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1 onium) or a drop in arterial blood pressure (nitroglycerine).
2 phenylephrine, phentolamine, labetalol, and nitroglycerine.
3 ial pacing induced vasoconstriction, whereas nitroglycerine administration resulted in significant va
4 e injected into the portal circulation after nitroglycerine administration, they penetrated more dist
5 explosives including PETN, tetryl, RDX, and nitroglycerine along with various compositions containin
7 r (nifedipine), and splanchnic vasodilators (nitroglycerine, calcitonin gene-related peptide [CGRP],
16 hemodynamic instability, shock, intravenous nitroglycerine, left-ventricular hypertrophy, sustained
17 n blood flow before and after application of nitroglycerine (mean difference, 0.035; 95% CI, 0.008-0.
20 reased, whereas treatment with L-arginine or nitroglycerine patches decreased AR activity and sorbito
21 tment with L-arginine (a precursor of NO) or nitroglycerine patches prevented sorbitol accumulation.
22 ministration of phentolamine, labetalol, and nitroglycerine prevented the phenylephrine-induced chang
26 f TNT is sufficiently different from that of nitroglycerine so that unambiguous detection of TNT with
28 icacy parameters including angina frequency, nitroglycerine usage, exercise time, and Canadian Cardio
30 impairment in response to acetylcholine and nitroglycerine was prevented by AG treatment (P < 0.005)
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