rstitial cells of Cajal (ICCs), might detect nitrergic signals to indirectly regulate smooth muscle t
100  of the canine gastric fundus in response to nitrergic stimulation by NO donors and electrical field
101 uction in [Ca2+]i and tension in response to nitrergic stimulation of the canine gastric fundus does
102 uanylyl cyclase, also blocked the effects of nitrergic stimulation on [Ca2+]i and tension, without af
103  reductions in [Ca2+]i and tension caused by nitrergic stimulation were blocked.
104    Block of hyperpolarization in response to nitrergic stimulation with tetrapentylammonium chloride
105 he neuronal areas of the general population, nitrergic subpopulation and (VIP) varicosities were redu
106 he neuronal areas of the general population, nitrergic subpopulation and (VIP) varicosities were redu
107 odenum, the HFD increased the density of the nitrergic subpopulation and decreased the area of nitrer
108           In the jejunum, the density of the nitrergic subpopulation was increased and the neuronal a
109 m, the density of the general population and nitrergic subpopulation were increased and the neuronal
110  the electrical excitability of MNCs via the nitrergic system and strengthen the importance of HCN ch
111 s a conserved pattern of organization of the nitrergic system from this primitive fish group to amnio
112                          We investigated the nitrergic system of two cladistian species by means of s

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