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1 ontinuous exposure to nitroglycerin or other nitrovasodilators.
2 vo antiplatelet effects of clinically useful nitrovasodilators.
3 he in vivo administration of clinically used nitrovasodilators.
4 0 micrograms) to test responses to exogenous nitrovasodilators.
6 cular relaxation by nitric oxide and related nitrovasodilators and inhibits vascular smooth muscle ce
9 bal NO-related metabolism of the cornerstone nitrovasodilator, glyceryl trinitrate (GTN; 0.1-100 mg/k
11 rated that the dilator response to exogenous nitrovasodilators is exaggerated in the setting of endot
12 ne whether enhanced sensitivity to exogenous nitrovasodilators is present in the coronary arteries of
13 ay disease and 2) inhaled SOD/mimetics or NO/nitrovasodilators may be therapies for the treatment of
14 er the NO donor diethylamine/NONOate nor the nitrovasodilator nitroglycerin had an appreciable effect
17 data may contribute to our understanding of nitrovasodilator resistance, a phenomenon resulting from
18 continuous exposure of SMC in culture to the nitrovasodilators S-nitroso-N-acetylpenicillamine (SNAP)
19 er, remodeled LCs were hypersensitive to the nitrovasodilator sodium nitroprusside (at day 7) and exh
22 e and OVX animals to sodium nitroprusside, a nitrovasodilator that generates NO, were reduced by > 50
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