ライフサイエンスコーパス: no role

1 No role for LKB1 in foam cell formation has previously been r

2 For the 22 no role cases, malignancies accounted for most fatalities.

3 There was also no role for lysosomal destabilization or neutrophil elastase

4 mary role in 68 (64%), a contributory role in 15 (14%), and no role in 22 (21%); 2 had insufficient data.

5 For contributory and no role cases, we assigned a primary cause of death.

6 k H3K79me2, a decrease of the repressive H4K20me3 mark, but no role for the generally strong transcription-related modifi

7 ever, variance in pulmonary gas exchange played essentially no role in determining peak VO2.

8 However, we found no role for F-actin in Cdc42 concentration, even in rdi1Delta

9 In contrast to hyphae, we found no role for neutrophil calprotectin in uptake or killing of i

10 In contrast, we found no role for regulatory T-cell-, programmed death receptor-, a

11 We found no role of MRP4 in ADP dense-granule storage, but MRP4 redist

12 Intervention teams had no role in data collection and the data collection teams were

13 s AtRALF1, is essential for root growth inhibition, and has no role in AtRALF1 alkalinization.

14 at its respective substrate phosphorylation by deltaPKC has no role in the response to cardiac ischemia and reperfusion.

15 ecific to tissue culture conditions, suggesting that it has no role in pathogenesis.

16 1p-activated genes in a carbon source-dependent manner, has no role in the Mxr1p-mediated activation of ACS1 expression.

17 ilencing experiments revealed that glioma-secreted MCSF has no role in autocrine functions and M2 polarization of macroph

18 ed treatments such as methadone, suggesting that CDDCs have no role in the treatment of opioid-use disorders.

19 ed treatments such as methadone, suggesting that CDDCs have no role in the treatment of opioid-use disorders.

20 atmospheric forcing and that ocean circulation changes have no role in causing the AMO.

21 ent action that enables self-cure (CCL5), and (vi) may have no role in responsiveness to chemotherapy.

22 that the relative energies of the states of the motor have no role in determining the directionality, stopping force, or

23 MRP2 appeared to have no role.

24 There is no role for the routine use of induction therapy before chemo

25 nd categorized as DILI having a primary, a contributory, or no role in the fatality.

26 ajority of this transformation, with MoaC playing little or no role in the pyranopterin formation.

27 contacts not present in the folded structure play little or no role in the self-assembly mechanism.

28 wever, this does not imply that self-control abilities play no role, for self-control abilities do influence whether indi

29 ocus harbouring all the genes dedicated to Tfp biology play no role in piliation or Tfp-mediated motility, (ii) two highl

30 discuss the possibility that the extrastriate cortices play no role in guiding exogenous attention so that they can be de

31 We conclude that UvrD and ppGpp play no role in TCR in E. coli.

32 a membrane, whereas the poorly conserved phi segments play no role in binding or protection.

33 considered the physiological form of Ca(2+) signaling, play no role in activating either NFAT protein.

34 Importantly, Y454 and Y474 play no role in A/E lesion formation on mucosal surfaces following

35 le pressure whereas nutrients or community diversity played no role in invasion success.

36 Furthermore, TLR2 signaling played no role in the clearance of bacteria from the spleen.

37 In previous exogenously driven SOC models, adaptation plays no role, and no optimization occurs.

38 e activity, controls proliferation and migration, but plays no role in chondrocyte intercalation.

39 ly, whereas donor T cell- or bone marrow-derived CD70 plays no role in GVHD, host-derived CD70 inhibits GVHD as CD70(-/-)

40 Some data and models suggest that DG plays no role in retrieval; others encourage the opposite conclusio

41 hat glycosylation of the extracellular domain of hCAR plays no role in CVB3 receptor function and that CAR-D2 is not spec

42 re study supported that extracellular hyperosmolarity plays no role in promoting autophagy in the NP.

43 We also found that moesin plays no role during endocytosis and recycling to the plasma membra

44 e hydrolases/cyclases of the MonB family, but SalBIII plays no role in the conventional cascade of ring opening/closing.

45 Whereas Setd1a plays no role in oogenesis, we report that Setd1b deficiency causes

46 The TL plays no role in pause recovery remaining unfolded during a pause.

47 ured by the excision repair-sequencing (XR-seq), UvrD plays no role in TCR genome-wide.

48 A new study suggests instead that pha-1 actually serves no role in development and instead is a component of a selfis

49 ormation, a previous study reported that Alr2 has little to no role in germination of C. difficile spores in rich medium.

50 t palliative care is synonymous with end-of-life care, with no role earlier in the course of ILD, has created a culture o