ライフサイエンスコーパス: nitrogen dioxide

1 and nitrogen oxides (e.g., peroxynitrite and nitrogen dioxide).

2 hey obtained similar results for exposure to nitrogen dioxide.

3 adical, ozone, the nitrate radical (NO3) and nitrogen dioxide.

4 s nitrous and nitric acid, nitric oxide, and nitrogen dioxide.

5 ity in the lavage of mice acutely exposed to nitrogen dioxide.

6 ent mechanisms involving the intermediacy of nitrogen dioxide.

7 idation of nitric oxide by dioxygen to yield nitrogen dioxide.

8 ozone, carbon monoxide, sulfur dioxide, and nitrogen dioxide.

9 e (NO(2-)) to a radical species, most likely nitrogen dioxide.

10 sures to fine particulate matter, ozone, and nitrogen dioxide.

11 oxide intermediates such as peroxynitrite or nitrogen dioxide.

12 reacts with activated peroxynitrous acid or nitrogen dioxide.

13 ozone, and 1.079 (95% CI: 1.065, 1.093) for nitrogen dioxide.

14 ), the ORs were 0.84 (95% CI, 0.76-0.92) for nitrogen dioxide; 0.85 (95% CI, 0.74-0.97) for ozone, 0.

15 6% per 10 parts per billion; 1.35-3.38), and nitrogen dioxide (1.70% per 10 parts per billion; 1.25-2

16 absolute decrease in prevalence of 1.8% for nitrogen dioxide, 1.7% for ozone, 2.2% for PM10, and 2.3

17 carbon monoxide: 1.048; 95% CI, 1.026-1.070; nitrogen dioxide: 1.011; 95% CI, 1.006-1.016; sulfur dio

18 n dioxide (13%), carbon monoxide (0.68%) and nitrogen dioxide (1000 ppm) in air.

19 artile-range increase in average exposure to nitrogen dioxide (13.6 parts per billion) during pregnan

20 c oxide concentration of 40 ppm, the highest nitrogen dioxide (4.47 ppm) concentration was found at t

21 sion processes (nitric oxide, nitrous oxide, nitrogen dioxide, ammonia, hydrazine, hydroxylamine, nit

22 Here we discuss the removal of nitrogen dioxide, an important toxic industrial chemical

23 t ventricular outflow tract obstructions and nitrogen dioxide and between hypoplastic left heart synd

24 secondary free radical intermediates such as nitrogen dioxide and carbonate radicals.

25 n detect aggressive oxidizing vapors such as nitrogen dioxide and chlorine at 250 and 500 ppb, respec

26 sociations were observed between exposure to nitrogen dioxide and coarctation of the aorta and pulmon

27 the association between maternal exposure to nitrogen dioxide and fine particulate matter (aerodynami

28 omine oxide and iodine oxide, as well as for nitrogen dioxide and formaldehyde.

29 In contrast, combination of nitrogen dioxide and guanine neutral radicals generated

30 rement of the blackness of PM2.5 filters and nitrogen dioxide and nitrogen oxide levels.

31 ts of meteorological data and air pollution (nitrogen dioxide and ozone).

32 Regional exposure measures of nitrogen dioxide and particulate matter less than 2.5 an

33 olling for the traffic-related co-pollutants nitrogen dioxide and particulate matter with an aerodyna

34 trous acid (HONO) yield for reaction between nitrogen dioxide and the hydroperoxyl-water complex and

35 the reaction between electronically excited nitrogen dioxide and water vapor is an important atmosph

36 less than 10 microm in aerodynamic diameter, nitrogen dioxide, and carbon monoxide, while evidence of

37 diameter less than 2.5 mum, sulfur dioxide, nitrogen dioxide, and carbon monoxide.

38 ergens (dust mite, cockroach, cat, and dog), nitrogen dioxide, and mold with symptoms of wheeze and p

39 ate matter <2.5 mum in aerodynamic diameter, nitrogen dioxide, and nitric oxide) and woodsmoke were e

40 or equal to 2.5 mum in aerodynamic diameter, nitrogen dioxide, and nitric oxide, were estimated at ea

41 ach 25-ppb increase in average nitric oxide, nitrogen dioxide, and nitrogen oxide levels, respectivel

42 ased estimates of exposures to nitric oxide, nitrogen dioxide, and nitrogen oxides were assigned base

43 modeled estimates of levels of nitric oxide, nitrogen dioxide, and nitrogen oxides were used to asses

44 Measured PM2.5, nitrogen dioxide, and ozone concentrations were spatiall

45 ; ambient concentrations of carbon monoxide, nitrogen dioxide, and ozone were obtained from state mon

46 e of ambient exposures to ozone, acid vapor, nitrogen dioxide, and particulate matter.

47 ciations were observed with carbon monoxide, nitrogen dioxide, and PM(10).

48 Ambient fine particulate matter, nitrogen dioxide, and preterm birth in New York City.

49 ess (PC20) with ozone, carbon monoxide (CO), nitrogen dioxide, and sulfur dioxide concentrations in 1

50 mic diameter (PM10), ozone, carbon monoxide, nitrogen dioxide, and sulfur dioxide) and cardiac autono

51 </=10 mum and </=2.5 mum in diameter, ozone, nitrogen dioxide, and sulfur dioxide), derived from part

52 ter of <10 microm (PM(10)), carbon monoxide, nitrogen dioxide, and sulfur dioxide-but not ozone-were

53 late matter (PM), gaseous pollutants (ozone, nitrogen dioxide, and sulphur dioxide), and mixed traffi

54 -day moving average concentrations of ozone, nitrogen dioxide, and the organic carbon fraction of par

55 ed with autism during gestation (exposure to nitrogen dioxide: AOR, 1.81 [95% CI, 1.37-3.09]; exposur

56 d during the first year of life (exposure to nitrogen dioxide: AOR, 2.06 [95% CI, 1.37-3.09]; exposur

57 response of a copper phthalocyanine film to nitrogen dioxide are used as an input example for a desi

58 ect of this reagent by in situ generation of nitrogen dioxide as a radical on aromatic compounds to g

59 elieve that the observed carbon monoxide and nitrogen dioxide associations can probably be attributed

60 rals, massicot, and litharge, are exposed to nitrogen dioxide at different relative humidity.

61 re to ambient volatile organic compounds and nitrogen dioxide at relatively low concentrations is ass

62 However, adjustment for nitrogen dioxide attenuated the full-pregnancy-particula

63 gents including superoxide anion radical and nitrogen dioxide can react with GXXXXGK(S/T)C motif-cont

64 2.5 microm in aerodynamic diameter (PM2.5)), nitrogen dioxide, carbon monoxide, and ozone increases r

65 ants (particles <2.5 mum in diameter, ozone, nitrogen dioxide, carbon monoxide, and sulfur dioxide).

66 -range increase in the time-weighted average nitrogen dioxide concentration (4.8 ppb) yielded an odds

67 The mean inspired nitrogen dioxide concentration in inhaled NO patients wa

68 increased with each 10-ppb increase in mean nitrogen dioxide concentration in the first trimester (o

69 Similarly, measured nitrogen dioxide concentration was associated with persi

70 large and small fluctuations in the ambient nitrogen dioxide concentration.

71 or for the monitoring of ambient atmospheric nitrogen dioxide concentrations (0-200 ppb).

72 a citywide low-emission zone failed to bring nitrogen dioxide concentrations under control.

73 Increased 4-month average nitrogen dioxide concentrations were associated with red

74 ng of NO2 in macrophage cells treated with a nitrogen dioxide donor.

75 ciated with interquartile-range increases in nitrogen dioxide during cumulative lag 3 (average of the

76 suicide risk was associated with exposure to nitrogen dioxide during the spring/fall transition perio

77 ns have experienced significant increases in nitrogen dioxide emissions from new trucks equipped with

78 From these incomplete measures, long-term nitrogen dioxide exposure and its effect on health must

79 IQR increase for LUR-based nitric oxide and nitrogen dioxide exposure estimates.

80 whereas among 2,136 boys, sulfur dioxide and nitrogen dioxide exposure in utero, during infancy, and

81 e, and smoking, an interquartile increase in nitrogen dioxide exposure increased the risk of membersh

82 The effect of nitrogen dioxide exposure on various measures of lung fu

83 usted odds ratio for the highest quartile of nitrogen dioxide exposure was 1.7 (95% confidence interv

84 ccurate measure of household-level long-term nitrogen dioxide exposure was available.

85 nuously over many years, but household-level nitrogen dioxide exposure was measured only during two 2

86 age at ROM associated with PM2.5 absorbance, nitrogen dioxide exposure, and nitrogen oxide exposure d

87 These ozone and nitrogen dioxide findings were fairly stable after adjus

88 cally more robust predictors of eNO than was nitrogen dioxide, for which associations were highly sen

89 l of these home exposures (indoor allergens, nitrogen dioxide, fungi) and to prospectively measure th

90 of nitric oxide with oxygen, an increase of nitrogen dioxide has to be taken into account.

91 ies such as nitric oxide, peroxynitrite, and nitrogen dioxide have been implicated in the pathophysio

92 er (PM10), nitric oxide, sulfur dioxide, and nitrogen dioxide in different growth phases with clinica

93 examined, the quantity of carbon dioxide and nitrogen dioxide in particular were close to or even abo

94 ological particles are nitrated by ozone and nitrogen dioxide in polluted air.

95 Adjusted estimates for nitrogen dioxide indicated that for each 10-ppb increase

96 Nitrogen dioxide is a common air pollutant with growing

97 Nitrogen dioxide is a highly toxic reactive nitrogen spe

98 he conclusion that generation of the mutagen nitrogen dioxide is peculiar to cell culture systems and

99 rs examined whether air pollution at school (nitrogen dioxide) is associated with poorer child cognit

100 V s)), chlorine (K0 = 2.24 cm(2)/(V s)), and nitrogen dioxide (K0 = 2.25 cm(2)/(V s)).

101 Photoionization of nitrogen dioxide led to the formation of the nitrate ion

102 )) and 10 mum (PM(10)) or less and ozone and nitrogen dioxide levels 7 days before Feno measurement w

103 udy, Huls et al. show an association between nitrogen dioxide levels in outdoor air and number of len

104 above 1,170 m, a 10-ppb increment in modeled nitrogen dioxide levels was associated with current asth

105 llution (calibrated to a 10-ppb increment in nitrogen dioxide levels) were associated with a 2.4% dec

106 llution (calibrated to a 10-ppb increment in nitrogen dioxide levels) were associated with pulmonary

107 onceptual recall memory after adjustment for nitrogen dioxide levels.

108 formation recall memory after adjustment for nitrogen dioxide levels.

109 cid, suggesting that the reactive agent is a nitrogen dioxide-like species that results from the one-

110 ted the associations of maternal exposure to nitrogen dioxide, nitrogen oxides, and particulate matte

111 pectral features suggesting the formation of nitrogen dioxide NO(2).

112 ation via oxidation of nitrite (NO(2)(-)) to nitrogen dioxide (NO(2)(*)).

113 Street-level concentrations of nitrogen dioxide (NO(2)) and particulate matter (PM) exc

114 c-related air pollution (TRAP), specifically nitrogen dioxide (NO(2)) and particulate matter (PM), an

115 ffects of indoor particulate matter (PM) and nitrogen dioxide (NO(2)) concentrations on COPD morbidit

116 Formation of nitrogen dioxide (NO(2)) during the reaction between NO

117 ty for the dynamic production of mixtures of nitrogen dioxide (NO(2)) in nitrogen (N(2)) based on con

118 We developed LUR models for nitrogen dioxide (NO(2)) using measurements conducted at

119 diacy of protein-free radicals, ferryl heme, nitrogen dioxide (NO(2)), and hydrogen peroxide (H(2)O(2

120 ynamic diameter < 2.5 microm (PM(2).(5)) and nitrogen dioxide (NO(2)), and modelled estimates of ozon

121 utants considered here include nitric oxide, nitrogen dioxide (NO(2)), carbon monoxide, formaldehyde,

122 the simultaneous measurement of atmospheric nitrogen dioxide (NO(2)), ozone (O(3)), and relative hum

123 10 (PM(10)) and less than 2.5 mum (PM(2.5)), nitrogen dioxide (NO(2)), ozone, and sulfur dioxide (SO(

124 nual exposure to five air pollutants: ozone, nitrogen dioxide (NO(2)), sulfur dioxide, particulate ma

125 sts that the one-electron oxidation product, nitrogen dioxide ((*)NO(2)), is the primary species form

126 sine and nitrite to form tyrosyl radical and nitrogen dioxide ((.)NO(2)), respectively, reactive inte

127 osylated by direct reaction of Cys(118) with nitrogen dioxide (*NO(2)), a reaction product of NO with

128 k carbon, total nitrogen oxides [NO(X)], and nitrogen dioxide [NO(2)]) were measured at 10 school sit

129 have previously shown that both NO/O(2) (via nitrogen dioxide, (*)NO(2)) and superoxide radical anion

130 llutants such as particulate matter (PM) and nitrogen dioxide (NO2 ) are independently associated wit

131 A new type of nitrogen dioxide (NO2 ) gas sensor based on copper phtha

132 mic diameter <2.5microm (PM2.5) (1999-2004), nitrogen dioxide (NO2) (2006), and ozone (O3) (2002-2004

133 Exposure to traffic related nitrogen dioxide (NO2) air pollution is associated with

134 ation systems contributed to satellite-based nitrogen dioxide (NO2) and fine particulate matter (</=

135 sia to evaluate associations with changes in nitrogen dioxide (NO2) and fine particulate matter (PM2.

136 s via three different channels, forming: (1) nitrogen dioxide (NO2) and hydroxyl radical ((*)OH), (2)

137 reactive nitrogen species peroxynitrite and nitrogen dioxide (NO2) and in vivo by drugs that damage

138 lso calculated photolysis rate constants for nitrogen dioxide (NO2) and nitrate radicals (NO3) in the

139 (LUR) model estimates of nitric oxide (NO), nitrogen dioxide (NO2) and nitrogen oxides (NOx).

140 s was PM2.5 absorbance and concentrations of nitrogen dioxide (NO2) and nitrogen oxides.

141 ta-analyses of studies examining exposure to nitrogen dioxide (NO2) and NOx and its association with

142 ions of average pollutant concentrations for nitrogen dioxide (NO2) and NOX in GMCs of 6.4 and 21.7 p

143 fleet was also observed in on-road ratios of nitrogen dioxide (NO2) and NOX.

144 O3) with nitrogen monoxide (NO) resulting in nitrogen dioxide (NO2) and oxygen (O2).

145 in the development of exposure surfaces for nitrogen dioxide (NO2) and ozone (O3).

146 Our aim was to evaluate LUR models for nitrogen dioxide (NO2) and particulate matter (PM) compo

147 ether exposure to elevated concentrations of nitrogen dioxide (NO2) and particulate matter with aerod

148 A link between exposure to the air pollutant nitrogen dioxide (NO2) and respiratory disease has been

149 trument (OMI) were used to examine trends in nitrogen dioxide (NO2) and sulfur dioxide (SO2) over a l

150 Peroxynitrite and nitrogen dioxide (NO2) are reactive nitrogen species tha

151 fy key controls on concentrations of ambient nitrogen dioxide (NO2) at a local-scale within a central

152 anuary and July) localized concentrations of nitrogen dioxide (NO2) by 28% (-2.33 ppbV) in highway gr

153 Ambient and home exposure to nitrogen dioxide (NO2) causes asthma symptoms and decrea

154 We analyzed satellite observations of nitrogen dioxide (NO2) columns by the Ozone Monitoring I

155 PM2.5, PMcoarse, and PM10, respectively) and nitrogen dioxide (NO2) concentrations at place of reside

156 d function and estimated residential outdoor nitrogen dioxide (NO2) concentrations from the Multi-eth

157 PM) concentrations, ultrafine particles, and nitrogen dioxide (NO2) concentrations were measured.

158 essment of airborne nicotine, endotoxin, and nitrogen dioxide (NO2) concentrations.

159 osures to ambient fine particles (PM2.5) and nitrogen dioxide (NO2) during the first, second, and cum

160 chniques to examine the long-term effects of nitrogen dioxide (NO2) exposure on children's lung funct

161 lso had more asthma symptoms associated with nitrogen dioxide (NO2) exposure than normal-weight parti

162 Mean annual residential nitrogen dioxide (NO2) exposure was estimated for curren

163 Including satellite observations of nitrogen dioxide (NO2) in land-use regression (LUR) mode

164 (PM10) and < 2.5 mum in diameter (PM2.5) and nitrogen dioxide (NO2) in relation to PD risk.

165 is highly reactive and rapidly converted to nitrogen dioxide (NO2) in the presence of ozone, making

166 Chronic exposure to indoor nitrogen dioxide (NO2) is a public health concern.

167 Nitrogen dioxide (NO2) is an environmental air pollutant

168 lobal exposure estimates we created a global nitrogen dioxide (NO2) land use regression model for 201

169 ial NRP exposure and regional ozone (O3) and nitrogen dioxide (NO2) levels in Los Angeles County.

170 many of which involve chemistry that reduces nitrogen dioxide (NO2) on some time scale.

171 erodynamic diameter </= 10 mum (PM10) and to nitrogen dioxide (NO2) on specific airway resistance (sR

172 lysis was used on 11-year time series of all nitrogen dioxide (NO2) pixels from the Ozone Monitoring

173 Nitrogen dioxide (NO2) released primarily from combustio

174 tellite-based measurements of urban form and nitrogen dioxide (NO2) to explore relationships between

175 r </= 2.5 mum (PM2.5), black carbon (BC), or nitrogen dioxide (NO2) variations that were independent

176 Levels of nitrogen dioxide (NO2) were estimated at subject residen

177 r (PM10; </=10 mum in aerodynamic diameter), nitrogen dioxide (NO2), and carbon monoxide (CO) and ris

178 amic diameter (PM2.5), sulfur dioxide (SO2), nitrogen dioxide (NO2), and carbon monoxide (CO) concent

179 ended particles (TSP), sulfur dioxide (SO2), nitrogen dioxide (NO2), and carbon monoxide (CO), and on

180 atter less than 2.5 mum in diameter (PM2.5), nitrogen dioxide (NO2), and ozone (O3) in the "moderate"

181 ly mean levels of particulate matter (PM10), nitrogen dioxide (NO2), and ozone (O3) were obtained fro

182 nts [PM10, ozone (O3), carbon monoxide (CO), nitrogen dioxide (NO2), and sulfur dioxide (SO2)] in sin

183 idence intervals, for long-term exposures to nitrogen dioxide (NO2), black smoke (BS), PM2.5 (particu

184 te associations between maternal exposure to nitrogen dioxide (NO2), dietary intake of methyl nutrien

185 Indoor air pollutants [sulfur dioxide (SO2), nitrogen dioxide (NO2), hydrogen sulfide (H2S), formalde

186 nitrite ion and its first oxidation product, nitrogen dioxide (NO2), in AN solution proceeds via the

187 s of a transportation-related air pollutant, nitrogen dioxide (NO2), in the United States.

188 However, environmental pollutants, including nitrogen dioxide (NO2), may promote adaptive immune resp

189 aerodynamic diameter (PM2.5), formaldehyde, nitrogen dioxide (NO2), nicotine, carbon dioxide (CO2),

190 ations of traffic related pollutants such as nitrogen dioxide (NO2), nitrogen oxides (NOx) and partic

191 ers to provide site-specific measurements of nitrogen dioxide (NO2), sulfur dioxide (SO2) and volatil

192 PM2.5), particulate matter </=10 mum (PM10), nitrogen dioxide (NO2), sulfur dioxide (SO2), and ozone

193 evel concentrations of carbon monoxide (CO), nitrogen dioxide (NO2), sulfur dioxide (SO2), ozone (O3)

194 ts in removing O3 and one of its precursors, nitrogen dioxide (NO2), the cost effectiveness of using

195 ctance of PM2.5), nitrogen oxides (NOx), and nitrogen dioxide (NO2).

196 they are highly selective in reactions with nitrogen dioxide (NO2).

197 tants: ozone, particulate matter, acids, and nitrogen dioxide (NO2).

198 Matter, 2.5 micrometers or less (PM2.5) and nitrogen dioxide (NO2)] predictors, we conducted a nonli

199 xture of benzene (0.6 ppm), toluene (1 ppm), nitrogen dioxide (NO2; 2 ppm) and sulfur dioxide (SO2; 5

200 -based reaction of the GDP guanine base with nitrogen dioxide (*NO2).

201 l-NO2, probably involving the combination of nitrogen dioxide (.NO2) and tyrosyl radical.

202 ugh formation of nitryl chloride (NO2Cl) and nitrogen dioxide (.NO2) by reaction with the inflammator

203 d ring core), effecting O-O cleavage, giving nitrogen dioxide (.NO2) plus a ferryl compound [(P(Im))F

204 on oxidation of thiols, possibly mediated by nitrogen dioxide (.NO2), and the subsequent reaction of

205 ated air pollutant exposures (represented by nitrogen dioxide; NO2) from a dispersion model, using re

206 , associations were strongest with ozone and nitrogen dioxide; observed associations of asthma/wheeze

207 NFA -308G>A modified the action of ozone and nitrogen dioxide on lung function, asthma risk, and symp

208 After adjustment for levels nitrogen dioxide or particulate matter with an aerodynam

209 overy cohort; for example, 5-day averages of nitrogen dioxide (OR = 0.68; 95 % CI: 0.53-0.88), and pa

210 l (CI): 0.99, 1.13) and a 10-ppb increase in nitrogen dioxide (OR = 1.08, 95% CI: 1.03, 1.13) during

211 rticle filters, but little change is seen in nitrogen dioxide over the period from 1995 to 2015.

212 oach for predicting fine particulate matter, nitrogen dioxide, oxides of nitrogen, and black carbon (

213 PM2.5), black carbon (BC), nitrogen oxides, nitrogen dioxide, ozone (O3), and carbon monoxide measur

214 n gaseous (carbon monoxide, sulphur dioxide, nitrogen dioxide, ozone) and particulate (diameter <2.5

215 y maximum concentrations of carbon monoxide, nitrogen dioxide, ozone, and sulfur dioxide and 24-hr me

216 al components, PM10, PM2.5, carbon monoxide, nitrogen dioxide, ozone, and sulfur dioxide for births i

217 Average concentrations of nitrogen dioxide, ozone, particulate matter with an aero

218 exposure to air pollutants, carbon monoxide, nitrogen dioxide, ozone, sulfur dioxide, and particulate

219 FVC were associated with declining levels of nitrogen dioxide (P<0.001 for FEV1 and FVC) and of parti

220 h of FEV(1) were associated with exposure to nitrogen dioxide (P=0.005), acid vapor (P=0.004), partic

221 Associations between nitrogen dioxide, particulate matter 2.5 (PM2.5) mass, P

222 Short-term effects of carbon monoxide, nitrogen dioxide, particulate matter less than 10 mum in

223 m ozone and 24-hour average carbon monoxide, nitrogen dioxide, particulate matter with an average aer

224 mmunities with high or low concentrations of nitrogen dioxide, particulate matter, and inorganic-acid

225 associations between short-term exposure to nitrogen dioxide, particulate matter, and sulfur dioxide

226 traffic-related pollutants (carbon monoxide, nitrogen dioxide, PM2.5 elemental carbon) were associate

227 Point estimates for associations between nitrogen dioxide, PM2.5 mass, and PM2.5 absorbance with

228 Exposure to traffic-related air pollution, nitrogen dioxide, PM2.5, and PM10 during pregnancy and d

229 Nitrogen dioxide production was not influenced by the fl

230 while generating hydroxyl radical (*OH) and nitrogen dioxide radical (*NO2), both more reactive than

231 ation of NO2- by an SOD-bound oxidant to the nitrogen dioxide radical (*NO2).

232 isomerization of fatty acids is possible by nitrogen dioxide radical, a product of NO and nitrite ox

233 ound oxidant results in the formation of the nitrogen dioxide radical, which was measured by monitori

234 he protein nitrating agents peroxynitrite or nitrogen dioxide radicals significantly decreased comple

235 ns in DNA associated with the combination of nitrogen dioxide radicals with 8-oxo-7,8-dihydroguanine

236 The combination of nitrogen dioxide radicals with the 8-oxoGua and G radica

237 on originates from water molecules, not from nitrogen dioxide radicals.

238 t are trapped by nitrate anions thus forming nitrogen dioxide radicals.

239 L showed reactivity toward both hydroxyl and nitrogen-dioxide radicals.

240 ndoor culturable Penicillium or total fungi, nitrogen dioxide, rodents (nonoccupational), feather/dow

241 mic diameter </=2.5 mum, or PM(2.5)), ozone, nitrogen dioxide, sulfur dioxide, and carbon monoxide in

242 Daily concentrations of ozone, nitrogen dioxide, sulfur dioxide, carbon monoxide, and p

243 sure (air pollution, ozone, carbon monoxide, nitrogen dioxide, sulfur dioxide, PM(10), and PM(2.5)) a

244 as a far greater impact on concentrations of nitrogen dioxide than of PM2.5.

245 ectroscopy (XPS) shows that upon exposure to nitrogen dioxide the surface of PbO particles reacts to

246 For nitrogen dioxide, the odds ratio (OR) for bronchitic sym

247 Nitrogen dioxide, the proximal nitrating species formed

248 nty of 1.4% (coverage factor k = 2) based on nitrogen dioxide titration values.

249 ive reactions, via the homolytic addition of nitrogen dioxide to a double bond or via the formation o

250 th diesel particle filters; raising the mean nitrogen dioxide to oxides of nitrogen ratios from less

251 lture studies results from the generation of nitrogen dioxide via the autoxidation of nitric oxide, a

252 The formation of nitrogen dioxide was <1 ppm at the level of the endotrac

253 ring a nitric oxide concentration of 10 ppm, nitrogen dioxide was always < 0.6 ppm.

254 er </= 2.5 mum (PM2.5), carbon monoxide, and nitrogen dioxide was associated with a 1-5% decrease in

255 Nitrogen dioxide was associated with HOMA-IR, glucose, i

256 Exposure to benzene, hydrocarbons, and nitrogen dioxide was estimated using land-use regression

257 Rather, community-level nitrogen dioxide was measured continuously over many yea

258 onoxide and a 20-parts-per-billion change in nitrogen dioxide were associated with symptom odds ratio

259 /=2.5 microm (PM2.5), black carbon (BC), and nitrogen dioxide were interpolated for each mother's res

260 Lags in carbon monoxide and nitrogen dioxide were positively associated with both me

261 The associations with carbon monoxide and nitrogen dioxide were the most robust in two-pollutant m

262 culate matter, 48-hr black carbon, and 96-hr nitrogen dioxide) were collected at each school for 16 w

263 ed sulfates, sulfur dioxide, ozone (O3), and nitrogen dioxide-were related to 1977-1992 mortality in

264 Nitrogen dioxide, which could form from nitrite under th

265 ed the association of particulate matter and nitrogen dioxide with asthma.

266 h aerodynamic diameter less than 2.5 mum and nitrogen dioxide with birth weight, restricting the popu

267 Heterogeneous chemistry of nitrogen dioxide with lead-containing particles is inves

268 that the reaction of electronically excited nitrogen dioxide with water can be an important source o