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1 treating hydrazinoazines with (15)N-labeled nitrous acid.
2 sor to ammonia, hydrazine, hydroxylamine and nitrous acid.
3 ophila HS by selective depolymerization with nitrous acid.
4 cts of chemical reactions between indole and nitrous acid.
5 at is formed directly by the condensation of nitrous acid.
6 dylinositol-specific phospholipase C but not nitrous acid.
7 oth cellular resistance to and mutability by nitrous acid.
8 These tetrasaccharides were analyzed using nitrous acid and enzymatic degradation combined with mat
10 ctive nitrogen species involved derives from nitrous acid and is most probably the nitronium cation.
11 lysis mainly sustains the observed levels of nitrous acid and nitrogen oxides at midday under typical
12 idence for rapid recycling of nitric acid to nitrous acid and nitrogen oxides in the clean marine bou
15 d much of the resistance of alpha- spores to nitrous acid and restored full resistance of alpha- spor
17 is would have been converted into nitric and nitrous acids and delivered to the early oceans as acid
18 to reduce the nitro groups and cyclized with nitrous acid, and the corresponding 4-position was funct
19 heat, formaldehyde, HCl, hydrogen peroxide, nitrous acid, and UV radiation than wild-type spores did
21 cells was released from plasma membranes by nitrous acid, as expected for GPI and its PI-PLC resista
22 gradients of DO, pH, free ammonia, and free nitrous acid, associated with aerated and nonaerated pha
24 dition to providing the first structure of a nitrous acid cross-link in DNA, these studies could be o
25 data available for lesion 2 originating from nitrous acid cross-linked DNA to synthetic 2 supports it
26 identical to that of lesion 1 obtained from nitrous acid cross-linked DNA, thus providing a proof of
27 , we described the solution structure of the nitrous acid cross-linked dodecamer duplex [d(GCATCCGGAT
28 ce to the understanding of the mechanisms of nitrous acid cross-linking and mutagenicity, as well as
29 sequence context effect on the structure of nitrous acid cross-links in [d(CG)]2 and the factors all
31 results of the disaccharide analysis of the nitrous acid-degraded [35S]HS suggested that 3-OST-2 tra
32 or quantification of both enzyme-derived and nitrous acid depolymerization products for structural an
33 e utility of PGC-MS for quantification of HS nitrous acid depolymerization products for structural an
34 a novel pretreatment strategy based on free nitrous acid (FNA or HNO2) to enhance methane production
35 5-45 mg of N/L) that was established by free nitrous acid (FNA)-based sludge treatment was not higher
37 o-N-2-phenylcyclopropyl-N-methylaniline with nitrous acid gave 4-chloro-N-methyl-N-nitrosoaniline (76
38 a pH lower than 7, implicating formation of nitrous acid (HNO2) and NO, that adds NO equivalents to
39 zing bacteria were not further detected, but nitrous acid (HNO2) was still removed through chemical d
40 in the plasma phase and the solution lead to nitrous acid (HNO2), nitric acid (HNO3), and hydrogen pe
43 hich the nitrosonium ion (NO+)and water form nitrous acid (HONO) and a hydrated proton cluster in the
45 labile nighttime radical reservoirs, such as nitrous acid (HONO) and nitryl chloride (ClNO(2)), contr
47 adiated nitrophenols can produce nitrite and nitrous acid (HONO) in bulk aqueous solutions and in vis
53 (HA) is thought to promote NO2 conversion to nitrous acid (HONO) on soil surfaces during the day.
54 orbed to indoor surfaces reacts with ambient nitrous acid (HONO) to form carcinogenic tobacco-specifi
58 HOx production rates from the photolysis of nitrous acid (HONO), hydrogen peroxide (H2O2), ozone (O3
61 yl radicals (OH) in the gas phase to produce nitrous acid, HONO, but essentially nothing is known abo
62 as been synthesized, and their reaction with nitrous acid in aqueous acetic acid at 0 degrees C was e
63 of the aniline into an aryl diazonium, using nitrous acid in aqueous conditions, was performed in sit
65 ion events leading to high concentrations of nitrous acid in the atmosphere contributed to an observe
66 ntaining mixtures, HgCl2-mediated release of nitrous acid in the presence of 2,3-diaminonaphthalene i
67 ants or the double mutant nor any changes in nitrous acid-induced mutagenesis for rdgB mutant strains
68 ontaneous mutagenesis spectra, as well as in nitrous acid-induced mutagenesis spectra, in wild-type c
73 recycling route reproduces levels of gaseous nitrous acid, NO, and NO2 within the model and measureme
74 treating the [3H]heparan sulfate chains with nitrous acid or bacterial heparin lyases, which cut the
75 may be formed either by the condensation of nitrous acid or by the autooxidation of nitric oxide, bo
78 ction of 3-amino-5-nitro-1,2,4-triazole with nitrous acid produces the corresponding diazonium salt.
79 le formed between 2,3-diaminonaphthalene and nitrous acid released from S-nitrosothiols by treatment
80 reaction between 2,3-diaminonaphthalene and nitrous acid released from the S-NO bond by HgCl2 approa
83 onase, NaOH/NaBH(4), heparinase I, or low pH nitrous acid showed that each HS-glycosaminoglycan regio
84 at internal N-sulfoglucosamine residues with nitrous acid then creates a set of fragments of defined
87 contrast, mutagenesis of nongrowing cells by nitrous acid was unaffected by an nirB mutation, suggest
88 ompletely abolished following treatment with nitrous acid, which digests heparan sulfate glycosaminog
89 f nitrate is converted in the acid medium to nitrous acid, which leads to the nitrosation of the indo
90 The reaction of 6 with in situ generated nitrous acid yielded the primary explosive bis(4-diazo-5
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