ライフサイエンスコーパス: nociceptive response

1 AF receptors decreased the late-phase of the nociceptive response.

2 are known to inhibit dural trigeminovascular nociceptive responses.

3 sure and salt and water balance and modulate nociceptive responses.

4 melastatin-8 (TRPM8) channel without evoking nociceptive responses.

5 n injection and showed sustained increase in nociceptive responses.

6 of Sp5C, without affecting cutaneous-evoked nociceptive responses.

7 ile-pain interactions employed heat to evoke nociceptive responses.

8 in higher level cognitive processes or basic nociceptive responses.

9 ed PACAP-induced CeA neuronal activation and nociceptive responses.

10 adiol alters injury- or inflammation-induced nociceptive responses after carrageenan administration i

11 n-injected mice showed significantly reduced nociceptive responses after injection of C terminus of m

12 tribution of heteromeric P2X2/3 receptors to nociceptive responses and mechanosensory transduction wi

13 In the absence of verbal report, these nociceptive responses are used as measures of pain sensa

14 GABAergic systems may contribute to aberrant nociceptive responses associated with inflammatory and n

15 of stress exhibit larger amplitude cortical nociceptive responses, but this is not reflected in thei

16 inflammation, indicating that modulation of nociceptive responses by amygdala ERK activation is spec

17 PRL enhances nociceptive responses by rapidly modulating channels in

18 hase 1 do not contribute to the magnitude of nociceptive responses during Phase 2.

19 lesions significantly decreased inflammatory nociceptive responses during the prolonged, tonic portio

20 large, dose- and time-related reductions in nociceptive responses following administration into the

21 algesia after L5 ligation or on inflammatory nociceptive responses following formalin injection in th

22 odulation of cutaneous-evoked C-fiber spinal nociceptive responses from the brainstem.

23 al and/or cutaneous facial trigeminovascular nociceptive responses, from the brainstem ventrolateral

24 may block spinal NMDA receptors to attenuate nociceptive responses, however, our data cannot exclude

25 of BN 52021 decreased the late-phase of the nociceptive response in a concentration-dependent manner

26 nanoparticles (NM0127) showed a strong anti-nociceptive response in multiple assays of evoked and on

27 lacement attenuated TMJ inflammation and the nociceptive responses in a dose-dependent manner in the

28 These data suggest that the enhanced nociceptive responses in AS model mice are due to loss o

29 EA-1021 showed equally potent attenuation of nociceptive responses in both animal models of pain.

30 e not only headache-associated pain but also nociceptive responses in extracranial tissues.

31 Morphine failed to significantly reduce nociceptive responses in hot plate or tail flick tests o

32 In the present study, we compared nociceptive responses in inflammatory and neuropathic mo

33 ifferences in the effects of KOR agonists on nociceptive responses in males and females.

34 -induced inflammation and pain, we evaluated nociceptive responses in mice that carry a null mutation

35 of chronic pain and nociceptive, but not non-nociceptive responses in normal animals.

36 consumption and provoked apathy and reduced nociceptive responses in rats.

37 ctions of improgan were evaluated on thermal nociceptive responses in rats.

38 ve NMDA receptor/glycine site antagonist, on nociceptive responses in the tail flick and formalin tes

39 levels and consequently alters inflammatory nociceptive responses in vivo.

40 isense oligodeoxynucleotide (ODN) on bladder nociceptive responses induced by intravesical acetic aci

41 The nociceptive response is limited by enkaphalin-expressing

42 ously found that TIP39 affects several acute nociceptive responses, leading us to now investigate its

43 , injection of FcgammaRIII siRNA reduced the nociceptive response of rats with an arthritic TMJ and r

44 Neither drug altered the nociceptive responses of sham-operated controls.

45 mine whether local inhibition of FAAH alters nociceptive responses of spinal neurons in the spinal ne

46 n the potency of opioid agonists to suppress nociceptive responses of the contralateral, uninflamed h

47 st, two sequential doses of WB4101 increased nociceptive responses on both the tail-flick and foot-wi

48 us, in addition to somatotopically organized nociceptive responses, orofacial deep tissue injury also

49 hat MOR ligands directly modulate SP-induced nociceptive responses primarily at postsynaptic sites, r

50 ember 1), but the mechanisms underlying this nociceptive response remain poorly understood.

51 c administration of ACEA-1021 attenuated the nociceptive responses solely in the formalin test.

52 Comparative studies assessed behavioral nociceptive responses, spinal cytokine mRNA and protein

53 thod, the mGluR(5) antagonist did not affect nociceptive responses, suggesting that thalamic receptor

54 d hot plate tests for spinal and supraspinal nociceptive responses than wild-type mice.

55 reas the contribution of PLCbeta1 to thermal nociceptive response thresholds requires a critical conc

56 tial vanilloid 1 (TRPV1), which transduces a nociceptive response to capsaicin.

57 n, constitutes an important component of the nociceptive response to CO(2).

58 ats depleted of dopamine displayed increased nociceptive responses to chemical stimulation of the fac

59 n spinal slices and in inhibiting behavioral nociceptive responses to heat.

60 ally to mice, CGRP8-37-cholestanol inhibited nociceptive responses to intraplantar injection of capsa

61 ventrolateral periaqueductal gray modulates nociceptive responses to noxious heating of the feet by

62 at are different from those that inhibit the nociceptive responses to noxious heating of the feet.

63 t are different from those that inhibits the nociceptive responses to noxious heating of the feet.

64 e ventrolateral periaqueductal gray inhibits nociceptive responses to noxious heating of the tail by

65 e ventrolateral periaqueductal gray inhibits nociceptive responses to noxious heating of the tail by

66 at are different from those that inhibit the nociceptive responses to noxious heating of the tail.

67 Nociceptive responses to select noxious thermal and mech

68 stimuli and morphine fails to attenuate the nociceptive responses to thermal stimuli in these animal

69 The nociceptive response (visceromotor response [VMR]) to CR

70 sion in DRGs neurons and to evaluate whether nociceptive responses were affected in AS model mice (gl

71 d in male and female AS model mice; however, nociceptive responses were not altered by the conditiona

72 Nociceptive responses were quantitated in the rat by mea

73 Kappa opioid receptor (KOR) ligands alter nociceptive responses when applied to the rostral ventro

74 ascular neuronal tone and Adelta-fiber dural-nociceptive responses, which differs from the way the br