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1 e structure of the acyl group of the acyloxy nitroso compound.
2 ites because of the increased formation of N-nitroso compounds.
3 he acidic medium of stomach to form N- and C-nitroso compounds.
4 nous production of mutagenic aldehydes and N-nitroso compounds.
5 ma by reducing the endogenous formation of N-nitroso compounds.
6 excess carboxylic acid gives various acyloxy nitroso compounds.
7 Processed meats contain N-nitroso compounds.
8 , we report that appropriately substituted C-nitroso compounds act solely as donors of neutral nitric
9 Calculated results reveal that the unbound nitroso compound acts as a better proton transferring ag
10 es to conjugated dienes in the presence of a nitroso compound (additive) was studied quantum mechanic
11 ar hetero-Diels-Alder cycloadduct of an acyl nitroso compound and a 9,10-dimethyl anthracene derivati
16 ction of nitroaromatics to the corresponding nitroso compounds and, subsequently, to hydroxylamine pr
18 combined forming the corresponding nitro and nitroso compounds, and the most reactive cis/syn isomer
20 ay increase cancer risk as HCAs, PAHs, and N-nitroso compounds are carcinogenic in animal models.
29 addition of nitrile oxide to form a bicyclic nitroso compound, followed by a retro-ene reaction of th
32 cient condensation of sulfinic acid and aryl nitroso compounds has been transformed into a chemoselec
33 exogenous alkylating agents, particularly N-nitroso compounds, has been associated with increased in
41 ncludes the observed nitroxide radical and C-nitroso compound intermediates has been forwarded to exp
43 The most common method to assay biological S-nitroso compounds is to chemically or photochemically re
45 g-down detection allows the measurement of S-nitroso compound levels from pico- to nanomole amounts.
46 as new species that react similarly to acyl nitroso compounds, making them useful synthetic intermed
50 iron, heterocyclic amines, and endogenous N-nitroso compounds (NOC) are proposed to explain this eff
51 a precursor in the endogenous formation of N-nitroso compounds (NOC), which are possible bladder carc
53 been linked to the formation of genotoxic N-nitroso compounds (NOCs) and lipid peroxidation products
55 e exposed to both endogenous and exogenous N-nitroso compounds (NOCs), and many NOCs can be metabolic
57 t appeared that the moderate basicity of the nitroso compound plays a crucial role to reduce the acti
58 e, via rearrangement of the O-nitrene to a C-nitroso compound (R'ON --> O=NR'), and subsequent tautom
63 exposure may also lead to the formation of N-nitroso compounds that can act as alkylating agents.
64 agents and highly acidic conditions, form N-nitroso compounds that have been found to be teratogenic
66 ree radicals, consists of using a nitrone or nitroso compound to "trap" an unstable free radical as a
67 The lower rate of conversion of the (C7-R)-nitroso compound to the corresponding oximes compared wi
69 leophilic addition of the protein thiol to a nitroso compound to yield an N-hydroxysulfenamide, which
71 yzed radical addition with in situ-generated nitroso compounds to prepare sterically hindered amines
72 EPR spectroscopic evidence showed that the nitroso compound trapped other transient intermediates t
75 adicals and leads initially to a diamagnetic nitroso compound, which is transformed into a persistent
77 cs of Diels-Alder reactions for a variety of nitroso compounds with dienes were explored with density
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