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1 d methods for detection of superoxide (e.g., nitroblue tetrazolium).
2 ADPH-dependent reduction of cytochrome c and nitroblue tetrazolium.
3 ll surface antigens, as well as reduction of nitroblue tetrazolium.
4 so inhibited the NADH-dependent reduction of nitroblue tetrazolium.
5 nectary tissues, nectaries were stained with nitroblue tetrazolium.
6 cceptors for NADH oxidoreductase (0.3 mmol/L nitroblue tetrazolium and 0.1 mmol/L ferricyanide) inhib
7 hrome c (Cyt c), assays for the reduction of nitroblue tetrazolium, and assays for the chemiluminesce
8 preparation: the Cyt c assay to cytosol, the nitroblue tetrazolium assay to plasma membrane, and the
10 on of NADH on nitroprusside was inhibited by nitroblue tetrazolium, ferricyanide, and diphenyliodoniu
11 NO release was inhibited by the presence of nitroblue tetrazolium, ferricyanide, and diphenyliodoniu
12 , HO2( *)) were detected by the reduction of nitroblue tetrazolium into formazan, and hydroxyl radica
13 ease (CGD), which is diagnosed by use of the nitroblue tetrazolium (NBT) and Fc-Oxyburst assays that
14 were detected spectrophotometrically using a nitroblue tetrazolium (NBT) assay in cells treated with
15 timulated isoniazid oxidation as measured by nitroblue tetrazolium (NBT) reduction and O2 consumption
17 tion and immunocompetence measured using the Nitroblue Tetrazolium (NBT) test were significantly diff
18 , superoxide production, as monitored by the nitroblue tetrazolium (NBT) test, was detected in up to
19 ion as measured by (1) the ability to reduce nitroblue tetrazolium (NBT), (2) the expression of Mac-I
22 4 subclones MR-2 and R4 cells as measured by nitroblue tetrazolium reduction and tRA-inducible genes
23 OD and tcvSOD recombinant proteins inhibited nitroblue tetrazolium reduction of superoxide anion gene
24 entiation, as measured by growth inhibition, nitroblue tetrazolium reduction, nuclear body relocaliza
30 eutrophil function in the proband, including nitroblue tetrazolium tests, myeloperoxidase assays, neu
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