ライフサイエンスコーパス: nitric

1 A method using digestion with diluted nitric acid and inductively coupled plasma optical emiss

2 egy is described in which nitrogen oxides or nitric acid are directly employed in photocatalyzed hydr

3 y to the quantification of Pu(IV) unless the nitric acid concentration is known and separate calibrat

4 tion of Pu(IV) without a priori knowledge of nitric acid concentration.

5 significant spectral variation with changing nitric acid concentration.

6 and heavy metals were digested using closed-nitric acid digestion and Rijksinstituut voor Volksgezon

7 Recently a dilute nitric acid extraction (0.43 M) was adopted by ISO (ISO-

8 d speciated using both a quantitative dilute nitric acid extraction and speciation (DNAS) and an in v

9 lurries were prepared using Triton X-100 and nitric acid for direct analysis of Pb using graphite fur

10 ed digestion step using sodium hydroxide and nitric acid in combination to digest all organic materia

11 e degree of sensitisation (DoS) as judged by nitric acid mass loss testing (ASTM-G67-04), and discuss

12 Decalcification with 7% nitric acid proceeded more rapidly than with 0.5 M EDTA

13 ly but incompletely displaced, likely by the nitric acid produced largely by the heterogeneous uptake

14 e extracting aqueous solution (0.1 mol.L(-1) nitric acid) on the sample.

15 ile species found in the atmosphere, such as nitric acid, ammonia, and water.

16 Popularly utilized oxidation media, via nitric acid/sulfuric acid mixtures, are too corrosive an

17 m reaction of superoxide anion (O2(*-)) with nitric oxide (.NO) and has been suggested to form an as-

18 Exhaled nitric oxide (eNO) is a biomarker of airway inflammation

19 ociation between baseline fractional exhaled nitric oxide (FeNO) and the response to inhaled corticos

20 On the other hands, the fraction of exhaled nitric oxide (FENO) is a useful noninvasive marker of eo

21 re examined for their analytical response to nitric oxide (NO(*)).

22 Nitric oxide (NO) activates the NO-sensitive soluble gua

23 tosis was accompanied by decreased levels of nitric oxide (NO) and inducible NO synthase (iNOS) in ce

24 mined the kinetics of growth and turnover of nitric oxide (NO) and N2 O at low cell densities of Nitr

25 ll removed through chemical decomposition to nitric oxide (NO) and nitrate.

26 g bacteria (AOB) emit substantial amounts of nitric oxide (NO) and nitrous oxide (N2O), both of which

27 potassium channels (KIR ), and synthesis of nitric oxide (NO) and prostaglandins (PG).

28 Although the wound healing effects of nitric oxide (NO) are known, the mechanism by which NO m

29 mic analysis in endothelial cells identified nitric oxide (NO) as major mediator of this phenotype in

30 f these heme-silk films for the detection of nitric oxide (NO) at nanomolar levels in the presence an

31 Hepatic and systemic hemodynamic, nitric oxide (NO) bioavailability, LF, HSC activation, a

32 ABSTRACT: Endothelial nitric oxide (NO) controls cardiac oxygen (O2 ) consumpt

33 KEY POINTS: In the heart, endothelial nitric oxide (NO) controls oxygen consumption in the wor

34 an plasma and cells serve important roles in nitric oxide (NO) dependent as well as NO independent si

35 ypothesized that Trpc6 would help to mediate nitric oxide (NO) dysregulation and that this would be m

36 Nitric oxide (NO) exerts pleiotropic effects on plant de

37 ssive degradation of its [4Fe-4S] cluster on nitric oxide (NO) exposure.

38 oxide synthase (NOS) uncoupling with reduced nitric oxide (NO) formation and greater oxidative and ni

39 nd direct approach to investigate sources of nitric oxide (NO) formed in soils.

40 Nitric oxide (NO) has been known to promote physiologica

41 und that Cavin-2 regulates the production of nitric oxide (NO) in endothelial cells by controlling th

42 The importance of nitric oxide (NO) in many biological processes has garne

43 els regulate flow-induced vasodilatation via nitric oxide (NO) in mouse mesenteric resistance arterie

44 cible nitric oxide synthase (iNOS) generates nitric oxide (NO) in myeloid cells that acts as a defens

45 Nitric oxide (NO) is a key messenger in plant stress res

46 Nitric oxide (NO) is a regulatory molecule in the vascul

47 Nitric oxide (NO) is able to lower intraocular pressure

48 Nitric oxide (NO) is an important regulator of inflammat

49 Nitric oxide (NO) is an intercellular messenger involved

50 Nitric oxide (NO) is an intermediate of the nitrogen cyc

51 itrification, a respiratory process in which nitric oxide (NO) is an intermediate.

52 Nitric oxide (NO) is generated from arginine and oxygen

53 Host nitric oxide (NO) is important in controlling TB infecti

54 Nitric oxide (NO) is inactivated by cell-free hemoglobin

55 The signaling molecule nitric oxide (NO) is synthesized in animals by structura

56 INTRODUCTION: Increased exhaled nitric oxide (NO) levels in asthma are suggested to be t

57 mammalian globin cytoglobin (CYGB) regulates nitric oxide (NO) metabolism and cell death.

58 he relationship between circulating arginine-nitric oxide (NO) metabolites and CTRCD remains unstudie

59 x species and it elucidates the influence of nitric oxide (NO) on sulfur trioxide (SO3) generation.

60 the reactive ammonia-oxidation intermediates nitric oxide (NO) or hydroxylamine (NH2OH) for N2O produ

61 , we demonstrate that bdMphi fail to produce nitric oxide (NO) or upregulate inducible nitric oxide s

62 his; however, the association of endothelial nitric oxide (NO) pathways with disease severity is unkn

63 a sigma (sigma) receptor pharmacophore and a nitric oxide (NO) photodonor.

64 Nitric oxide (NO) produced by endothelial cells in respo

65 ed to lower blood pressure, possibly through nitric oxide (NO) production in skin.

66 pendent activation of nitric oxide synthase, nitric oxide (NO) production, NO-dependent S-nitrosylati

67 ty is associated with elevated intracellular nitric oxide (NO) production, which promotes nitrosative

68 mulating these glutamate receptors increases nitric oxide (NO) production, which stimulates matrix me

69 and significant inhibitory activity against nitric oxide (NO) production.

70 otes is linked through a single mechanism to nitric oxide (NO) sensing [3, 4].

71 he inhibitory phosphorylation of endothelial nitric oxide (NO) synthase (eNOS) at Thr497 (eNOS(pThr49

72 Neurons expressing nitric oxide (NO) synthase (nNOS) and thus capable of sy

73 m the nitric oxide produced by the inducible nitric oxide (NO) synthase of macrophages.

74 n transport chain, and uncoupled endothelial nitric oxide (NO) synthase.

75 bryonic fibroblasts via mechanisms involving nitric oxide (NO) synthesis and posttranslational modifi

76 hemoglobin from ongoing hemolysis scavenges nitric oxide (NO) to create an NO deficiency which can a

77 asing evidence for the potential function of nitric oxide (NO) to overcome MDR.

78 yte colony-stimulating factor (G-CSF) and of nitric oxide (NO) upon challenge with corynebacterial gl

79 parent activity in the ready eliminateion of nitric oxide (NO) via facile redox reactions, and the el

80 emical sensor for the selective detection of nitric oxide (NO), a critical cancer biomarker.

81 troglycerin-mediated dilation (GMD), urinary nitric oxide (NO), and inflammatory markers were measure

82 production of pro-inflammatory cytokines and nitric oxide (NO), in response to LPS and alpha-synuclei

83 NOS), which generates the potent vasodilator nitric oxide (NO), is decreased.

84 e synthase (eNOS) and its bioactive product, nitric oxide (NO), mediate many endothelial cell functio

85 Reactions between nitric oxide (NO), nitrite (NO2-), and unsaturated fatty

86 Multiple lines of evidence point to impaired nitric oxide (NO)-cGMP bioavailability as playing a cent

87 s on glucose and lipid metabolism, has shown nitric oxide (NO)-dependent vasodilator properties in ex

88 KEY POINTS: Combining nitric oxide (NO)-mediated increased blood flow with ang

89 ferentially to neovascularization induced by nitric oxide (NO)-mediated vasodilatation, by comparing

90 e accompanied by increased soil emissions of nitric oxide (NO).

91 idamide, dihydrochloride (AAPH) radicals and nitric oxide (NO).

92 rdiovascular system, many of which depend on nitric oxide (NO).

93 several HDACs are subjected to regulation by nitric oxide (NO); in plants, however, it is unknown whe

94 ulfur limits so questions were posed whether nitric oxide (NOx) and particulate matter (PM) emissions

95 nction, respiratory symptoms, exhaled breath nitric oxide [eNO], exhaled carbon monoxide [eCO], and h

96 tor reversibility >/=12%, fractional exhaled nitric oxide [FeNO] >/=35 parts per billion, and peak ex

97 Modulation of nitric oxide activity through blockade of CD47 signaling

98 In addition, MSCs secreted nitric oxide after Mtb infection, and inhibition of NO b

99 e guanylyl cyclase (sGC) is the receptor for nitric oxide and a highly sought-after therapeutic targe

100 eover, we highlighted the cross-talk between nitric oxide and H2S in several bilogical contexts.

101 Aortic nitric oxide and Nos2 levels were higher in Adamts1-defi

102 reactivity in OZR reflects a loss in venular nitric oxide and PGI2 bioavailability, associated with t

103 indicating downstream signaling through both nitric oxide and prostaglandins.

104 ing lipopolysaccharide (LPS)-stimulated ROS, nitric oxide and TNFalpha production, which translated i

105 eral vascular resistance and the increase in nitric oxide are impaired or absent in salt sensitivity,

106 ic studies demonstrated that adiponectin and nitric oxide are released after activation of adipocyte-

107 Since then, the discovery of nitric oxide as a biological effector and better underst

108 NEET or Asp-123 to Val in Miner1 facilitates nitric oxide binding in the [2Fe-2S] cluster, indicating

109 ss spectrometry analyses further reveal that nitric oxide binds to the reduced [2Fe-2S] clusters in M

110 y contested given their ability to transduce nitric oxide bioactivity within the microcirculation.

111 dative stress (94.7 +/- 6.2%) and normalised nitric oxide bioavailability (115.6 +/- 22.3%) in the fe

112 e 5 expression (167 +/- 13.7%) and decreased nitric oxide bioavailability (54.7 +/- 6.1%) in the feta

113 s benefits on dilatory function and baseline nitric oxide bioavailability.

114 nted in ECSHIP2(Delta/+) mice, as was aortic nitric oxide bioavailability.

115 SOV of FAs reflects the local production of nitric oxide by the endothelium in response to luminal s

116 The nitric oxide concentration and nuclear factor-kappa B (N

117 Nitric oxide contributes to protection from tuberculosis

118 inine synthesis in urea cycle and citrulline-nitric oxide cycle.

119 on-PN intermediate in the catalytic cycle of nitric oxide dioxygenase (NOD) enzymes, which facilitate

120 The identity of the specific nitric oxide dioxygenase (NOD) that serves as the main i

121 n of ADP-induced platelet aggregation by the nitric oxide donor sodium nitroprusside and the phosphod

122 onal responses, detectable within minutes of nitric oxide exposure, typically took several hours to m

123 The nitric oxide formation from selected N-nitrosamines was

124 esidues in proteins are readily disrupted by nitric oxide forming a protein-bound dinitrosyl iron com

125 cuss novel approaches to improve endothelial nitric oxide generation and preserve its bioavailability

126 onchodilator response and fractional exhaled nitric oxide had good sensitivity and specificity for pr

127 ur proteins are among the primary targets of nitric oxide in cells.

128 sicians, and the fractional concentration of nitric oxide in exhaled air (FeNO) was measured.

129 ractions stimulate FA endothelium to release nitric oxide in response to elevated shear stress second

130 The results suggest that binding of nitric oxide in the CDGSH-type [2Fe-2S] clusters in mito

131 Binding of nitric oxide in the reduced Miner2 [2Fe-2S] clusters pro

132 Placebo (nitrogen) or inhaled nitric oxide initiated at 20 ppm was decreased to 10 ppm

133 s sputum eosinophilia and fractional exhaled nitric oxide levels, along with oral corticosteroid use,

134 inophils, as well as high fractional exhaled nitric oxide levels, exacerbation rates, and oral cortic

135 Pronounced elevations in trough levels of nitric oxide metabolites occurred with KNO3 (visit 2: 19

136 crophage function, we assessed cytotoxicity, nitric oxide or reactive oxygen species production, and

137 tility of pharmacological stimulation of the nitric oxide pathway as a therapeutic strategy.

138 ipids, indicate that both the urea cycle and nitric oxide pathways are dysregulated at early stages i

139 In contrast, we report that nitric oxide primarily protects mice by repressing an in

140 ve nitrogen species (RNS) generated from the nitric oxide produced by the inducible nitric oxide (NO)

141 synthesis, restored cellular BH4 levels and nitric oxide production and decreased radiation-induced

142 Blocking CX3CR1 or nitric oxide production during G-CSF treatment reduces e

143 g either CD23 upregulation or CD23-dependent nitric oxide production eliminated the enhanced antifung

144 educed by 30% cytokine-induced apoptosis and nitric oxide production in INS-1E cells.

145 pression profiles, oxygen consumption rates, nitric oxide production levels, shear stress responses,

146 ne stimulation in these cells also activated nitric oxide production that was blocked by sigma1-recep

147 lcium signals in primary cells that activate nitric oxide production to increase ciliary beating and

148 a stronger bactericidal activity with higher nitric oxide production, a more proinflammatory polarize

149 and Ca(2+) influx, which activates low-level nitric oxide production, increases apical membrane Cl(-)

150 euronal nitric oxide synthase activation and nitric oxide production.

151 ation, reduced HO-1 expression and increased nitric oxide production.

152 to HAT as implausible for the reaction with nitric oxide radical in all the solvents investigated.

153 l-1,3-butadiene) carrying nearly unperturbed nitric oxide radical to form [((R)DDB)Fe(NO)2((*)NO)](+)

154 se, one contained genes for both nitrate and nitric oxide reductase, and one had nitrate and nitrite

155 ights regarding the mechanism of flavodiiron nitric oxide reductases.

156 g as the terminal components to dioxygen and nitric oxide reductive scavenging pathways in these orga

157 city, oxygen radical absorbance capacity and nitric oxide scavenging assays.

158 are genetic variants in 2 genes that mediate nitric oxide signaling (Nitric Oxide Synthase 3 [NOS3] a

159 A genetic predisposition to enhanced nitric oxide signaling is associated with reduced risks

160 Pharmacological stimulation of nitric oxide signaling may prove useful in the preventio

161 chondrial protein Miner2 may represent a new nitric oxide signaling mode in cells.

162 pact of a genetic predisposition to enhanced nitric oxide signaling on risk for cardiovascular diseas

163 Nitric oxide signaling plays a key role in the regulatio

164 tion studies, we examined the effect of this nitric oxide signaling score on cardiometabolic and othe

165 lves a non-canonical mechanism of retrograde nitric oxide signalling, which is triggered by Ca(2+) in

166 on the cell surface appear to also modulate nitric oxide signalling; both signalling pathways affect

167 han in generating an immediate resistance to nitric oxide stress, suggesting that survival of M. tube

168 t-translational modifications of endothelial nitric oxide synthase (eNOS) lead to impaired nitric oxi

169 dysfunction, which is caused by endothelial nitric oxide synthase (eNOS) uncoupling, is an initial s

170 Instead, expression of endothelial nitric oxide synthase (eNOS), which generates the potent

171 OPE also had higher NO, inducible nitric oxide synthase (iNOS) and cyclooxygenase (COX-2)

172 complex 1 (mTORC1), HIF1alpha and inducible nitric oxide synthase (iNOS) coordinates DC metabolism a

173 of the NF-kappaB subunit p65; and inducible nitric oxide synthase (iNOS) expression.

174 by genetic deletion of inducible isoform of nitric oxide synthase (iNOS) from VCP TG mouse and by ph

175 Inducible nitric oxide synthase (iNOS) generates nitric oxide (NO)

176 ported that Sal-1 targets cellular inducible nitric oxide synthase (iNOS) in a miRNA manner, leading

177 lyze the role of NADPH oxidase and inducible nitric oxide synthase (iNOS) in a murine model of A. act

178 ce nitric oxide (NO) or upregulate inducible nitric oxide synthase (iNOS) mRNA following Toll-like re

179 aB p65-responsive genes, including inducible nitric oxide synthase (iNOS), interleukin-6 (IL-6), and

180 1 (CB1R) and inhibitory effect on inducible nitric oxide synthase (iNOS).

181 proximately 60% of which expressed inducible nitric oxide synthase (iNOS).

182 teins cyclooxygenase-2 (COX-2) and inducible nitric oxide synthase (iNOS).

183 s and markedly decreased levels of inducible nitric oxide synthase (iNOS).

184 SC were treated with inhibitors of inducible nitric oxide synthase (L-NAME and NCX-4016).

185 6) and decreased the expression of inducible nitric oxide synthase (M1 marker).

186 el of human enteric neuropathy, the neuronal nitric oxide synthase (nNOS(-/-)) deficient mouse model,

187 he nitric oxide synthesizing enzyme neuronal nitric oxide synthase (nNOS) in nerve fibers of the muri

188 Inhibition of neuronal nitric oxide synthase (nNOS) is a promising therapeutic

189 Neuronal nitric oxide synthase (nNOS) is a target for development

190 oduction from the amino acid l-arginine, via nitric oxide synthase (NOS) enzymes, research in recent

191 ytotoxic effects, which are inhibited by the nitric oxide synthase (NOS) inhibitor L-NIO, and genetic

192 e dystrophin-sarcoglycan complex delocalizes nitric oxide synthase (NOS) to alter its signaling, and

193 Oxidative stress and nitric oxide synthase (NOS) uncoupling are thought to co

194 This imbalance leads to nitric oxide synthase (NOS) uncoupling with reduced nitr

195 hat protein kinase Cbeta (PKCbeta) and brain nitric oxide synthase (NOS1), both identified by proteom

196 the metalloproteinase ADAMTS1 and inducible nitric oxide synthase (NOS2) as therapeutic targets in i

197 racellular adhesion molecule 1 (ICAM-1), and nitric oxide synthase (NOS2), developed leukostasis and

198 crosis factor-alpha (P = 0.04) and inducible nitric oxide synthase (P = 0.02) in skin biopsy specimen

199 uM(vgat/vglut2)) and another also expressing nitric oxide synthase (SuM(Nos1/Vglut2)).

200 h is located at 11p14, potentially affecting nitric oxide synthase 3 (NOS3) expression, as shown by m

201 2 genes that mediate nitric oxide signaling (Nitric Oxide Synthase 3 [NOS3] and Guanylate Cyclase 1,

202 d indomethacin and inhibition of endothelial nitric oxide synthase abolished the effects of therapeut

203 nd isoform-specific endothelial and neuronal nitric oxide synthase activation and nitric oxide produc

204 nucleotide phosphate-oxidase 2 and inducible nitric oxide synthase and enhanced lung concentrations o

205 SIRT1-regulated Akt, endothelial nitric oxide synthase and GLUT4 levels were also induced

206 ased glutathione levels, increased inducible nitric oxide synthase and heme-oxygenase 1 expression, a

207 ased glutathione levels, increased inducible nitric oxide synthase and heme-oxygenase 1 expression, a

208 is attributable to preservation of neuronal nitric oxide synthase and sarcoplasmic reticulum Ca(2+)

209 by the sigma1-receptor antagonist or various nitric oxide synthase blockers.

210 mitogen-activated protein kinase (MAPK), and nitric oxide synthase deactivation.

211 n, shedding of procoagulant MPs, endothelial nitric oxide synthase downregulation, and reduced nitric

212 rotyrosine formation and increased inducible nitric oxide synthase expression.

213 emodelling by over-expression of endothelial nitric oxide synthase in the fat pad of the adult rat me

214 ing loss of ROV and was eliminated following nitric oxide synthase inhibition.

215 Importantly, infusion of the nitric oxide synthase inhibitor l-N(G)-monomethyl-l-argi

216 tric tissues and these were inhibited by the nitric oxide synthase inhibitor L-NNA.

217 dichlorofluorescein diacetate dye, inducible nitric oxide synthase levels determined by Western blot,

218 activation by fluvoxamine triggered the Akt-nitric oxide synthase signaling pathway, resulting in ti

219 capillaries via the NMDA receptors-neuronal nitric oxide synthase signaling pathway.

220 volves the activation of the Akt-endothelial nitric oxide synthase survival pathway, and the inhibiti

221 tion that could be restored by inhibition of nitric oxide synthase via the inhibitor N-nitro-l-methyl

222 e dismutase were increased, whereas those of nitric oxide synthase were decreased, in 7- to 10-week-o

223 c contractions persisted until inhibition of nitric oxide synthase with N(omega) -nitro-l-arginine me

224 triggers calmodulin-dependent activation of nitric oxide synthase, nitric oxide (NO) production, NO-

225 dimerization and phosphorylation of neuronal nitric oxide synthase, sarcoplasmic reticulum Ca(2+) rel

226 actility, tetrahydrobiopterin, the dimers of nitric oxide synthase, sarcoplasmic reticulum Ca(2+) rel

227 ry cytokine tumor necrosis factor, inducible nitric oxide synthase, the M1 activator interferon gamma

228 effects alter the expression of endothelial nitric oxide synthase, the stability of atherosclerotic

229 ssion of proinflammatory proteins (inducible nitric oxide synthase, tumor necrosis factor-alpha, and

230 adhesion molecule-1 (VCAM-1) and endothelial nitric oxide synthase, whereas PDEs had significantly hi

231 )) and triple-negative breast cancer (TNBC), nitric oxide synthase-2 (NOS2) and cyclooxygenase-2 (COX

232 ediators including cytokines, chemokines and nitric oxide synthase-2.

233 st, affects development of DN in endothelial nitric oxide synthase-knockout db/db mice.

234 rons ( approximately 1%) expressing neuronal nitric oxide synthase.

235 obese type 2 diabetic mice by an endothelial nitric oxide synthase/Akt/vascular endothelial growth fa

236 in capillary/arteriole density, endothelial nitric oxide synthase/Akt/vascular endothelial growth fa

237 e availability of BH4 leads to uncoupling of nitric oxide synthases and production of highly oxidativ

238 t radiation disrupted BH4, which resulted in nitric oxide synthases uncoupling and augmented radiatio

239 terin (BH4) is an essential cofactor for all nitric oxide synthases.

240 itric oxide synthase (eNOS) lead to impaired nitric oxide synthesis.

241 P (SP), neuropeptide tyrosine (NPY), and the nitric oxide synthesizing enzyme neuronal nitric oxide s

242 nd loss of function of amacrine cells (brain nitric oxide synthetase/tyrosine hydroxylase expression)

243 for IgE levels (268 IU), fraction of exhaled nitric oxide values (14.5 ppb), and blood eosinophil cou

244 ormed spirometry and had fraction of exhaled nitric oxide values measured twice during the school yea

245 siveness to mannitol and fraction of exhaled nitric oxide values were measured.

246 tinuous inotropic support for >/=14 days, or nitric oxide ventilation for >/=48 hours.

247 However, macrophage ability to produce nitric oxide was defective.

248 On day 4 plasma and urine nitric oxide was increased by 244 +/- 89% and 450 +/- 36

249 ever, after challenging M. tuberculosis with nitric oxide we found that the rapid transcriptional res

250 To carry and deliver nitric oxide with a controlled redox state and rate is c

251 fied human mitoNEET and Miner1 fails to bind nitric oxide, a single mutation of Asp-96 to Val in mito

252 ural sleep included tidal breathing, exhaled nitric oxide, and multiple breath washout measures.

253 f blood eosinophil count, fractional exhaled nitric oxide, Asthma Control Questionnaire, medication u

254 ee known small molecule bioregulators (SMBs) nitric oxide, carbon monoxide, and hydrogen sulfide were

255 ounds, oxygen, carbon dioxide, ethylene, and nitric oxide, change during the submergence of plant org

256 balance between various mediators, including nitric oxide, endothelin, and prostanoids, among others.

257 P2 receptors on endothelial cells to produce nitric oxide, endothelium-derived hyperpolarizing factor

258 mission from rotational-vibrational bands of nitric oxide, hydroxyl and molecular oxygen as signature

259 Inhaled nitric oxide, initiated at 20 ppm on postnatal days 5 to

260 , the transient one-electron reduced form of nitric oxide, is a significant challenge owing to its hi

261 percent predicted FEV1, fraction of exhaled nitric oxide, or asthma symptoms.

262 a combination of adenosine triphosphate and nitric oxide, respectively.

263 ted with markers of systemic vasodilatation (nitric oxide, rho = -0.66, P = 0.06; diastolic blood pre

264 y inflammation, including fractional exhaled nitric oxide, serum IgE, periostin, and blood and sputum

265 than or equal to 10 mum in diameter (PM10), nitric oxide, sulfur dioxide, and nitrogen dioxide in di

266 cid arginine is a physiological precursor to nitric oxide, which is a key mediator of embryonic survi

267 type 5 inhibitor, potentiates the actions of nitric oxide, which leads to vasodilatation of the uteri

268 l-venous delivery gradients of intravascular nitric oxide, with deoxyhemoglobin-mediated reduction id

269 effects of organic nitrate therapy, via both nitric oxide-dependent and -independent mechanisms.

270 ect on T-cell responses that was mediated by nitric oxide-dependent depletion of l-arginine.

271 itrosylation is therefore revealed here as a nitric oxide-dependent host strategy involved in plant i

272 and distribution of GABAergic parvalbumin or nitric oxide-expressing and cholinergic interneurons wer

273 c oxide synthase downregulation, and reduced nitric oxide-mediated inhibition of platelet aggregation

274 quired for VSMC recruitment during increased nitric oxide-mediated vasodilatation and angiopoietin si

275 lium-dependent vascular relaxation through a nitric oxide-related mechanism.

276 itive balance between heme-free, ferric, and nitric oxide-sensitive ferrous sGC in cells and tissues,

277 required to allow beneficial effects of both nitric oxide-sensitive guanylyl cyclase activation and i

278 osolic calcium, reactive oxygen species, and nitric oxide.

279 cellular cascades involved Ca(2+) stores and nitric oxide.

280 cture may switch mitoNEET and Miner1 to bind nitric oxide.

281 associated with an increase in production of nitric oxide.

282 hondrial O2(. -), and diminished endothelial nitric oxide.

283 , oxidative stress, and limited bioavailable nitric oxide.

284 onmental signals such as oxygen, nitrate and nitric oxide.

285 nd subsequent chemiluminescence detection of nitric oxide.

286 ants (50.2% male [n = 115]) received inhaled nitric oxide.

287 ulosis infections in response to hypoxia and nitric oxide.

288 tile or rapidly diffusing substances such as nitric oxide.

289 against the antiviral effector mechanism of nitric oxide.

290 ers in Miner2, with each cluster binding one nitric oxide.

291 guanylyl cyclase (sGC), a key enzyme in the nitric oxide/cGMP signaling pathway.

292 Endothelial nitric-oxide synthase (eNOS) and its bioactive product,

293 he stability and activity of the endothelial nitric-oxide synthase (eNOS) and that Cavin-2 knockdown

294 s by mitochondria, NADPH oxidase, and type 2 nitric-oxide synthase (NOS-2) and resulted in S-nitrosyl

295 ry cytokines and the mRNA encoding inducible nitric-oxide synthase in both LRP1-expressing and -defic

296 of pro-inflammatory cytokines and inducible nitric-oxide synthase induction in BV2 cells in a concen

297 of capon (C-terminal PDZ ligand of neuronal nitric-oxide synthase protein), apoptosis-associated spe

298 Intriguingly, eNOS activity is regulated by nitric-oxide synthase trafficking inducer (NOSTRIN), whi

299 glial fibrillary acidic protein and neuronal nitric-oxide synthase, and Abeta and p-Tau(Ser-202) also

300 In contrast, inhibition of nitric-oxide synthase, p38 MAPK, and G9a abrogated H3K9m